knee presentations Flashcards
Patellar fx epidemiology
1% of all fxs
most common 20-50 yo
Males 2x>F
>50% non-displaced
Patellar fx hx
MOI: fall on A. knee
sudden quad activation
Patellar fx sxs
pain/inability to extend knee
A. knee pain
Patellar fx exam
-palpable gap at fx site (may or not)
-local tenderness
-painful RT> AROM for ext
-painful end range flex ROM
-antalgic gait
Pittsburgh knee decision rule
- hx blunt trauma/fall
- inability to WB x4 steps immediately and in ED
- age <12 y/o OR >50
Ottawa knee decision rule
TTP head of fibula
inability to WB x4 steps immediately or in ED
Age >/= 55
inability to flex knee 90
isolated TTP patella
Tendon rupture: patellar and quad epidemiology
patellar: <40 yo commonly
quad: >40 yo commonly
quad: M 4-8x>F
Patellar and quad tendon rupture rfs
-local steroid injection
-prolonged corticosteroid use
-RA
-lupus
-connective tissue diseases
-infectious diseases
-arteriosclerosis
-DM
-hyperthyroidism
Patellar and quad tendon rupture hx
-eccentric overload extensor mechanism/trauma
-sudden onset from fall, hemarthrosis common
—-quad: related to regaining balance/rapid quad contraction
—-patellar: jump landing common
-hx degen. tendinopathy
-hx TKA
-ACL reconstruction (patellar tendon graft)
Patellar and quad tendon rupture sxs
anterior knee pain
Tendon rupture exam
-absent active knee ext OR painful active knee ext (complete/ partial)
-painful knee flexion ROM
-palpable defect
-antalgic gait OR unable to ambulate
Osgood Schlatter disease
apophysitis of tibial tubercle
M>F
ages: M: 10-15, F: 8-13
repetitive loading of knee into flexion
radiology: calcification of tibial tubercle
Osgood Schlatter hx
adolescent athlete
common bilaterally
Osgood Schlatter sxs
anterior knee pain
aggravated w/ activity/ resisted knee ext
Osgood Schlatter exam
-local TTP
-prominent tibial tubercle observed visually
-pain end-range knee flex ROM
-painful RT w/ knee ext>AROM
-possibly pain w/ tuning fork
Articular cartilage deficits
lesion prevalence: 60-70%
32-58% non-contact trauma MOI
Articular cartilage healing
Loss of proteoglycans: matrix reaches loss that is irreversible (matrix cannot replenish)
Mechanical: blunt trauma, penetrating injury, friction abrasion, sharp concentration of joint forces
-Healing depends on extent: chondral, chondral/subchondral, subchondral, cystic
Articular cartilage healing: Chondral vs subchondral
Chondral: limited response bc no inflammatory response
Subchondral: extends to blood supply, fills in w/ fibrocartilage, fibrin clot at 48hrs, 2 months kinda looks like normal cartilage, erosive changes at 6 months
Osteochondritis Dissecans
-articular cartilage defect
-separation of cartilage from subchondral bone
-juveniles
Osteochondritis dissecans rfs
lateral aspect of medial condyle most common
-M>W
greatest 10-20 yo
active individuals
commonly bilat
Osteochondritis dissecans hx
-traumatic MOI vs insidious
-hemarthrosis w/in 2 hrs
Osteochondritis dissecans sxs
non-specific knee pain
aggravated w/ activity, improves w/ rest
stiff/swell with activities
grinding, locking, catching
Osteochondritis dissecans exam
TTP femoral condyle/ medial or lateral joint lines
antalgic gait
knee effusion
limited/ painful knee ROM
Articular cartilage defects -surgery interventions
-arthroscopic lavage and debride
-microfracture
-autologous osteochondral mosaicplasty grafting
-ACI (implant)
-OAT procedure (autograft transfer)
-Allograft transplantation
-post op mx
Meniscus lesion epidemiology
-incidence
-concomitant ACL injury common
Meniscus lesion hx
-contact vs noncontact injury vs degenerative
-audible pop in directional change
-delayed effusion (6-24 hrs following injury)
meniscus lesion sx
catching/ locking/ giving way
local knee pain
meniscus lesion exam
-pain at end range knee EXT
-pain/limited flex ROM
-pain/weak flex and ext RT
-joint line tenderness
+McMurray’s, Thessaly, Appley’s, dynamic test (lateral meniscus)
Varus or valgus test (not for meniscus but may be +)
Tissue healing: Ligament
1st degree: minor tear of fibers
2nd: partial tear of structure
3rd: complete rupture
Ligament heal P1
acute inflammation and reaction (3 days) hematoma formation
Ligament P2
repair and regeneration (2-3 days post injury to ~6 wks)
fibroblasts produce collagen
matrix disorganized
Ligament P3
remodeling and maturation (>=12mnths post injury)
collagen fibers more parallel
increase tissue concentration and tensile strength
Ligament healing requirements
-disrupted tissue must remain in continuity
-controlled forces necessary to facilitate collagen synthesis
-protection from harmful stresses on tissues
ACL lesion epidemiology
VERY common
knee OA incidence high following ACL injury
increase risk for injury to other stabilizers of knee
ACL lesion correlations
-Females 2-9x>M
(jump landing mechanics, Q angle, narrower intercondylar notch, hormones and laxity)
-decreased hamstring or core strength
-duration of activity/fatigue
-dry/artificial turf
-high BMI