Idiopathic Flashcards
What is the most common population with Trigeminal Neuralgia?
elderly but can happen in the young
What is the etiology of Trigem neuralgia?
mechanical compresion of CN V nerve root
or
demylination plaques due to MS
What artery is most likely pathophysiologically at fault in Trigem Neuralgia?
Superio Cerebellar artery compress CN V root
What is the pathophysiology for Trigeminal Neuralgia?
peripheral sensory neurons of Trigem ganglia affected or central sensitization of Spinal trigeminal nucleus (espeically in final syndrome)
What is the presentation of trigeminal neuralgia
1) abrupt onset
2) electric shock, stabbing pain in regions of face
3) unilateral, rarely Bilateral
4) triggers can be area outside of area of pain, nonnoxious stimuli, cervical region triggers
What is the etiology of Trigeminal neuralgai?
idiopathic. some suggest familial but likely mulitfactorial compression or irritation of CN V root or ganglion
What is the etiology of Complex regional pain syndrome (causalgia)?
idiopathic often associated with post-trauma
What increases the liklihood of getting Complex regional pain syndrome?
sciatic nerve damage or distal nerve lesion
women are 3x as likely as men
mean age is early 40’s
What is the most common event that preceeds CRPS?
trauma 77% (sprain then post surgery then crush or compression)
-colles fracture, peripheral nerve lesion, sciatic nerve damage
What are some suggested pathologies for CRPS?
abberant healing, exaggerated inflammatory process, protective disuse, dysfunctional nervous system, myofascial, CNS abnormalities
What is the presentation for CRPS?
Can progress in 3 stages. Most cases present with burning pain and paresis, skin temperature and color change, limited ROM, hyperpathy, hyperesthesia, hypoesthesia, edema, alter hair and nail growth
What is the presentation of stage 1 CRPS?
acute onset after 1 month of trauma
1) ache, burn pain aggravated by touch or emotion
2) bony pain and allodynia is possible
last 3 months post injury
What is the presentation of stage 2 CRPS?
dystrophic 1)spontaneous radiating burning pain 2) hyperpahty 3) trophic change to hair, skin 3-6 months post trauma
What is the presentation of stage 3 CRPS?
Atrophic 1) diminishing pain 2)cooling skin 3) subcutaneous atrophy and wasting 6-12 months post trauma
What is the treatment for CRPS?
NSAID, corticosteriods
Sympathetic blockage, placebo block
Gabapentin, clondine
TCA’s
Ca channel blockers, beta blockers, calcitonin
Surgical ablation and dorsal column stimulation