Interactionist approach to schizophrenia Flashcards

1
Q

Interactionist approach to schizophrenia

parts

A

Explaining the interactionist approach: diathesis-stress model
Treatment according to the interactionist model

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2
Q

interactionist approach

definition

A

a broad approach to explaining schizophrenia
└acknowledges that a range of factors (e.g. biological, psychological) are involved in the development of schizophrenia

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3
Q

biological factors

A

genetic vulnerability, neurochemical/neurological abnormality

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4
Q

psychological factors

A

stress (e.g. from life events)

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5
Q

Explaining the interactionist approach: diathesis-stress model

A

└diathesis stress model: an interactionist approach to explaining behaviour
└schizophrenia is explained as being the result of an underlying vulnerability (diathesis) and a trigger
└both necessary for the onset of schizophrenia
└earlier models: vulnerability= genetic, triggers= psychological
└now: diathesis= genes/trauma, stress= psychological/biological

diathesis stress model says both a vulnerability to schizophrenia and a stress trigger are necessary to develop the condition

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6
Q

diathesis

A

vulnerability

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7
Q

stress

A

negative psychological experience

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8
Q

Meehl’s model

A

└Meehl (1962)
└original diathesis stress model- diathesis was entirely genetic (result of a single schizogene). This led to a development of a biologically based schizophrenic personality (one characteristic is sensitivity to stress).
└chronic stress through childhood and adolescence can result in the condition
└e.g. schizophrenogenic mother

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9
Q

Modern understanding of diathesis

A

└Ripke et al (2014)
└many genes appear to slightly increase genetic vulnerability
└no single ‘schizogene’
└Ingram and Luxton (2005)
└psychological trauma as a diathesis
└Read et al (2001)
└proposed a neurodevelopmental model in which early trauma (e.g. child abuse) alters the developing brain
└e.g. the hypothalamic-pituitary-adrenal (HPA) system can be overactive
└=person more vulnerable to later stress

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10
Q

Modern understanding of stress

A

└Houston et al (2008)
└modern definition of stress relating to the diathesis stress model= anything that risks triggering schizophrenia
└cannabis use can be a stressor
└increases risk of schizophrenia up to 7 times
└interferes with dopamine system
└most people that take it don’t develop schizophrenia
└must be one or more vulnerability factors

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11
Q

Treatment according to the interactionist model

A

└ acknowledges both biological and psychological factors in schizophrenia
└=compatible with both biological and psychological treatments
└combines antipsychotic medication and psychological therapies like CBT
└Douglas Turkington et al (2006)
└can still practice CBT to relieve psychological symptoms in biological cases of schizophrenia
└internationalist treatment more common in UK than USA
└CBT, family therapy ad use of token economised are usually carried out along with antipsychotics

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12
Q

Interactionist approach to schizophrenia
strengths
summary

A

Evidence for the role of vulnerability and triggers- Pekka Tinerari et al (2004)

Support for effectiveness of combination treatments- Turkington et al (2004)

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13
Q

Interactionist approach to schizophrenia
strengths
Evidence for the role of vulnerability and triggers

A

└Pekka Tinerari et al (2004)
└investigated the combination of genetic vulnerability and parenting style (the trigger)
└children adopted from 19,000 Finnish mothers with schizophrenia between 1960 and 1979 were followed up
└adoptive parents assessed for child rearing style
└rates of schizophrenia were compared to control group of adoptees without any genetic risk
└child rearing style characterised by high levels of criticism and conflict and low levels of empathy → development of schizophrenia
└only in children with a high genetic risk
└not in the control group
└suggests that both genetic vulnerability and family-related stress are important in the development of schizophrenia
└genetically vulnerable children are more sensitive to parenting behaviour

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14
Q

Interactionist approach to schizophrenia
strengths
Support for effectiveness of combination treatments

A

└Turkington et al (2004)
└315 patients randomly allocated to a medication +CBT group, medication + supportive counselling or a control group (medication only)
└patients in the 2 combination groups showed lower symptom levels than control group
└no difference in hospital readmission
└practical advantage to interactionist approach
└better outcomes

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15
Q

Interactionist approach to schizophrenia
limitations
summary

A

Diathesis stress model too simple/reductionist - James Houston et al (2008)

Don’t know exactly how diathesis and stress work

Treatment-causation fallacy - Turkington et al

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16
Q

Interactionist approach to schizophrenia
limitations
Diathesis stress model too simple/reductionist

A

└ classic model of single schizogene and schizophrenic parenting style as the major source of stress is now regarded to be over simple
└multiple genes increase vulnerability to schizophrenia, no single schizogene
└stress comes in many forms e.g. dysfunctional parenting
└vulnerability and stress don’t have a single source
└vulnerability can be from early trauma, stress can be biological
└James Houston et al (2008)
└childhood sexual trauma= vulnerability factor
└cannabis= trigger

17
Q

Interactionist approach to schizophrenia
limitations
Don’t know exactly how diathesis and stress work

A

we have evidence that vulnerability + stress can lead to schizophrenia
└suggestions on how this may lead to symptoms
└don’t understand the mechanisms by which symptoms of schizophrenia appear and how both vulnerability and stress produce them

18
Q

Interactionist approach to schizophrenia
limitations
Treatment-causation fallacy

A

Turkington et al
└argue that there is a good logical fit between the interactionalist approach and using combination treatments
└just because combination treatments are more effective than separate treatments doesn’t mean the interactionist approach to schizophrenia is correct
└just because drugs help doesn’t mean it’s of biological origin
└treatment-causation fallacy