Lecture 1-intro Flashcards

1
Q

Ligaments and tendons

A
  • composed of dense, organised connective tissue
  • 78% water, 20% collagen and 2 % GAGs
  • collagen forms 70% of dry weight
  • extreme tensile strength with great resistance to pulling forces with minimal elongation
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2
Q

What are the phases of connective tissue healing?

A
  1. Reaction phase
    -lasts up to 72hrs
    Vasodilation, oedema, stimulation of pain fibres
  2. Regeneration phase
    - lasts from 48hrs to 6 weeks
    - elimination of debris, revascularization, fibroblast proliferation
    - reduces pain and inflammation
  3. Remodelling phase
    - lasts from 3 weeks to 12 months
    - contraction and slow maturation of collagen for increase in tensile strength
    - increase ROM, strength and proprioception
    -
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3
Q

What are the classifications of connection tissue ligament trauma / injury?

A
First degree (mild)
- microscopic stretching or minimal tearing ( classic strain)
- painful but minimal loss of integrity
- full recovery 10 days - 2 weeks
Second degree (moderate)
- moderate tearing
- some loss of structural integrity
-2-3 weeks of activity modification and rehab with up to 3 months full recovery
Third degree (severe)
- complete tear
- up to 6 months for full recovery
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4
Q

What is the grading used for a connective tissue injury?

A

Grade 1: pain following activity
Grade 2: pain during activity
Grade 3: pain interferes with activity
Grade 4: continuing pain even between activities
Grade 5: pain interferes with activity and daily living

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5
Q

What are the aims of treatment in muscle injury?

A
  • minimise further damage
  • reduce pain and spasm
  • control haemorrhage and oedema
  • promote healing
  • reduce scaring formation
  • regain strength
  • regain flexibility
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6
Q

What are the classifications of a muscle injury?

A
  1. Exercise induced muscle injury
  2. Muscle strain
  3. Contusion
  4. Avulsion
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7
Q

Exercise induced muscle injury

A

• Also referred to as post-exercise soreness
• Manifests 24 – 48 hours following unaccustomed or intense
exercise
• Excessive eccentric loading magnifies the response
• Pain is related to damage of muscle cells and associated inflammation
• Can result in acute compartment syndrome, usually in LL
• Significant bacterial or viral infection, particularly if associated with fever or myalgia may result in inflammatory myopathy with associated rhabdomyolysis (muscle breakdown).
• Can result in secondary renal damage due to myoglobinuira.

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8
Q

Muscle Strain

A

In turn classified as:
1. First degree (mild)
2. Second degree / partial tear (moderate)
3. Third degree / complete tear (severe)
• Presentation: Trauma, significant loss of ROM and function, tense and painful swelling
• Tears of muscle belly heal more rapidly than the musculotendinous junction
• Better prognosis associated with mild-moderate strains and minimal bleeding
• Poorer prognosis associated with re-tear, severe strain esp. located in a compartment with excessive bleeding

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9
Q

Muscle strain- risk of complication

A

Degree of injury correlates with long-term disability as well as risk of complications such as:
*Myositis ossificans uProlific scarring
*Slow resolution
*Risk of re-injury
As the degree of the muscle strain increase (grade I to III) there is :
Increase Pain and spasm
Increase Swelling and bruising
Increase Defect
Increase Loss of ROM and Function
Increase Recovery time

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10
Q

Contusions

A

Contusion
• Common in contact sports
• Mild contusion – more haematoma than with a
comparable strain
• Moderate to severe – hard to distinguish from tears (moderate – severe strains)
Severe contusions may predispose

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11
Q

Contusions may either be?

A
  • Intermuscular
    • Along large intermuscular septa / fascial sheaths (eg. thigh)
    • Facilitates gravitational tracking of extravasated blood which minimizes inflammatory response and potential scarring – early resolution!
  • Intramuscular
    • 2-3 times longer to heal than intermuscular
    • Haemorrhage more confined and palpable
    • Inflammation greater
    • Risk of Myositis Ossificans, compartment syndrome and scarring greater to significant scarring
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12
Q

Avulsion

A
Often commonly occurs in specific anatomic locations:
ASIS with Sartorius
AIIS with Rectus Femoris
Ischial tuberosity with Hamstring 
Olecranon process with Triceps
Patella with Quadriceps tendon
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13
Q

