Interdisciplinary Management Flashcards

1
Q

Difference between medical assessment and PT assessment

A

Medical — more reliance on imaging and structural diagnosis while PT is more functional and biomechanical assessment.

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2
Q

Indications for epidural injections

A
  • Lumbosacral disc herniation with radicular pain
  • Cervical radicular pain
  • Spinal stenosis with radicular pain
  • Compression fracture of spine with radicular pain

GOAL = reduce radicular pain

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3
Q

MOA for epidural injections

A
  • Inflammation of nerve root in epidural space provoked by leakage of disc material
  • Compression off nerve root vasculature
    And/or
  • Irritation of dorsal root ganglia from spinal stenosis
  • Pain relief due to reduction in inflammatory process of afflicted nerve roots
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4
Q

Efficacy of epidural injections

A

Symptoms of back pain/radiculopathy of shorter duration have more sustained relief

Chronic cervical radicular pain = may benefit from epidural injections but data is weak and inconsistent

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5
Q

Risk and complications of epidural injections

A

Total dose of steroids limited due to negative effects of steroids on bones/joints/soft tissues, postural puncture headache, backache, bleeding, infection, nerve root injury

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6
Q

Indications for facet joint and SI joint injections

A

Facet joints — focal tenderness over facet joints, post laminectomy syndrome, post spinal fusion pain, low back pain with normal radiological findings

SI joint — focal tenderness over SI joint, positive provocative SI joint tests.

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7
Q

Efficacy for facet joint injections in chronic pain

A
  • no clear differences between intra-articular facet joint injection with corticosteroid versus saline in pain or function.
  • glucocorticoid injections into facet joint have NOT been shown to be effective treatment in low back pain
  • American pain society guideline recommends against facet joint injection use
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8
Q

Efficacy for SI Joint injections

A

Poor evidence for short term and long term relief from Inter articular steroid injections, periarticular injections with steroids or botulin toxin

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9
Q

What is facet medial branch blocks and facet radio frequency ablation (RFA)

A

Medial branch block — Performing a temp block with lidocaine to see if patient experiences pain relief

If successful, proceed with RFA which is a high frequency electrical current runs through an insulated needle. Heat from the tip of the RFA device is targeted to create a small lesion within a nerve.

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10
Q

Efficacy for RFA in chronic pain

A

Discogenic pain = statistically significant pain reduction but no long term follow up or return to function assessment has been done

Chronic LBP = little additional benefit from normal treatment like regular exercise and psychological support

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11
Q

What are concerns for RFA with chronic pain

A

Medial branch also innervates muscles that provide stabilization and protection of spine so loss of sensation there do we really want that? Long term the nerve can repair and regrow but that regrowth can be painful

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12
Q

Indications for sympathetic block

A

Complex regional pain syndrome
Phantom limb pain
Vasomotor dysfunction
Swelling
Allogynia
Hyperalgesia
Skin trophic changes

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13
Q

What is a sympathetic block

A

Blocks are performed at lower third L2 or upper third of L3 and the injection of a local anesthetic drug around the nerve temporarily blocks the function of the nerve

Leads to vasodilation and improvement in blood flow to the limb.

Referral to PT is usually same day for exercise and mobs while patient is experiencing some of that pain relief

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14
Q

Efficacy of sympathetic block in chronic pain

A

Low quality and conflicting evidence
Not great

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15
Q

Indications for spinal cord stimulators

A

Failed back surgery
Complex regional pain syndrome
Peripheral neuropathy
Phantom limb pain

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16
Q

What is a spinal cord stimulator

A

Low level electrical impulse, delivered directly into SC through the spinal cord stimulator that is inserted in the epidural space, interfere with the direct transmission of pain signals traveling along the spinal cord to the brain

Patient uses external transcutaneous telemetry device to turn stimulator on and off

Like tens but inside you = gate control theory

17
Q

Efficacy for spinal cord stimulator for chronic pain

A

Mod-quality evidence supporting use of SCS in patients with persistent radicular pain due to failed back surgery

Pain relief with SCS tends to decrease over time

Lots of risks though

18
Q

What is opioid tapering

A

Getting someone off opioids SLOW AND GRADUAL. Minimizes withdrawal symptoms

  • lots of patient ed
  • determine treatment goal
  • must be individualized tapering plan for each person
19
Q

Pharmacotherapy goals for addiction services referral

A

Suppress opioid withdrawal, block effects of illicit opioids, reduce opioid craving and stop use of illicit

Promote and facilitate patient engagement in recover-oriented activities

20
Q

NSAIDS MOA

A

Block prostaglandin production by inhibiting COX 1 OR 2

Cox 1 — tummy issues

21
Q

What are 2 black box warnings on NSAIDS

A
  1. GI risks
  2. Cardiovascular risks
22
Q

Antidepressants

A

SSRIs — duloxetine and venlafaxine
- increase NE and serotonin available in synaptic cleft at supraspinatus and spinal levels
- inhibition of pain transmission via descending pathway
- decrease excitability of neurons in the CNS and PNS

SE = BP elevation, GI, insomnia/sedation, weight gain, sexual dysfunction

23
Q

Skeletal muscle relaxants

A

Cyclobenzaprine most common
- typically should be used only for short-term relief of muscle spasms NOT recommended past 3 weeks
SE = drowsiness, urinary retention, dry mouth, constipation

24
Q

Antispasticity agents

A

Benzos — diazepam most common
- GABA receptor agonist, presynaptic inhibition of SC
- extensive adverse -effect profile, abuse potential, and likelihood for multiple drug interactions
- high risk of overdose if combined with opioids

25
Q

Anticonvulsants

A

Gabapentin
- prescribed for neuropathic pain, post-herpes, HIV, related neuropathy, fibromyalgia, peripheral nerve changes
- selectively bind calcium channel subunits in the dorsal root ganglia of SC
- thought to down regulate calcium channels in SC and lead to pain attenuation

26
Q

Topical things - capsaicin

A

Capsaicin (hot chili pepper oil) — selectively binds to receptor on a-delta and C nerve fibers and in CNA, releasing substance P and causing transient depolarization, chronic exposure causes receptor desensitization resulting in depletion of substance P at afferent endings

27
Q

Topical things - menthol

A

Counter-irritant of sensory nerve endings offsets pain served by same nerves, reddening of skin via vascular dilation, warm soothing feelings

28
Q

Topical things - lidocaine

A

Local anesthetic blocks sodium channels, decreased pain transduction and decreasing peripheral sensitization

29
Q

What is pain school

A

12 week health education programming based on the bio psychosocial spiritual approach to pain management
Goal is self management in chronic pain

30
Q

What 2 group options to patients have in pain school

A
  1. Cognitive behavioral therapy — problem focused coping skills which address things we can control
  2. Acceptance and commitment therapy — emotion focused coping skills which address the things we cannot control
31
Q

What is acceptance and commitment therapy

A

ACT — clinical behavioral analysis treatment that uses acceptance and mindfulness strategies mixed with commitment and behavior change strategies to increase psychological flexibility

32
Q

What is recreational therapy

A

RT - embraces a definition of health as enhancement of physical, cognitive, emotional, social, and leisure development so individuals may participate fully and independently in chosen life pursuits

Goal — to maintain their therapeutic outcomes to their life after the healthcare team is no longer involves = greater health maintenance over time.