Intracranial haemorrhage Flashcards

1
Q

Extradural haemorrhage (epidural)

A

between the skull and the dural membrane- often as a result of head trauma, source of blood often arterial

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2
Q

extra dural haemorrhage symptoms

A

beware of deteriorating consciousness after head injury that originally produced no loss of consciousness.
Increasingly severe headache, vomiting, confusion, and seizures. If bleeding continues- ipsilateral pupil dilates, coma deepens, bilateral limb weakness develops, death due to resp arrest

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3
Q

Most common artery causing extra dural haemorrhage

A

middle meningeal artery- trauma to temple

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4
Q

Tests for extra dural haemorrhage

A

CT shows haematoma

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5
Q

management of extradural haematoma

A

neurosurgical unit for clot evacuation +/- ligation of the bleeding vessel

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6
Q

Subdural haematoma

A

between dura mater and arachnoid mater

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7
Q

when to consider subdural haematoma

A

conscious level fluctuating, having an evolving stroke- esp if on anticoagulants
most subdural come from trauma but it is often long forgotten - minor

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8
Q

where is bleeding from in subdural haematoma

A

bridging veins between cortex and venous sinuses

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9
Q

who is most susceptible to subdural

A

elderly- brain atrophy makes bridging veins more vulnerable. Also- alcoholics, anticoags

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10
Q

symptoms and signs of subdural

A

fluctuating levels of consciousness, insidious physical or intellectual slowing, sleepiness, headache, personality change, and unsteadiness

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11
Q

Imaging for subdural

A

CT/MRI shows clot +/- midline shift

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12
Q

what should you look for on imagine for subdural

A

crescent shaped collection of blood over one hemisphere

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13
Q

management of subdural

A

reverse clotting abnormalities urgently

clots >10mm or >5mm with midline shift need evacuating- via craniotomy or burr hole

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14
Q

SAH symptoms

A

Sudden onset excruciating headache, typically occipital (thunderclap), vomitting, collapse, seizures and coma often follow.

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15
Q

SAH signs

A

neck stiffness, kernigs sign (hip flexed, knee extension beyond 90 degrees painfull) takes 6h to develop, focal neurology at presentation may suggest site of aneurysm

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16
Q

Causes of SAH

A

berry aneurysm rupture (80%)- common sites junctions of posterior communicating with the internal carotid or anterior communicating with ant cerebral or bifurcation of MCA
Arteriovenous malformations

17
Q

risk factors for SAH

A

previous aneurysmal sah, smoking, alcohol misuse, increased BP, bleeding disorders,

18
Q

conditions associated with berry aneurysms

A

polycystic kidneys, aortic coarctation, Ehlers danlos

19
Q

tests

A

urgent CT
consider LP if- CT negative but history very suggestive of SAH- needs to be done >12hr after onset of headache to allow breakdown of RBCs so that a positive sample is xanthochromatic

20
Q

Management

A

maintain cerebral perfusion
nimodipine- reduces vasospasms
Surgery- endovascular coiling vs surgical clipping - coiling preferred where possible

21
Q

complications of SAH

A

rebleeding, cerbral ischaemia, hydrocephalus, hyponatraemia