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Physical Dx > L9: Cardiovascular > Flashcards

L9: Cardiovascular Flashcards

1
Q

Precordium

A

Precordium: anterior chest wall overlying the heart

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2
Q

Apical Impulse

What is it also called?

What is it?

A

Apical Impulse, AKA Point of Maximal Impulse (PMI)

the point on the precordium farthest outwards (laterally) and downwards (inferiorly) from the sternum at which the cardiac impulse can be felt.

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3
Q

Jugular venous distension (JVD)

What is it?

A

occurs when the pressure inside the vena cava, a large vein that carries deoxygenated blood back to the heart, causes a bulging neck vein

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4
Q

Deformity of the Chest Wall

Pectus excavatum & Pectus carinatum

What are they?

Common/Uncommon?

A

Pectus excavatum (common): sternal depression

Pectus carinatum (uncommon): sternum protrudes

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5
Q

Skin Abnormalities:

Herpes Zoster

What can it mimic?

A

Can often mimic MI

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6
Q

Anatomy Review

Apex & Base of Heart

Location?

A

Base: Superior aspect of the heart, where the great vessels originate

Apex: Inferolateral tip of the left ventricle

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7
Q

Stethoscope: Bell & Diaphragm

Which is best for high- or low-pitched sounds?

Firm or light pressure?

A
  • Use both the diaphragm & the bell
  • Diaphragm
    • Best for high-pitched sounds of S1 and S2
    • Press firmly against the chest wall
  • Bell
    • Best for low-pitched sounds of S3 and S4
    • Apply lightly, with just enough pressure to make a seal against the chest wall
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8
Q

Auscultation of Cardiac Areas

What are the the four key areas?

Where are they located?

What is an additional location?

A
  • Four key areas:
    • Aortic – 2nd ICS, RSB
    • Pulmonic – 2nd ICS, LSB
    • Tricuspid – 4th & 5th ICS, LSB
    • Mitral (apex) – 5th ICS, MCL
  • Others as needed
    • Second Pulmonic – 3rd ICS, LSB
    • “Inch” stethoscope along
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9
Q

Angle of Louis

What is it?

How to find it?

A

The sternal angle (angle of Louis) is the anterior angle formed by the junction of the manubrium and the body of the sternum which varies around 162 degrees in males.

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10
Q

Auscultation Rate

Normal, Fast, Slow

What is considered a normal/slow/fast rate?

What is a normal/slow/fast rate called?

Types?

A
  • Normal
    • 60 – 100 bpm
    • Sinus rhythm or flutter with normal rate, or second-degree AV block
  • Fast
    • >100 bpm
    • Tachycardia - sinus, supraventricular, ventricular, or flutter
  • Slow
    • <60 bpm
    • Sinus bradycardia, second- degree AV block, complete heart block
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11
Q

Auscultation: Irregular Rhythm

Types of irregular rhythms?

A
  • Irregular (arrhythmia or dysrhythmia)
    • Rhythmically irregular
    • –Sporadically irregular (occasional vs. frequent)
    • Irregularly irregular
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12
Q

Valves

What ar the atrioventricular (AV) valves?

What are the semilunar valves?

What structures do they connect?

A
  • Atrioventricular (AV) valves
    • Tricuspid
      • R atrium → R ventricle
    • Mitral
      • L atrium → L ventricle
      • Semilunar valves
    • Aortic
      • L ventricle → aorta → body
    • Pulmonic
      • R ventricle → pulmonary artery → lungs for oxygenation
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13
Q

What is the path of blood through the heart?

A
  • Venous blood arrives in R atrium
  • Through tricuspid valve → R ventricle
  • Through pulmonary valve → pulmonary artery → lungs
  • Oxygenated blood returns to L atrium via pulmonary veins
  • Through mitral valve into L ventricle
  • Through aortic valve into aorta → to rest of body
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14
Q

Cardiac Cycle:

“Lub-Dub”

When is each sound heard?

Diastole/systole is associated with which sound?

A
  • “Lub”
    • Closure of AV valves (tricuspid & mitral) very soon after systole begins
  • “Dub”
    • Beginning of diastole upon closing of semilunar valves (aortic & pulmonary)
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15
Q

First Heart Sound (S1)

Systole or Diastole?

What is the sound?

What causes the sound?

A
  • Systole:
    • Closure of mitral & tricuspid (AV) valves produce the S1 sound → “Lub”
      • Louder than S2 at the apex
      • Contraction of ventricles
    • Aortic & pulmonic valves forced open, and blood is ejected into the arteries
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16
Q

Ventricular Systole

What occurs during ventricular systole?

