Initial survey Flashcards

1
Q

Define triage

A

Triage is the process of “sorting out” patients on initial present to the emergency centre. Triage requires a focused on assessment of the “major body systems” to determine the urgency of the situation. All patients presenting for emergency treatment should be triaged immediately with the aim of identifying those requiring immediate emergency treatment and those who are stable and can wait for veterinary attention.

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2
Q

Examples of patients that should be triaged as urgent (6)

A
Examples of patients that should be triaged as urgent include those with:
• Major trauma
• Head trauma
• Haemorrhage
• Altered mentation
• Cluster seizures or status epilepticus
• Inability to walk or stand
• Snake bite
Any patient assessed as unstable, should be immediately brought to the treatment room for rapid assessment by a veterinarian.
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3
Q

Define and describe primary survey

A

This first evaluation is sometimes termed the ‘primary assessment’ or ‘major body system assessment’. The aim of the primary assessment is to evaluate “what will kill the patient first” in order to initiate life-saving treatment as soon as possible. Major body system assessment includes evaluation of cardiovascular, respiratory and neurological stability in addition to abdominal palpation (+/- AFAST* examination)
and rectal temperature. Once a primary assessment has been performed and any life threatening issues addressed, then a secondary (more thorough) assessment can take place.

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4
Q

What is MBSA

A

Rapid assessment of CVS, RESP, NEURO, ABDO palp, RECTAL TEMP

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5
Q

Describe MBSA - CVS

A

Cardiovascular system evaluation: Heart rate and rhythm, mucous membrane colour, CRT and pulse quality including synchronicity with HR should all be assessed. Any haematological or cardiovascular condition that leads to reduced DO2 warrants immediate treatment.
Assessing pulses
Pulse pressure is the difference between systolic and diastolic pressure. Poor pulse quality is reduced or loss of palpable pulse pressure.
Pulses may be described in reference to their height (tall or short) and in reference to their width (wide or narrow). Various other terms have been used to describe pulse quality, e.g., bounding, thready or weak. The term ’weak’ typically refers to short, narrow pulses. The term ‘bounding’ generally refers to tall, narrow pulses. Pulse pressure varies with stroke volume and compliance of the artery. Dogs and cats have little variation
in compliance therefore changes in pulse pressure are likely to reflect stroke volume. A tall wide pulse reflects an increased stroke volume such as occurs in a tachycardic euvolaemic animal. A tall narrow pulse occurs with rapid ejection of a reduced stroke volume such as occurs in early hypovolaemia. A short narrow pulse reflects a greatly reduced stroke volume such as occurs with advanced hypovolaemia or cardiac failure.

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6
Q

Equation for MAP?

A

Mean arterial pressure (MAP) = [1/3(SAP – DAP)] + DAP
where SAP = systolic arterial pressure and DAP is diastolic arterial pressure. Therefore, MAP is approximately 1/3 of the pulse pressure.

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7
Q

Describe MBSA - resp

A

Respiratory system evaluation: Respiratory rate, breathing effort and pattern should be assessed; any abnormal noises such as stertor or stridor should also be noted. Respiratory instability again warrants immediate treatment. Instability includes rapid respiratory rate, laboured or shallow breathing, open mouth breathing, orthopnoea and any other abnormal patterns such as paradoxical movement of the abdomen.

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8
Q

Desc MBSA - Neuro

A

Neurological system evaluation: Level of mentation or consciousness and ability to stand or ambulate should be assessed. Instability includes any alterations of mentation or consciousness, acute limb paresis or paralysis, current seizures or recent seizure activity.

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9
Q

Desc MBSA - abdominal assessment

A

Abdominal assessment: The patient’s abdomen should be palpated for pain; excessive pressure; typany; masses; a fluid wave. An AFAST exam will rapidly rule in or out abdominal effusion.

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10
Q

Desc MBSA - rectal temp

A

Rectal temperature is an important part of the primary assessment of a patient. Hyperthermic patients may present with obstructive breathing or heat stroke, while a low rectal temperature may be indicative of shock.

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11
Q

What is FAST exam?

A

FAST examination is becoming increasingly used as part of the primary assessment of emergency patients (both with and without trauma). Thoracic FAST (TFAST) exams allow recognition of pericardial effusion and tamponade, pleural effusions, pneumothorax, increased left atrial to aortic ratio and subjective evaluation of cardiac contractility. Abdominal FAST (AFAST) exams can identify peritoneal or retroperitoneal effusions, allowing rapid detection of abnormalities that may be key to diagnosis of the underlying disorder. E.g. haemorrhagic effusions would identify haemorrhage as the cause of hypovolaemic shock, modified transudates indicate third spacing of fluid which can cause hypovolaemic and possibly distributive shock if volumes are high, and identifying septic effusions would indicate sepsis as the cause of shock.

