Interactionist approach to schizophrenia Flashcards

1
Q

interactionist approach definition

A

way of explaining the development of behaviour in terms of a range of factors, including both biological and psychological ones, most importantly such factors don’t simply add together but combine in a way that can’t be predicted by each on separately (they interact)

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2
Q

diathesis stress model definition

A

interactionist approach to explaining behaviour. for example, schizophrenia is explained as the result of bothe na underlying vulnerability (diathesis) and trigger (stressor), both of which are necessary for the onset of schizophrenia, in early versions of the diathesis-stress model, vulnerability was genetic and triggers were psychological. nowadays both genes and trauma are seen as diathesis, and stress can be psychological or biological in nature

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3
Q

what is the interactionist approach sometimes called

A

the biosocial approach

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4
Q

why is the interactionist approach sometimes called the biosocial approach

A

acknowledges that there are biological, psychological and social factors in development of schizophrenia

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5
Q

what do biological factors in interactionist approach include

A

genetic vulnerability, neurochemical and neurological abnormality

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6
Q

what do psychological factors in in interactionist approach include

A

stress such as from life events and daily hassles such as social factors and poor quality interactions in the family

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7
Q

what is one way to present in interactionist approach

A

diathesis-stress

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8
Q

what does diathesis-stress mean

A

-diathesis means vulnerability
-stress (in this context) means a negative experience

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9
Q

what does the diathesis-stress model say

A

both a vulnerability to schizophrenia and a stress-trigger are necessary in order to develop the disorder. one or more underlying factors make a person particularity vulnerable to schizophrenia but the onset is triggered by stress

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10
Q

who did original diathesis-stress model

A

Meehl’s

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11
Q

what was Meehl’s diathesis-stress model

A

diathesis was entirely genetic, as a result of one schziogene. this led to the idea of biologically based schizotypic personality, one characteristic of which is sensitivity to stress. according to Meehl, if a person does not have the schziogene no amount of stress would lead to schizophrenia. however, in carriers of the gene, chronic stress through childhood and adolescence, in particular the presence of a schizophrenogenic mother could result in development of the disorder

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12
Q

how has modern understanding of diathesis changed from Meehl’s model

A

clear that many genes each appear to increase genetic vulnerability only slightly, there is no single schziogene (Ripke et al)

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13
Q

what do modern view of diathesis also include

A

a range of factors beyond the genetic, including psychological trauma (Ingram and Luxton)- so trauma becomes the diathesis rather than the stressor

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14
Q

what did Read et al propose

A

neurodevelopmental model

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15
Q

what was Read et al neurodevelopmental model

A

early trauma alters the developing brain. early and severe trauma, such as child abuse, can seriously affect many aspects of brain development. for example the hypothalamic-pituitary-adrenal system can become overactive, making a person much more vulnerable to later stress

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16
Q

what side of the nature-nurture debate was the original (Meehl’s) model on

A

psychological in nature, in particular related to parenting

17
Q

what is the modern definition of stress

A

anything that risks triggering schizophrenia (houston et al)

18
Q

is psychological stress still important

A

yes, including that which results from parenting is still considered important, but the modern definition of stress includes anything that risks triggering schizophrenia

19
Q

what is a recently researched factor into development of schizophrenia

A

cannabis use

20
Q

role of cannabis in the diathesis-stress model

A

cannabis is a stressor as it increases the risk of schizophrenia by up to 7 times according to dose

21
Q

why does cannabis increase risk of schizophrenia

A

interferes with the dopamine system

22
Q

why do most people that smoke cannabis not develop schizophrenia

A

do not have the requisite vulnerability factors

23
Q

what types of treatments is the interactionist model compatible with

A

biological and psychological as acknowledges both biological and psychological factors

24
Q

what particular treatments are associated with the interactionist approach

A

combining antipsychotics and psychological therapies (most commonly CBT)

25
what did Turkington et al point out
possible to believe in biological causes of schizophrenia and still practice CBT to relieve psychological symptoms. however, this does require adopting an interactionist approach- it is not possible to adopt a purely biological approach and tell people diagnosed with schizophrenia that their condition is purely biological and there is no psychological significance to symptoms, and then to simultaneously treat them with with CBT
26
what is the increasingly standard practice for treating schizophrenia in britain
combination of antipsychotic drugs and CBT
27
how is schizophrenia treated in the US
more of a history of conflict between psychological and biological models of schizophrenia which may have led to a slower adoption of the interactionist approach
28
how does standard treatments in US and UK change treatments
medication without accompanying psychological treatment is more common in the US and the UK
29
strength of the interactionist approach to schizophrenia - support for vulnerability and triggers
-evidence supporting the role of both vulnerability and triggers -in a large scale study by Tienari et al, the impact of both genetic vulnerability and psychological trigger (dysfunctional parenting) was investigated. the study followed 19000 Finnish children whose biological mothers had been diagnosed with schizophrenia. in adulthood this high genetic risk group were compared to a control group of adoptees without a family history of schizophrenia (low genetic risk). adoptive parents has been assessed for child-rearing style and it was found that high levels of criticism of schizophrenia, but only in the high genetic risk group --> shows that a combination of genetic and vulnerability and family stress can lead to greatly increased risk of schizophrenia
30
limitation of the interactionist approach to schizophrenia - diathesis and stress are complicated
-original diathesis stress model is oversimplified -now clear that the original model that portrayed diathesis as a single schziogene and portrayed stress as schizophrenogenic parenting is hopelessly simplistic. multiple genes in multiple combinations influence diathesis. stress also comes in many forms, including but not limited to dysfunctional parenting. in facts, diathesis can also be influenced by psychological factors and stress can be biological as well as psychological. this is shown in a a study by Houston et al in which childhood vulnerability to schizophrenia and cannabis as the major trigger --> means multiple factors, both biological and psychological, affecting both diathesis and stress, supporting the modern understanding of both diathesis and stress
31
strength of the interactionist approach to schizophrenia - real-world application
-in combination of biological and psychological treatments -practical application of acknowledging biological and psychological factors in schizophrenia has been combination of drug treatments and psychological therapies. studies show that combining treatments enhanced their effectiveness. Tarrier et al randomly allocated 315 participants to either medication + CBT, medication + counselling ot the control group of just medication. participants in two combination groups showed lower symptoms following the trail than the medication-only group, though there was no difference in hospital readmission --> means clear practical advantage to adopting an interactionist approach to schizophrenia in terms of superior treatment outcomes
32
limitation of the interactionist approach to schizophrenia - counterpoint to real-world application
Jarvis and Okami point out that saying that a successful treatment for mental disorder justifies a particular explanation is the logical equivalent of laying that because alcohol reduces shyness, shyness is caused by a lack of alcohol. this logical error is called treatment causation fallacy --> cannot automatically assume that success of combined therapies means interactionist explanations are correct
33
evaluation of the interactionist approach to schizophrenia - urbanisation
-schizophrenia is more commonly diagnosed in urban than rural areas. this is sometimes used to justify the interactionist position, as it assumes that urban living is more stressful than rural and therefore city living acts as a trigger -however, it may simply be that schizophrenia is more likely to be diagnosed in cities, or that people with diathesis for schizophrenia (teenagers abused as children) tend to migrate to cities