Intro To Func Rest Flashcards

1
Q

Two approaches for locomotor system pain and dysfunction

A

Structural

Functional

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2
Q

Rooted in anatomy, biomechanics
Visualized with imagery or surgery
Diagnosed by clinical tests

A

Structural approach

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3
Q

Structural approach repaired through

A

Immobilization, surgery, rehabilitation

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4
Q

Basis for most medically-oriented education and practice

A

Structural approach

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5
Q

Lesions cannot be observed directly with structural tools

Must be visualized virtually by understanding interactions of structures and systems

A

Functional approach

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6
Q

Everything must work together - the sensory motor system - muscles
Recognizes the funciton of all processes and systems within the body, rather than focusing on a single site of pathology

A

Functional approach

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7
Q

Janda’s theory

A

Joints, muscles, nervous system functionally integrated
Sensory and motor systems function together as sensorimotor system
Muscular system often reflects status of sensorimotor system

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8
Q

Kinetic chain is made up of

A

The soft tissue system (muscle, ligament, tendon, and FASCIA), neural system and articular system

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9
Q

Overuse injuries may be traced to improper technique that puts too much stress

A

Somewhwere on the kinetic chain

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10
Q

Relationship between kinetic chain structure and function

A

Each component system within the kinetic chain works interdependently to allow structural and functional efficiency
If any systme does not work efficiently compensation adn adaptations occur in the other systems

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11
Q

Compensations and adaptations lead to

A

Tissue overload, decreased performance, predictable patterns of injury

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12
Q

3 subsystems in stabilizing the spine

A

Passive musculoskeletal subsystem
Active musculoskeletal subsystem
Neural and feedback subsystem
Need all to have good spinal stability

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13
Q

Passive musculoskeletal subsystem

A

Spinal column

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14
Q

Active musculoskeletal subsystem

A

Spinal muscles

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15
Q

Neural and feedcback subsystem

A

Neuromuscular control unit

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16
Q

**Basic biomechanical functions of the spinal system

A

To allow movements between body parts
To carry loads
To protect the spinal cord and nerve roots

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17
Q

Normal function of the spinal stabilizing system

A

Provide sufficient spinal stability to match instantaneously varying demands due to
Postural changes, static loads, dynamic loads

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18
Q

Degradation of spinal stabilizing system results from dysfunction in any of 3 subsystems

A

Injury
Degeneration
Disease

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19
Q

Any abnormal loading conditions including overload and immobilization can produce

A

Tissue trauma and/or adaptive changes that may result in disc degeneration

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20
Q

Adverse mechanical conditions can be due to

A

External forces or may result from impaired neuromuscular control of the paraspinal and abdominal muscles

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21
Q

If and when pain is on board your body

A

Is unable to respond in a timely fashion due to loading you won’t have appropriate muscle timing/sequencing

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22
Q

Movement of arm in patients with neck pain indicates

A

Significant deficit in teh automatic feedforward control of the cervical spine

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23
Q

People with recurrent LBP respond differently to trunk loading despite

A

Remission from symptoms

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24
Q

Neuromuscular function in athletes following recovery from a recent acute low back injury

A

Objective measures of neuromuscular function indicated altered muscle response pattern to sudden trunk loading in athlets following recovery