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12
Q

Why is rapid assessment of shock important?

A

Patients in shock are unstable patients and warrant immediate attention. Early recognition of shock allows for timely intervention and an increased chance of halting progression into potentially irreversible shock. Shock can ultimately result in cell death and, left unchecked, can cause organ failure and patient death. However, re-perfusion and renewed delivery of oxygen to the cell can also cause cell death due to the formation of reactive oxygen species (ROS). It is therefore clear that prompt and effective treatment of shock is vital for patient wellbeing.

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13
Q

Clinical parameters in the assessment of shock?

A
Patients in shock will generally present with some or all of the following, depending on the degree of cardiovascular instability or cause of shock:
• Increased or decreased heart rate
• Pale or injected mucous membranes
• Inappropriately fast (< 1 sec) or slow (> 2 sec) CRT
• Poor pulse quality
• Tachycardia
• Tachypnoea
• Dull mentation
• Cool extremities and/or hypothermia
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14
Q

What is the shock index?

A

Shock Index = heart rate/systolic blood pressure
The shock index is a measure that designed act as a tool for triage and assessment of the severity of shock. Unfortunately, indirect blood pressure measurement is not reliable in hypotensive veterinary patients and heart rate can vary widely. There is no clear advantage to using a shock index over traditional clinical parameters for triage or assessment of shock.

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15
Q

Which type of shock is important to differentiate and why? What are some PE indicators of this type of shock?

A

Initially, it is most important to differentiate cardiogenic shock from other forms of shock, as the mainstay of treatment for most forms of shock is aggressive fluid therapy, which would be fatal to a patient suffering cardiogenic shock. Pneumothorax is another exception to the fluid rule, where correction of shock requires removal of the air that is compressing venous return and causing distributive shock.
Patients presenting in cardiogenic shock will have specific indicators of cardiac dysfunction. Auscultation of a cardiac murmur or gallop, palpation of pulse deficits, presence of arrhythmias on ECG, documentation of a pericardial effusion or increased left atrial to aortic ratio on ultrasound exam are indicative of underlying cardiac disease. Patients with congestive heart failure may present with dyspnoea, tachypnoea, cyanosis, pulmonary crackles or pleural effusion.

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16
Q

What is an emergency database?

A

The emergency database is an integral part of assessing the emergency patient. Ideally, blood for an emergency database is collected and analysed during the primary assessment. It is useful to draw the required blood sample from an intravenous catheter. The emergency database can aid in the recognition of shock and help diagnosis of the underlying cause. Commonly run tests are PCV/TS (packed cell volume and total solids), electrolytes, glucose, lactate and venous blood gases. This allows for immediate recognition of hypo and
hyper glycaemia, anaemia, haemoconcentration, acute haemorrhage, hyperlactataemia, hypoventilation, alterations in pH, and electrolyte derangements.

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17
Q

How can you detect acute haemorrhage?

A

Detecting acute haemorrhage can be tricky. Total solids will always decrease before the PCV as splenic contraction elevates the PCV in the initial stages of haemorrhage. If TS is less than ≤ 50 g/L then this raises the suspicion of acute haemorrhage.

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18
Q

Why is assessment of glucose important as part of our initial database?

A

Detecting acute haemorrhage can be tricky. Total solids will always decrease before the PCV as splenic contraction elevates the PCV in the initial stages of haemorrhage. If TS is less than ≤ 50 g/L then this raises the suspicion of acute haemorrhage.

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19
Q

Why is assessment of acid base status important as part of our initial database?

A

ilst arterial blood gases are the gold standard for assessment of hypoxaemia, it is uncommon to have access to an arterial sample when an emergency patient first presents. More commonly, venous blood gases are assessed. Venous blood gases give us information on the acid-base status of the animal as well as ventilator function. Low venous oxygen may increase the index of suspicion of arterial hypoxaemia or increased oxygen extraction ratio, however venous oxygen should be interpreted with care, as it can be unreliable. Peripheral venous oxygen depends on cardiac output, oxygen consumption by tissues and arterial oxygen content. A change in any of these three components can cause venous oxygen to be low. Normal PvO2 is approximately 40 mm Hg. A PvO2 below 30 mm Hg should prompt investigation.

