lecture 1 Flashcards
frontal lobe
control/action planning
parietal lobe
awareness of body state/location, object recognition and reading
temporal lobe
interpreting information/comprehension
occipital lobe
vision/reading
EEG/MEG
changes with damage
CT Scan
structure of brain, atrophy
MRI
large scale anomalies, tissue atrophy
fMRI
structure and activity
fNIRS
oxygenated and deoxygenated blood
post mortem
cadavers
what are the assumption s of neuropsych
fractionation, anatomical modularity, functional modularity, universality and subtractivity/transparency
fractionism
cognitive functions can be explained in simpler components; brain damage -> selective impairment of specific components
functional modularity
informational encapsulation, domain specificity, anatomical specificity; lesion to module affects a specific cognitive process
anatomical modularity
specific brain regions are responsible for certain processes
universality
all brains are highly similar
subtractivity/transparency
total cognitive system - components impaired by lesion = performance of patient
association
inability to perform on task 1 and 2; tasks might use the same process or tasks are mediated by areas adjacent to each other affected by the lesion
dissociation
patient impaired with task 1 but not task 2; tasks involve different systems or one task is harder than the other
double dissociation
opposite dissociations in two patients; reflect involvement of different systemsr
group studies
patients grouped according to classical systems
group studies CONS
heterogeneity, too large of a filter makes averaging too lenient. functional homogeneity not guaranteed + information lost in averaging process
case studies
one patient studied extensively, patients overlap with one symptom but differ in others, each patient a different test for a cognitive theory
case studies CONS
replication (look for converging evidence); generalistion: universality
