labour and puerperium Flashcards

1
Q

What are the 5 indications for induction of labour

A
  • prolonged gestation (offered between 40 and 42 weeks)
  • premature rupture of membranes (delay IOL if <34 weeks unless fetal distress etc)
  • maternal health problems (common egs: HTN, pre- eclampsia, diabetes, obstetric cholestasis)
  • Fetal growth restriction
  • intrauterine fetal death (if mother well with intact membranes)
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2
Q

Describe 4 absolute and 3 relative contraindications for induction of labour

A
Absolute:
- cephalopelvic disproportion
- major placenta praevia
- vasa praevia
- cord prolapse
- transverse lie
- active primary genital herpes
- previous classical c section (unless consultant assessed and says otherwise)
Relative:
- breech presention
- triplet or more
- two or more previous low transverse c sections 
Contraindications generally same as for vaginal delivery
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3
Q

What are the three methods of inducing labour?

A
  • vaginal prostaglandins
  • amniotomy
  • membrane sweep
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4
Q

What is the preffered method of induction (according to NICE) and how does it work

A
  • vaginal prostaglandins (tabel/ gel or pessary regimes)
  • prostaglandins prepare the cervix for labout by ripeining it and also have role in smooth muscle contraction of uterus
  • can take multiple days
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5
Q

When is a amniotomyused?

A
  • Only when the cervix has been deemed as ripe under the bishop score
  • often given with an oxytocin infusion to increase strength and frequency of contractions
  • should not be used as a primaru method of induction unless prostaglandins are contraindicated eg high risk of uterine hyperstimulation
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6
Q

When is a membrane sweep used?

A
  • offered at 40-41 weeks to nulliparous women and 41 weeks to multiparous women
  • classified ad adjunct- increases likelyhood of spontaneous delivery and reduces need for formal induction
  • a gloved finger is inserted through the cervic and rotated against the fetal membranes to try separate the chorionic membrane from the decidua, releasing natural prostaglandins in an attempt to kick start labour
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7
Q

What is a bishop score used to asses, what features score points?

A
  • cervical ripeness- score >7= ripe and high change of response to interventions made to induce labour, score <4 suggests labour unlikely to progress naturally and prostaglandins will be required
  • dilation,cervix length, station relative to ischial spines, consistency and position of cervix (posterior- anterior/mid) are assessed
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8
Q

When should CTG be used during induction of labour?

A
  • prior to induction
  • after initiation when contractions begin, use it continiously until a normal heart rate is confirmed then assess using intermittent auscultation
  • of oxytocin infusion is started then monitor using continuous CTG throughout pregnancy
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9
Q

Give 5 complications of induction of labout

A
  • failure of induction: offer further prostaglandins or c section
  • uterine hyperstimulation (contractions last too long (>2mins) or are too frequent (>4 in 10), leading to fetal distress. manage with tocolytic agents such as terbutaline)
  • cord prolapse (amniotomy esp if fetal head is high)
  • infection
  • pain (IOL often more painful than spontaneous and epidural anaesthetic often required)
  • increased rate of further intervention eg emergency c section or instrumental delivery)
  • uterine rupture (rare)
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10
Q

What instruments are most commonly used in operative vaginal deliveries?

A
  • ventouse: attaches cup to fetal head by vacuum. two types, ‘kiwi’ can be used in all positions and rotational deliveries
  • forceps: double bladed instruments, 3 types, higher risk of tears, less often used to rotate and dont require maternal effort
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11
Q

What are the indications for a operative vaginal delivery?

A
  • decision made in 2nd stage of labour
  • inadequate progress: nulliparous women- should expect delivery after 2 hrs of active pushing , multiparous women should deliver within one hr of active pushing
  • maternal exhaustion
  • maternal medical conditions that mean active pushing or prolonged exertion should be limited eg intracranial pathology, heart diseases, severe HTN
  • suspected fetal compromise in 2nd stage of labour
  • clinical concerns eg significant antepartum haemorrhage
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12
Q

Give 5 contraindications of operative vaginal delivery

A
  • unengaged fetal head in singleton pregnancies
  • incompletely dilated cervix in singleton pregnancies
  • true cephalo- pelvic disproportion (where fetal head is too large to pass through maternal pelvis)
  • breech and face presentations, and most brow presentations
  • preterm gestation (<34 weeks), for ventouse
  • high likelyhood of any fetal coagulation disorder for ventouse
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13
Q

Give 5 pre- requisites for instrumental/ operative delivery

A
  • fully dilated
  • ruptured membranes
  • cephalic presentation
  • defined fetal position
  • fetal head at least at level of ischial spines and no more than 1/5 palpable per abdomen
  • empty bladder
  • adequate pain relief
  • adequate maternal pelvis
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14
Q

What are the 3 classifications of operative vaginal delivery and what is the significance of the class?