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25
Asymptomatic athletes with a recent history of LBP were slower during performance of teh timed 20 m shuttle run than atheletes wihtout Athletes with resolved LBP were
Slower than a matched group of normal atheltes without LBP
26
Athletes with history of lower extremity injury
Significantly slower response time
27
Kinetic chain deficits may exist long after symptomatic recovery from injury resulting in functional deficits which may be missed on a
Standard physical assessment
28
Believed that muscles are at functional crossroad between CNS and PNS
Janda theory
29
Motor system acts as window into
CNS function
30
Reflexes influence
Muscle balance and function
31
Chronic musculoskeletal pain and dysfunction is functional pathology mediated by
CNS
32
Janda’s muscle imbalanace paradigm
Impaired relationship between muscles prone to tightness vs inhibition/weakness
33
Muscles predominantly static, tonic, postural have tendency to become
Tight
34
Muscles predominantly dynamic, phasic have tendency toward
Weakness, inhibition
35
If patient has upper and lower crossed syndrome
Layer syndrome
36
7 muscles prone to hypertonicity (tightness) when the iliopsoas is tight and the gluteals have become inhibited
``` Quadratus lumborum Thoracolumbar fascia Piriformis Hamstrings Iliotibial band/tensor fascia lata Adductors Gastroc/soleus ```
37
Mobility and stability joint by joint
``` Foot should be stable Ankel mobile Knee stable Hip mobile Lumbar stable Thoracic mobile Cervical stable C0-C2 mobile Hand stable Wrist mobile Elbow stable Shoulder mobile Clavicle stable ```
38
Pronation distortion syndrome weak muscles
``` Posterior tibialis Anterior tibialis Vastus medialis oblique Biceps femoris Gluteus medius ```
39
Pronation distortion syndrome tight muscles
``` Peroneals Adductors Medial hamstrings TFL/ITB Psoas ```
40
Pronation distortion syndrome arhtrokinematic dysfunctions
1st MTP Subtalar joint Tibiotalar joint SI joint/ISjoint/PS joint
41
Pronation distortion syndrome neuromuscular dysfunction
Decreased pronation control of the foot and ankle Decreased frontal and transverse plane control at the knee Increased compensation in the lumbopelvic hip complex
42
The force transmission between muscle and its surroundings, passing via the outer limits of muscle-tendon complexes (epimysium)
Epimuscular myofascial force transmission
43
The effects of myofascial force transmission have a major impact of our understanding of
In vivo muscle function
44
Integrative approach for myofascial force transmission
To combine knowledge of functional properties of isolated elements of the locomotor system with the knowledge of effects of nearby structures belonging to a higher level of organization and their interactions
45
Myer’s myofascial meridians: the lateral line
``` Myofascial tracks Peroneal muscles Anterior ligament of fibular head Iliotibial tract TFL Gluteus maximus Abdominal obliques Intercostals Splenius capitis/SCM ```
46
Myer’s myofascial meridians: the spiral line
``` Splenius capitis, cervicis Rhomboids Serratus anterior External oblique Abdominal aponeurosis, linea alba Internal oblique TFL, iliotibial tract Tibialis anterior Peroneus longus Biceps femoris Sacrotuberous ligament TLF, erector spinae ```
47
Back chain
``` Tuberosity of tibia Subpatellar tendon/patella Vastus lateralis Shaft of femur Gluteus maximus Sacrum Sacral fascia, lumbodorsal fascia, lat dorsi Shaft of humerus ```
48
Front chain
``` Linea aspera of femur Adductor longus Pubic tubercle and symphysis Lateral sheath rectus abdominus 5th rib and 6th rib cartilage Lower edge pec maj Shaft of humerus ```
49
Model for chiro
Macroinjury, microinjury, hypomobility, hypermobility, chronic stress - inflammation - connective tissue fibrosis - abnormal mechanical and nociceptive afferent input to CNS - altered motor patterns - abnormal tissue stress - more abnormal mechanical and nociceptive input to CNS and fibrosis - chiro specific adjusting, soft tissue mobilization and anti-inflammatory protocols - remodeling of connective tissue fibrosis - improvement of tissue mechanical function and improvemnt of mechanical and nociceptive neurological afferentation to CNS
50
Assessment for kinetic chain dysfunction
Myofascial balance assessment Sensorimotor system assessment Spinal stabilization assessment
51
Functional approach to treatment
Restore/improve proprioceptive input Restore myofascial balance Facilitation of afferent system and sensorimotor training
52
Rehabilitation efforts that attempt to maximize the extent of cortical neuroplastic changes stand to provide the greatest potential for
Rehabilitation success
53
Assessment of muscle length and movement patterns
Postural muscles | Phasic muscles
54
Postural muscles
Responsible for maintaining posture esp during gait Type I slow twitch Tend to become short and tight - not necessarily weak Stabilizers
55
Phasic muscles
Antagonistic to postural muslces Type II Tend to become weak/inhibited Mobilizers
56
Upper crossed syndrome weak muscles
``` Rhomboids Medial/lower trapezius Serratus anterior Teres minor/infraspinatus Posterior deltoid Longus colli/capitis ```
57
Upper crossed syndrome tight muscles
``` Pec major/minor Levator scapulae Upper trapezius Latissimus dorsi Subscapularis SCM Rectus capitus/scalenes ```
58
Upper crossed syndrome arthrokinematic
``` C0-C1 Cervico-thoracic Thoracic/rib AC joint SC joint ```
59
Upper crossed syndrome neuromuscular dysfunction
Excessive cervical protraction Scapular winging Early/excessive scapular elevation
60
Lower crossed syndrome weak muscles
``` Lower abdominals Multifidus Deep erector spinae Gluteus maximus Biceps femoris ```
61
Lower crossed syndrome tight muscles
Psoas Superficial erector spinae Rectus femoris Adductors
62
Lower crossed syndrome arhtrokinematic dysfunctions
``` SI joint Iliosacral joint Iliofemoral joint Proximal tibio-fibular joint Subtalar joint ```
63
Lower crossed syndrome neuromuscular dysfunctions
Altered hip extension Decreased frontal plane stabilization Increased lumbar extension
64
Muscle length assessment of tightness-prone muscles
``` Triceps surae Hip flexors Hip adductors Hamstrings Piriformis Pec major Upper trapezius Levator scapula ```
65
Triceps surae
Make hook with 5th MCP and hold calcaneus between hook and thenar eminence Distract calcaneus distally until reach barrier to fix inserion of triceps surae Apply pressure to sole of foot to passively dorsiflex ankle without inversion/eversion Normal is 90 degrees dorsiflexion
66
Ankle dorsiflexion test - triceps surae differentiation
Flex pt knee while maintaining calcaneal distraction and dorsiflexion Increase in dorsiflexion following kene flexion indicates tight gastrocnemius No increase in dorsiflexion following knee flexion indicates tight soleus
67
Muscle length assessment of tightness-prone muscles: hip flexors and adductors
Pt contacts table with ischial tuberosities and pulls knee on non-tested side to chest, hold and rolls back to lie supine on table Examiner stablizes the patinet non-tested leg and observe position of tested leg Normal = thigh on tested side should lie horizontal with leg vertical
68
Pec major lower sternal
150
69
Pec major mid sternal/clavicular
90
70
Functional screening sequence
Assessment of quality of stereotypical movements Obsrevation with light palpation Look for alterations in muscle firing - selection, timing, intensity
71
Functional screening sequence muscles
``` Hip extension Hip abudction Trunk curl-up Cervical felxion Shoulder abduction Wall angel Apley’s Stright leg raise Deep squat SFMA ```
72
Hip extension screen indicators
Decreased gluteus maximus bulk Increased hamstring bulk Observation of spinal horizontal grooves or creases Anterior pelvic tilt Increased or asymmetrical paraspinal bulk Decreased trailing limb posture at terminal stance during gait
73
Hip Abduction screen indicators
Lateral shift or rotation of pelvis Asymmetrical height of iliac crest Adducted hips or varus position Positive result on single-leg stance test Trendelenburg sign or increased lateral pelvic shift during loading response during gait
74
The SFMA top tier movements
``` Cervical movement patterns UE movement patterns Multi-segmental flexion Multi-segmental extension Multi-segmental rotation Single leg stance Squatting pattern ```
75
Cervical movement patterns
Flexion, extension - feet together teeth together
76
UE movment patterns
Apley’s | Inf shouldn’t get winging
77
Multi-segmental flexion
Patient should be able to touch her toes, should have uniform spinal curve and sacral base angle of 70 at least
78
Multi-segmental extension
Feet together, ASIS should clear the toes, spine of scapula should clear heels, uniform spinal curve, GH joint should maintain at least 70-90 of humeral flexion
79
Multi-segmental rotation
50 deg hips, | 50 deg torso
80
Single leg stance
10 sec eyes open 10 sec eyes closed Leg raised should be to 90 degrees and no loss in height when doing this
81
Top Tier movements are categorized by functional or dysfunctional and then non-painful or painful
FN, DN, FP, DP
82
Dysfunctional non-painful movments
Are further broken down
83
How do you break down a test
Fully loaded with full influence of gravity (dys due to mobility, stability, and/or motor control) Partially loaded with partial influence of gravity (dys could be due to mobility, stability, and/or motor control) Unloaded, PROM (dys most likely due to mobility)
84
Mechanics of breathign
Abdomen expands outward during inspiration and inward during expiration Not anterior-posterior plane movement Cylindrical like filling a balloon Belly breathing often encourages movement in 1 plane
