IM Final Red Boxes - Sheet1 Flashcards
Total peripheral resistance of maternal
Goes down because 2 parallel circuits
Maternal blood volume increases how much
35%
Maternal plasma volume increases how much
45%
Maternal erythrocyte volume increases how much
20%
Maternal platelets increase how much
0
Maternal minute ventilation
^50%
Maternal alveolar ventilation
^70%
Maternal tidal volume
^40%
Maternal respiratory rate
^15%
Maternal closing volume
unchanged or slightly decreased
Maternal Arterial pH
Unchanged
Maternal arterial PO2
^10mmHg
Maternal arterial PCO2
v10mmHg
Maternal airway resistance
v36%
Maternal vital capacity
unchanged
Maternal inspiratory lung capacity
unchanged
Maternal frc
v20%
Maternal tlc
unchanged
Maternal erv
v20%
maternal rv
v20%
maternal O2 consumption
^20%
Fetal heart rate
120-160
Fetal stress
vperfusion, hypoxia, vpH
NST
Nonstress test
NST looks at
FHR and movement over 15-60min period
want to see with NST
veriability and accelerations=negative NST
use vibrator for
transabdominal stimulation to increase FHR
BPP
Biophysical Profile
BPP monitors
fetal breathing, body movements, tone, heart rate reactivity, and amniotic fluid volume
CST
Contraction stress test
CST monitors
FHR over 10mins with 3 contractions(induced vs noninduced)
CST wants to see
variability and accelerations=negative CST
FSpO2
Fetal oxygen saturation
FSPO2 checked with
fetal scalp probe
concerning fetal SaO2
<30%
Normal fetal scalp blood gas pH
7.25-7.35
Normal fetal scalp blood gas SO2
30-50%
Normal fetal scalp blood gas PO2
18-22mmHg
Normal fetal scalp blood gas PCO2
40-50mmHg
pH vs APGAR
normal to high pH associated with higher apgar
SpO2 vs APGAR
normal to high SpO2 associated with higher APGAR
Classes of fetal heart rate variability
no, reduced, normal, increased
No FHR variability
range undetectable
Reduced FHR variability
0-<5beats/min
Normal FHR variability
5-10beats/min
Increased FHR variability
> 15beats/min
tocograph
contraction graph
FHR accelerations
well being
FHR early decelerations
non pathological, normal, not from hypoxia
FHR late decelerations
uteroplacental insufficiency, decreased uterine bf, hypoxia, chemoreceptors fire resulting in vagal discharge
FHR variable decelerations
vagal firing in response to cord compression or sustained head compression
APGAR
Activity, pulse, grimace, appearance, respiration
APGAR min and max
0-10
first question, who’s the painter
Gustov
Accuracy of peristaltic pump
”+/-5%”
Accepted accuracy of infusion pumps
”+/-5%”
BARD infusion accuracy for infusion and bolus
”+/-3%”
How do you set up an infusion on a general pump?
no idea
Most accurate pump
syringe pump
BET scheme
Bolus elimination transfuse
Target controlled infusion
they took our jobs
Advantages of patient controlled analgesia
Pt autonomy, rapid pain relief, dosage tailored to requirements
Key points to PCA system
route of administration, type of administration, ease of programming, ease of priming, power source, safety, security, portability, display, printout
Where is POC useful
ER, ICU, CCU, Ob Suites, NICU, Burn unit, Trauma unit, OR
Issues influencing intro of POC testing
Personnel and training, QC, proficiency testing, calibration, certification, records and doc, integration with central lab
Analyzer
Evaluates blood permanently withdrawn from pt
Monitor
In vivo or Ex vivo
Preanesthesia check for offsite/mri
Emergency backup power to ensure pt protection for unforeseen circumstances. 10) Adequate means to illuminate the pt, anes machine, and monit equip. 13) If you’re giving gas, have equip to monitor everything just like in the OR
Depth for esophageal stethoscope
30cm
Depth for esophageal temp probe
38-42cm past teeth
Depth of nasal temp probe
tragus
Temp where regulation is lost
28C
Lower limit of survival
23-28C
Skin contributes what percent to control of thermoregulatory defense
20%
Major complications of mild perioperative hypothermia
surgical wound infection, morbid cardiac events, myocardial damage, duration of postanesthetic recovery, adrenergic activation, mortality after major trauma
Potential benefits of mild perioperative hypothermia
dec mortality after brain trauma, inc glasgow score 12mos after brain trauma, neurologic outcome after cardiac arrest
spinal anesthesia does to threshold of sweating and vasoconstriction/shivering
inc sweating threshold and dec vasoconstriction/shivering
Skin and core temp relation
directly related
Difference between temps of shivering and vasoconstriction
as little as .2 deg
Heat loss
RACEC
Radiation loss
65%
Convection loss
25%
Evaporation/respiration loss
10%
Conduction heat loss
min
Heat loss after 30min
1C
Heat loss after 1hr
1.6C
Heat loss levels after
3C loss after 3hr
Change of shivering point gas vs spinal
Gas decreases shiv thresh more than spinal
Temp variation in esphagus
”+/-4C
Measured temps highest to lowest(excl axillary and forehead)
Rectal-Bladder. Everything else is about the same
Axilary and forehead temps compared to rest
2-3C lower but same trend
Cooling and warming occur most rapidly where
esophagus and nasopharynx
Dec temp of 2 L of RT fluid
.5C
Dec temp of 4 L of RT fluid
1C
Dec temp of 2 L of 4C fluid
1C
Dec temp of 4 L of 4C fluid
2C
Measures heat dissipation
heated wire anemometer
Measures pressure gradient
pseumotachometer
All ventilators display what 3 things
volume, rate, and MV
where to place pressure flow sensors
B&D on picture, both inspiratory and exiratory limbs
why respirometer on expiratory limb?
So you can detect a disconnect early, even a ett leak
Why place respirometer in positions A&B on exp limb?
So you can detect reverse flow and a malfunction unidirectional valve
Pressure control flow volume loop shape
parallelogram