A
  • outlet: skull reached pelvic floor, scalp visible or fetal head on perineum
  • Low
  • midline
  • lower class= less risk of classifications as less rotation needed and fewer pulls
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15
Q

Give 4 fetal complications of operative vaginal delivery

A
  • neonatal jaundice
  • scalp lacerations
  • cephalhaematoma
  • subgaleal haematoma
  • facial bruising
  • facial nerve damage
  • skull fractures
  • retinal haemorrhage
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16
Q

Give 4 maternal complications of operative vaginal delivery

A
  • vaginal tears of 3rd/ 4th degree: 4x more likely in ventouse delivery and 10x more likely (1 in 10) if forceps
  • VTE
  • incontinence
  • PPH
  • shoulder dystocia
  • infection
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17
Q

What is the definition of premature rupture or membranes?

A
  • rupture of membranes at least 1 hr prior to onset of labour at >37 weeks gestation
  • preterm premature rupture (P-PROM): rupture of membranes at <37 weeks
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18
Q

What is thought to cause premature rupture of membranes?

A
  • early activation of normal physiological processes (higher than normal levels of apoptotic markers and MMPs in amniotic fluid)
  • infection- 1/3 women with P-PROM have +ve amniotic fluid cultures
  • genetic predisposition
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19
Q

Give 5 RFs for PROM and P- PROM

A
  • smoking (esp at <28 weeks)
  • previous PROM/ preterm delivery
  • vaginal bleeding during pregnancy
  • lower genital traction infection
  • invasive procedures eg amniocentesis
  • polyhydramnios
  • mutliple pregnancies
  • cervical insufficiency
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20
Q

Describe the clinical features of PROM

A
  • painless popping sensation followed by gush of watery fluid from vagina
  • sometimes can be gradual leakage of watery fluid and damp underwear/ pad
  • on speculum: fluid draining from cervix and pooling in posterior fornix , fluid may be expelled if you ask them to cough
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21
Q

How should premature rupture of membranes be investigated?

A
  • USS if diagnosis unclear from examination and hx
  • high vaginal swab for GBS
  • ferning test can be used to confirm PROM- if no vaginal pooling on speculum examination
  • actim prom: swab to look for IGFBP-1 in vaginal samples
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22
Q

How should PROM be managed if >36 weeks

A
  • most will start labour spontaneously
  • monitor for clinical signs of chorioamnioitis
  • give penicillin if GBS isolated
  • wait for 24hrs then consider induction of labour
  • steroids
  • IOL and delivery recommended if >24hrs but they can wait up to 96hrs
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23
Q

how should P- PROM from 34-36 weeks be managed?

A
  • monitor for chorioamnionitis and advise to refrain from sex
  • prophylactic erythromycin for 10 days
  • penicillin if GBS isolated
  • give corticosteroids
  • IOL and delivery is recommended if labour doesnt commence within 24hrs
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24
Q

How should P-PROM be managed if 24-33 weeks?

A
  • monitor for chorioamnionitis and advise to refrain from sex
  • prophylactic erythromycin for 10 days
  • penicillin if GBS isolated
  • give corticosteroids
  • give magnesium sulphate for neuroprotection incase they do go into labour
  • aim expectant management until 34 weeks
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25
Q

Give 4 complications of PROM

A
  • chorioamnionitis: inflammation of fetal membranes due to infection, risk increases the longer membranes the membranes remain ruptured and baby undelivered
  • oligohydramnios: if age <24 weeks as greatly increases risk of lung hypoplasia
  • neonatal death: due to prematurity, sepsis and pulmonary hypoplasia
  • placental aburption
  • umbilical cord prolapse
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26
Q

What are the major benefits of VBAC vs planned c section?

A
  • shorter hospital stay
  • lower risk of maternal death (4/100,000 vs 13/100,000)
  • good chance of future VBACs if successful
  • lower risk of transient resp difficulties for neonate
  • risk of still birth beyond 39 weeks whilst awaiting spontaneous labour
  • avoids anaesthetic risk, bleed risk, infection risk, risk to local structures (bladder, bowel), accidental risk to baby etc associated with c section
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27
Q

What are the major benefits to planned C section over VBAC?

