Intro to Blocks/Segna Flashcards

1
Q

full onset of nerve blocks may not occur for

A

30 minutes

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2
Q

benefits of blocks

A
  1. fast pain relief
  2. decreased risk of intraoperative bleeding, stroke, MI
  3. decreased intraoperative need for narcotics = less N/V, less constipation, faster wake up, discharge
  4. minimal risk - (dont get resp. depression) compared to tradiational pain managemnet
  5. may be able to avoid general anesthesia for certain cases (do entire cases under block)
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3
Q

rate of absorption in LAs

A

rate of absoprtion in LAs

  • depends on vascularity
  • increased vascularity will lead to faster systemic absorption, higher blood levels, shorter time to obtain peak levels,
  • also greater risk for toxicity

Intravenous > Tracheal > intercostal > caudal > epidural > brachial plexus > sciatic > subq

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4
Q

Patients with hypoalbuminemia

A

are at a higher risk of LAST because LAs have a high degree of protein binding and the increased free fraction of the drug will predispose them to LAST

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5
Q

Clearance of LA is affected by

A
  1. shorter half life
  2. lower protein binding - more free drug

elimination rate dependent on concentration of free fraction

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6
Q

systemic toxicity

A

time and dose dependent

  • more likely with higher serum levels/faster onset
  • main. targets = CNS/ heart
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7
Q

true allergy to LA

A

is extremely rare

  • esters -> PABA, cross rxn within group
  • amides -> may contain methylparaben.
  • no cross sensitivity between esters and amides
  • epinephrine often MISTAKE as allergy
  • patient should be fully monitored during entire block and at least 15 min after
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8
Q

Clinical Symptoms of LAST (Neuro)

A
  • Early:

Circumoral numbness, dizziness, tinnitus, blurred vision

  • restlessness, agitation, nervousness, seizures
  • CNS depression: slurred speech, drowsiness, unconsciousness
  • may bypass earlier symptoms with arterial (esp. vertebral) injections -> seizures
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9
Q

Clinical Symptoms of LAST (cards)

A
  • LA-induced dysrhythmias: sodium channel blocking effect of LA
  • prolongatoin of impulse conduction may lead to malignant arrhythmias
  • lidocaine: less toxic bc fast in/fast out
  • bupivacaine: more potent and toxic, fast in/slow out
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10
Q

treatment for LAST

A
  1. supportive (correct hypoxemia/acisosis)
  2. ACLS protocol - avoid lidocaine
  3. Intralipid:
  • 20% emulsion
  • 1.5mL/kg as initial bolus
  • 0.25 mL/kg/min for 30-60 min
  • bolus can be repeated 1-2 times for persistent asystole.
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11
Q

Commonly used block anesthetics and DOA

A
  1. ropivacaine: 12 hrs (pushed to 24 with adjuncts)
  2. bupiviaine: 15 hrs ~ 24 hrs
  3. lidocaine: 2 hrs
  4. mepivacine: 6 hours
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12
Q

supraclavicular block WITH neurostimulation has

A

a much higher rate of pneumothorax (5%) compared to other UE blocks

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13
Q

COPD is relatively contraindicaiton for

A

supraclavicular block because pleural apex may be higher

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14
Q

axillary nerve block has a high rate of failure for

A

single nerve injections

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15
Q

single injection ulnar block

A

block at elbow

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16
Q

single injection of median nerve

A

travels in antecubital fossa with brachial artery

17
Q

single injection of radial nerve

A

travels at lateral elbow within brachioradialis

18
Q

ischemia is an important component of

A

the bier block.

Blocks nerve conduction, may obtain anlagesia with normal saline in 20-25 minutes of ischemia.

19
Q

bier block: mechanisms of action

A
  1. direct action by LA + retrograde flow into endonerium, capillary plexus, eventuallly into nerve trunks
  2. compression of nerve trunks (slow component)
20
Q

Most common drugs for bier block

A
  1. 0.5% lidocaine or prilocaine
  2. NO epi added

Dosage ->

  • UE = 50 mL
  • LE = 100 mL
21
Q

in bier block, tourniquet should be kept inflated for a minimum of

A

20 minutes

22
Q

lumbar plexus is

  1. derieved from
  2. divdes into
A

lumbar plexus is

  1. derieved from L2,L3,L4
  2. divdes into:
  • femoral
  • lateral femoral cutaneous
  • obturator
23
Q

lateral femoral cutaneous

A

arises from L2-L3, part of lumbar plexus

Motor: NONE

sensory: lateral aspect of thigh

24
Q

femoral nerve

  • arises from
  • motor
  • sensory
A

femoral nerve

  • arises from: L2, L3, L4
  • motor:
    • anterior branch - sartorius
    • posterior branch: quads
  • sensory: anterior thigh
25
Q

obturator nerve

  • arises from:
  • motor:
  • sensory:
A

obturator nerve

  • arises from: L2, L3, L4
  • motor: hip adductors
  • sensory: distal inner thigh and part of hip
26
Q

larger branch of the sciatic nerve

A

tibial (L4-S3)

peroneal: L4-S2

27
Q

intercostobrachial nerve

A

arises from T2

Not anesthetized with any approah to the brachial plexus. Requires a field block.

28
Q

the stellate ganglion is located at

A

C7

also called “cerivothoracic ganglion”

29
Q

injection of LA into vertebral artery

A

as little as 1 mL of LA injected into vertebral artery can lead to seizure

30
Q

Vertebral artery injection is most likely with

A

interscalene block

31
Q

most common complication of supraclavicular block

A

pneumothorax

32
Q

for an intraclavicular block the needle must pass through

A

pectoral major and minor

33
Q

3

A