85
What constitutes good breathing
Nasal breathing pattern - rest tongue in roof of your mouth Increase in intra-abdominal pressure especially on left side and without moving into spine extension Should be authentic Relaxed inhalation followed by LONG exhalation (2-4x as long as inhalation) with a pause between breaths Try to keep xiphoid and pubic bone in approximation during exhale Get out of your neck!!!!
86
Ability of body to control the whole range of motion of a joint so that there is no major deformity, neurological deficit or incapacitating pain
Spinal stability
87
Spinal stability
Neural control subsystem - neural Passive subsystem - spinal column Active subsystem - spinal muscles
88
Osteo-ligamentous cervical spine shown to buckle with
10.5N of applied axial compression | 1/5 to 1/4 the weight of the average head
89
Osteo-ligamentous lumbar spine shown to buckle with
90N of applied axial compression | Normal loads in standing 2-3x body weight
90
Muscles provide
Support and stiffness necessary at intervertebral level to sustain forces commonly encountered in life
91
Requirement of spinal stability in neutral posture estimated to be 5-10% MVC co-contraction of
Abdominal and paraspinal
92
Endurance is more important than strength to maintain spinal stability
Strength reserve necessary for unpredictable activities
93
Segments damaged by ligamentous laxity or disc disease require greater muscle activation
Results in greater compressive force
94
Coordination of muscle activity to respond to both
Expected and unexpected forces
95
Must activate correct muscles in the right amount at the right time in order to
Protect spine from injury
96
Loss of both feedforward and feedback motor control seen in
Lumbar and cervical injury (pain) patients
97
Motor control shown to become dysfunctional post neck and low back injury Changes also seen in muscle structure
Transverse abdominis Multifidus Longus capitis Longus colli
98
Goal of assessing spinal stability is
To identify loss of stability, motor control and aberrent recruitment patterns
99
Results for assess spinal stability
Provide data for reeducation of faulty motor patterns | Creating dynamic stability in the presence of mechanical compromise
100
Spinal stabilization assessment
``` LPHC muscle imbalances Abdominal bracing Shear/prone instability Neuromuscular control (NMC) Endurance Force transfer from lower to upper extremities ```
101
Abdominal bracing
Contracting the muscles of the trunk in a hoop-like fashion without drawing the abdominal wall inward The level of contraction should be low = 10% of maximum Continue to breathe!
102
Lumbar shear stability positive
Pain with resting position that diminishes in active position
103
Sahrmann core stability test - level 1
Begin in supine, crook-lying position while abdominal bracing Slowly raise 1 leg to 100degree of hip flexion with comfortable knee flexion Opposite leg brought up to same position
104
Sahrmann level 2
From hip-felxed position, slowly lower 1 leg until heel contacts ground Slide out leg to fully extend the knee Return to starting flexed position
105
Sahrmann level 3
From hip-flexed position slowly lower 1 leg until heel is 12 cm above ground Slide out leg to fully extend the knee Return to starting flexed position
106
Sahrmann level 4
From hip-flexed position, slowly lower both legs until heel contacts ground Slide out legs to fully extend the knees Return to starting flexed position
107
Sahrmann core stability test level 5
From hip-flexed position, slowly lower both legs until heels 12 cm above ground Slide out legs to fully extend the knees Return to starting flexed position In order to attain next level of stabilization must maintain pressure change +/-mm pressure
108
In order to attain next level of stabilization must maintain pressure change
+/- 10mm pressure
109
Sahrmann core stability test explanation
Pt braces, needle can move 10 mmHg either way but shouldn’t move at all. Pt is able to abdominal brace and not change pressure of biofeedback unit under lumbar spine. Then take pt through various positions, can pt hold their own legs without abherrent movement in the needle.
110
Pressure biofeedback placed under the spine with the subject in sidelyihgn position and inflated until the lumbar curve was straight to determine target pressure
Hip abduction test
111
Hip abduction test, pressure changes of 5 mmHg from the target pressure are allowed to accomodate
Changes induced by breathing
112
Hip abduction test explanation
Bladder between iliac crest and ribs in love handle region. This time bladder is pumped up so pt is now in spine neutral then ask patient to abduct the leg. Should not see any chnges in the needle moving.
113
Pressure biofeedback device folded in 3, fastened and placed behind the neck at the occiput. Inflated until pressure is stabilized on the baseline of 20 mmHg Pt attempts to nod the head to inc cushion pressure by 2mm and hold 6-10 sec Progressive inc attempted up to 30mm
Craniocervical flexion test
114
Craniocervical flexion test positive
Inability to achieve deesired pressure change
115
Craniocervical flexion test indicates
Decreased activation of deep segmental cervical stabilizing musculature
116
Craniocervical flexion test corrective action
Reactivation of deep neck flexors via craniocervical flexion exercise training
117
Assessment for deep neck flexor endurance
Pt supine, tuck chin, lift head 2 cm and hold | Test terminated when chin tuck no longer maintained
118
Mean endurance capacity for deep neck flexors
``` Males = 18.2 sec Females = 14.5 sec ```
119
Modified biering sorenson test
Turnk extensor endurance time Trunk flexor endurance time Trunk lateral flexor endurance time
120
Normal modified biering-sorenson test
Trunk extensor endurance greater than flexor and/or lateral flexor endurance
121
Modified biering-sorenson Test extensor endurance time
Pt prone with lower body fixed to table at ankles, knees, and hips upper body on floor or stool Exertion Beginning of exertion, upper limbs held across chest with hands resting on the opposite shoulders Upper body lifted off teh floor until upper torso horizontal Pt instructed to maintain horizontal position as long as possible Endurance time recorded in seconds from point at which pt assumes horizontal position until upper body comes in contact with support surface
122
Modified Biering-Sorenson Test Flexor Endurance Time
Pt sits on table with upper body against a support with an angle of 60degrees and knees and hips flexed to 90. Pt arms folded across the chest with the hands placed on the opposite shoulder and teos were placed under toe straps. Pt instucted to maintain the body posiiton while supporting wedge pulled back 10cm Test terminated when the upper body fell below 60d angle
123
Modified Biering-Sorenson Test Lateral Flexor Endurance Time
Pt sidelying on a table with legs extended Top foot placed in front of lower foot support Pt instructed to lift hips off table to maintin a straight line over their full body length supported on one elbow and both feet. Uninvolved arm held across the chest with hand placed on the opposite shoulder Test ended when the hips returned to the table.
124
Modified Biering-Sorenson Test | Extensor
``` Norm time male = 146 sec Norm time female = 189 sec Norm ratio male = 1.0 sec Norm ratio female = 1.0 sec NWNL ratio = N/A ```
125
Modified Biering-Sorenson Test flexor
``` Norm time male = 144 sec Norm time female = 149 sec Norm ratio male = .99 sec Norm ratio female = .79 sec NWNL = >1.0 ```
126
Modified Biering-Sorenson Test Side Bridge, Right
``` Norm time male = 94 sec Norm time female = 72 sec Norm ratio male = .64 sec Norm ratio female = .38 sec NWNL ratio = >0.75 or side-to-side difference >0.05 ```
127
Modified Biering-Sorenson Test Side Bridge, Left
``` Norm time male = 97 sec Norm time female = 77 sec Norm ratio male = .66 sec Norm ratio female = .40 sec NWNL = >0.75 or side-to-side difference >0.05 ```
128
Corrective action for NWNL ratio =
Spinal stabilization exercise training to improve balance in endurance times
129
Form closure
Cut out of white boxes can easily hold black on like a shelf
130
Force closure
Pick up niece or nephew by ears - hands cupping either side and lift them off the ground
131
We are a combination of form and force closure
SI joints cut at an angle and sacrum sits in that. Muscles force that together
132
Myofascial slings contributing to SI joint force closure: posterior oblique system
Glute with opposite latissimus
133
Myofascial slings contributing to sI joint force closure: anterior oblique system
Anterior oblique with piriformis
134
Supine active leg raise (ASLR)
Assesses force transfer from lower extremities to upper extremities through pelvic girdle Pt supine, flex hip and elevate leg off table noting degree possible right versus left; ease of performance (subjective and objective); compensatory pelvic or trunk rotations
135
Corrective for ASLR form ligamentous
Greater trochanteric belt
136
Form closure augmentation in ASLR
Passive compression of SI joints with medially-directed force applied to lateral innominate as patient attempts ASLR
137
ASLR form vs force dysfunction improvement in any assessment criteria indicates
Positive test
138
Corrective action form vs force closure dysfunction in ASLR
Temporary applicaiton of pelvic (trochanteric) belt | Core stabilization training with emphasis on anterior oblique system
139
Force closure augmentation ASLR
Activation of anterior oblique sling with patient reaching UE toward opposite knee against tester resistance as patient attempt ASLR
