Jasmonates

Question 1

(Hemi)Biotrophic vs necrotrophic

Answer 1

Bio/hemi: microorganisms living within their host tissue without causing immediate death. Triggers salicylate production.

Necr: microorganisms cell death accompanies or precedes colonization. Triggers jasmonates production.

Question 2

Participation of Jasmonates

Answer 2

Plant defenses to insects spreading disease and necrotrophs. Accumulates in the circadian pattern in phase with herbivory.

Contribute to developmental and growth controls.

The dominant smell of jasmine flowers

Question 3

Jasmonate synthesis

Answer 3

Regulated and induced by wounding.

JA is Oxylipins (derived from the oxidation of free fatty acids)

Occurs in the plastid, peroxisome, and conjugation by JAR1 in the cytosol.

JA-Ile is the most active compound, whereas MeJA may be a
transport form,

JA-Ile is degraded by cytochrome P450 oxidases.

Question 4

Jasmonate WIPK and SIPK

Answer 4

Contribute to rapid activation of JA synthesis enzymes.

WIPK: wound-induced protein kinase.

SIPK: salicylic acid-induced protein kinase.

Question 5

Proteinase

Answer 5

Induced Inhibitors that deter and weaken herbivores via interference within the digestion.

Question 6

Jasmonate Signaling

Answer 6

1. JA-lle binds to SCF^COL1 and JAZ protein
2. JAz ubiquitinated and degraded by 26S proteasome
3. Degradation of repressor permits transcriptional activation by MYC2 and other transcription factors

Question 7

JAZ

Answer 7

Repressor degradation

transcriptionally upregulated

JAZ ZIM domain interacts with NINJA, which interacts with TOPLESS, which represses transcription without hormones.

Question 8

Auxin signaling

Answer 8

1. Auxin binds to SCF^TIR1 and aux/IAA
2. AUX/IAA ubiquitinated and degraded by 26S proteasome
3. Degradation of repressor permits transcriptional activation by ARF transcriptional factors

Question 9

jin1is deficient in

Answer 9

JA-induced transcription and encode MYC2.

MYC2 upregulates JAZ genes and JA synthesis, and Wound response