A
  • negates risk of uterine rupture
  • no risk of anal sphincter injury
  • lower risk HIE
  • lower risk still birth
  • generally thought to be lower risk if multiple pregnancy, macrosomia, older maternal age
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28
Q

Give 5 RFs for uterine rupture in vaginal delivery after c section

A
  • previous c section- classical (vertical) incisions carry highest risk
  • previous uterine surgery eg myomectomy
  • induction or augmentation of labour
  • obstruction of labor
  • multiple pregnancies
  • multiparity
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29
Q

If vaginal birth after c section is opted for, how should it be managed?

A
  • in hospital setting with facilities for emergency c section and advanced neonatal resus
  • continuous CTG monitoring
  • beaware of needing additional analgesia as may indicate impeding uterine rupture
  • avoid induction where possible (if need todo it, risk is lower with mechanical techniques than prostaglandins)
  • after 39 weeks an elective c section is recommended delivery method
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30
Q

Give 2 absolute and 2 relative contraindications for vaginal birth after c section

A
Absolute:
- classical c section scar
- previous uterine rupture 
- anything else that contraindicates vaginal delivery eg palacenta praevia 
Relative:
- complex uterine scars
- >2 lower segment c sections
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31
Q

What is shoulder dystocia?

A

Where, after delivery of the head, the anterior shoulder of the fetus becomes impacted on the maternal pubic symphysis, or less commonly, the sacral promontory.
It is a obstetric emergency with an incidence of 0.7% of all pregnancies.

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32
Q

Give 3 RFs for shoulder dystocia

A
prelabour:
- previous shoulder dyscotia
- macrosomia
- diabetes
- maternal BMI >30
- induction of labour
Intrapartum:
- prolonged 1st stage of labour
- secondary arrest (where initially good progress in labour but then progress stops, usually due to malposition of baby)
- prolonged second stage of labour (fully dilated and pushing bit)
- augmentation of labour with oxytocin
- assisted vaginal delivery
33
Q

Describe the clinical features of shoulder dystocia

A
  • delay in delivery of shoulders following the head following the next contraction after using traction
  • failure of restitution (fetus remains in occipital- anterior position and doesnt turn to side)
  • turtle neck sign: fetal head retracts slightly back into pelvis, so neck is no longer visible
34
Q

How should shoulder dystocia be managed?

A
  • call for help (need sr obstetrician, sr midwife and paeds)
  • tell mum to stop pushing
  • avoid downwards traction
  • consider episiotomy (can make access for manoeuvers easier)
  • 1st line manoeuvres: mcroberts (knees to chest position) +/- suprapubic pressure (sustained or rocking fashion to apply pressure behind the anterior shoulder and disimpact it)- success rate 90%
  • 2nd line: ‘internal manoeuvres’- grab the posterior arm and deliver that 1st or internal rotation, move pt onto all 4s and repeat
  • 3rd line (rarely used in uk): cleidotomy, symphsiotomy, zavenelli (push head back in and do c section)
35
Q

What are the potential complications of shoulder dystocia

A
  • maternal: 3rd or 4th degree tears (3-4%), PPH (11%)

- fetus: clavicle or humerus fractures, brachial plexus injury (may be permenant), hypoxic brain injury

36
Q

Describe the clinical features of amniotic fluid embolism

A

Sudden onset:

  • hypoxia/ resp arrest
  • hypotention
  • fetal distress
  • seizures
  • shock
  • confusion
  • cardiac arrest
  • DIC (1st sign in some, but nearly all well develop this within 4 hrs)
  • physiology is similar to anaphylaxis and severe sepsis
37
Q

How should suspected amniotic fluid embolism be investigated and managed?

A
  • A-E resus
  • bloods (fbc, u+e, clotting, abg)
  • ECG
  • CXR
  • contact ITU
  • contact haematology if DIC
  • if baby not yet delivered and pt stable, continuous fetal monitoring w/ view to imminent delivery
  • if cardiac arrest or severe maternal compromise then perimortem section is indicated to facilitate CPR of mum
  • definitive diagnosis only post mortem, w/ fetal squamous cells along with debris in pulmonary vasculature
38
Q

What is umbilical cord prolapse?

A

Where the cord descends through the cervix. Can be complete (overt- cord is below presenting part of fetus) or incomplete (occult- cord is alongside presenting part of fetus))

39
Q

Why does fetal hypoxia occur due to cord prolapse?

