kettering section d: special procedures Flashcards
Kettering rough criteria for selecting mode of transport for inter-hospital transport.
- Ambulance (ground):
– Traveling 0-80 miles for usual patient
– < 30 miles if patient in critical condition - Helicopter (rotary wing aircraft):
–Traveling 80-150 miles - Airplane (fixed wing aircraft)
–Traveling over 150 miles
Kettering–How does clinician decide whether or not to transport patient?
Transport–when benefit of transport outweighs risk of death.
Kettering on risks of transporting patients
- Complications/adverse events that occur during transport
- Different/new healthcare providers
- Back-end discontinuation of care
- Failure to follow-up on previously identified problems
Kettering on benefits of transporting patients
- To give patient access to specialists
- To give patient access to diagnostic equipment
- To give patient access to otherwise unavailable treatment
- To maximize patient opportunity to receive “best possible care”
- To give patient chance to identify undiagnosed problem, allowing appropriate care
Kettering on prevention of adverse events during in-house transports.
- Stabilize patient before transport
- Prepare patient thoroughly before transport
- Communicate well between personnel and departments
- Use checklists and protocols
- Check equipment frequently during transport
- Sedate patient appropriately
- Design hospitals to minimize distance for typical transports
Kettering list of major adverse events during transports.
- Cardiac arrest
- Pneumothorax
- Death
Kettering list of minor adverse events during transports.
- Oxygen desaturation
- Hypotension
- Acute changes in heart rate (tachycardia or bradycardia)
- Patient/ventilator asynchrony
- Accidental extubation
- Physiologic instability calling for medication–e.g., vasopressors
- Movement of PA catheter
Kettering: Why avoid BVM ventilation during transports?
- BVM leaves no control over PIP or Vt (could result in hypo-/hyperventilation–with respiratory acidosis/alkalosis
- BVM could exacerbate lung injury (which could lead to ALI/ARDS–better to use lung protective ventilation)
- BVM increases risk of hemodynamic compromise–resulting in increased WOB in patients breathing spontaneously
- Avoiding BMV consistent with AARC clinical practice guidelines
Kettering: Why might transport increase incidence of VAP?
- Supine positioning during transport or diagnostics
- Manipulation of endotracheal tube or circuit may raise risk of microaspiration
Kettering: How are ventilators monitored safely during transport?
- Require standard, real-time monitors and alarms
- Maintain equivalent level of monitoring at all times
- Check equipment frequently during transport
Kettering: How does the AARC stand on using ventilators during transports?
AARC, through a position statement, recommends using ventilators for transports when possible.
Kettering: What areas are at issue when using a ventilator to transport a patient?
- Running out of oxygen
- Running out of battery power
- Maintaining ventilator settings equivalent to ICU
Kettering notes on oxygen use during ventilator transport.
- Calculate tank duration prior to transport
- Determine true flow requirements by accounting for patient Ve and ventilator bias flow
How does is that tank duration calculated again?
PSIG x tank factor / L/m of flow
What are the popular tank factors?
0.28 for e cylinder
3.14 for H/K tank
Kettering: What factors will influence battery duration during transport?
- Battery type
- Operating characteristics
- Drive mechanisms: continuous vs. variable speed turbine or compressor
Kettering–What ventilator settings and capabilities influence quality of ventilation during transport?
- Mode
- Rate
- Trigger
- Graphics
- FiO2
Kettering on issues that arise from ventilators for MRI transports.
- Ferrous/ferric effects of metal
- The types of ventilator
–MRI safe ventilator poses no hazard
–MRI conditional ventilator is suitable only under specific conditions
–MRI unsafe cannot be use in MRI suite
Kettering on Issues MRI ventilation raise for ventilators delivering consistent performance for patients.
- Patient movement in and out of MRI machine requires long ventilator circuit
- Expect to increase Vt to account for volume lost in circuit
- Calculate lost volume using tubing compliance factor
- Correct for volume lost
Kettering notes on iNO during patient transport.
- May be used while transporting patients–for example, en route to OR
- Plan either for acquiring specialized transport iNO or for handling bulky standard equipment
Kettering on pulmonary artery catheter during transport.
Swan-Ganz catheters should be monitored with pulmonary arterial pressure (PAP) waveform during transport.
Kettering notes on capnography during transport
- Standard of care in ED, PACU, ambulance, intubations, and for cpr effectiveness
- Use capnography to confirm intubations
- Use capnography for all ventilator transports
- Attend closely to capnography for patients needing tight control fo PaCO2, such as patients with traumatic brain injury
Kettering definition of ventilator-associated pneumonia.
PNA that develops 48 hours after a patient is placed on mechanical ventilation.
What about hospital acquired pneumonia? What’s that?
K–HAP occurs 48 or more hours after admission to hospital and results from an infection that was not incubating at time of admission. Note that VAP is a version of HAP.
What does K say is healthcare-associated pneumonia?
Pneumonia in a patient who resides in a long-term care facility
or
In a patient in an acute care hospital for a specified time before developing pneumonia in the LTC
What does K say is a ventilator-associated event?
