lec 1 Flashcards

1
Q

T or F: stuttering disorders are completely psychological

A

false – also biological

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

what is a core behaviour?

A

the actual stuttering events

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

what’s the diff bw repetitions, prolongations, blocks, and interjections?

A
  • repetitions: repetition of sounds or syllable(s)
  • prolongations: airflow continues but articulators stop moving
  • blocks: stoppage of airflow and articulators
  • interjections: adding extra words/sounds such as um, uh, like etc
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

what are secondary behaviours? examples?

A
  • extra words and behaviours
  • examples: blinks, eyebrow furrowing, jaw sliding
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

whats the diff bw escape behaviours and avoidance behaviours?

A
  • escape: attempt to terminate a stutter and finish the word
  • avoidance: attempt to prevent stuttering
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

T or F: repetitions, prolongations, and blocks are secondary behaviours

A

false – RPB = core behaviours

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

whats the diff bw incidence and prevalence? what is each statistic for stuttering?

A
  • incidence: how many people have stuttered at some point in their lives? 5-8%
  • prevalence: how many people are stuttering at one point in time? <1%
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

T or F: some cultures have lower incidence rates than others

A

true – competitive cultures tend to have higher than non-competitive cultures

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

what is the average age of stuttering onset?

A

2-3.5 years

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

do spontaneous recoveries from stuttering happen often in children?

A

yes ~85%

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

what is the average time of recovery for stuttering?

A

18-24 months from onset

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

why does the sex ratio of stutterers change over time? i.e., 4:1 males for females but 1:1.2 at onset

A

bc females start to stutter earlier, recover earlier and recover more often

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

what are the big 6 predictors of stuttering persistence?

A
  1. family history
  2. male
  3. onset later than 3.5 years
  4. duration over a year (= worse prognosis)
  5. other speech/lang/motor/cognitive delays
  6. pattern of stuttering over time
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

what is the best predictor of recovery?

A

recovery within 12 months of onset

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

if a parent brings a 4 year old stuttering child to an SLP, should they wait and monitor or begin therapy? why?

A
  • begin therapy
  • treatment most effective during preschool years bc brain plasticity
How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

name 5 fluency enhancing conditions

A
  1. delayed auditory feedback
  2. masking noise
  3. choral speech
  4. rhythmic speech
  5. reinforcements
17
Q

T or F: stuttering is a neurodevelopmental disorder

A

true

18
Q

does stuttering have a strong genetic component? which gene is responsible?

A
  • yes (but not purely genetic)
  • polygenetic
19
Q

T or F: if someone has the stuttering gene(s), that means they will definitely stutter

A

false – genetics = predisposition NOT determination (think light switch)

20
Q

T or F: your environment can cause stuttering

A

false – can only trigger predisposition (ie need the gene in the first place)

21
Q

what are some congenital factors that are associated with stuttering onset? (3)

A
  • infectious diseases
  • head injuries
  • anoxia
22
Q

which 3 areas of the inferior frontal gyrus are involved in the formation of an articulatory plan? (brodmann # + name)

A
  • BA 44, 45, 47 (broca’s area)
  • BA 4 (primary motor cortex)
  • BA 6 (premotor area)
23
Q

what do each of the following parts of the brain do regarding speech production:
a) premotor area – ventral part
b) premotor area – dorsal part
c) primary motor cortex

A

a) interfaces between planning + execution
b) supplementary motor area (tells you “go!”)
c) speech execution

24
Q

the ___ and ___-___ systems are in constant communication with the cortical areas via connective ___ ___ ___ of the cortico-striatal loops and the internal capsule.

A
  • cerebellar
  • basal-ganglia
  • white matter fibres
25
Q

what would the white matter connections in someone who stutters look like relative to someone who does not stutter? what purpose do these connections serve?

A
  • reduced in density
  • connect sensory, planning, and motor areas for speech
26
Q

what does it mean if “the left premotor cortex is hypo-perfused” in someone who stutters?

A

that the premotor cortex is not recruited enough for speech planning and production

27
Q

differences in perfusion for left vs right hemisphere of someone who stutters?

A
  • left: often hypoperfusion
  • right: more likely hyperperfusion, compensating
28
Q

T or F: after treatment, compensation by the left hemisphere speech regions,rather than by the right, may be more effective in bypassing the structural anomalies and improving the efficiency of signal transmission

A

true

29
Q

T or F: people who stutter have trouble getting their ideas together

A

false – it’s an issue of timing motor programs

30
Q

how might temperament influence stuttering?

A

someone with a laid back temperament may handle the emotional impacts of stuttering more effectively

31
Q

main points of environmental theories: parents/environmental stressors? (3)

A
  • parent characteristics = stuttering (eg low SES, anxious, demanding)
  • life events = stuttering (eg divorce, moving)
  • studies show this is FALSE
32
Q

main points of environmental theories: diagnosogenic? (3)

A
  • early speech disfluencies are not stuttering
  • stuttering develops after it has been diagnosed by parent
  • led to reduced early intervention
33
Q

main points of environmental theories: anticipatory struggle? (3)

A
  • 1) frequent communicative failure (eg delayed language), 2) heavy communicative pressure (eg parent standards), and 3) high vulnerability to communicative pressures (eg child weakness) = stuttering
34
Q

main points of environmental theories: capacities and demands? (3)

A
  • child’s capacity = bucket
  • internal and environmental demands (eg temperament, interactions) = water
  • overflow = disfluency
35
Q

main points of cerebral dominance theory? (3)

A
  • left hemisphere should be dominant for speech
  • for stuttering, the hemispheres are competing = asynchronous nerve impulses
  • forced children to be right-handed – did not help
36
Q

main points of DIVA model? (3)

A
  • feedback pathway: slow and consciously controlled + directed by the cortex and cerebellum.
  • feedforward pathway: quick and automatic + directed by the basal ganglia and sensory motor area.
  • stuttering = impaired feedforward pathway.
  • people who stutter should use feedback pathway to achieve fluency.
37
Q

DIVA model: implications of relying on the feedback pathway? (2)

A
  • requires lots of cognitive resources
  • unrealistic for children to self-monitor
38
Q

which 3 theories are most useful?

A
  1. capacities and demands
  2. DIVA model
  3. two stage model
39
Q

main points of two-stage model? (2)

A
  • primary stuttering: inherited or congenital causes = structure/function issues in speech planning pathways.
  • secondary stuttering: factors can promote recovery or cause stuttering to persist (eg temperament, conditioning).