A
  • occlusion: presenting part of fetus presses onto the umbilical cord, occluding blood flow
  • arterial vasospasm: the exposure of the umbilical cord to the cold atmosphere results in ubilical artery vasospasm, reducing blood flow to the fetus
40
Q

Give 5 RFs for cord prolapse

A
  • breech presentation
  • unstable lie (where presentation of fetus changes between transverse/ oblique/ breech and back, if >37 weeks consider inpatient admission until delivery)
  • artificial rupture of membranes- esp when presenting part of fetus is high in pelvis
41
Q

Describe the clinical features of cord prolapse

A
  • always consider in non reassuring fetal heart rate patterns and absent membranes
  • confirmed by external inspection or on digital vaginal examination
  • fetal heart rate patterns may be subtle eg decelerations w/ contractions or bradycardia
  • no vaginal bleeding- suspect placental aburption or vasa praevia if this is the case
42
Q

How should cord prolapse be managed?

A
  • avoid handling the cord to prevent vasospasm
  • if in community, fill bladder with 500ml warmed normal saline via catheter and transfer to hospital
  • manually elevate the presenting part
  • encourage left lateral position with head down and pillow under left hip or knee- chest position
  • consider tocolysis with terbutaline- if delivery not imminently available / to save time for prep for c section
  • emergency c section unless: fully dilated and vaginal delivery appears imminent OR at threshold for viability (23- 24+6 weeks)- discuss expectant management due to significant maternal morbidity w/ c section and poor fetal outcomes
43
Q

What is eclampsia?

A

Seizures occurring in pregnancy or within 10 days of delivery, with at least two of the following documented within the last 24 hrs:

  • HTN
  • proteinuria
  • thrombocytopenia
  • raised transaminases
44
Q

Describe the clinical features of eclampsia?

A
  • new onset tonic clonic type seizures
  • in presence of pre- eclampsia (HTN + proteinuria after 20 weeks gestation)
  • headache
  • hyper reflexia
  • n+v
  • generalised odema
  • RUQ pain + jaundice
  • visual disturbance
  • change in mental state
45
Q

Give 5 complications of eclampsia

A
maternal:
- HELLP syndrome
- DIC
- AKI
- ARDS
- cerebrovascular haemorrhage
- permanent CNS damage
- death
Fetal complication:
- IUGR
- prematurity
- infant respiratory distress syndrome
- intrauterine fetal death
- placental abruption
46
Q

Give 5 differentials for seizures in pregnancy

A
  • hypoglycaemia
  • pre existing epilepsy
  • head trauma
  • haemorrhagic stroke
  • meningitis
  • medication induced
  • brain tumour
  • cerebral aneurysm
  • septic shock
  • ischaemic stroke
  • metabolic disturbance
47
Q

How should eclampsia be investigated?

A
  • blood glucose
  • fbc
  • u+e
  • lft
  • clotting studies
  • abdo USS- rule out abprution which can complicate
  • CTG monitoring
  • full neuro workup inc CT/ MRI head may be needed of atypical features
48
Q

How should eclampsia be managed?

A
  • resus
  • stop seizures (magnesium sulphate 4g in 100ml NS, then 1g hrly for 24hrs for maintanence, 2g bolus for recurrent seizures)
  • BP control: IV labetalol or IV hydralazine w/ continious CTG monitoring
  • prompt delivery of baby and placenta AFTER mother is stable, c section is ideal but seizures in established labour may need vaginal delivery, then HDU until shes stable
  • monitoring: fluid balance to prevent pulmonary odema and detect AKI, and for complications
49
Q

Describe the postnatal care and follow up for eclampsia

A
  • regular symptom rv
  • blood 72 hrs post partum
  • pre ceonceptual counselling (reduce RFs and prophylaxis for future pregnancies)
  • CT head if persistent neuro deficit
  • BP check 2 weeks post partum
  • follow up appt 6 weeks post partum (check BP, proteinuria, FBC, lft, creatinine)
50
Q

What are the two types of uterine rupture?

A
  • incomplete: peritoneum overlying the uterus is intact, uterine contents remain within the uterus
  • complete: peritoneum is also torn, uterine contents can escape into the peritoneal cavity. Tear can extend
51
Q

What are the clinical features of uterine rupture

A
  • sudden severe abdo pain which persists between contractions
  • shoulder tip pain
  • vaginal bleeding
  • may have regression of presenting part
  • scare tenderness and palpable fetal parts
  • hypovolaemic shock
  • fetal distress of absent heart sounds
52
Q

How is uterine rupture diagnosed?