A
  • Ventricles contract
  • Blood pushed against AV valves (causes them to close)
  • Contracting papillary muscles and chordae tendinae prevent valve claps from everting into atria
  • Semiluar valves (aortic & pulmonic) forced open → blood ejected into the arteries (pulmonary trunk & aorta)
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17
Q

Abnormal S1

Accentuated S1

Diminshed S1

Louder/softer?

Cause?

Examples?

A
  • Accentuated (Louder) S1:
    • Due to diseased AV valve or more forceful closure of AV valve
    • E.g., Tachycardia, fever, HTN, exercise, anemia, hyperthyroidism, or mitral stenosis
  • Diminished (Softer) S1:
    • Due to weak contraction of heart or reduced sound transmission
    • E.g., thick chest wall or emphysematous lungs
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18
Q

Second Heart Sound (S2)

Systole or Diastole?

What is the sound?

What causes the sound?

A
  • Diastole:
    • Closure of the aortic & pulmonic valves produce the S2 sound → “Dub”
      • Louder than S1 at the base
    • Relaxation of heart
    • AV valves open, allowing ventricles to start to refill passively
    • Atrial contraction (kick)
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19
Q

Ventricular Diastole

What occurs during ventricular systole?

A
  • Ventricles relax and fill with blood both:
    • Passively
    • Then by atrial contraction as AV valves remain open
  • During ventricular relaxation, some blood from the aorta & pulmonary trunk flows back toward the ventricles filling the semilular valve cusps → forcing them to close
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20
Q

Aortic Valve

What is the purpose of the cusps?

A
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21
Q

Which is longer, systole or diastole?

A

Diastole slightly longer than systole

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22
Q

Spliting of S2: Physiologic Splitting

What are the two components?

Heard during inspuration or expiration?

Which sound is heard first? Why?

A
  • May hear two discernible components of S2, during inspiration
    • A2 (aortic valve closure)
    • P2 (pulmonic valve closure)
  • Similar events occur on left & right sides of the heart
    • Right-sided pressures are lower than corresponding pressures on the left → sounds occur slightly later on the right
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23
Q

Spliting of S2: Pathologic Splitting

A
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24
Q

Third Heart Sound (S3)

What is it?

What is it caused by?

What is it also called?

A
  • Low-pitched sound created in early diastole by passive, rapid filling of the ventricles with blood from the atria
    • Produced by blood filling a chamber that is already volume overloaded, causing rapid distension of the ventricular walls, leading to vibration
    • S1 + S2 + S3 = ventricular gallop rhythm
  • Heard best with bell at apex
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25
Q

Third Heart Sound (S3)

When is it non-pathologic? Pathologic?

A
  • Non-pathologic
    • Children, healthy young adults, and pregnant women may have a non-pathological S3
  • Pathologic S3 (ventricular gallop)
    • Over age 40, usually pathologic
    • Due to heart failure, anemia, volume overload of ventricle, decreased myocardial contractility
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26
Q

Fourth Heart Sound (S4)

What is it?

What is it caused by?

What is it also called?

A
  • Low-pitched sound created by second phase of ventricular filling in diastole, as the atria contract and eject blood into the ventricles (during the “atrial kick”)
    • Produced by the rush of blood causing vibration of valves, papillary muscles, and ventricular walls
    • S1 + S2 + S4 = atrial gallop rhythm
  • Heard best with bell at apex
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27
Q

Fourth Heart Sound (S4)

When is it non-pathologic? Pathologic?

A
    • May be normal in trained athletes and some older individuals without other heart disease
  • Pathologic S4 (atrial gallop)
    • Over age 40, usually pathologic
    • Due to resistance to ventricular filling; stiffness of heart muscle (reduced compliance)
      • HTN, CAD, AS, cardiomyopathy
      • Right-sided S4 from pulmonary hypertension or pulmonary stenosis
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28
Q

Murmur

What is a murmur?

What causes murmur?

A
  • Murmur: prolonged heart sound made by blood rushing through
    • Narrowed valve
    • Leaking valve
    • Wall between chambers of the heart
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29
Q

Cardiac Chamber Pressures

Which has a higher/lower pressure?

Atria vs ventricles

Left vs right side

A
  • Higher pressure on left side
  • Higher pressue in ventricles
30
Q

Gradations of Murmurs

What are the 6 grades of murmurs?