140
Corrective action ASLR force closure
Core stabilization training with emphasis on anterior oblique system
141
Prone active straight leg raise (ASLR)
Assesses force transfer from lower extremities to upper extremities thorugh pelvic girdle
142
Prone active straight leg raise (aSLR) | Pt prone, extend hip and elevate leg off table noting
Degree possible right vs left Ease of performance (subjective and objective) Compensatory pelvic or trunk rotations
143
Form vs force closure: posterior sling
Raise leg as high as can while keeping stright, then other leg Check form closure first, greater trochanters squeeze then have raise legs same as before Improved height = less abherrant problems = form closure problem = greater trochanteric belt and corrective exercises - lats and glutes Check force closure put one arm by side, extends and hold 45 away from body, push down on arm while she holds, raise leg if does better = force closure = lats and glutes exercises Focusing on posterior oblique ssytem
144
Form vs force closure: anterior oblique sling
Pt lies supine - looking at ant oblique chain Raise leg, then other one with foot flexed Form = passive Force = active Check form - use your muscles to push SI together as hard as can have pt raise both legs - if SLR improves when pushing SI joint together = form closure problem = greater trochanteric belt Check force - pt hand on opposite shoulder, 1/2 sit up, then raise leg - if improves = hyperactivating obliques = need to focus on corrective exercises specifically the obliques
145
Lumbar shear stability test
ASIS laid on table, hanging on to table, feet still on ground Start at L1, pincer grip press in PA - any pain? Go down level by level asking if pain If says yes - then have pt raise legs - press in again - if now says no = turns on extensors = positive test = corrective exercises for extensor group
146
Apley’s (FMS)
Looking for quality of movement Measure hand from distal wrist crease to tip of chiro finger 1.5 hand length total = ok Feet together, arms out, thumbs in palms, measure Look to see if have scapular winging or loss of height from jutting head forward, no spinal deviation Asymmetry from L to R is predictor of future injury - depending on what she has going on as a pt might tease that out - need abduction, external rotation and elbow flexion superior - GH extension, internal rotation, and elbow flexion for inferior If pt does not have good thoracic extension, won’t be able to get into this apley’s position To gain thoracic extension - chiro adjustment
147
Wall angel test
Feet 4-6 inches from the wall, spine leaning against the wall, get rid of lumbar space - shoulders should be able to be against wall without moving. EOP should be against wall too. Don’t let patient extend neck to get against wall - tight SCM, suboccipital in addition with problem with anterior head carriage Pt stuck in ant flexion pulls head off the wall as well - they’re gonna tilt back again. Pt abducts shoulders to 90 degrees - double check to make sure stays against wall - really tight = terrible thoracic extension Ask pt to ext rotate both hands - if can’t get to full ext rotation = tightness in the subscap
148
Janda’s trunk flexion test
Not common test done often Pt hands on thighs, does crunch, shoulder blades up off the table. Doc stands near feet with hands underneath heels If heels comes off table that’s not good - should be able to move trunk without heels moving - positive test
149
Cervical flexion test
C0-C1 neck flexion - hold for 10 sec - watch to see if have any abherrant movment patterns Let go slowly and have pt maintain that position, if they break position and starts to chin jut toward ceiling that’s tightness in suboccipital or SCM - pt shaking = fail. Pt head shifts to one side or another or rotate = maybe levator scap issues Should be able to hold for whole 10 seconds
150
Muscle length assessment upper trap and levator scap
Pt supine, bring her into FULL cervical flexion - we’re assessing the left upper trap, lateral flex away and rotate her toward the upper trap you’re assessing - hand goes on AC articulation and give some muscle play For levator scap on left - bring pt to FULL cervical flexion, go lateral flex away from levator and cervical rotate away from levator checking - go to AC articulation and give SI pressure to see if have spring If doesn’t have spring have tightness in levator scap and/or upper trap
151
Muscle length assessment hamstring: passive and active
Passive first - start with opposite knee flexed in hookline position - 45 hip flexion, 90 knee flexion - try to raise other leg should be 70-90 degrees passive hip flexion Ask pt feet together, toes to nose, raise leg as high as can go, now bend other knee which will put that iliopsoas in slack and have them actively raise again. If now can do it - need to maybe look at putting some length on the opposite iliopsoas Pt bend elbows to 90 degrees and drive them into the table, feet together toes to nose, raise leg If get length gain = hyperactive core
152
Muscle length assessment piriformis
Pt supine bring them into little knee flexion and int rotation and look for a little springy feel Alternative way - prone - bring legs into 90 degrees and let legs fall outward - feel tighter than the other one? Should be able to see which is not rotating as well - this means you’re able to see active ROM easier
153
Muscle length assessment pec major: clavicular vs sternal division
Elbo flexed at 90, abduct GH to 90 and ext rotate - your hand tractioning sternum pointing toward shoulder - should drop below or be parallel to the floor - clavicular division Sternal = abduct to 150 deg - should drop below table again with humerus or parallel to floor Pec major is like a chinese fan - fanning out - by doing this both clavicular and sternal you’re getting all the fibers
154
Muscle length assessment adductors: 1 vs 2 joint
Doc in between pt’s legs Should be able to abduct leg into 45 deg If get stuck before that, differentiate between 1 or 2 joint adductors Now bend knee and abduct more - problem tightness is in 2 joint adductor If meet resistance and then bend knee but no more abduciton = 1 joint adductors are tight
155
Modified thomas: iliotibial band and adductors
Ischial tube is barely on edge of table, bring one knee to chest then lay back - brace foot of bent knee on oblique of you, if thigh is already in abduction = IT band might already be tight. Try to pull into adduciton, if not good springiness = tightness in IT band/TFL Adductors - push into abduction - if already in adduction could already have adductors tight. If don’t have length won’t move nicely in abduction.
156
Modified thomas: iliopsoas and rectus femoris
Ischial tubes barely on table - one knee to chest then lie straight back, brace as in above Looking for thigh to be parallel or closer to floor - if high then tight iliopsoas If doesn’t have springiness = tight iliopsoas Rectus femoris - tibia should be 90 deg relative to femur - if doesn’t meet that then come in and see If have springiness = tight rectus femoris (attaches AIIS to tibial tuberosity)
157
Half kneeling ankle dorsiflexion
Proposal position - looking for knee to come over toe by 4 inches or 10 cm - normally shoes off - keep heel on ground
158
Janda’s hip abduction
Side lying - straighten them out, bend bottom knee so they’re more stable, bottom arm cups head for stability - raise top leg towards ceiling - looking for at least 45 deg of hip abduction - if when raising leg she hip hikes first it’s dominance of quad lumborum. If as raises leg abducts and sneaks into hip flexion that’s tight TFL. If raises up and starts to ext rotate = tight piriformis. Looking for firing pattern of glutes, quad lumborum, TFL, pure hip motion into abduction
159
Janda’s hip extention
Pt prone Keep one leg stright and raise to ceiling while doc has one hadn on glutes and one on mid lumbar region - looking for glutes or hamstrings to go first and erectors to go 3rd or 4th - if not then that’s a problem If we get knee flexion - adding in hamstring - don’t want that Goes into lumbar lordosis before raising that’s a problem
160
Progressive model for rehabilitation of physically active individuals
Physical exam Control swelling, control pain, restore integrity of injured tissue Restore ROM, restore control of volitional contractions Restore strength and endurance, restore reflex reactions Restore control of complex functional movements Return to functional activities
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4 principles of functional rehabilitation
Discovery of type of injury present Determination of method of presentation of injury Complete and accurate diagnosis of injury Plan of treatment of injury: short term goals, long term goals, progression and return-to-play criteria
162
Framework for funcitonal rehabilitation
Type of injury Method of presentation How to rehabilitate Knowledge of injury type and presentation method determine what needs to be rehabilitated
163
Macrotrauma
Due to spcific event Time, place mechanism of injury (MOI) usually clear Single event resulting in previously normal anatomical structures becoming suddenly and distinctly abnormal after injury
164
Microtrauma
Chronic, repetitive injuries Usually a process resulting from failure of homeostasis of cellular mechanisms and tissue constituents to maintain integrity of structures subjected to demand of physical activity over time Fairly long process Clinically evident adaptive changes in flexibility, balance, strength, biomechanics, performance occur with continued sports participation