20
Q

Two types of hyperlactataemia?

A

Type A & Type B

21
Q

Describe type A hyperlactataemia

A

Increased serum lactate concentrations may indicate tissue hypoperfusion (this is called Type A hyperlactataemia).

From this is can be seen that the by-products of anaerobic glycolysis are lactate and H+. Acidaemia results when the body’s buffer systems are overwhelmed. Hence the occurrence of lactic acidosis. The term lactic acidosis, should not be confused with lactic acid (the conjugate acid of lactate ion). Lactate remains completely dissociated at a physiological pH so acts as a strong ion. Metabolism of lactate (occurring primarily in the liver) consumes a hydrogen ion. This is the reason that the lactate in Lactated Ringers Solution (Hartman’s) is able to act as a buffer.

22
Q

Describe the basic biochemistry behind Type A hyperlactataemia

A

Glycolysis produces 2 moles of pyruvate and 2 moles of ATP for every mole of glucose used.
Glucose—› 2pyruvate+2ATP+NADH+2H+
In the presence of oxygen, pyruvate is converted to acetyl CoA and enters the citric
acid cycle (also called the Krebs cycle). Whilst the hydrogen ions produced by glycolysis are taken up by the mitochondria and used in the electron transport chain for oxidative phosphorylation.

In the absence of oxygen, pyruvate cannot be converted to acetyl CoA so the products of glycolysis (NADH pyruvate and H+) build up within the cytoplasm. The production of lactate from pyruvate serves to regenerate NAD+ from NADH* and allows continued removal of pyruvate from the cytoplasm. Removal of pyruvate is necessary in order for glycolysis to continue uninhibited.

*NADH is reduced nicotinamide adenine dinucleotide and NAD+ is oxidised nicotinamide adenine dinucleotide
Pyruvate + H+ + NADH —› lactate + NAD+

The ATP produced by glycolysis in the absence of oxygen is used by the cell and is hydrolysed within the cytoplasm. Hydrolysis of ATP produces H+. This means that glycolysis in the absence of oxygen produces H+ due to cytosolic hydrolysis of ATP.
2 ATP + H20 —› 2 ADP + 2 HPO4 + 2 H+

23
Q

What are the byproducts of anaerobic metabolism?

A

From this is can be seen that the by-products of anaerobic glycolysis are lactate and H+.

24
Q

What causes academia?

A

Acidaemia results when the body’s buffer systems are overwhelmed.

25
Q

What is lactic acidosis?

What fluid type and why is useful with lactic acidosis?

A

Acidaemia results when the body’s buffer systems are overwhelmed. Hence the occurrence of lactic acidosis. The term lactic acidosis, should not be confused with lactic acid (the conjugate acid of lactate ion). Lactate remains completely dissociated at a physiological pH so acts as a strong ion. Metabolism of lactate (occurring primarily in the liver) consumes a hydrogen ion. This is the reason that the lactate in Lactated Ringers Solution (Hartman’s) is able to act as a buffer.

26
Q

What is type B hyperlactataemia?

A

The generation of lactate without acidosis occurs when there is increased glycolysis in the presence of oxygen and functioning mitochondria. This type of hyperlactataemia is called Type B. Hyperlactataemia in this circumstance is mild to moderate and will not usually result in lactate levels over 6 mmol/L. When moderate to severe hyperlactataemia occurs (> 3 mmol/L) it is almost always the result of anaerobic glycolysis. Examples of Type B hyperlactataemia include extreme tissue energy demands such as seizures, mitochondrial dysfunction and increased cortisol or catecholamine levels.

27
Q

Why is it important to measure lactate?

A

Because lactate is a biomarker for anaerobic glycolysis (and thus poor cellular perfusion), the monitoring lactate concentrations during fluid resuscitation can allow you to determine the adequacy of fluid resuscitation (i.e. improved perfusion and thus oxygen delivery).

28
Q

Describe monitoring in the emergent patient?

A

Like the emergency database, monitoring equipment can supplement the knowledge that is gained from the clinical examination of an emergency patient. Basic monitoring equipment should be set up as soon as a patient is triaged to be unstable. This should including ECG, non-invasive blood pressure monitoring (NIBP), and pulse oximetry.

29
Q

What is blood pressure, how and why do we measure it?