An event triggered by sustained increase in FiO2 or sustained increase in PEEP after a period of stability in a patient receiving invasive ventilation.
What is a healthcare/hospital-acquired infection?
An infection that began after hospitalization.
What classes of bugs are behind ventilator-associated pneumonia?
- Usually bacteria
- Sometimes fungi
- Rarely viral–if so, from epidemic–SARS (K)
How is the timing of VAP categorized? How does timing affect otcomes?
- Early onset pneumonia–48-72 hours after intubation
- Late-onset pneumonia– >72 hours after intubation, with a worse outcome than early-onset
What is the impact of VAP (K)?
- Prolonged hospital stay (increases length of stay)
- Increased healthcare costs
- Mortality rates 25-50%
- VAP accounted for 60% of all hospital-associated infections
What is incidence of VAP (K)?
- 8-28% for all intubated patients
- 100,000 lives compaign by Heathcare Inprovement (IHI) has decreased occurrence rate
With what phenomena is VAP linked? (K)
- Oropharyngeal secretions
- Esophageal or gastric contents
How should VAP influence our cuff pressure practices? (K)
- Minimal leak technique or minimal occluding volume should not be used
What specific bug is most often behind VAP? (K)
Most often a gram positive bacilli–MRSA most common
What bugs are behind VAP for specific co-morbidities?
COPD: H. influenzae, Staph pneumonia, Moraxella catarrhalis
Cystic fibrosis: pseudomonas, Staph aureus
Head trauma or diabetes: MRSA
What pharmacology is associated with VAP? (K)
- Concurrent steroid therapy
- Excessive sedation and paralytics
- Inappropriate antibiotic therapy
- HIstamine antagonists
- Antacids for gastric protection
What non-pharmacological factors increase risk of VAP? (K)
- Artificial airway (ETT or tracheostomy tube–while mask NPPPV, LMA, obturators do not cause VAP because the do not pass the vocal cords)
- Ventilator circuits
- Humidifiers
- Respirometers
- Nebulizers
- Reusable ventilator probes (e.g. temperature probe)
- Bronchoscopes
- Suction equipment
- Endoscopes
Why is VAP difficult to diagnose accurately?
- Sputum sample may have contaminates
- Previous antibiotics can alter results
- CXR infiltrates may arise from other complications
K: After 48 hours of mechanical ventilation, two or more of what findings suggest VAP?
- Presence of new or persistent lung opacity on CXR
- Fever > 38.3ºC
- Hypothermia < 36ºC
- WBC > 10,000 mm3 or < 5000mm3 (leukocytosis or leukopenia
- Purulent endotracheal aspirate
What sampling can be performed to support a suspicion of VAP?
- Bronchoalveolar lavage
- Protected specimen brush
- Mini-BAL
Order gram stain and C&S on aspirate
K’s odd requirement for BAL.
“Requires Carlens (double lumen) ET tube.” ??
K’s description of protected specimen brush (PSB) method
- Insert catheter with sealed plug down bronchoscopes
- Remove plug
- Obtain sample
- Can be done without a bronchoscope
K description of Mini-BAL technique
- Insert small catheter (usually a plugged telescopic catheter) into trachea
- Instill fluid
- Suction fluid back for sample
Where does K say Mini-BAL is often done?
In the ED to rule out VAP
How does K describe blind bronchial sampling?
- Wedge blindly a catheter into a distal bronchus
- Instill fluid
- Aspirate fluid immediately for sample
K’s measures to prevent VAP
- Keep head of bed at 30-45 degrees
- Gastric prophylaxis
- Perform regular oral care
- Use lung protective strategy
- Use weaning protocols
- Use heated wire circuit to reduce condensation
Does doe K describe weaning protocols to prevent VAP?
- RT- & RN-driven weaning
- Daily sedation vacation/sedation holiday
- Initiate weaning when RSBI < 100
- Spontaneous breathing trial
How K describes spontaneous breathing trial
- AKA Spontaneos Awake Trial (SAT)
- 30-120 minutes is minimum and maximum for SBT
- If patient fails SBT, then rest patient for 24 hours–don’t repeat SBT “in a few hours”
K types of capnography.
- Colormetric or chemical capnography
- Capnograph
- Volumetric capnography
K on colormetric capnography
- Brand name EasyCap II
- Chemically changes color from purple (no CO2) to yellow (increased CO2)
K on capnograph
- Quantifies PetCO2
- Shows or does not show capnogram waveform
- Measures exhaled carbon dioxide values using infrared absorption
K on volumetric capnography
- Integrated with ventilator volume delivery to measure volumes along the capnograph curve
- Use volumes at a given PetCO2 to calculate values such as VD/VT, VCO2, etc.
K’s highlights of AARC CPG for capnography/capnometry
- Use continuous waveform capnography to monitor correct placement of ETT (colormetric if waveform not available)
- Use PetCO2 to guide management of patients requiring mechanical ventilation
- Use continuous capnography during transport of patients receiving mechanical ventilation
- Apply volumetric capnography to assess CO2 elimination & VD/VT to optimize mechanical ventilation
How is quantitative capnography used in intubated patients during CPR?
