Lecture 2: Osteopathic Consideration for Obstetric Patient Flashcards

1
Q

Which chronic MSK condition may have improved sx’s with pregnancy and which may be aggravated?

A
  • Improved = rheumatoid arthritis
  • Aggravated = ankylosing spondylitis (due to ↑ stress on SI joints)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

What changes occur to the SI joints and pubic symphysis during pregnancy?

A

Widening and ↑ mobility

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

What changes occur to the pelvis, lower back, shoulders, and neck during pregnancy?

A
  • Anterior tilt of pelvis
  • Exaggerated lordosis of lower back
  • Forward flexion of neck
  • Downward movement of shoulders
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

Excessive CT stretching and microtrauma during pregnancy can cause LBP during pregnancy, where is the pain typically localized?

A

SI region

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

What are some characteristics of the posterior pelvic pain due to the mechanical stress associated with pregnancy (i.e., location, radiation, exam findings)?

A
  • Distal and lateral to lumbosacral jct
  • Radiates down posterior thigh to knee
  • NO muscle weakness or sensory impairment + normal +2/4 reflexes
  • Pain may be due to relaxation of ligaments
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

List 3 alarm features of LBP in pregnancy which would necessitate referral for further evaluation?

A
  • Severe pain that interferes w/ function, particularly non-positional persistent pain AT NIGHT
  • ↑ pain w/ cough, sneezing, valsalva
  • Neuro deficits: either by hx or on exam
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

The majority of cases of radicular pain during pregnancy are due to what?

A
  • Herniated disc (40%)
  • Bulging disc (10%)
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

How would radicular pain due to a bulging or herniated disc present in a pregnant pt?

A
  • As paresthersias in ilioinguinal and genitofemoral nerve distribution
  • “Lightning pains”
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

How large of an increase in interstitial fluids is seen during pregnancy?

A

6.5 L over the course

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

How do increased estrogen, progesteronem and adrenal hormones affect the lymphatic and venous stress seen w/ pregnancy?

A

Promote fluid retention (tissue edema)

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

What occurs to SVR and CO during the 1st trimester?

A
  • SVR progressively ↓
  • CO progressively ↑
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

What happens to CO, SV, and HR while in the supine position during the 3rd trimester?

A
  • CO + ↓ SV + ↑ HR
  • Enlarged uterus can compress the IVC and ↓ venous return to heart
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

What are 3 causes of decrease lymphatic flow during pregnancy?

A
  • Fascial torsions
  • Organ hypertrophy
  • Diaphragm restriction
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

What are some signs/sx’s associated with decreased lymphatic flow during pregnancy and during which trimester are they most often seen?

A
  • Hemorrhoids
  • Vulvar and LE varicosities due to sluggish venous return
  • Most sx’s in 3rd trimester
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

The decreased lymphatic flow in pregnancy is due to a less effective what?

A

Less effective pressure gradient

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

Motion of what structure helps drive the pressure gradient for venous return?

A

Thoracic cage motion

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

Venous stresses during pregnancy can lead to CNS congestion which causes what signs/sx’s?

A

HA, nausea, and light-headedness

18
Q

How can venous stresses during pregnancy cause delayed LBP (occurring during the evening)?

A
  • Dependent edema moves back into vasculature + direct pressure on IVC by uterus
  • ↓ flow in pelvis —> stagnant hypoxia of neural and vertebral tissue
19
Q

Pregnant women incapacitated by LBP have higher levels of what hormone?

A

Relaxin

20
Q

Which hormone leads to a change in the mechanical configuration of the thoracic cage during pregnancy?

A

Progesterone

21
Q

What are 2 relative contraindications for OMM in the OB patient?

A
  • Premature rupture of membranes
  • Premature labor
22
Q

List 7 absolute contraindication for OMM in the OB patient?

A
  • Undiagnosed vaginal bleeding
  • Prolapsed umbilical cord
  • Placental abruption
  • Ectopic pregnancy
  • Placenta previa
  • Threatened or incomplete abortion
  • Severe pre-eclampsia/eclampsia
23
Q

Which factor if established before pregnancy can decrease the likelihood of a Mom suffering LBP during pregnancy?

A

Established exercise routine

24
Q

The effect of touch when treating a pregnant patient with OMM can be classified by using which of the 5-models?

A

Behavioral

25
Q

What are 2 areas of focus when treating hyperemesis gravidarum with OMM during the first trimester?

A

Tx areas C2 and T5-9

26
Q

Recommending prenatal vitamins and treating hyperemesis gravidarum fits under which of the 5-models of OMM?

A

Metabolic-energetic-immune

27
Q

Which type of pelvic torsion should you expect to find during the second trimester (14-26 weeks) when evaluating a pregnant patient?

A

Pelvis rotating anterior about right/left axis (forward torsion)

28
Q

There may be a compensatory increase of the thoracic kyphosis during the second trimester of pregnancy causing what type of strain?

A

Cervical strain

29
Q

OMM during the second trimester should focus on what?

A
  • Fascial release (indirect/direct)
  • Tx sacrum and pelvis (any modality that is comfy to pt)
30
Q

Which syndrome may arise during the second trimester due to the edematous state?

A

Carpal Tunnel Syndrome

31
Q

What are 2 focuses of tx during the 2nd trimester using the metabolic-energetic-immune model?

A
  • Prenatal vitamins
  • Constipation: pelvic diaphragm release, stool softeners, laxatives
32
Q

What are the viscerosomatic levels for the adrenals, ovaries, and uterus which should be trargeted with OMM during the 3rd trimester?

A

T10-L2

33
Q

Which viscerosomatic levels for the bladder should you focus OMM towards during the 3rd trimester?

A

S2-4

34
Q

Using the behavioral model what are some recommendations for dealing with the hypotension and GERD associated w/ the 3rd trimester?

A
  • HoTN: drink plenty of fluids
  • GERD: elevate head of bed, dietary modifications
35
Q

Which OMM tx may help regulate uterine contractions during labor?

A

Thoracic spine ST via sympathetic innervation

36
Q

What are some of the signs/sx’s associated with rupture of the pubic symphysis?

A
  • Acute pain radiating to back and/or thighs
  • Palpable gap w/ local tissue edema
  • Waddling gait —> ↑ pain on gait or bending
37
Q

What are some components of the conservative tx approach to rupture of the pubic symphysis?

A
  • Bed rest –> lateral recumbent
  • Pelvic binder –> reduces separation
  • OMM –> spine and pelvis (indirect)
38
Q

Infant and lithotomy position post-partum encourages what type of sacral and cranial SD?

A

Anterior sacral base (flexion) w/ cranial extension

39
Q

The infant and lithotomy position assoc. w/ an anterior sacral base (cranial extension) may cause what sx’s in the patient?

A

Fatigue, depression, and low energy

40
Q

Utilizing OMM in the first visit post-partum is important before which changes occur?

A

Tx prior to resolution of hormonal changes on ligamentous structures (i.e., relaxin)

41
Q

When should the second screening for SD’s occur during the post-partum period?

A

4 weeks post-partum

42
Q

What are 2 pregnancy specific relative contraindications to aerobic exercise?

A
  • Intrauterine growth restriction (IUGR) in current pregnacy
  • Unevaluated maternal cardiac arrhythmia