A

Blood pressure is a measurement of the pressure exerted at a particular time and point
on a vessel wall. Blood pressure monitoring allows recognition of hypotension (i.e. systolic arterial pressure of < 90 mm Hg or mean arterial pressure of < 60 mm Hg). Hypotension results in inadequate tissue perfusion and requires immediate intervention.

30
Q

What use does blood pressure assessment in shock have?

A

Falling blood pressure
is a late change in the continuum of shock as it reflects failing of these compensatory mechanism. In addition to hypotension being a late change, indirect blood pressure measurements in hypotensive patients tend to be inaccurate. For these reasons, the measurement of blood pressure should not be relied on as a sole guide for resuscitation of hypotensive shock.

31
Q

Desc pulse oximetry monitoring

A

Pulse oximetry (SpO2) estimates the saturation of haemoglobin with oxygen (SaO2). It can however, be unreliable in patients in shock if peripheral perfusion is poor. For the pulse oximetry reading to be considered reliable, it is important that the pulse rate detected matches that of the patients pulse and that the plethysmogram shows a reliable pulse wave.

32
Q

Desc EDG monitoring

A

Electrocardiograph monitoring allows hands off assessment of heart rate and rhythm. In particular, intermittent arrhythmias and trends in heart rate may be missed if continuous ECG monitoring is not used.

33
Q

What are advanced monitoring parameters in the critically ill patient

A

Critically ill patients that are hypotensive can be complex to monitor. They will often
be suffering from more than one type of shock. For instance, a patient with systemic inflammatory response syndrome (SIRS) may be suffering from hypoxaemic, hypovolaemic, metabolic and distributive forms of shock all at once. Invasive monitoring may be required in the case of septic shock or critically ill patients. Placement of arterial catheters for invasive blood pressure monitoring and serial PaO2 measurements and/or central venous catheters for central venous pressure (CVP) monitoring and measurement of central venous oxygen saturation (ScvO2) can be done in the ICU setting to further advance patient monitoring with the aim of more accurately tailoring treatment.
The following parameters may be used in the critical care setting in order to monitor for and guide for shock:
• Direct blood pressure monitoring
• Urine output
• CVP
• ScvO2
• Cardiac output

34
Q

Desc direct ABP measurement

A

Direct arterial blood pressure monitoring is considered the gold standard for blood pressure monitoring. It gives the most accurate measurement of blood pressure and allows minute- to-minute changes to be observed.

35
Q

What is normal UOP?

What is the minimum UOP goal in shock resuscitation?

A

Normal urine output is approximately 1-2 mL/kg/hr. A minimum urine output of >0.5 mL/ kg/hr should be aimed for in shock resuscitation. Many critical care patients have a urinary catheter that will allow this parameter to be monitored.

36
Q

What is CVP?

A

The use of CVP to guide fluid therapy is controversial as individual measurements can vary greatly. Even so, if CVP is low it is likely that the patient is hypovolaemic and may benefit from additional fluid therapy. Ideally, CVP should be > 0 cm of water.

37
Q

What is central venous oxygen saturation?

A

Central venous oxygen saturation (ScvO2) is a measure of the percentage saturation of haemoglobin with oxygen in the cranial vena cava. It is used as a surrogate marker for oxygen saturation of mixed venous blood (SvO2). Mixed venous oxygen saturation is a measure of the percentage saturation of haemoglobin with oxygen in the pulmonary artery. This parameter is thought to representative of global oxygen utilisation. Blood in the pulmonary artery is a mix of blood returned from the entire body, while blood in the cranial vena cava is reflective of blood returning from the upper body and head with less influence from mesenteric, renal, coronary and other peripheral vascular beds. An ScvO2 of >70%
is a recommended end point goal of resuscitation. Central venous oxygen saturation is decreased if oxygen delivery to tissues is decreased (decreased cardiac output, hypoxaemia or anaemia) or if cellular utilisation of oxygen is increased (seizures, fever). Central venous oxygen saturation is increased in the hyperdynamic phase of sepsis (cytopathic hypoxia of sepsis). Decreasing ScvO2 may be an early indicator of clinical deterioration.
One human study showed that there was no benefit to the use of ScvO2 as a resuscitation goal over and above the use of lactate clearance6.

38
Q

How to we monitor CO?

A

Cardiac output monitoring is commonly used in human critical care patients. Cardiac output monitoring aims to further define cardiovascular status and therefore guide fluid, pressor and inotrope therapies over and above what can be achieved with standard monitoring. Even so, there is currently no evidence to support its routine use in veterinary patients. This is likely to change with the advancement of technology available to measure cardiac output. Cardiac output monitoring will be further discussed in Subject 4 of this course.