- To assess cardiopulmonary status
- To assess effectiveness of chest compressions
- To monitor for return of spontaneous circulation
In broad strokes, how does K say PetCO2 reflects PaCO2?
- Both are directly influenced by VCO2 (CO2 production)
- VCO2 is a component of basic metabolic rate (BMR)
- PetCO2 lags below PaCOs by 4-6 mmHg
K–What increases basic metabolic rate?
- Fever
- Sepsis
- Shivers
- Seizures
- Hyperthyroidism
K–What decreases basic metabolic rate?
- Sedation
- Hypthermia
- Inadequate nutrition/starvation
K–How do normal numbers for PaCO2 and PetCO2 compare?
PaCO2 is normally 40 mmHg
PetCO2 is normally 34-36 mm Hg
K–What is one European convention for expressing PetCO2?
- As exhaled CO2 percent
- Normal PetCO2 is 3-5%
K–Under what conditions does PetCO2 decrease (suggesting a decrease in PaCO2)?
- Hyperventilation
- Hypocapnia
- Respiratory alkalosis
K–Under what conditions does PetCO2 increase (suggesting an increase in PaCO2)?
- Hypoventilation
- Hypercapnea
- Respiratory acidosis
K–What does low PetCO2 immediately after intubation suggest?
Esophageal intubation, which must be immediately corrected.
K–What does PetCO2 decreasing toward zero indicate?
A leak or disconnect with the ventilator.
K–What can capnography be used to monitor?
- Severity of pulmonary disease
- Response to therapies
- Correct ETT placement
- Ventilator/ETT leaks or disconnects
- Both pulmonary and cardiac blood flow
- The effect of neuromuscular blocking agents
K–How does capnography show response to therapies?
- Improved VD/VT
- Improved V/Q matching
K–How does capnography monitor pulmonary and cardiac blood flow?
- Decreased cardiac output results in sudden, low PetCO2 (indicating early cardiac failure/MI/CHF
- Decreased pulmonary blood flow results in sudden low PetCO2 (as an early indication of pulmonary embolism)
K–How does capnography monitor neuromuscular blocking agents?
As NMBA wears off, PetCO2 increases.
K–How can volumetric capnography be used for monitoring?
- Increasing VCO2 may indicate patient fever or NMBA is wearing off
- Decreasing PetCO2 may indicate VD/VT, cardiac failure, or decreased cardiac output
- Increasing VA shows improving ventilator status–patient may be ready for weaning
- Decreasing VD/VT shows improving ventilator status–Patient may be ready for weaning if VD/VT <60%
How should capnography be used to confirm ETT intubation?
AHA/ACLS Guidelines recommend quantitative waveform capnography.
How would waveform capnography detect ventilator circuit or ETT leak or disconnect?
PetCO2 would suddenly decrease to zero.
How would capnography monitor cardiac blood flow?
Sudden lowering of PetCO2 could be an early indicator of cardiac failure/MI/CHF.
How would capnography monitor pulmonary blood flow?
A sudden lowering of PetCO2 could indicate pulmonary embolism.
What may increasing VCO2 indicate?
- Fever
- NMBA wearing off
What might decreasing VCO2 indicate?
- Decreasing VD/Vt
- Decreasing cardiac output
- Cardiac failure
What might increasing VA show?
- Improving ventilator status
- Readiness for weaning
What does decreasing VD/VT show?
- Improving ventilator status
- Readiness for weaning if VD/VT < 60%
K: What is nutrition?
The interaction between diet and metabolism.
K: What is malnutrition or undernutrition?
A condition where dietary intake does not meet metabolic demand
K: Some ways undernutrition increase morbidity and mortality
- Five days of starvation reduces muscle strength
- Respiratory muscles can be compromised
- Undernutrition may contribute to failure to wean patient from ventilator
K: How does carbohydrate consumption affect the respiratory system?
- Increases CO2 requiring clearance (increased VCO2)
- Increases oxygen demand (decreased VO2)
K: How do excessive calories from any nutritional source affect respiratory system?
Excess calories bring excess CO2 production.
K: Which nutritional component has the highest energy yield?
Lipids
K: How does malnutrition arise from serious illness?
Through abnormal nutrient processing.
K: In critically ill patients, how do nutrients serve as toxins?
5% of glucose is normally metabolized into lactate.
In acutely ill patients, up to 85% of glucose is metabolized into lactate.
K: What is a reasonable caloric target?
25 kcal/kg/day
For a 70 kg patient: 70 x 25 = 1750 kcal/day
K: How quickly should nutritional therapy be initiated after ICU admission?
24-48 hours
K: Name three relevant complications of malnutrition.
- Protein wasting
- Respiratory muscle catabolism
- Hypoglycemia
K: What are two common tools to assess nutritional status?
- Serum albumin concentration
- Nutritional studies using anthropometric data
K: What common anthropometric calculation is used to assess nutritional status?
Body Mass Index