A
  • USS if pre labour

- often diagnosed during C section indicated by fetal distress

53
Q

how is uterine rupture managed?

A
  • A-E resus

- C section and then uterus either repaired or removed

54
Q

What is the difference between minor and major primary PPH

A

Minor: 500ml-1000ml
Major= >1000ml (moderate 1L-2L, Massive if >2L or >150mls/ min)
Per vagina and within 24 hrs of delivery

55
Q

What are the 4 causes for primary post partum haemorrhage

A

4 Ts:

  • tone
  • tissue (retention of placental tissue, placenta praevia/ abruption if not picked up// on delivery after discovering it)
  • trauma (eg from c section, episiotomy, instrumental delivery)
  • thrombin (coagulopathy eg VWD, Haemophilia, DIC, HELLP) or vascular pathology (abruption, HTN, pre- eclampsia)
56
Q

What is uterine atony and what are the 4 major RFs

A
  • where uterus doesnt contract adequatly following delivery, is the commonest cause of primary PPH
  • Maternal profile (age >40, BMI >35, asian)
  • uterine over distension (multiple pregnancy, polyhydramnios, fetal macrosomia)
  • labour (induction, prolonged >12 hrs)
  • placenta problems (placenta praevia, abruption, previous PPH)
57
Q

Describe the clinical features of primary post partum haemorrhage

A
  • PV bleeding
  • dizziness, palpitations and SOB
  • prolonged cap refill tachycardia, hypotension
  • abdo exam pay show signs of uterine rupture
  • speculum may show sites of local trauma
  • examination of placenta to ensure the placenta is complete (a missing cotyledon or ragged membranes could both cause PPH
58
Q

How should primary PPH be managed generally?

A
  • involve appropriate ppl eg midwives, obstetricians, anaesthetists, blood bank, haematologist
  • investigations and monitoring (fbc,clotting, xmatch, u+e, RR, O2, sats, HR, BP, temp)
  • consider central venous line
  • obs every 15 mins
  • warmed crystalloid infusion
  • consider catheter
  • definitive management depends on cause
59
Q

How is uterine atony managed?

A
  • bimanual compression to stimulate uterine contraction (first inside anterior fornix then compress uterus against it from above
  • pharmacological management to increase myometrial contraction (ergometrine, synotocinon infusion, carboprost into myometrium, mistoprostal rectually)
  • surgical: intrauterine balloon tamponade, haemostatic suture around uterus (b- lynch stitch), bilateral uterine or internal iliac artery ligation, hysterectomy as last resort
60
Q

How should trauma and tissue and thrombin causes of primary PPH be managed?

A
  • repair laceration. laparotomy and repair or hysterectomy for uterine rupture
  • Tissue: IV oxytocin, manual removal of placenta with regional or general anaesthetic and prophylaxtic abx in theatre. Start Iv oxytocin infusion after removal
  • thrombin: correct coagulation abnormalities with blood products under advice of haematology
61
Q

What drugs are used in PPH?

A
  • syntocinon (synthetic oxytocin)- IM/IV 15-30 mins duration, no contraindications
  • ergometrine -IM, 1-2 hrs duration, contraindicated by cardiovascular disease
  • carboprost (prostaglandin analogue)- IM, 1-2 hrs action, CVD contraindicates
  • misoprostol (prostaglandin analogue)- oral, 1-2 hrs no contraindicationsn
62
Q

How is PPH risk reduced?

A
  • in 3rd stage of labour:
  • women delivering vaginally get 5-10 units IM oxytocin prophylactically
  • women delivering via c section should get 5 units IV oxytocin prophylactically
63
Q

How should depression during and post pregnancy be managed?

A

Social support and psychological treatments are recommended. When considering antidepressant treatment, it is important to acknowledge that no antidepressant is without risks in pregnancy and breastfeeding. Published guidelines recommend different antidepressants for use in pregnancy, so it may be best to seek advice from pharmacy colleagues or specialist Perinatal Psychiatry Services.

64
Q

What is the definition of post natal depression?