A

Grade 1/6: Barely audible in quiet room

Grade 2/6: Quiet, but clearly audible

Grade 3/6: Moderately loud

Grade 4/6: Loud, associated with thrill

Grade 5/6: Very loud, heard with stethoscope partially off chest; obvious thrill

Grade 6/6: Very loud, heard with stethoscope entirely off the chest, obvious thrill

31
Q

Systolic Murmurs

What are the types of systolic murmurs?

Sound type?

Cause?

32
Q

Aortic Stenosis

Cause of stenosis?

What type of murmur does it cause?

Where best heard?

Cause of sound?

A
  • Aortic Stenosis
    • Narrowed valve: congenital or calcific disease process
  • Type:
    • Mid-systolic crescendo-decrescendo murmur
  • Heard best at
    • base along right & left sternal borders
  • Cause of sound:
    • From LV, blood flows through aortic valve, into aorta → murmur gets louder* → aortic pressure increases as more blood flows into it from LV, which creates resistance to further inflow from the LV → murmur *softens due to reduced blood flow
    • From RV, blood flows through pulmonic valve, into pulmonary artery, etc.
      • Pressure is not as great as in left-sided system, but similar principles apply
33
Q

Innocent Systolic Murmur

What is it?

Common in?

Physiologic in…

Chracteristic features?

A
  • Common in children & young adults
  • Physiologic in pregnancy, anemia, fever hyperthyroidism (increased flow across valve)
  • Characteristic features:
    • Grade ≤2 intensity
    • Softer when sitting (vs. supine)
    • Short systolic duration (not holosystolic or diastolic)
    • Minimal radiation
    • Musical/vibratory quality
34
Q

Atrial Septal Defect (ASD)

What is an ASD?

What type of murmur does it cause?

A
  • Atrial Septal Defect: Congenital anomaly resulting in left-to-right shunting of blood (LA → RA), with RV enlargement & increase flow through pulmonic valve
  • Midsystolic murmur (2nd ICS)
  • Wide, fixed splitting of S2
35
Q

Mitral/Tricuspid Regurgitation

Cause of regurgitation?

Type of murmur caused?

A
  • Pressure in the left & right ventricles is significantly higher than in the atria
  • When ventricles contract, leakage of blood from LV to LA (or RV to RA) continues throughout systole
    • There is no resistance to the flow
36
Q

Ventricular Septal Defect (VSD)

Cause of defect?

Type of murmur caused?

Sound heard?

A
  • Pansystolic (or holosystolic) murmur
  • Left-to-right shunt (LV → RV)
  • Harsh or blowing pansystolic murmur at LLSB
  • LLSB Thrill
37
Q

Diastolic Murmurs

What are the types of diastolic murmurs?

Sound type?

Cause?

38
Q

Aortic Regurgitation

Cause of regurgitation?

Type of murmur caused?

Where best heard?

A
  • Early diastolic decrescendo murmur
  • Leaking aortic valve
  • Heard best at apex
39
Q

Mitral/Tricuspid Stenosis

Cause of stenosis?

Type of murmur caused?

A
  • Middiastolic Murmur
  • Narrowed valve
40
Q

Mitral Stenosis

Cause of stenosis?

What type of murmur does it cause?

A
  • Opening snap & diastolic rumble
  • Narrowed valve
    • Obstructs flow from the LA to LV
41
Q

Systolic-Diastolic Murmur

What is it?

A
  • aka “To-and-fro” murmur
  • Aortic stenosis with aortic regurgitation
    • Obstruction to outflow due to a narrowed valve and
    • Failure of complete closure of the aortic valve during diastole, with leakage of blood back into the left ventricle
42
Q

Continuous vs. “To-and-fro” Murmurs

A
  • Continuous
    • Patent ductus arteriosus (machinery-like)
    • –Failure of channel between aorta & pulmonary artery to close after birth
  • “To-and-fro” (systolic-diastolic murmurs)
    • Aortic stenosis/regurgitation
    • Severe aortic regurgitation
43
Q

Specialized Exam: Ausculate at cardiac base, leaning forward

How to perform?

What are you listening for?

A
  • Patient in seated position:
    • Ask patient to lean forward, exhale completely & hold breath in expiration
    • Listen at the base
    • Best for hearing soft murmurs at the base:
      • –Aortic regurgitation
      • –Pulmonic regurgitation
    • Remember to let the patient breathe
44
Q

Valsalva

What is it?

A

The Valsalva maneuver is a breathing method that may slow your heart when it’s beating too fast. To do it, you breathe out strongly through your mouth while holding your nose tightly closed. This creates a forceful strain that can trigger your heart to react and go back into normal rhythm.

