L10 Ear Infections Flashcards
Why are infants more likely to get ear infections?
Their eustachian tube is more horizontal (nose to ear drainage)
“Swimmers ear”
otitis externa
Cause of otitis externa
heat/moisture leads to swelling and maceration of EAC allowing bacteria to enter, trauma, skin diseases (eczema, psoriasis, seborrheic dermatitis)
Etiology of Otitis Externa
Bacterial (most common) or fungal
Bacteria causing otitis externa
pseudomonas aeruginosa (38%), staph epidermidis (9%), staph aureus (8%)
Fungus causing otitis externa
Asperigillus niger or Candida albicans
Clinical presentation of otitis externa
ostalgia (worse with movement of tragus and pinna), pruritus (mild/severe), discharge, EAC may be erythematous and edematous, decreased hearing or swelling (conductive due to d/c or swelling)
Yellow discharge with OE
staph infection
Green discharge with OE
pseudomonas infection
Fluffy white/black “bread mold” discharge with OE
fungal infection
Otomycosis
fungal OE
Tx for OE
Cortisporin otic suspension (polymixin B, neomycin, hydrocortison), Floxin otic solution (ofloxacin), ciprodex or CiproHC (ciprofloxacin + glucocorticoid)
First line treatment for OE
Cortisporin otic solution
Treatment of OE with perforated TM
floxin otic solution (ofloxacin)
Side effects of Cortisporin otic solution
allergic rxn.- contact dermatitis, DON’T USE WITH TM PERFORATION
Why are otic suspensions better than solution?
less acidic (pH 5 vs. pH 3.4), so less irritation to infected tissues
Tx for fungal OE
Clotrimazole 1% solution (BID x 14 days), Acidifying solution (acetic acid)
Tx for marked swelling in EAC
ear wick (Oto-wick)- apply medication to wick TID-QID; remove wick after 48-72 hours and continue meds as directed
Management of OE
pain control, keep canal dry (no swimming 7-10d, cotton in ear when bathing)
How soon do OE usually resolve
5-7 days
If no improvement of OE in 48-72 hours what do you consider?
noncompliance, otomycosis, periauricular cellulitis, or malignant otits externa
Prevention of OE
prophylaxis: 2% acetic acid (Vosol), vinegar soluiton, isopropyl alcohol or hair dryer; bathing cap or custom ear plugs
Malignant OE
necrotizing external otitis
Cause of MOE
infection spreads from skin of EAC to skull base (pseudomonas)
Who is most at risk for MOE
elderly diabetics & immunocompromised patients
Clinical presentation of MOE
otalgia and otorrhea not responsive to OE tx; pain nocturnal and with chewing; red granulation tissue in EAC; possible periauricular lymphadenopathy, edem and trismus; watch for CN involvement (indication of progressive osteomyelitis)
Dx of MOE
CT!!! (bone erosion), ESR and CRP elevated; MRI
Tx of MEO
ADMIT, C&S of ear discharge, IV Ciprofloxacin, possible debridement
Complications of MOE
intracranial spread leading to meningitis, brain abscess and sepsis; mortality 10-20%
OME
otitis media w/ effusion
other names of OME
serous, secretory or nonsuppurative OM
Middle ear effusion in the absence of acute sxs
Otitis Media w/ Effusion
acute illness with middle ear fluid and s/sx of middle ear inflammation
Acute otitis media (AOM, suppurative otitis media)
Chronic infection of middle ear with non-intact TM and otorrhea
Chronic otitis media
Sx of OME
ear fullness and decreased hearing; usually painless
Etiology of OME
recent AOM, URIs, allergies, eustachian tube dysfunction, barotrauma, or nasopharyngeal carinoma
Clinical presentation of OME
patient afebrile, amber-colored (straw) fluid behind TM, air-fluid levels and bubbles, neutral or retracted TM; possible conductive hearing loss, TM not moving with pneumatic otoscopy, Tympanogram (Type B Pattern)
Recurrent OME could be a sign of what
nasopharyngeal carcinoma - REFER TO ENT TO RULE OUT
Type B tympanogram
little to no movement of TM; suggests OME
Tx of OME
usually resolves spontaneously, re-evaluate in 4-6 weeks (“watchful waiting,” intranasal steroids if underlying allergic rhinitis; refer to ENT for tympanostomy “T-tubes” if: persistent fluid and/or hearing loss >3 months, child considered at risk for speech, language or learning problems