Implant Complications, Peri-Implantitis and Treatment Flashcards
BIOLOGY OF IMPLANT
Epithelial Attachment
(3)
2mm
Long junctional epithelium attached implant
Via basal lamina and hemidesmosomes
BIOLOGY OF IMPLANT
Connective Tissue
(3)
Parallel, circular “cuff-like” fiber bundles
Seal with a space of a 20nm wide proteoglycan layer
1-1.5mm high
Supracrestal connective tissue attachment
for Implants
– mm
3-4
Soft Tissue Assessment
(3)
Dimensions of the papilla
Probing
Dimensions of the buccal soft tissue
Osseointegration vs PDL
(2)
Periodontal mechanoreceptors
Higher stress at the neck of the screw/implant
PDL space ~
(2)
0.2mm
Sensory feedback
Timed occlusal contacts
Teeth opposing teeth: – microns
Implant opposing teeth: – microns
Implant opposing implant: – microns
20
48
64
Vascularity
(3)
Limited in peri-implant gingival mucosa
Sources are from alveolar bone and the connective tissue
Same inflammatory response to plaque
Proximity limitations:
Vertical soft tissue limitations:
Tooth-tooth
Tooth-implant
Implant-implant
1mm 5mm
1.5mm 4.5mm
3mm 3.5mm
NEW CLASSIFICATION
Peri-implant Diseases and Condition
(4)
Peri-implant health
Peri-implant mucositis
Peri-implantitis
Peri-implant hard and soft tissue deficiencies
Peri-implant mucositis
Prevalence:
–% of patients
–% of implants
79
50-90
Peri-implant mucositis
(4)
Caused by plaque accumulation.
Presence of inflammation.
Reversible condition.
Precursor of peri-implantitis.
Peri-implantitis
Prevalence:
–% of patients
–% of implants
20
10-56
Peri-implantitis
(4)
Caused by plaque accumulation.
Presence of inflammation.
Loss of supporting bone.
Non-reversible condition.
Peri-implant hard and soft tissue deficiencies
Contributing factors:
(6)
tooth loss, trauma, periodontitis, thin
soft tissue, lack of keratinized mucosa,
implant malposition, etc.
The microbiome may be different
although the opportunistic
periodontal pathogens can be
identified in —
patients
peri-implantitis
Stronger inflammatory response was
around
implants than teeth; need
longer time to complete reverse
peri-mucositis than gingivitis
Peri-implantitis contained larger
proportions of (2) than in periodontitis
neutrophil granulocytes
and osteoclasts
Peri-implantitis
risk factors/indicators
(7)
micromovement
Poor plaque control
Lack of regular
maintenance
Tissue quality: thin
phenotype, bone
deficiency
Iatrogenic factors:
malpositioning, poor
design of emergency
profile, inadequate
abutment/implant
seating
Excessive cement
Occlusal overload
Titanium particles:
implant corrosion,
Peri-implantitis risk
indicators/modifiers
(4)
History of
periodontal disease
Smoking
DM
Genetic factors/
systemic condition
Disease presentation
(4)
Inflammation:
redness, swelling
Pain
Suppuration
Bone loss
CLINICAL EXAMINATION
(3)
Peri-implant tissue
Occlusion and mobility
Plaque, probing depth, BOP, exudates
Peri-implant probing
CLINICAL EXAMINATION
Diagnostic Procedures
Variables in peri-implant probing:
- Probe Positioning
- Presence of Inflammation (BoP, Exudates)
Plastic or Metal?
Occlusal overload
(3)
Loosening of abutment screws
Implant failure
Prosthetic failure