165
Acute
Injury episode easily recalled | Activity halted or curtailed
166
Chronic
Usually microtrauma with gradual symptom onset Pain may be widespread Activity still ongoing although at reduced performance level
167
Acute exacerbation of chronic injury
Previous injury apparently successfully treated; symptom resolution does not equal normal function Acute exacerbation occurs with return to acitivity History of previous injury and rehab plan give clues to remaining underlying deficits: inflexibiilities, strength deficits/imbalances, biomechanical faults
168
Subclinical adaptations to athletic activity
Maladaptations to training Asymptomatic strength, flexibility, biomechanical changes that predispose to future injury Need to screen for kinetic chain dysfucntion prior to implementing strength and conditioning program
169
Clinical alteration
Clinical symptom complex
170
Anatomic alteration
Tissue injury complex | Tissue overload complex
171
Physiologica na dmechanicl alteration
Functional biomechanical deficit complex | Subclinical adaptation complex
172
Framework for functional rehab clnical alteraltion
Frequently occurs in presence of subclinical alterations May be present with acute injury or may be produced as a result of acute injury Clinical symptom complex
173
Clinical symptom complex
Pain, swelling, decreased ROM
174
Tissue injury complex
Actual tissue that has been damaged
175
Tissue overload complex
Tissues that have been stress/overloaded | Contribute to or exacerbate injury
176
Functional biomechanical deficit complex
Alterations in activity performance mechanics: caused by abnormalities in strength, strength/muscle balance, flexibility
177
Subclinical maladaptation complex
Substitute motions Altered recruitment patterns Synergistic dominance
178
Dec pain and restor normal joint motion Restore balance in muscle system - length, strength, endurance Improve proprioceptive input - local facilitation, peripheral stimulation Re-educate movement patterns and posture on an automatic basis
Good quality good control of motion and posture
179
Philosophy of exercise design
Training for health versus performance Integration of prevention and rehabilitation strategies Continuous improvement in function/pain reduction ADL journal Ensuring the progressive positive slope Patient lifestyle changes
180
Training for health vs performance
Emphasizes muscle endurance, motor control perfection, maintenance of spine stability during ADLs
181
Integration of prevention and rehabilitation strategies
Must reduce source that exacerbates tissue overload | Exercise enhances prevention and rehabilitation outcomes
182
Continuous improvement in function/pain reduction
Return of function and reduction of pain can be slow process | Patients have good and bad days during reconditioning process
183
ADL journal
Documenting back pain/stiffness essential in identifying link with mechanical stresses
184
Ensuring the progressive positive slope
Initiate reconditioning process with limited number of exercise Add new exercises one at a time after positive slope established Add/remove exercises based on positive slope changes
185
Patient lifestyle changes
Must change patterns that result in tissue loading in excess of threshold
186
Guidelines for core stabilization training
Develop sound basis for exercise prescription
187
Basic issues core stabiliztion training
Flexibility Strength Endurance
188
Flexibility
Generally spine flexibility should not be emphasized until spine has stabilized and undergone endurance and strength conditioning Lumbar problem = hypermobile lumbar. Hypomobile thoracics or hips
189
Strength
Appears to have little relationship with spine health | Perturbed ration of flexor-to-extensor strength ration may have effect on low back health
190
Endurance
Diminished trunk extensor endurance linked to low back injury
191
Aerobic exercise
Appears to enhance effects of spine-specific exercise
192
Order of exercises within a session
Prior activity can modulate biomechnicas of spine in subsequent activity Motion exercises performed first to reduce spinal tissue viscosity
193
Breathing
Must learn to maintain spinal muscle stiffness during torque demands adn breathing patterns
194
Time of day for exercise
No forward bending exercise in first 1-2 hours after rising with low back pain
195
Our discs rehydrate
Overnight
196
Stage 1 of patient progression
Awareness of psine position and muscle contraction
197
Awareness of spine position and muscle contraction
Distinguishing hip flexion from lumbar flexion Maintaining mild abdominal contraction Learning abdominal bracing
198
Stage 2 patient progression
Stabilization exercises to groove stabilizing motor patterns and build endurance
199
Stabilization exercises to groove stabilizing motor patterns and build endurance
Key is to determine optimum starting level For chronic patients may want to undershoot Once positive improvement slope estabilished can increase rehab challenge
200
Stage 3 of patient progression
Ensuring stabilizing motion patterns and muscle activation during ADLs
201
Ensuring stabilizng motion patterns and muscle activation during ADLs
Clinician must identify range of ADLs for which patient must prepare Rehearse spine-sparing strategies and appropriate motor patterns related to ADLs
202
Methodology of sensorimotor training
Establish key postural stability points
203
Key postural stability points
Foot SI joint Cervical spine
204
Stages of progression
Static Dynamic Functional
205
Static phase senosrimotor training
Emphasis on stable core Maintenance of postural stability on progressively unstable surfaces Progression methods - weight shift, eyes closed, adding head moevment
206
Dynamic phase sensorimotor training
Addition of arm and leg movements Use of progressively unstable surfaces External resistance applications - manual, elastic, isotonic
207
Functional phase sensorimotor training
Performance of functional movements - squats, lunges | Integrate use of unstable surfaces and external resistance - resisted lunges on labile surfaces
208
Active self-care and functional reactivation
``` Spine-sparing strategies: Hip hinge Safe squatting ADL modification Slump posture self-care Micro-breaks ```
209
Active self care hip hinge
Pole/broomstick behind back, hold above head and behind near butt and hip hinge with back stragith against pole
210
Sit to stand using hpi hinge
Practice from chair
211
Wall squat
Hands against wall above head, squat while keeping hands on wall
212
Ball squat
Ball behind back, sit down like in wall sit
213
ADL modification brushing teeth
Use stool and put one foot up
214
Slump posture self-care
Bruegger relief position - sit on very edge, hands against chair Wall lean - sit on very edge of setat arms against wall Wall slides - stands against wall feet about 6 inches forward hands in ext rotation 90 degrees, slide down wall
215
Postures of development
``` Supine Prone Quadraped Sitting Kneeling Vertical stance ```
216
4-6 months old before can
Sit and diaphragm starts to act as spinal stabilizer
217
Super important how we originally develop some of our stability and moviblity movements from
When we were a child | We crawl, then push up, then pull up
218
Patterns of developemnt
``` First thing when baby is born = takes a breath Breathign and gripping is our first pattern of development Reaching Head movement Rolling and crawling Hinging and rocking Squatting Pulling up/pushing down Gait ```
219
Flexibility
Ability of soft tissue structures to elongate through available joint ROM
220
Often single structure is cause of movement restriction | Not uncommon to have concurrent limitations from more than 1 structure
Flexibility
221
Limitation from structural involvement can be caused by trauma, surgery or lack of stretching or lack of general appreciation of what we’re supposed to be able to do
Flexibility
222
Pain associated with disruption to tissue or joint swelling may inhibit ability to
Actively and passively generate joint movement
223
types of muscle hypertonicity****
``` Limbic system dysfunction Interneuron dysfunction MTrPs Reflex spasm Muscle tightness ```
224
Limbic system dysfunction
Caused by psychological stress Inc muscle tone in cervico-thoracic-shoulder complex, low back, pelvic floor muscles Headache, LBP, dysmenorrhea, dyspaneuria, urinary frequency
225
Caused by aberrant afferent info sent by spinal or peripheral joint dysfunction (subluxation) - like when you get adjusted, walk in tight and then feel less tight
Interneuron dysfunction
226
Hypertonicity in segmentally-related muscles that can spread beyond involved segments
Interneuron dysfunction
227
Interneuron dysfunction prone to
Form MTrPs Muscle imbalance including reciprocal inhibition, synergistic dominance Faulty movement patterns established and perpetuated
228
MTrPs
Myofascial trigger points
229
Hyperirritable spot usually within taut band of skeletal muscle or in the muscle’s fascia, that is painful upon compression and can give rise to characteristic referred pain, tenderness and autonomic phenomena
Myofascial trigger points
230
MTrPs formed as a result of dysfunction
``` Sustained shortened position Sustained lengthened position Acute overload (muscle strain) ```
231
Zone of intense pain in a hardened muscle band that refers (triggers) pain distantly when stimulated
Myofascial trigger points
232
Central TrPs
Develop in muscle belly at endplate zone (motor point) | Primary
233
Attachment TrPs
Enthesopathy that develops at each end of involved fibers | Secondary due to sustained tension
234
Active TrPs
