Lec 3- Gait and Motor Activation Flashcards
Muscular Activation in Gait Definitions
Isotonic: length changes in muscle
Concentric: isotonic contraction where muscle is shortening (generation of speed, force)
Eccentric: isotonic contraction where muscle is lengthening (controlling joint), shock absorption, deceleration, control
Reciprocal Inhibition: allows joint to freely move w/o antagonist activating
Co-Activation: simultaneous contraction of agonist and antagonist muscle groups work to stabilize the joint
Muscular Synergies: groups of muscles across joints that work together to perform activity
Why is walking efficient and smooth?
-Muscular activity is delivered at spinal cord level during routine walking
-Genetic blueprint and years of training
-Finely tunes on and off muscular activation
Reciprocal Inhibition
-is governed at the SC level
-is a system created by a stimulus activation (activation and reciprocal inhibition)
-occurs across muscle groups in both limbs
-allows for on and off cycle of muscle activation
-allows flexibility
-is a mechanism to govern motor function
Reciprocal Inhibition occurs with activation of…
muscular contraction (prevents agonist and antagonist activation)
An example of reciprocal inhibition
Terminal Swing
Ankle: Anterior Tibialis is activated and Gastroc/Soleus is not activated
-allows ankle DF not to be impeded
-allows for optimal joint function thru DF
An example of muscular co-activation
Terminal Stance
Knee: Quadriceps are concentrically activating and hamstrings are eccentrically
-controlling knee extension
-purpose is to control, decelerate down knee extensor activity
-absorb ground shock
Neurobiology of Gait: CPGs
-brain to spinal cord
-SC coordinates on and off timing of motor activity during walking
-controlled by CPGs in SC (control limb segments, ipsilaterally and contralaterally)
How does the CPG work?
Rhythm Network: method of activating muscle groups or motor pools of flexors then extensors
-combination of sensory/feedback (touch, pressure from environment & forces within muscle) to provide feedback to pattern formation and provide motor activity
-sensory input adjusts rhythm and walking pattern
Muscular Activation in Walking
-organized by swing phase (flexors) and stance phase (extensors)
-force generation emerges from stance phase
-timing of muscle groups is rhythmical and speeds up/slows down based on indv. pace
-similar across human species
Initial Contact
Hip: glute max and add. magnus active to prep for LR
Knee: Quad extend knee, HS contact to counteract contact torque
Ankle: TA eccentric control and pre tibial muscles
Loading Response
Hip: glute max (low fibers), add. magnus, & HS are activated to counteract flexor torque. TFL, glute med., and glute min., and glute max (upper fibers) stabilize frontal plane
Knee: Quad eccentric activity (knee flexed absorbs shock)
Ankle: EHL, EDL, TA eccentrically contract. TA peaks activity (eccentric). TP stabilizes t/o stance, gastroc-soleus activates late loading response.
Midstance (SLS)
Hip: No sagittal plane muscle activity except glute max. (upper fibers), glute med. stabilizes frontal plane, TFL co-activates for stability
Knee: Quads stabilize dynamic knee stability up until midstance eccentric
Ankle: Gastroc-soleus restrains tibia for forward progression. Gastroc-soleus eccentrically control forward progression of tibia
Where does gait break most often?
Loading Response & Midstance
-LR: quads eccentrically control for shock absorption
-Midstance: gastroc-soleus controls tibia advancement over fixed foot
Midstance Frontal Plane Activation
-Glute med. provides frontal plane stability at hip
-TFL co-activates for stability across hip and knee
Terminal Stance (Trailing Limb)
Hip: anterior TFL still on (from LR)
Knee: No quads activation. Bicep fem. activation to prevent knee extension
Ankle: Gastroc-soleus peaks to prevent tibial collapse (provides propulsion), TP FL & FB stabilize