Stages of muscle healing

A

uInitial inflammation which promotes phagocytosis
uEarly healing: initial focal deposits of collagen represent early scarring and contribute to tissue stiffness (replacement of contractile with non-contractile tissue)
uEarly ROM exercises are essential here to lengthen the immature plastic scar, minimize adhesion formation and assist with collagen orientation
uModalities such as heat and IFC may assist here
uEstablished healing (days 6-14): early, gentle rehabilitation to re-establish strength and ROM, muscle regeneration is facilitated by activity
uCare must be taken here not to re-injure

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14
Q

Common types of fractures

A
  1. Stable: broken ends of the bone are aligned (good prognosis for healing)
  2. Simple (closed): no skin piercing; as opposed to –
  3. Compound (open): skin is pierced either by bone or trauma to the region, bone may be visible (Concern for development of osteomyelitis)
  4. Transverse: Horizontal fracture line
  5. Oblique: Oblique fracture line
  6. Comminuted: 3 or more fragments; often referred to as “shattered” in lay terms
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15
Q

Childhood fractures

A
  • The zone of hypertrophy (ZOH) is the weakest structural layer in bone, therefore most prone to fracture – and therefore ideally is less likely to have growth implications*
  • Fractures in children are classified as Salter-Harris Types I – V (crush injury)
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16
Q

Describe the 5 types of salter-Harris fractures

A

o uTypes I & II: mostly through ZOH, heal rapidly and are usually ready for full stress by 6-8 weeks. Long- term complications and deformity are minimal.
o uType III: may extend into the joint, leading to problems of joint incongruity
o uType IV: directly cross from metaphysis to epiphysis through the growth plate
o uTypes IV and V may result in growth retardation and/or deformity

17
Q

Stages of fracture healing

A
  1. Inflammation (
18
Q

Factories influencing fracture healing

A

Patient age
Circulation
Mechanical factors

19
Q

Red flags in MSK medicine

A

uWhat is a RED FLAG?
uCommon (general) red flags in MSK medicine:
uAge >50
uProgressive neurological deficit
uPathological changes in bowel or bladder function
uUMN signs – remember these?
uHistory of cancer
uRecent significant trauma
uLong-term corticosteroid use
uPresence of constitutional symptoms – remember these?

20
Q

Severity & Character of the Pain

A

This gives us clues as to which structures are involved too, as well as to what extent!
Remember this?
Visceral pain: eg. hepatitis
uContraction or distention of viscera
uGnawing, cramping or aching
uOften difficult to localize
Parietal pain: eg. appendicitis
uInflammation of viscera which affects parietal peritoneum uMore severe than visceral
uUsually easy to localize
Referred pain: eg. acute cholecystitis
uOriginates at different sites but shares embryological innervation from the same spinal level
uUsually dull, aching pain
uDifficult to differentiate from MSK pain at times!

21
Q

Differential Diagnoses

A
Using a universal list: “VINDICATE” uV ascular
Infection
N eoplasm
D egenerative
I nflammatory
C ongenital
A rthritic
T raumatic
E ndocrine
22
Q

Active, passive and resisted isometrics

A

Active:
1. Contractile tissues will be painful with contraction or
stretching
2. Non-contractile tissues will be painful when stretched or ‘pinched’
Passive:
1. Contractile tissues will be painful with stretch
2. Non-contractile tissues will be painful when stretched or ‘pinched’
Resisted isometrics:
1. Contractile tissues will be painful with contraction; stretching not likely to occur due to contraction preventing end-range movement
2. Non-contractile tissues not likely to be stretched or ‘pinched’ due to muscular contraction preventing end- range movement

23
Q

What is tested during active, passive and resisted isometrics?

A

Active:
-Contractile & Non-contractile tissues tested, if any pain or limitation
Passive:
-Non-contractile tissues predominantly tested
Resisted isometrics:
-Contractile tissues predominantly tested

24
Q

Stress fractures

A
  • Usually in response to repetitive stress, often due to accelerated, unaccustomed, intense activity with insufficient recovery period
  • Occurs in elderly, osteoporotic, but also healthy young athletes
  • Common regions include: femur, tibia, tarsals and metatarsals
  • Distinguish from pathological fracture – therefore, if due to osteoporosis can be considered pathological. Other pathological causes include tumors or bone cyst.
25
Q

Clinically a patient with a stress fracture may present with these different signs:

A
  1. Point tenderness of bone
  2. Soft tissue swelling
  3. Alteration of gait
  4. Muscular atrophy
  5. Full and painless ROM
  6. Painless resisted active movement of joint
  7. Hairline radiolucency , periosteal callus
  8. Palpation of callus with time
  9. Active:
26
Q

What is the cycle injury cycle?

A

Trauma–> inflammation—> pain AND/OR spasm—> healing OR chronicity