IHD and heart failure Flashcards

1
Q

cardiac causes of chest pain

A

Acute coronary syndrome (unstable angina and myocardial infarction).
Stable angina.
dissecting thoracic aneurysm, pericarditis, cardiac tamponade, myocarditis, acute congestive cardiac failure, or arrhythmias.

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2
Q

resp causes of chest pain

A

Pulmonary embolus, pneumothorax or tension pneumothorax, community-acquired pneumonia, asthma, or pleural effusion.

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3
Q

other causes of chest pain - non cardiac or resp

A

Gastroenterological causes, such as acute pancreatitis, oesophageal rupture, peptic ulcer disease, gastro-oesophageal reflux, oesophageal spasm, or oesophagitis.
Musculoskeletal causes, such as rib fracture, costochondritis, spinal disorders (disc prolapse, cervical spondylosis, facet joint dysfunction), rheumatoid or psoriatic arthritis, fibromyalgia, or osteoporotic fracture.
Cancer (for example, lung cancer); herpes zoster; Bornholm disease; precordial catch (Texidor twinge); or psychogenic or non-specific chest pain.

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4
Q

assessment referral

A

urgent same day - suspected ACS and pain free with chest pain in last 12 hours and normal ECG, no chest pain and no complications, or chest pain in last 12-72 hours
2 week - suspected ACS and pain free with chest pain >72 hours ago and no complications, suspected malignancy, lung collapse, or pleural effusion
routine - stable angina, chest pain of unknown cause

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5
Q

angina causes

A

coronary artery disease
valvular disease (aortic stenosis), hypertrophic obstructive cardiomyopathy, or hypertensive heart disease

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6
Q

define stable angina

A

physical exertion or emotional stress and relieved with mins of rest of GTN spray

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7
Q

define unstable angina

A

(usually within 24 hours) onset angina, or abrupt deterioration in previously stable angina, often occurring at rest. Unstable angina usually requires immediate admission, or referral to hospital.

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8
Q

stable angina management

A

smoking cessation
cardioprotective diet
increased physical activity
limit alcohol consumption

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9
Q

pharmacological management stable angina

A

GTN spray
beta blocker or CCB (second line - isosorbide mononitrate, ivabradine, nicorandil)
can be on 3rd drug while waiting for specialist assessment

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10
Q

secondary preventions of CVD events

A

for STEMI or NSTEMI
dual antiplatelet - low dose aspirin and…(ie rivaroxaban or clopidogrel)
ACEi (coexisting hypertension, heart failure, asymptomatic left ventricular dysfunction, chronic kidney disease, or previous myocardial infarction or DM)
statin
beta blocker

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11
Q

non drug treatments ACS

A

PCI - Primary PCI (if within 12 hours of symptom onset and within 120 minutes of the time when fibrinolysis could have been given) is the preferred strategy for most patients.
or CABG

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12
Q

initial treatment ACS

A

GTN
IV opioids - morphine
loading dose of aspirin
if low o2 sats - oxygen
monitor for hyperglycaemia
when in hospital - second antiplatelet (prasugrel) - unless high bleeding risk so aspirin alone

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13
Q

HF summary

A

https://www.nice.org.uk/guidance/ng106

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14
Q

define HF

A

progressive clinical syndrome caused by structural or functional abnormalities of the heart, resulting in reduced cardiac output. It is characterised by symptoms such as shortness of breath, persistent coughing or wheezing, ankle swelling, reduced exercise tolerance, and fatigue. These symptoms may be accompanied by signs such as elevated jugular venous pressure, pulmonary crackles, and pulmonary oedema.

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15
Q

risk factors for HF

A

men
smokers
diabetic
age
african or afro-caribbean
pre existing co-morbidites - hronic kidney disease, atrial fibrillation, hypertension, dyslipidaemia, obesity, diabetes mellitus, and chronic obstructive pulmonary disease

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16
Q

complications HF

A

CKD
afib
depression
cachexia
sexual dysfunction
sudden cardiac death

17
Q

reduced ejection fraction HF

A

the left ventricle loses its ability to contract normally and therefore presents with an ejection fraction of less than 40%

18
Q

preserved ejection fraction HF

A

the left ventricle loses its ability to relax normally therefore the ejection fraction is normal or only mildly reduced

19
Q

non pharm management HF

A

Smoking cessation, reducing alcohol consumption, increasing physical exercise if appropriate, weight control, and dietary changes such as increasing fruit and vegetable consumption and reducing saturated fat intake
Pts encouraged to weigh themselves (any weight gain of >1.5-2kg in 2 days)
salt intake <6g per day
dilutional hyponatraemia - fluid restriction
management if pregnancy - specialist
psychological support
Implantable cardioverter defibrillators and cardiac resynchronisation therapy are treatment options recommended in patients with heart failure and a reduced ejection fraction of less than 35%
pneumococcal and influenza vaccine

20
Q

HF with reduced ejection fraction

A

HF+ angina = amlodipine, other rate limiting drugs avoided
diuretics - loop (furosemide or bumetanide)
ACEi and beta blocker
if HF sx worsen or persist - spironolactone unless hyperkalaemia
then after if sx persist - HF specialist, can try digoxin if in sinus rhythm
possible anticoag

21
Q

preserved ejection fraction tx

A

heart failure specialist
low to medium dose loop diuretic

22
Q

advanced heart failure sx

A

breathlessness
…not oxugen unless also copd/other requirements

23
Q

define afib

A

results from irregular, disorganized electrical activity in the atria, leading to an irregular ventricular rhythm. The ventricular rate of untreated AF often averages between 160–180 beats per minute

24
Q

common causes of afib

A

ischaemic heart disease, hypertension, valvular heart disease, and hyperthyroidism

25
Q

complications of afib

A

Stroke and thromboembolism.
Heart failure.
Tachycardia-induced cardiomyopathy and critical cardiac ischaemia.
Reduced quality of life

26
Q

complications of afib

A

Stroke and thromboembolism.
Heart failure.
Tachycardia-induced cardiomyopathy and critical cardiac ischaemia.
Reduced quality of life

27
Q

management of afib

A

Admitting people who have severe symptoms or a serious complication.
Identifying and managing any underlying causes.
Assessing stroke risk using the CHA2DS2VASc assessment tool.
Assessing the risks and benefits of anticoagulation and starting treatment if appropriate — the ORBIT assessment tool should be used to assess the risk of major bleeding.
Treating the arrhythmia: a rate-control treatment (beta-blocker, rate-limiting calcium channel blocker, or digoxin) is recommended for most people with AF. Referral for rhythm-control treatment (cardioversion), in addition to rate-control treatment, may be appropriate if the person has AF with a reversible cause (for example a chest infection); heart failure thought to be primarily caused, or worsened, by AF; or new-onset AF.
Follow-up

28
Q

referral to cardiologist

A

rhythm control appropriate
Rate-control treatment fails to control the symptoms of AF (prompt referral within 4 weeks).
The person is found to have valvular disease or left ventricular systolic dysfunction on echocardiography.
Wolff–Parkinson–White syndrome or a prolonged QT interval

29
Q

more on management afib

A

haemodynamically stable + <48 hrs - LMWH, rate or rhythm control - amidodarone of flecainide for rhythm and beta blockers or CCB for rate or DC conversion
>48 hours - electrical cardioversion but delayed until fully anticoagulated for 3 weeks or TOE
haemodynamically unstable - electrical cardioversion urgent(synchronsied to R wave)

if cardioverted - anticoag for 4 weeks after