A
  • a depressive episode within the first 12 weeks postpartum
  • distinguished from baby blues: low mood and irritabilith which starts 3-4 days after birth and lasts for about a week, doesnt need treatment
65
Q

describe the clinical features of post partum/ puerperal psychosis

A
  • can develop rapidly over a few hrs
  • more common if previous mental health problems
  • more likely if theyve had it in their last pregnancy
  • may become quiet and withdrawn or agitated and distressed
  • may express bizzarre ideas, grandiose or paranoid delusions
  • auditory hallucinations
  • mania
  • sleep disturbance
66
Q

How should post partum psychosis be managed?

A
  • thorough risk assessment and usually need inpatient admission in specialist mother and baby units
  • usually need antipsychotics and/ or mood stabilisers
  • prognosis is usually good but can take 6-12 months to recover
  • need to monitor closely in further pregnancies as recurrance rate is 50%
67
Q

What are the 4 main causes of secondary PPH

A
  • uterine infection (endometritis- RFs: c section, premature rupture of membrane, long labour)
  • retained placental fragments or tissue
  • abnormal involution of placental site (inadequate closure and sloughing of spiral arteries)
  • trophoblastic disease (very rare)
68
Q

What examination findings suggest endometritis and retained placenta?

A

endometritis: foul smelling iochia (normal uterus discharge) and lower abdo tenderness
retained placenta: uterus may be high

69
Q

What investigations are needed for secondary PPH

A
  • bloods (fbc, u+e, clotting, crp, coagulation, g+s, blood cultures)
  • high vaginal swab
  • USS- can be used to diagnose retained placental tissue
70
Q

How should secondary pph be managed?

A
  • antibiotics: ampicillin and metronidazole
  • gent also if endomyometritis (tender uterus) or over sepsis
  • uterotonics (syntocinon (oxytocin), misprostol, carboprost etc)
  • balloon catheter insertion may be effective
  • if massive haemorrhage, resus and surgical interventions to stop bleeding will be indicated
71
Q

What can SSRI use in pregnancy cause?

A

pulmonary htn and cardiac defects - can still use despite this

72
Q

What 3 factors need to be considered when assessing for delay or prolonged labour?

A

Power: could be due to poor or uncoordinated uterine contractions
Passenger: large fetal head, mal presentation (brow, face or shoulder or malposition), twin pregnancy
Passage: bony (contracted or deformed), soft tissue (adhesions, full bladder, pelvic tumour, scars)

73
Q

What fluids are given in PPH and when?

A
  • crystallioid- for all, warmed
  • blood- give o-ve then group specific as soon as possible
  • FFP if PT or APTT prolonged and haemorrhage ongoing OR 4 units of PRBCS have been givem
  • Platelet conentrates-1 pool if haemorrhage on going and platelet count <75
  • Cryopreciptate- 2 pools if haemorrhage ongoing and fibrinogen <2
74
Q

How should reduced fetal movements be managed?

A
  • auscultate fetal heart
  • if not present-> USS to diagnose IUFD
  • if present –> CTG to exclude imminent fetal compromise
  • If CTG suspicious manage as per protocol
  • if CTG ok and no risk factors for still birth, reassure
  • if CTG but RFs, do USS for amniotic fluid volume and fetal growth
75
Q

What is HELLP syndrome and how is it managed?

A
  • disease characterised by haemolysis, elevated LFTs and low Platelets
  • carries risk of DIC, AKI, abruption
  • associated with pre eclampsia
  • management is to deliver the baby and reduce the mothers BP
76
Q

How should preterm labour with intact membranes be diagnosed?

A
  • if <30 weeks- on clinical assessment alone
  • if >30 weeks- do TV USS, if cervix >15mm then not labour, if <15 mm then it is
  • can do fetal fibronectin test as alternative to TV USS (>50ng/ml= positive and in preterm labour)
77
Q

How should preterm labour with intact membrane be managed?

A
  • consider nifedipine for tocolysis if between 24 and 34 weeks
  • oxytocin receptor antagonists for tocolysis if nifedipine contraindicated
  • Consider ‘rescue’ cervical cerclage for women between 16+0 and 27+6 weeks of pregnancy with a dilated cervix and exposed, unruptured fetal membranes, contraindicated by contractions, active vag bleeding, infections
  • maternal corticosteroids for 24-48 hrs if <34 weeks and in suspected or established labour, planned labour or PPROM
  • Corticosteroids if going for c section<39 weeks
  • MgSO4 for 24hrs or until birth if in established preterm labour at <34 weeks
78
Q

What are early, late and variable decelerations on CTG associated with?

A
Late= fetal hypoxia
variable= cord compression
Early= head compression