Lec 5 Flashcards

1
Q

force - velo relationship

A

as we increase force we decrease speed.
eccentric contractions provide the most force

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2
Q

motor unit

A

the nerve and all the muscles its connected to are recruited to exert force

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3
Q

musclr fibre ratio

coordinationo movement compared to size of muscle

A

the ratio of fibres innervated by the motoro neuron is determined by the precision accuracy and coordination of the moveemn, not the size of the muscle group

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4
Q

size principle

A

the body only recruits muscle units that it needs ot complete the job

in order from 1 - 2A-2X

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5
Q

muscle and neural adaptaiton to resistance training

A

we see that most traiing studies stop before they reach peak strenght and hypertrophy in the person at 8-12 weeks but we see that at this time is when neural adaptation beigins to peak and plateua.

most studies end at 8-12 weeks, serious strenght trainers peak after this

exogenous anabolic hormone use can break the hypertrophy plateau

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6
Q

majority. of population is what percent slow twitch

A

60%

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7
Q

fibre typing

A

using a biopsy provides moderate results

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8
Q

what determines contractile speed

A

myosin atpase

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9
Q

which fibres produce more lactate quicker

A

fast twtich fibres

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10
Q

swithvc from 2x to 2a

A

need 20 week 6-8 reps till failure

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11
Q

switch from 2a to 2x

A

21 week detraining

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12
Q

bone remodeling requires the right

A

stimulus and nutrients

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13
Q

horizontal both growth

A

shin kicks provide a stimulus horizontally allowing the bone to grow horizontally

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14
Q

BMD decreases with

A

age and if in space - no stimulus of tension

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15
Q

after one day of bed rest

A

we lose immense BMD

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16
Q

tibia BMD loss

A

38% in 60 days

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17
Q

BMD peaks and is created when

A

from 10 -20 is created peaks at 20

18
Q

osteoporosis

A

2 standard deviations from normal BMD

19
Q

suboptimal lifestyle factors include and what do they do

A

include lack of nutrients, vit d,, reduced physical activity

these decrease the peak of of BMD

20
Q

muscular hypertropy

A

muscular enlargment due to increase in muscle cross sectional area

the net increase in contractile proteins within the myofibril and the increase and myofibrils in the muscle

21
Q

sarcoplasmic hypetrophy

A

due to increase in volume in the uscle, from fluid swelling and glycogen

which can becoem a stimulus for growh

22
Q

3 ways to stimuluate mTOR

A

leucine
Resistance Training
Insulin

23
Q

what is the onjly direct stim of mTOR

A

eatong EAAs

24
Q

insulin

relaxation

A

insulin causes the relaxation of smooth muscle , which cause release of NO3 which vasodialates - which increases blood flow, which is driectly proportional to MPS

25
Q

sarcoplasmic expansion hypothesis

A

this jsut says that the sarcoplasmic volume. increases allowing mroe roomn for ribosmomes and the constant swelling triggers fusion with sasatellite cells whihc birngs mre contractile proteins

26
Q

myofribril expansion hypothesis

A

says that ribosomes increase tranascription during resistance traiing iand create more contractile proteins

27
Q

tapering of myofibril hypertrophy but muscles still look bigger

A

we see that overtime myofibril hypertrophy tapers off and sarcoplasmic hypertrophy continues swelling - this saccounts for big muscles adn not always big strenght

28
Q

is there an anabolic window

A

no 24 hours and 3 hours and 1 hour after exercise didnt chanfe the amount of MPS

29
Q

the limit for mTOR

A

mTOR can only be stimmed for a an hour 60minutes until it taps out, then the refracotry period is about 4 hours which is why we should space our diet to be 3-4horus inbetween meals

30
Q

how long is MPS stimulated after exercises?

A

48 hours so eat properly in the next 48 hours but to optimize hit same muscle 3 times a week

31
Q

carbs effect on insulin

A

carbs stim inuslin response- 3 hr post eating shows a non-catabolic effect rather than a anabolic effect

32
Q

carbs and piss with aa

A

when you eat carbs and aa after workout you excrete less aa acid in piss

33
Q

why can’t you train both endurance and RT and see gains

A

AMPK inhbiits mTOR but is turned on when enery is low (AMPK monitors our energy status), no anabolism, only catabolism to get us enrgy from carbs fat and some protein

34
Q

AMPK-Akt master switch

A

during RT - akt stims mTOR to turn on and MPS - inhibiting AMPK

during endurance training, AMPK senses low energy levels (glycogen) and stims TSC2 to inhbit mTOR for

35
Q

interference model

A

the interference model says

ig we want neural adaptation we need high load and if we want cardiovascular adaptation we need high intesity

once we start doing both we are in the zone of interference

75% of HRmax = HIhg intesity
5RM hihg load

dont train weight room shit on the field adn vice versa

36
Q

hormone release after RT

A

the relase of hormones after RT are usually acute and trnasient not significantly effecting muscle growth

37
Q

rest intervals inbetween sets

A

larger rest intervals past 120secons increase the amount of testosterone released and if you wiat 5 minutes you will get more atp then you had before the set even started (Supercompensation)

38
Q

natural hormone fluctioation form RT

A

doesnt lead to increaesed mnuscle growth

39
Q

frequency and volume of workouts

A

if we keep the volume constant, the amount of time we workout does not significantly change the amount of muscular hypertrophy

40
Q

training till failure

A

it does not matter the amount of weight you are lifting, when training till failure we see the saem muscle growth

41
Q

1rm boost if

A

you train heavy at low reps, physiological factor working at those heavy weights