45
Q

Effects of Maneuvers:

Standing (or strain phase of valsalva)

Squattting (or release phase of valsalva)

On

Ventricular volume

Vascular tone

BP

PVR (peripheral vascular resistance)

Exception?

46
Q

Extra Sounds:

Ejection Sound

What is it?

What is the sound?

Cause?

Heard best with…

A
  • Aortic or pulmonic ejection click
    • High-pitched; indicates valve disease or dilated aorta or pulmonary artery, or pulmonary hypertension
  • Heard best with diaphragm of stethoscope
47
Q

Extra Sounds:

Systolic Click(s)

What is it?

Cause?

Sound?

How common?

A

Mitral Valve Prolapse

  • Ballooning of mitral leaflet(s) into the left atrium during systole
  • Mid-late systolic click(s) are often present
  • Variable pitch
  • Mitral regurgitation may also occur, with late systolic murmur
  • Common condition: over 5% of general population; usually benign
48
Q

Extra Sounds:

Extracardiac Sounds

Venous Hum

Pericardial Friction Rub

What are they?

Cause?

Sound?

A
  • Venous hum
    • Turbulent blood flow through jugular veins
    • Both systolic and diastolic sounds
    • Common in children
  • Pericardial friction rub
    • Inflammation of pericardial sac
    • 3 component (triphasic)
      • Scratchy/squeaky, intermittent
49
Q

Jugular Venous Pressure (JVP)

What is it?

What is it used to evaluate for?

A
  • Indication of pressure in the right atrium (central venous pressure), related to intravascular fluid volume
    • Mainly reflects right heart function
  • Used in the evaluation of heart failure
  • Best assessed from pulsations in the right internal jugular vein (or external jugular vein)
    • Deep to SCM muscles in neck
  • Identify surface pulsations
    • Dominant movement is inward (carotid pulse is outward)
50
Q

Internal Jugular vs. Carotid Pulsations

Palpable?

Describe the pulsation

Are pulsations eliminated by pressure on the veins?

Does height of pulsations change with position?

Does height of pulsations change with inspiration?

51
Q

Specialized Exam: Measurement of JVP

How to measure?

A
  • Stand on patient’s right, exam table at 30°
  • Patient’s head turned slightly to the left
  • Find the highest point of oscillation (meniscus) in the right internal jugular vein
  • Measure vertical distance above sternal angle
    • Add 5cm
    • Sum = JVP
  • In each position, the sternal angle remains roughly 5 cm above the right midatrium
52
Q

Abnormal JVP

Elevated

Decreased

Definition?

Cause?

Hypervolemia or Hypovolemia?

A
  • Elevated (>8 cm above the RA)
    • Heart failure
    • Pulmonary HTN
    • Increased venous vascular tone
    • Pericardial tamponade
    • Hypervolemia: anticipate ↑ JVP, raise head of bed
  • Decreased
    • Blood loss
    • Hypovolemia
    • Decreased venous vascular tone
    • Hypovolemia: anticipate ↓ JVP, lower head of bed
53
Q

Specialized Exam: Hepatojugular (abdominojugular reflex)

How to do the test?

What is a positive test?

A
  • Patient is supine, exam table at 30°
  • Apply firm, sustained pressure (~10 sec) to the abdomen in the RUQ over the liver region
  • Observe the neck for an increase in JVP, followed by a decrease as the hand is released
  • JVP will transiently increase in all patients with this maneuver, but it is exaggerated in right heart failure
  • > 3 cm increase or remains elevated is abnormal (positive)
54
Q

Thrills

Define

Cause?

What to do if present?

A
  • Thrill: Buzzing or vibratory sensation
    • May be caused by vigorous blood flow through any narrowed opening (e.g., aortic stenosis, ventricular septal defect, etc.)
  • If present, auscultate area for murmur
55
Q

Lifts (or heaves)

Define

Cause?

A
  • Lift (or Heave): Vigorous cardiac impulse that can be seen/felt through the chest wall
    • May be caused by ventricular hypertrophy or hyperdynamic ventricular activity
56
Q

Where to check for lifts (heaves) and thrills?

A

Left Sternal Border (LSB) and Base

57
Q

Where to palpate for apical impulse?