39
Q

Resuscitative goals in the treatment of hypotensive shock? (overall goal, not individual parameters)

A

he ultimate goal of the treatment of shock is to normalise oxygen delivery to tissues. Normalisation of vital signs and laboratory values indicate that therapeutic interventions are working and homeostasis is being restored. When monitoring shock resuscitation, it
is important to assess the patient as a whole. No one parameter can guide resuscitation efforts. An animal that was originally tachycardic may have a heart rate that falls to be within normal range for a period before progressing to bradycardia followed by cardiac arrest. This example demonstrates the importance of checking a number of parameters in order to assess a response to treatment.

40
Q

Resuscitative shock parameters?

A
The following parameters should be assessed repetitively during resuscitation of shock:
• Mentation
• Heart rate and rhythm
• Mucous membranes
• Pulse rate and pressure
• Systolic and mean arterial pressure
• Respiratory rate and effort and SpO2
• Temperature
• PCV/TS
• Serum lactate
41
Q

Pulse palpation as a resuscitative parameter

A

Both femoral and peripheral pulses should be monitored. Peripheral pulses will be absent whilst femoral pulses are still present. Improvement in pulse quality is a subjective measure and there are no absolute measurement values when appreciating pulses; however,
an improvement in pulse quality height and width should be evident during effective resuscitation.

42
Q

Describe blood pressure as a resuscitative goal in hypotensive shock?

A

It is important not to rely on blood pressure measurements, as stated above; they can be inaccurate in our patients. The range for normal systolic arterial pressure in the dog is 130 – 170 mm Hg and in the cat is 120 – 170 mm Hg. The range for normal mean arterial pressure values in both species are approximately 95 – 115 mm Hg. Targeting normal blood pressure during resuscitation is not necessarily aim. Rather the aim is to target a blood pressure that will ensure cerebral perfusion. Cerebral perfusion occurs when MAP is > 60 mm Hg, and SAP > 90 mm Hg. Hypotensive resuscitation is a term applied to resuscitation that specifically targets lower than normal blood pressure values. Hypotensive resuscitation is recommended for patients with haemorrhagic shock (i.e. hypovolaemic shock secondary to acute haemorrhage). Judicious crystalloids to restore the SAP to no greater than 90 – 100 mm Hg. The aim of hypotensive resuscitation is to prevent disruption of haemostatic clots and to prevent excessive dilution of coagulation factors. Hypotensive resuscitation is a temporary measure that ensures cerebral perfusion whilst haemostasis is achieved

43
Q

RR, effort and Pulse oximetry as a resuscitative parameter

A

Normal values are between 20-30 breaths per minute in healthy patients. Respiratory rate and effort can be increased, decreased or normal in animals presenting in shock. Increased respiratory rate may reflect hypoxaemia or acidosis. While pulse oximetry measurements may be unreliable, it is unusual for a normal SpO2 reading to be erroneous.

44
Q

Temperature as a resuscitative parameter

A

Normalisation of temperature in cardiac patients can be a good marker that peripheral perfusion is being restored. Animals with hypovolaemic shock whom are receiving large volumes of cold fluid therapy may have a further drop in body temperature; this should not be interpreted negatively.

45
Q

PCV/TS as a resuscitative parameters

A

Packed cell volume and total solids will fall with fluid resuscitation. Monitor PCV/TS every 15-30 minutes while infusing shock rate fluids in order to detect signs of haemorrhage or developing anaemia. Rapidly falling PCV/TS values may indicate ongoing haemorrhage that requires hypotensive resuscitation, haemostatic control and blood product administration

46
Q

Lactate as a resuscitative parameter

A

Serial lactate measurements are used during shock resuscitation as a marker of tissue perfusion and thus efficacy of shock treatment. In early shock resuscitation, lactate values can also increase as blood flow is restored to tissues and lactate enters the circulation.

47
Q

What is goal directed therapy?

A

Goal-directed therapy refers to the use of specific targets (goals) to guide resuscitation
of patients in shock. The chosen goals are predetermined and linked to a management algorithm that the treating clinician follows. The use of goal directed therapy remains controversial in human medicine. Some studies in have shown improved patient outcomes when goal-directed therapy is used early in the treatment of shock4. Whilst more recently, investigations in human septic shock patients have not shown improved outcomes5.