Causes clinical pain complaint | Always tender
235
Latent TrPs
More common than active TrPs Pain free unless palpated Alter muscle activation patterns
236
Sensory abnormality characterized by primarily by pain
Can be local to site of taut band and distant (referred) to another part of body Autonomic dysfucntions - abnormal sweating, lacrimation, salivation, pilomotor activity Clinical attributes of TrPs
237
Clinical attributes of TrPs | Proprioceptive dysfunctions
Imbalance, dizziness, baragnosis | Motor dysfunction characterized by constant, discrete hardness within muscle
238
Motor dysfunction characterized by constant, discrete hardness within muscel
Taut band or nodule within belly of muscle Constant feature of active TrP Can be present in absence of pain Primary abnormality that develops in response to stressors that activate TrP
239
Development of TrPs | Acute overload
Unaccustomed eccentric exercise or eccentric overload (muscle strain) Eccentric exercise in unconditioned muscle Maximal or submaximal concentric exercise leading to muscle fiber damage adn hypercontraction within muscle fiber Like going to the gym at the start of january
240
Chronic overload devleopment of TrPs
Sustained or repetitive postures and/or movements - us sitting all day long or doing the same movment all day long like a hairstylist
241
We should not stretch myofascial trigger points,
It won’t fix them, might just make them
242
Scalene MTrPs
Medial border of scapula over shoulder lateral arm doewn lateral forearm into first and second digit on dorsum of hand, 2 points above nipple on same side
243
Trapezius MTrPs
TrP1 = behind ear down neck, angle of mandible, behind eye TrP2/3 = top of shoulder, back of occiput on either side of time TrP 4/5/6 = top of GH joint, medial border of scapula, T3-6 on side of spine
244
This patient with sinus infections with ear pain
SCM or suboccipital MTrPs
245
SCM trigger points
Behind ear, top of head, in EAM, path from in front of EAM up over eyebrow, below end of SCM, forehead above eye from one side to teh other
246
Suboccipital MTrPs
Behind ear up to behind eye
247
Quadratus Lumborum MTrPs
Top of iliac crest and near greater trochanter, sacrum and ischial tube, front lower ab next to iliac crest
248
Iliopsoas MTrPs
Next to lumbar spine, anterior thigh
249
Gluteus minimus MTrPs
Anterior portion: butt lateral side of thigh through pes anserine through to back of calf Posterior portion: butt into iliac crest, back of thigh, popliteal fossa, back of calf
250
Reflex spasm
Muscle spasm in response to nociception
251
Reflex spasm frequently acts as
Splinting mechanism
252
Once underlying pain process resolves
Hypertonicity often remains
253
If left uncorrected reflex spasm leads to
MTrP formation and faulty movement patterns
254
Myopathological and neuropathological state where muscle becomes hyperactive and shortened
Muscle tightness
255
Most commonly from overuse esepcially in postural function | Results in reciprocal inhibition, synergistic dominance
Muscle tightness
256
Over time muscle tightness leads to formation of
Joint dysfunction (Subluxation), MTrP formation, aberrant movement pattern
257
Techniques to restore flexibility
``` Ballistic (dynamic) stretching Static stretching Proprioceptive neuromuscular facilitation (PNF) Self-myofascial release (SMFR) Neurodynamic stretching ```
258
Ballistic (dynamic) stretching
Bouncing movement in which repetitive contractions of agonist work to stretch antagonist muscle
259
Static stretching
Stretch to point of discomfort and hold at that point for period of time
260
Proprioceptive neuromuscular facilitation (PNF)
Involves alternating contractions and stretches
261
Self-myofascial release (SMFR)
Gentle force application to adhesion or knot within the fascial ssytem in the body
262
Neurodynamic stretching
Gentle technique to release adhesions within nervous system fascia
263
Muscle relaxation techniques (MRT) basis in
Proprioceptive neuromuscular facilitation (PNF)
264
Used to relax overactive muscles and associated fascia | Involve manual resistance of patient’s isometric or isotonic muscular effort
Muscle relaxation techniques
265
Utilize post-contraction inhibition and reciprocal inhibition (RI) Sometimes referred to as postisometric relaxation (PIR) and neuromuscular stretching
Muscle relaxation techniques
266
Neurophysiology of MRT | 2 aspects to MRT
Ability to relax overactive muscle | Ability to inc extensibility of shortened muscle or fascia when connective tissue changes are present
267
Ability to relax overactive muscle
Increased neuromuscular tension Spasm Myofascial trigger points
268
Post contraction inhibition
After a muscle contracts it is a brief latent/inhibitory states - 25-30 seconds with agonist or antagonist contraction vs 10 seconds with static stretching
269
Reciprocal inhibition
Sherington’s law of reciprocal inhibition | When a muscle contracts its antagonist is automatically relaxed
270
Essential to relax neuromuscular component of a muscle prior to attempting forceful stretching
Inhibits stretch reflex | Prevents sarcomere damage adn reduces patient pain in the presence of MTrPs
271
Muscle length changes can be caused by
Neuromuscular factors Connective tissue factors Both
272
If a muscle lengthens spontaneously after application of MRT then probably a
Primary neuromuscular cause | If not porobably primary connective tissue
273
Clinical application of PIR
Bridge between passive adn active care
274
Complementatry to chiro adjustment
Main application is in directly treating muscular component to enhance efficacy of adjustment May be used to relax tension in muscles before adjustment May be used to stretch chronically shortened muscle or fascia after adjustment
275
require patient participation and are less likely to result in dependency can be used in place of deep massage in areas with hypersensitivity to pressure Easily tolerated in all phases of healing
MRT
276
Engaging the barrier
Muscle elongated to extent that full resting length attained Wind-up muscle taking out slack in all planes Barrier is point at which further lengthening would cause initiation of stretch reflex Must engage barrier but not go past
277
Use of isometric contraction
Least amount of force necessary is used Gentler contraction tried first in order to isolate TrP Duration usually 4-10 seconds, can be as long as 30-60 seconds
278
Use of breathing and eye movements
Most muscles facilitated with inhalation and inhibited with exhalation Some muscle facilitated with eye movement in certain direction and inhibited with opposite eye movment
279
Feeling the release
After isometric contraction released wait for tension to release Not a stretch Guide muscle until new barrier is reached and repeat process
280
Hamstring PIR
Pt supine, straight leg raise up | Push above knee toward feet and at ankle toward head
281
Iliopsoas PIR
Pt on edge of table, knee up to chest, lean back push knee down into chest and down into floor OR Side lying - one knee bent, other pulled back
282
Quadratus lumborum PIR
Pt side lying, have them hip hike, then relax you lean on above iliac crest
283
Erector spinae PIR
Pt side/back lying, top leg off table behind them, bottom leg bent, pull shoulder forward and push iliac crest back
284
Upper trapezius PIR
Pt supine, lat flex head to one side, push shoulder toward feet on other side
285
Levator scapula PIR
Head lat flex push it forward while pushing shoulder down
286
Scalene PIR
Anterior scalene middle scalene: head off table, laterally flex over
287
Pec Minor PIR
Arm off table push down on GH joint
288
Self-myofascial release (SMFR) focuses on
The fascial system in the body
289
Gentle force application to the adhesion or knot Elastic collagenous fibers are manipulated from a bundled position (that causes the adhesion) into an alignment that is straighter with the direction of the muscle and/or fascia
SMFR
290
SMFR
Self-myofascial release
291
Also assist in releasing the knot by stimulating the golgi tnedon organ and thus create autogenic inhibition
SMFR
292
Find the tender spot (this indicates an adhestion) and sustain pressure on that spot for a minimum of 20-30 seconds to activate the autogenic response
SMFR
293
Possibly stimulates fascial Ruffini end-organs creating gel-to-sol effect
SMFR
294
Helps restore the body back to an optimal level of function and performance by resetting teh soft tissue proprioceptive mechanisms
SMFR
295
Use prior to static stretchign for postural distortion patterns and/or activity as well as a useful cool-down
SMFR
296
Mechanism of myofascial release
Practitioner’s manipulation stimulates intrafascial mechanoreceptors CNS repsonse includes change in tonus of related striated muscle fibers and autonomic nervous system effects: altered global muscle tonus, change in local vasodilation and tissue viscosity, lowered tonus of intrafascial smooth muscle cells
297
Resotring myofascial balance
Foam roller self-myofascial release
298
PIR
25% effort | 4-10 seconds
299
Adductor PIR
Looking for inferior ilium or IT band - inc bulk at upper 1/3 of medial thigh, genu valgus stress in midstance, femoral acetabular discomfort - lower crossed syndrome,
300
High ilium side is
Tight quadratus lumborum
301
High ilium Diffuse pain Janda’s abduction test will hip hike then abduct Treatment is PIR quad lumborum
Quad lumborum
302
Femoral acetabular joint falls anterior to malleoli, butt is flat
Hamstrings PIR option
303
Pt gets headache crawling up the side of the neck
Upper trapezius PIR
304
Do some of this before cervical adjustment, could make adjustment go easier
Levator scapula PIR
305
Posterior scalene