A

Palpate Apex

  • Place right hand on chest as pictured, with heel of hand on lower sternum and fingertips at apex
  • Palpate for apical impulse
    • PMI (point of maximal impulse)
      • Represents pulsation of left ventricle
    • Note size (5th ICS in MCL)
      • Should not exceed 2.5 cm in diameter one intercostal space
      • Cardiac impulse lateralto MCL or largerthan normal suggests left ventricular enlargement
  • Most prominent impulse may be in the xiphoid or epigastric area due to right ventricular hypertrophy in patients with COPD
58
Q

Specialized Exam: Cardiac Percussion

What is this test for?

How to test?

What is it useful?

A
  • Limited value
  • Estimate cardiac size (when PMI not palpable)
    • Begin at about 5th ICS in the midaxillary line and percuss medially, listening for onset of dullness
    • More useful, evident with cardiac pathology → cardiomegaly, pericardial effusion, etc.
59
Q

Specialized Exam: Auscultate at cardiac apex with bell, left lateral decubitis

How to examine?

What is this test for?

A
  • Patient in left lateral decubitus position
  • Listen at the apex
  • Best for hearing low-pitched filling sounds:
    • Gallops (S3, S4)
    • Murmurs
      • E.g., mitral stenosis
60
Q

Grading Peripheral Pulses

How are they graded?

A

3+ Bounding

2+ Brisk, expected (normal)

1+ Diminished, weaker than expected

0 Absent or unable to palpate

61
Q

Pulse Pressure

What is it?

How is pulse pressure calculated?

A

Systolic minus diastolic pressure

(Pulse pressure is the difference between the upper and lower numbers of your blood pressure. This number can be an indicator of health problems before you develop symptoms. Your pulse pressure can also sometimes that you’re at risk for certain diseases or conditions)

62
Q

Carotid Artery Palpation

  • Location of carotid arteries?
  • Carotid upstroke?
    • Duration?
    • Character/quality/describe? When?
  • Abnormal carotid upstroke?
    • What conditions is it seen in?
    • What is the abnormality?
A
  • Palpate (one at a time!)
    • Just inside medial border of relaxed SCM muscle, roughly adjacent to cricoid cartilage
      • Avoid pressing on carotid sinus
        • Adjacent to top of thyroid cartilage
  • Carotid upstroke:
    • Brisk
    • Smooth, rapid, almost immediately follows S1
  • Presence of thrills
  • Abnormal carotid upstroke
    • Cardiogenic shock: small, thready, or weak
    • Aortic Regurgitation: bounding
    • Aortic stenosis: delayed
63
Q

Bruits

A

Bruits: a murmur-like sound arising from turbulent arterial blood flow

64
Q

Where to check brachial, radial & ulnar arteries?

A

Brachial: At the bend in elbow, medial to biceps tendon

Radial: Lateral flexor surface

Ulnar: Medial flexor surface

65
Q

Specialized Exam: Allen Test

How to do?

What does it test for?

What is an abnormal test?

A
  • Used to evaluate patency of ulnar artery
    • Prior to puncturing the radial artery for blood gas evaluation
  • Ask patient to make a tight fist
  • Compress both radial & ulnar arteries
  • Ask patient to open hand (pale)
  • Release pressure over ulnar artery
  • If patent (normal), palm flushes within 3-5 sec
66
Q

Abdominal Aorta

How to palpate?

What is an abnormal size?

A
  • Use two hands, above umbilicus
  • Slightly left of midline
  • Identify aortic pulsations
  • Estimate size
    • Adults >50 years (abnormal: >3 cm)
67
Q

Abdominal Bruits

Where to auscultate for abdominal bruits?

A
  • Auscultate for bruits:
    • Aorta
    • Renal arteries
    • Iliac arteries
    • Femoral arteries
68
Q

Femoral, popliteal, dorsalis pedis, & posterior tibial arteries

Where to palpate?

A

Femoral: Below inguinal ligament, midway between ASIS & symphysis pubis

Popliteal: Medially behind femur

Dorsalis Pedis (DP): Dorsum of the foot, just lateral to extensor tendon of great toe

Posterior Tibial: Behind medial malleolus of ankle

69
Q

Swelling & Edema

What to do if swelling or edema is present?

A
  • If swelling or edema is present, palpate for pitting edema
    • Press firmly, but gently with thumb at least 2 seconds
      • Over dorsum of each foot
      • Behind each medial malleolus
      • Over the shins
70
Q

Specialized Exam: Homan Sign

What does it test for?

How to do exam?

A
  • Calf pain on passive dorsiflexion of the foot
  • Unreliable for presence of DVT
71
Q

Where do the following veins run?

Femoral vein

Great saphenous vein

Small saphenous vein

Perforating vein

Physical Dx (11 decks)

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