Joint Mobilizations Flashcards

1
Q

What is physiologic movements?

A

Motion that can be created volitionally

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2
Q

What is non-physiologic movements?

A

Motion that does not occur in isolation voluntarily

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3
Q

What are component movements?

A

Occur in combination with physiologic motion
-Ex: clavicular rotation during arm elevation

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4
Q

What is joint play?

A

Occur only in response to an outside force

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5
Q

What are accessory motions?

A

Joint play needed for normal physiologic range

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6
Q

What is joint mobilization?

A

-Skilled passive movement
-Low velocity
-Does not go beyond end range
-Can be graded I-III or I-IV

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7
Q

What is joint manipulation

A

-Low amplitude
-High velocity
-Thrust
-Intentionally moves beyond end range
-Grabe V mobilization

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8
Q

What are the 2 different grading systems of joint mobilizations?

A

-Maitland
-Nordic/Kalltenborn

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9
Q

What is the grading system for Maitland?

A

-I-IV and all are oscillatory with either small amplitude (6-12/sec) or large amplitude (3-6/sec) and are before or after the first stop, but before the final barrier
-V is a high velocity, low amplitude thrust (HVLAT) intentionally moving beyond the final barrier
-Mostly for capsular restrictions

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10
Q

What is a grade I Maitland mobilization?

A

-Far before the first stop
-6-12/sec
-Barely unweighting the joint
-For pain relief

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11
Q

What is a grade II Maitland mobilization?

A

-Oscillates between 1st stop and relaxing the tissue
-3-6/sec
-For pain relief

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12
Q

What is a grade III Maitland mobilization?

A

-Oscillates between 2nd stop and 1st stop, does not let the tissue relax
-3-6/sec
-Increase mobility

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13
Q

What is a grade IV Maitland mobilization?

A

-Oscillates between 2nd stop and just before 2nd stop
-6-12/sec
-Increase mobility

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13
Q

What are the types Nordic/Kalltenborn mobilizations?

A

-Compression
-Traction
-Translatoric (gliding)

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13
Q

What is compression used for in Kalltenborn joint mobilizations?

A

It is a provocation assessment to see if the patients symptoms are reproduced from compression

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13
Q

What is grade I Kalltenborn mobilizations?

A

Just enough to nullify compression forces and is only used in traction mobilization

14
Q

What is grade II Kalltenborn mobilizations?

A

-Tissue slack is being taken up
-Surrounding joint tissues are being tightened to assess tissue response and subjective response

14
Q

What is grade III Kalltenborn mobilizations?

A

Actual tissue deformation by moving beyond 1st stop

15
Q

What are the Kalltenborn end feels?

A

-Soft: soft tissue or muscle stretch
-Firm: capsular or ligamentous or muscular
-Hard: bone or cartilage

16
Q

What are the Kalltenborn end feel quantities/grades?

A

0=ankylosed (not moving/stuck)
1-2= hypomobile
3= normal
4-5= hypermobile
6= unstable

17
Q

What are indications for passive joint motion/mobilizations?

A

-Pain and muscle guarding
-Stiffness and decreased joint mobility

18
Q

What are type I receptors (postural)? What are the neurophysiologic benefits of passive joint mobilization on type I receptors (postural)?

A

-Found in joint capsules
-Small diamter, myelinated fibers
-Low threshold, continually firing, slow adapting
-Both static and dynamic firing mechanoreceptors
-Small motions lead to increased firing
-Promotes body awareness in space
-Degenerate with age

19
Q

What are type II (dynamic) receptors? What are the neurophysiologic benefits of passive joint mobilization on type II receptors (dynamic)?

A

-Found in joint capsule and articular fat pads
-Medium diameter, myelinated
-Dynamic mechanoreceptors
-Low threshold, fast adapting
-Movement and direction
-Associated with marked discharge

20
Q

What are type III (inhibitive) receptors? What are the neurophysiologic benefits of passive joint mobilization on type III receptors (inhibitive)?

A

-Found in ligamentous structures, especially in spinal column
-Large myelinated fibers, identical structurally to the GTO
-Dynamic mechanoreceptors
-High threshold, slow adapting fibers
-Firing leads to reflex inhibition to surrounding muscles

21
Q

What are type IV (nociceptive) receptors? What are the neurophysiologic benefits of passive joint mobilization on type IV receptors (nociceptive)?

A

-Found in most joint structures
-Lattice like, unmyelinated fibers and free nerve endings
-Generally high thresholds for firing

22
Q

What are contraindications to passive joint mobilizations?

A

-Hypermobility/instability
-Recent fracture, ligamentous sprains, RA, osteroporosis
-Any active disease process
-Flu, infection, malignancies
-Conditions of acuity, substantial inflammation, reactivity
-Based on history/interview
-Presence of swelling, warmth, or muscle guarding

23
Q

What motions fire type I and type II receptors? What are the benefits of firing these receptors?

A

-Small, oscillatory movements
-Inhibits pain and muscle guarding

24
Q

What motions fire type III receptors? What are the benefits of firing these receptors?

A

-End range, dynamic thrusts
-Inhibits muscle guarding

25
Q

What is the benefit of grade I traction Kalltenborn mobilizations?

A

-Relieve pain and muscle guarding
-Move joint fluid

26
Q

What is the benefit of grade II traction Kalltenborn mobilizations?

A

-Relieve pain
-Assess joint play/reactivity

27
Q

What is the benefit of grade III traction Kalltenborn mobilizations?

A

Increase mobility

28
Q

What is the benefit of grade II gliding Kalltenborn mobilizations?

A

Assess joint play and reactivity

29
Q

What is the benefit of grade III gliding Kalltenborn mobilizations?

A

Improve mobility

30
Q

What are the Maitland principles?

A

-Use grade I and II to reduce pain
-Use grades III and IV to increase mobility
-End all joint mob. sessions with grade I and II to facilitate relaxation and relieve pain
-Initial mob. techniques should be performed in loose pack position

31
Q

What concept guides what direction to perform glides in?

A

-Convex-concave rule
-Ex: Humerus is concave and glenoid fossa is concave, so the roll and glide occur in OPPOSITE directions. If flexion is limited, a posterior glide should be performed