INTRODUCTION TO APHASIA Flashcards

1
Q

What we need to understand about patients
who are neurologically impaired

A

Neurological basis of speech and language
* Function-structure relationship
* Outcomes of brain damage related to language
areas
* Providing evidence-based intervention

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2
Q

Perisylvian Zone / Language Zone

A

Perisylvian Zone in the dominant hemisphere
includes …..
* Broca’s area
* Wernicke’s area
* Supramarginal gyrus
* Angular gyrus
* Major long association tracts e.g., arcuate fasciculus
* Perisylvian vs. Extrasylvian areas

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3
Q

Aphasia –
Root word
Which hemisphere is affected ?

A

Aphasia –
* root word (aphatos: not speak)
* In Greek, aphasia is defined as “the state of a man who had run out
of arguments”
* Aphasia is …
* An __acquire__________ disorder of _language___________
* A dysfunction of the hemisphere dominant for language (followed
by brain damage)
* Aphasia vs. dysphagia
* Dysphagia: symptoms of difficulty in swallowing
* Aphasia is a SYNDROME (a cluster of symptoms)
* a lot of severity and patterns of language difference

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4
Q

The definition Aphasia
Factors influencing language symptoms

A

Aphasia: acquired communication disorder that impairs a
person’s ability to process language
* Encompassed under the term aphasia are selective, acquired
disorders of reading (alexia) and writing (agraphia)
* Factors influencing language symptoms
* Physiological inefficiency
* Impaired cognition
* Sensory loss
* Motor impairments
* apraxia

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5
Q

Competence vs. Performance Question

Questions: If aphasia is a deficit of linguistic competence or
linguistic performance?
If it’s a competence problem

A
  • If competence problem, aphasia is a loss of linguistic
    knowledge -> Rehabilitation will focus on re-learning new
    words and rules of sentences
  • However …
    Many reasons to argue that aphasia is a competence and/or
    performance deficit (McNeil)
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6
Q

Competence vs. Performance

Aphasia is an access (performance) problem if .

A
  • Aphasia is an access (performance) problem if …
  • Symptoms are sometimes transient
  • Co-existing impairments can exacerbate linguistic
    symptoms
  • If a continuum exists from aphasic to typical
  • Stimulability of patients with aphasia
  • Variability of aphasic behavior
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7
Q

Aphasia demographics

A
  • Prevalence
  • at least 2,000,000 with aphasia in the States
  • 225,000 new cases annually in the States
  • Among stroke patients, 20~40% have aphasia (many cases
    get bypassed)
  • Mean survival following stroke: ~10 years
  • Handedness
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8
Q

Aphasia-related demographics

A
  • Gender
  • No difference between genders but men have a tendency
    (30% higher than women) to have more strokes
  • Age
  • Average age of onset is 55 years old
  • Aphasia Awareness Report
  • In a 2020 survey, only 7% of respondents had heard of
    aphasia and could identify it as a language disorder
    [NATIONAL APHASIA ASSOCIATION]
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9
Q

From practical standpoint,
why understand “What is aphasia?

A

Treatment of impairment depends on definition
* 2nd C.A.D.: Celsus believed that the tongue, not the brain, was
the source of speech disorders → Treatment: tongue massages
and gargles
* 1657: William Harvey was treated for his speech loss with a cut
in the frenulum of the tongue (to loosen it)
* Cupping, leeching, bleeding were accepted treatments for
aphasia into the 19th century.
* Despite recent advances, continuing to increase our
knowledge about language will enable us to develop
stronger treatment protocols.

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10
Q

Paul Broca

A

Paul Broca (1865)
* Presented the case “Tan” along
with other patients
* Autopsy presented basis of
localizing language to the LH in the
__inferior frontal__ lobe
* Acknowledged some language
functions on the RH as well
* Patients described as using “halting,
agrammatic speech”

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11
Q

Carl Wernicke

A

Carl Wernicke (1874)
* Described receptive aphasia
* First to identify
__auditory comprehension ___ problem
* Patient’s speech was fluent, but had no
informational value
* Associated with temporal lobe damage
in LH

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12
Q

Hughlings Jackson
Nonpropositional vs
Propositional

A
  • “To localize the damage which destroys speech and to
    locate speech are two different things” – A warning to
    localization
  • Emotional (automatic) vs. intellectual (controlled)
    language
  • _nonpropositional______ (emotional) language e.g.,
    idioms, proverbs, lyrics of songs, conversational formulas,
    counting numbers
  • _propositional_______ (intellectual) language: novel
    language that conveys messages and concepts
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13
Q

Impairment of Blood flow

A

Because of …
* Diseases that produce
alteration in blood
pressure
* Diseases of arterial walls
* Diseases that result in
blockage of the arterial
lumen

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14
Q

Stroke: Cerebrovascular accidents

A

Stroke: Cerebrovascular accidents (CVA)
* Occurs when blood supply to the brain is stopped
* If this happens for enough time, neurons will start to dies
because they will not get enough oxygen

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15
Q

Types of Stroke

A

Three main types of
stroke
* ___ischemic strokes_____
* ___Hemorrhogic stroke_____________
* Transient ischemic
attack (mini-stroke)

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16
Q

Causes of Stroke
* Ischemia (Blockage)
Types of ischemic strokes

A
  • Ischemia (Blockage)
  • Occlusive Vascular Pathology
  • Thrombosis: A blood clot in the brain or neck
  • Embolism: A blood clot from somewhere else that has moved and
    now blocks a blood vessel in the brain or neck
  • Lacunar strokes – small, numerous
17
Q

Causes of Stroke
Hemorrhage (Bleeding)
Types of hemorrhage strokes

A

Hemorrhage (Bleeding)
* Intracerebral hemorrhage – most common type
* Subarachnoid hemorrhage

18
Q

Causes of Stroke
* Transient Ischemic Attack (TIA)

A
  • Transient Ischemic Attack (TIA)
  • Temporal blockage of blood flow to the brain (neurological deficits
    lasting < 24hours
19
Q

Causes of Stroke
4

A
  • Ischemia (Blockage)
    Hemorrhage (Bleeding)
  • Transient Ischemic Attack (TIA)
    Arteriovenous malformations (AVM)
20
Q

Occlusive Vascular Pathology
* Arteriosclerosis

A
  • Arteriosclerosis
  • Hardening and occluding of arterial walls
  • Slow process—plaques, blood platelets, fatty
    deposits, lipids, accumulate along walls of blood
    vessels
  • Increased thickness of blood
  • Insufficient blood supply can lead to
     Ischemia
21
Q

Hemorrhage
* Hematoma

A

Hemorrhage
* Hematoma
* Localized blood pool forms between skull and brain
or within brain structures
* Accumulated blood needs to be removed (often
surgically)

22
Q

Hemorrhage
Aneurysm

A

Aneurysm
* Ballooning of artery due to weakness of vessel wall
or congenital arterial defect
* Makes vessel vulnerable to rupture/hemorrhage

23
Q

Arteriovenous Malformations
* AVMs:

A
  • AVMs:
  • Congenital condition
  • Tangled arteries and veins
    mass in a specific area
  • Typically have thin walls
    (vulnerable to rupture)
  • If they grow in size,
    increases risk of
    hemorrhage