Knee: non-acute Flashcards

1
Q

Knee OA definition

A

Deterioration of the articular cartilage and remodelling of the periarticular bone

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2
Q

Knee OA SE

A

• I/M deep knee pain/ache 1st with activity & stiffness after rest
• Onset: gradual, insidious
•Mechanism: nil or post trauma
• Progresses to constant & night pain (particularly if they have a flare up) &
AM stiffness (< 30 min)
• Agg: WB, squatting, twisting
• Ease: heat, rest

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3
Q

Knee OA OE

A

• Antalgic gait eg. limp
• Valgus/ varus deformity (often developed from walking with antalgic gait
and muscle imbalance)
• Swelling (usually small, cold swelling but with flare up progressed to hot
swelling) + TOP jt line
• ROM: F/E restricted, pain and stiffness during ROM (normal flexion =
about 160, OA = 90-110; normal extension = 0 -5 deg hyp, lack hyp)
• +/- crepitus (knee grates)
• +/- stiff patella accessory movements (because not moving leg through full
ROM)

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4
Q

Knee OA Management

A
• Active exercise
-range of movement -F/E
-strengthening eg quadriceps -stretching/imbalance-?hamstrings tight from prolonged flexion -proprioception
-gait re-education ?gait aid to reduce pain on WB
• Wt ↓ and self management education
• Gait aids
• Aquatic therapy (NWB exercises)
• NSAIDs (oral paracetomol or topical)
• Cold pack
• Tai Chi
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5
Q

Common sources of anterior knee pain

A

o Patellofemoral Joint Syndrome
o Patellar tendinopathy
o Patellofemoral instability & dislocation

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6
Q

Less common sources of anterior knee pain

A

o Fat pad impingement-fat pad behind patellar tendon
o Quadriceps tendinopathy
o Bursitis (pre-patellar, infra patellar are the most common in
anterior knee problem)
o Stress fracture patella
o Osgood Schlatters –inflamed patellar ligament at
insertion on tibial tuberosity (traction apophysitis)
o Johansson Larsen-osteochondritis (degeneration of cartilage)
inferior pole patellar
o Synovial plica (fold in synovium)-medial pain

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7
Q

PFPS SE

A
  • Onset: Gradual overuse or after injury (i.e. blow to knee) • Area: Vague ant/lat or ant/med pain
  • Often bilateral Nature:
  • i/m, deepish ache, +/- crepitus
  • Often irritable Behaviour:
  • Agg: ↑ PFJ forces (i.e. Squat, stairs, sitting)
  • Ease: ↓ PFJ force (i.e. rest, tape to realign)
  • Aetiology: young women, , patella malt racking, VMO vs V lat imbalance, hip abd weakness, abnormal foot mechanics, ITB tight
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8
Q

PFPS OE

A
• +/- slight swelling ( pick up with patella tap/palpation)
• Knee HE 
• Foot pronation
• Femoral IR/ add
• ↑ Q angle
• Wasted VMO
• Tilted patella
Functional:
• pain &amp; poor control on squat &amp; going ↓ stairs

ROM: usually FROM +/- pain @ EOR Flexion (when compress patella) PFJ: pain/creps on comp/ glides
RSC: E may cause P (positive result - RSQ compress patella, can be negative result)
MLT: tight ITB
Palpation: Patella usually lateral +/- tilt, Tender under surface of patella; crepitus

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9
Q

PFPS: Management

A
• ↓ pain and inflammation  EPAs
• Stretch tight lateral structures (eg. ITB)   DTF, Myofascial release, stretches
• Strengthen VMO
  Exercise program
  EMG biofeedback
• Patella mobilisation techniques
• Correct contributing factors
• Orthotics
• Training programme
• Strengthen hip ER/ abductors
• Patellar taping – tape medially
• Strengthen hips (glut med and min so don’t go into IR)
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10
Q

Patellar Tendinopathy SE

A
  • Onset: Gradual overuse, common in jumping sports
  • Area: Inferior pole of the patella; Superficial
  • Behaviour: Agg: Jumping, hopping, bounding; Ease: Rest, ice
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11
Q

Patellar Tendinopathy OE

A

•Knee ROM: Usually normal
•RSC: Q/S weakness in severe cases; may cause pain reproduction
•Palpation:
TOP attachment to inferior pole of patella. Thickening also common.
•PFJ: Normal, taping has less effect
•Functional: Decline squats ↑pain

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12
Q

Patellar Tendinopathy Management

A

• Local treatment i.e. ice/ EPA’s, TF

  • Address contributing factors: Quads/ HS tightness, LL biomechanics (e.g. foot position), PFJ (may not glide enough superiorly, stretch tendon), Core stability
  • ‘strengthening’ of PT then progressive eccentric exs
  • Eccentric treatment program: decline board, some level of discomfort, work up to 3 sets of 15 reps daily, modify sports activities
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13
Q

Patellar dislocation SE

A
  • Onset: Sudden, severe pain

* Mechanism: Jumping/twisting – knee gives way, +/- audible pop, Often reduces spontaneously, Immediate, gross effusion

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14
Q

Patellar dislocation OE

A

• Haemarthrosis (blood around patella)
• RSC: Q/S ↑pain
• Lateral apprehension test +ve (if you push in direction they dislocated
in, will spasm)

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15
Q

Patellar dislocation Management

A

Conservative – 4 weeks extension splint and quads rehab +++
Surgical:
• Lateral release (release structures on lateral side to stop patella being pulled laterally)
• VMO advancement or transfer of patellar tendon [limited evidence for efficacy]

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16
Q

Knee bursitis: causes, presentation and management

A

• Inflammation of the bursa
• Due to repetitive friction (from surrounding structures)
• Soft fluctuant swelling (extra-capsular)
• Locally painful-eg over knee cap or behind quadriceps tendon
(suprapatellar bursa) or behind patellar tendon (infrapatellar bursa)
• Inflammatory presentation i.e. constant pain, night pain, hot/ red/ swollen/
tender
• Rx: rest form aggravating activity, ice, elevation, medication, prevention

17
Q

ITB friction syndrome SE

A

• Ache over lateral knee
• Agg: running (worse with longer distances, ↓ hill or cambered/slope
surface)
• Ease: rest

18
Q

ITB friction syndrome OE

A
  • Weak kneeF&E
  • ↓ braking forces
  • Weak hip abductors (worse eccentrically) & hip F
  • TOP lateral femoral epicondyle (2-3cm ↑ jt line)
  • +/- tight & TrP in ITB, TFL, glutes
  • Biomechanical & foot posture contributors
19
Q

ITB friction syndrome Management

A
  • Activity modification
  • Symptom relief – EPAS, analgesics
  • Soft tissue release
  • ITB stretch
  • Correct biomechanical contributing factors