Intestinal Obstruction Flashcards

1
Q

Differences between Acute Obstruction vs Peritonitic

A

Acute obstruction:
•colic abdominal pain
•Soft, non- tender on palpation
Peritonitic
•Constant abdominal pain
•Tense and tender on palpation

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2
Q

What are the Zones of Head

A

The zones of the abdomen and the three embryological gut segments: foregut, midgut, and hindgut.

  1. Foregut Zones:
    • Epigastric Zone: This zone includes the lower oesophagus, stomach, duodenum, and pancreas, which are derived from the foregut.
  2. Midgut Zones:
    • Right Upper Quadrant (RUQ): The right part of the stomach and the majority of the small intestine (jejunum and ileum) are derived from the midgut.
    • Right Lower Quadrant (RLQ): The cecum, appendix, and the ascending colon are also part of the midgut.
  3. Hindgut Zones:
    • Left Lower Quadrant (LLQ): The sigmoid colon and the majority of the descending colon are derived from the hindgut.
    • Hypogastric Zone: The rectum and the distal part of the large intestine are part of the hindgut.

Conclusion:
In intestinal obstruction, Periumbilical pain suggests midgut origin, and suprapubic suggests hindgut origin

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3
Q

What is intestinal obstruction?

A

A condition in which there is impediment of cephalic- caudal propulsion of the intestinal contents by mechanical or non-mechanical
-One of the common causes of acute abdomen in our settings

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4
Q

Classification of IO, depending on aetiopathology

A
  1. Dynamic: peristalsis is working against a mechanical obstruction
    • Associated with colicky abdominal pain
    ■INTRALUMINAL:
    •Fecal impaction
    •Foreign bodies
    •Parasites: Round worms 🪱 turn into a ball after administration of antiparasitic medication
    •Bezoars
    •Gallstones

■MURAL: Strictures
♡Benign:
-Post anastomotic
-Schistosomiasis
-TB infection
-IBD: UC/Crohn’s disease
-Post radiation
Others
-Sigmoid valvulus
-Diverticulitis
-Intusssusception
-Polyps

◇Malignant
-Adenocarcinoma
-Sarcoma
-Neuroendocrine

■EXTRAMURAL
-Adhesions
-Hernias
-Infectious: tuberculosis
Malignancy:
-1⁰ Peritoneal Malignancy
-2⁰ Metastasis to the peritoneum

  1. Adynamic
    -Mechanical element is absent
    -Peristalsis may be absent (Paralytic ileus) or it might be present but in a non-propulsive form (Pseudo-obstruction)
    -In general, its not associated with pain

Causes:
●Peritonitis
●Electrolyte imbalance
●Postoperative
●Ischaemia
●Drugs
●Retroperitoneal causes

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5
Q

Pathophysiology of intestinal obstruction

A
  1. Accumulation of intestinal contents
  2. Increased frequency and amplitude of peristalsis ( at this moment, bowel sounds have high pitch and frequency)
  3. Progressive Accumulation of air and fluids.
    Amound of fluids produced per day
    ♤Saliva: 1-1.5 litres
    ♤Gastric juice: 2 - 3 litres
    ♤Pancreatic juice: 500- 1000 ml
    ♤Bile: 500-800 ml
  4. Third spacing of the fluid: It’s not participating in the physiological processes of the body
  5. Progressive bowel distension and oedema
  6. Obstruction, in the order: lymphatic, venous, and then arterial
  7. Arterial obstruction leads to bowel necrosis and perforation
  8. Weakening of gap junctions: bacterial translocation
    ●Proximal: 10⁴
    ●Midgut:10⁶
    ●Cololon 10^14
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6
Q

Pathophysiology of intestinal obstruction

A
  1. Accumulation of intestinal contents
  2. Increased frequency and amplitude of peristalsis ( at this moment, bowel sounds have high pitch and frequency)
  3. Progressive Accumulation of air and fluids.
    Amound of fluids produced per day
    ♤Saliva: 1-1.5 litres
    ♤Gastric juice: 2 - 3 litres
    ♤Pancreatic juice: 500- 1000 ml
    ♤Bile: 500-800 ml
  4. Third spacing of the fluid: It’s not participating in the physiological processes of the body
  5. Progressive bowel distension and oedema
  6. Obstruction, in the order: lymphatic, venous, and then arterial
  7. Arterial obstruction leads to bowel necrosis and perforation
  8. Weakening of gap junctions: bacterial translocation
    ●Proximal: 10⁴
    ●Midgut:10⁶
    ●Cololon 10^14
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7
Q

What are the cardinal signs of intestinal obstruction

A
  1. Pain
    - Tells that there is mechanical obstruction
    - where pain begins tells the level

Site: The location of the pain
Onset: When the pain started
Character: The quality or type of pain (e.g. sharp, dull, burning)
Radiation: Whether the pain radiates to other areas
Associated symptoms: Any other symptoms accompanying the pain
Time course: How the pain has changed over time
Exacerbating/Relieving factors:
Severity: The severity of the pain on a scale of 1 to 10

  1. Abdominal distension
    - Mainly in LBO
  2. Vomiting
    - In SBO, it is early and frequent
  3. Constipation
    - Occurs early in LBO
    -Obstipation: absolute, no flatus
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8
Q

Small bowel obstruction (SBO)

A

High SBO: Duodenum & Jejunum
●Early profuse vomiting and dehydration
■Causes:
-Adhesions
-Lipomas
-Leiomyomas
-Congenital

Low SBO: Ileum
● Vomiting delayed
●Pain and mild central distension
Causes
-Hernias
-Adhesions

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9
Q

Large bowel obstruction (LBO)

A

●Early pronounced distension + Constipation
●Mild pain
●Vomiting and dehydration occur later
◇Location:
-Anywhere in the large intestiine
-Caecum: high risk of perforation, thanks to LaPlace’s Law
3-6-9 rule
-caecum has the largest radius

Causes:
●Colorectal carcinoma
●Volvulus
●Tuberculosis Strictures
●Diverticulitis
●Congenital megacolon

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10
Q

Simple vs. Strangulated obstruction?

A

Simple:
- Blockage without vascular compromise
Strangulated: surgical emergency
-Vascular compromise
-Associated with: hernia, volvulus, closed loop obstruction, intussusception, mesenteric infarction, adhesions
Clinical features of strangulation
●Pyrexia >38⁰C
●Tachycardia
●Tachypnea
●Constant abdominal pain
●Rebound tenderness
●Shock

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11
Q

Clinical Examination of IO

A

●Introduction and explanation of the procedure
●General examination:
ABDOMEN
Inspection:
- end of the bed, closer to the bed
Symmetry
Distension
Umbilicus
Visible peristalsis
Visible mass
Distended veins

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