Lateral flexion and rotation away
306
Anterior scalene
Lat flex away, rotation towards. Shoulder elevate and reflex
307
Middle scalene
Hook left occiput area, shoulder shrug
308
Suboccipital PIR
Take head and make occiput go away from atlas like on a stick when cooking a pig. He looks backwards with his head and eyeballs. Pt has sensation of a double chin
309
Thoracic clean-up move. Person in VP area has bump
Cross their hands, put them on EOP, bring elbows together. Inferior hand goes on elbow, superior hand on his crossed fingers. Pt looks backwards with ehad and eyes
310
Pec Minor PIR
Shoulder protraction. Grab shoulder joint and move it like on an axel. If coracoid process is coming closer to the ribs, want to take shoulder like roll it back. Grab inferior angle of scapula, other hand is on GH area. Pt will rotate into your top hand, then when he relaxes you’ll push back up and around.
311
Self-myofascial release (SMFR) focuses on
The fascial system in the body
312
Gentle force application to the adhesion or knot Elastic collagenous fibers are manipulated from a bundled position (that causes adhesion) into alignment that is straighter with the direction of the muscle and/or fascia
SMFR - self-myofascial release
313
Assist in releasing the knot by stimulating the golgi tendon organ and thus create autogenic inhibition Possibly stimulates fascial ruffini end-organs creating gel-to-sol effect
SMFR
314
SMFR how to
Find tender spot (indicates adhesion) and sustain pressure on that spot for a minimum of 20-30 seconds to activate the autogenic response
315
Helps restore the body back to an optimal level of function and performance by resetting the soft tissue proprioceptive mechanisms
SMFR
316
Use prior to static stretchign for postural distortion patterns and/or activity as well as a useful cool-down
SMFR
317
Practitioner’s manipulation stimulates intrafascial mechanoreceptors
Mechanism of myofascial release
318
CNS response includes change in striated muscle fibers and autonomic nervous system effects - mechanism of myofascial release
Altered global muscle tonus Change in local vasodilation and tissue viscosity Lowered tonus of intrafascial smooth muscle cells
319
Functional anatomy of core musculature
Lumbar spine muscles Abdominals Hip muscles Cervical spine muscles
320
Transversospinalis group parts
``` Rotatores Interspinales Semispinalis Intertransversarii Multifidus ```
321
Poor mechanical advantage relative to movement production Primarily type 1 muscle fibers with high degree of muscle spindles (2-6x normal) Designed for stabilization and proprioception
Multifidus
322
Primarily responsible for providing proprioceptive information to CNS Inter/intra-segmental stabilization
Transversospinalis group
323
Segmental deceleration of flexion and rotation of spine during functional movements Must be trained to allow dynamic stabilization
Transversospinalis group
324
___ may be most important part of transversospinalis group Provides intersegmental stabilization in all positions
Multifidus
325
Erector spinae
Thoracic longissimus and iliocostalis Long extension moment arm with minimal compression
326
Most efficient lumbar extensors
Erector spinae
327
Lumbar longissimus and iliocostalis
Create posterior shear with lumbar flexion
328
Quadratus lumborum
Stabilizer in wide variety of tasks involving flexion, extension and lateral bending
329
Latissimus Dorsi
Largest moment arm of all back muscles therefore great effect on LPHC Any UE rehab has to pay attention to it and its impact on LPHC
330
Bridge between upper and lower extremities
Latissimus dorsi
331
Operate as a functional unit to help maintain optimal spinal kinematics
Abdominal musculature
332
Provide sagittal, frontal and transverse plane stabilization by controlling forces reaching LPHC
Abdominal musculature
333
Abdominal musculature
Rectus abdominis External oblique Internal oblique
334
Attaches to posterior layer of thoracolumbar fascia
Internal oblique
335
Contraction of TrA and internal oblique create traction and tension forces on
TL fascia
336
Enhances regional inter-segmental stability in LPHC
Abdominal musculature
337
Provide dynamic stabilization against rotational and translational stress Provide optimal neuromuscular control to entire LPHC
Transverse abdominis (TrA)
338
Contraction precedes activation of other abdominal muscles regardless of direction of reactive foreces
Transverse abdominis
339
Important for dynamic stabilization during all trunk movements
Transverse abdominis
340
Active during all trunk movements
Transverse abdominis and multifidus
341
Contributes to stability of lumbar spine during inspiration and expiration Involved in the control of postural stability during sudden voluntray movement of the limbs
Diaphragm
342
Normally in horizontal position in adults Cephalad posiition is inhibitory of normal function
Diaphragm
343
Posterior intersegmental cervical spine muscles
Multifidi and suboccipitalis
344
Deep cervical flexors
Longus capitis and colli Primary segmental stabilizer Feedforward contraction with arm movment
345
Lower cervical/upper thoracic extensors
Semispinalis cervicis and longissimus cervicis
346
Scapular mobilizers and stabilizers
Upper, middle, lower trapezius Levator scapula Pectoralis minor Serratus anterior
347
Injury mechanics in the lumbar spine
Too many repetitions of force and motion and/or prolonged postures/loads
348
Cumulative loading
Compression, shear, or extensor moment Injury mechanics in the lumbar spine
349
Axial torque with flexion or extension loading Cumulative exposure to unchanging work
Injury mechanics in the lumbar spine
350
Any abnormal loading conditions (including overload and immobilization) can produce
Tissue trauma and/or adaptive changes that may result in disc degeneration
351
Adverse mechanical conditions can be due to
External forces, or may result from impaired neuromuscular control of the paraspinal and abdominal muscles
352
Most important modifiable mediating factor for primary OA
Muscle dysfunction
353
Gute med exercise
Clam shells Side plank from knees - downside is getting glute med, you’re getting closed kinetic chain closed shoulder, neurodevelopmental
354
Reducing tissue damage
Reduce peak and cumulative spinal compressive loads Reduce repeated spine motion to full flexion Reduce repeated full-range flexion to full-range extension Reduce peak and cumulative shear forces Reduce slips and falls Reduce length of time in prolonged sitting especially exposure to seated vibration
355
Name one way to teduce repeated spine full motion to flexion
Hip hinge
356
Abdominal bracing
Tightening in a hoop like fashion - uses multiple muscles and we want that!
357
Ab hollowing is using one muscle
Transverse abdominus - suck belly button into spine as much as you can
358
Stiffening or tightening muscles of the midsection as if someone was about to strike you in the trunk
Abdominal bracing
359
Abdominal bracing
The level of contraction should be low about 10% maximum
360
Train core stabilizaing musculature without focus on any 1 muscle Minimize shear and compression McGills Big 3
Shown to train core stabilizing musculature with relatively low compressive loads
361
McGill’s big 3
Curl-up Side bridge Birddog
362
High level of rectus abdominis activation with posterior pelvic tilt High level of compression
Hanging knee-up
363
High compressive load (6000N) Extension load of posterior elements Potential damage to interspinous ligaments
Superman
364
High compressive load (4000N) Extension load of posterior elements Lumbar extensors not designed for powerful extenstion movements
Roman Chair Back Extension
365
Phases
Corrective Functional Performance
366
Corrective phases
Stability training
367
Functional phases
Functionally integrated training
368
Performance phases
Strength and power training
369
Corrective exercise training goals focus on
Postural control, muscle balance, pain reduction/centralization
370
Train coordination and endurance with
Safe, low-load exercises
371
Progress to complex activites and functional exercises once patient learns to
Move and position spine in fundamental ways
372
Program corrective design
At least 1 session a day!! Longer than 8 seconds could cause muscle trigger points in muscles
373
Cat camel
Warmup
374
Leg loading with
Biofeedback device
375
Dead bug progression
Back, knees at 30d One leg lifted Both legs lifted Both lifted, one at 90, one straight, one arm above head, one toward ceiling
376
Quadraped birddog
All fours One hand out One leg out One hand and opposite leg out
377
Sidelyign bridge beginner
Knees down, one or two hands
378
Sidelying bridge advanced
On feet only...then one leg lifted
379
Rotational bridge
Side plank, plank, other side plank, plank...etc
380
Abdominal curl up
One knee up, hands behind back | Lift head and shoulders
381
Supine bridge progression
Supine bridge | Then one leg in air, one on mat
382
Clamshell
On side, top knee up and down
383
Cranio-cervical flexion
Feet on table, kenes up, flex head
384
Stability ball hamstring training hip extension
Feet on exercise ball, lying on back, do supine glute bridge
385
Stability ball double leg curl
Feet on exercise ball, spine bridge, bring knees in while rolling ball
386
Stability ball hamstring training single leg curl
One leg on ball, supine bridge, one knee in air, bring other knee in
387
Stability ball bridge ball braidge
Supine bridge with feet on ball
388
Stability ball abdominal curl up
Back over ball, crunch
389
Core stability trained in exercises mimicking patients
ADLs SRAs DE
390
Training with movements that are within patients fucntional range while being as functional as possible. Progression continue until
Pt’s functional range includes ADLs, SRAs and DEs expected to be encountered
391
Functional exercise training program design Acute variables
2-3 exercises 2-3 sets x 10-12 reps Rest period approx 45 sec 2-4 sessions/week
392
Performance exercise training goals
High-level activites with narrow safety/stability margin Atheletic activity performance enhancement and innjury prevention
393
Built on a foundation of conscious-kinesthetic awareness of appropriate motor control
Performace exercise training goals
394
Sites of injury****
Soft tissue, osseous, fibro-osseous tunnels Sites of nervous system branching Sites of relative fixation to interface Areas with high possibility of friction forces from unyielding interface structures Tension points
395
Neurodynamic tensioners
Neurodynamic test that increases tension in neural structures
396
Relies on natural viscoelasticity of nervous system and does not exceed elastic limit Does not produce plastic deformation or damage
Neurodynamic tensioners
397
Median nerve tensioner
Arm out to side palm up, extend wrist as far as possible, laterally flex head away
398
Ulnar nerve tensioner
Like going to hit yourself in side of head with wrist extended and head laterally flexed away
399
Radial nerve tensioner
Arm out, palm towards back fully flexed, laterally flex head away
400
Neurodynamic sliders/flosser
Neurodynamic maneuver whose purpose is to produce a sliding movement of neural structures relative to their adjacent tissues
401
Sliders can be thought of as
Tensioners with one end put on slack
402
Nerve flossing
Neurodynamic sliders
403
Lower extremity neurodynamic tensioners
Hands behind back, leg out front, flex head in
404
Sequence of subject postures in slump test
Pt sits erect Pt slumps lumbar and thoracic spine while ex holds head in neutral Pt flexes head and neck Ex carefully applies overpressure to cervical spine as pt extends knee Pt dorsiflexes foot Pt extends head and neck If symptoms are reproduced at any stage, further sequential movements are not attempted
405
Femoral nerve neurodynamic test
Prone knee bend | Slump knee bend
406
Obturator nerve neurodynamic test
Slump SLY/KF/HE HAb obturator test
407
Peroneal nerve neurodynamic test
PR/IN/SLR | PF/IN/SLR via shoulder
408
Tibial nerve neurodynamic test
DF/EV/SLR and reversal
409
Sural nerve neurodynamic test
DF/IN/SLR
410
Balance is an essential function of
Locomotion
411
Freeman first to suggest training for peripheral sensory deficit following
Ankle sprains
412
Freeman about ankle sprains established importance of addressing
Proprioceptive deficit throughout locomotor system
413
Janda’s contributions to sensorimotor training
Believed that msucle imbalances led to movement impairments and altered motor programming
414
Treatment approach janda
Normalize peripheral proprioceptive structures Correct postural/muscle imbalance Faciliatate correct motor program
415
Stages of motor learning according to janda
Voluntary control of movement | Automatic control of movement
416
Voluntary control of movement requires
Cortical integration and patient concentration | Constant feedback from positive and negative experiences
417
Voluntary control of movement
Feedback motor control | Inefficient for creating motor programs
418
Automatic control of movement
Coordinated movement pattern programmed in subcortical region Requires less conscious processing, therefore quicker Feedforward motor control
419
Essential to protect joints for dynamic functional stability
Automatic control of movement
420
Indications for sensorimotor training****
``` Post-traumatic, postoperative Chronic neck, back pain Faulty posture especially with respiratory dysfunction General hypermobility and/or instability Muscle imbalance Prevention of falls in senirs Maintenance of general fitness ```
421
Key postural areas according to janda
Foot Pelvis espeically SI joint Cervical spine
422
Foot
Cutaneous and intrinsic muscle proprioceptive input | Small (short) foot
423
Pelvis esp SI joint
Proprioceptive input | Neutral lumbopelvic position
424
Cervical spine
Proprioceptive input
425
The small (short) foot
Attempt by patient to draw metatarsal heads toward calcaneus thus raising medial longitudinal arch and shortening foot wihout flexing toes
426
Progression small short foot
Tactile stimulation Passive remodeling NWB Active-assisted remodeling NWB Active remodeling NWB - partial WB - WB
427
Level 1 sensorimotor training - static phase
Maintain postural stability on progressively unstable surfaces
428
Exercises level 1 sensorimotor training staitc phase
Single leg balance eyes open Single leg balance eyes closed Single leg balance EO/EC on labile surfaces like balance board, wobble board, airex cushion, dynadisc, foam roller
429
Static phase level 1 program design
1-2 exercises 1-3 sets x 10-30 seconds or 10-20 repetitions Rest period 30 sec 3-5 sessions/week
430
Level 2 sensorimotor training dynamic phase
Add arm and leg movements while maintaining postural stability on progressively unstable surfaces
431
Level 2 exercises
Reaches on stable surface | Reaches on unstable surfaces
432
Level 2 sensorimotor program design
1-2 exercises 2-3 sets x 10-12 repetitions Rest period 30 sec 3-5 sessions/week
433
Level 3 sensorimotor training - fucntional phase
Perform functional movements on progressively unstable surfaces
434
Level 3 sensorimotor exercises
Single leg squat Single leg deadlift Single leg resisted movements Balance sandal training
435
Level 3 sensorimotor program design
1-2 exercises 2-3 sets x 10-12 repetitions Rest period 30 sec 3-5 sessions/week
436
Janda’s balance sandals
Sandals with balls on the bottom
437
Clinical application of balance sandals
Significant increases in gluteal activation and decreases in time to 75% MVC in 7 days Increased leg EMG activity particularly ankel evertors and invertors in 11.6 + or - 14.9 weeks Improved medial-lateral postural stability in stable and unstable ankles after 8 weeks of functional balance training
438
Janda balance sandal protocl
Initial stage Second stage Third stage
439
Jandas balance sandal initial stage
Stance training with support | Sandals in horizontal position
440
Second stage jandas balance sandals
Walking with support | Start with walking in place then progress to shoulder support only
441
Third stage jandas balance sandal
Short steps, a few meters forward and backward walking, sidestepping 1-2 minutes several times per day up to 15 minutes total
442
Patients in pain often worry that will cause more harm than good if
Active
443
Advice to let pain be your guide reinforces
Attitudes and beliefs that support pain-avoidance behavior
444
Clinicians goal in active care is to modify
Patient helath behavior in direction fo reactivation
445
Back pain traditionally viewed as
Acute, self-limiting condiiton
446
Now recognized as involving frequent reoccurrences and/or chronic course
Back pain
447
Many approaches for spine injury concerned only with
Diagnostic triage and pain managment
448
Pain-relief modalitis will always be accepted treatment Patient education about self-care through gradual reactivation rapidly gaining scientific traction Becoming
Standard of care for prevention of diability associated with spinal disorders
449
Keys to active self-care
Reassurance and advice Cognitive behavioral approach Multidisciplinary biopsychosocial approach
450
Reassurance and advice
Identify pt concerns and goals Reassurance regarding seriousness of condition Specific reactivation advice
451
Key points in initial report of findings reassurance and advice
Identify spine-related worries and fears Provide assurance that there is no serious disease Explain that injuries and degeneration can be pain precipitators but likely pain perpetuators are controllable factors Provide specific activity modification and reactivation advice
452
Cognitive behavioral approach
More structured approach involving cognitive behavioral classes/sessions
453
Cognitive behavioral approach
Address pt worries and fears Teach methods to reduce fear and apprehension May be appropriate for subacute patienst at heightened risk for chornic pain or chronic pain patients
454
Comprehensive, multidisciplinary approach that combines CB model with strategies that address return-to-work obstacles
Employer issues Compensation issues May be appropriate for chronic patients if steps 1 and 2 arenot successful
455
Patient education alone often not sufficient to
Engage patients in active self-care model
456
Must take patient-centered approach
Patient is not a diagnosis or label | Report of findings shifts model from biomedical/HCP-centered fix to biosocial/patient-centered cope and adapt model
457
Enhancing patient motivation to resume activity
Collaboratively establish functional goals Reassurance that the spine is not damaged Education that gradual reactivation will enhance recovery whereas rest with interfere with recovery Consistent verbal and written messages Make exercises simple enough to be performed at home without significant equipment needs Establish realistic expectations regarding possiblity/probability of flare-ups
458
Tips for enhancing patient compliance
Education that hurt does not necessarily equal harm Education that fitness is the key to prevention Make exercises simple enough to be performed at home without significant equipment needs Link exercises to specific fucntional deficits/goals Encourage patients to work at an exercise level that is somewhat hard for them Establish realistic expectations regarding possibility/probability of flare-ups