HYHO II - Acute Kidney Injury Flashcards

1
Q

You have a patient that you suspect has Acute Kidney Injury (AKI) what sort of data would help you to conclude that your diagnosis is correct?

A

An Increase in Serum Creatinine and BUN

Little Urine output (0.5 mL/kg/hour for more than 6 hours)

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2
Q

You suspect AKI, and want to rule out any obstructions – what would you look for that effectively rules out obstruction?

A

Hydronephrosis

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3
Q

You have a patient that has been on Lisinopril and Losartan for the past 3 years and has an increase in serum creatinine in the past 72 hours, and has had little to no urine output for more than 8 hours. Upon further evaluation you find that they have BP of 80/50 and you are starting to see some liver failure as well. What sort of problem are you thinking?

What sort of other things are associated with this dz?

A

Pre-Renal Acute Kidney Injury

Hypovolemia, CHF, NSAIDS, ACEi, ARBs, Cyclosporine

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4
Q

You have a patient that is presenting with an elevated serum creatininne within the past 48 hours, and has little urine output for the past 7 hours. Your patient is noted to have frequent kidney stones, – this makes you worried about what?

A

Post-Renal AKI

(Bladder outlet obstruction, Obstructions!)

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5
Q

Your attending wants to know some of the major forms of Intrinsic forms of AKI. What are some of the major forms?

A

Acute Glomerulonephritis

Damage/Ischemia to the Tubules and Intersitium

Vasculitis (ANCA dz’s)

TTP-HUS

Malignant HTN

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6
Q

Associate the following with their potential associated disease process?

Blue Toes

Appearance of a Drug Rash

Volume Contraction

Volume Overload

Jaundice/Ascites

A

Blue Toes –> Cholesterol Emboli

Drug Rash –> Acute Interstitial Nephritis

Vol Cont –> Dehydration

Vol Overload –> Cardiorenal Synd

Jaundice/Ascites –> Liver Failure/Portal HTN

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7
Q

What are some of the other associated symptoms/signs you need to be worried about in your patient after you have diagnosed them with AKI?

A

Decreased Urine Output

Worsening Dyspnea/Orthopnea/PND

Ascites/Liver Distention

S3 –> HF

Hypotension

JVD

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8
Q

You are a physician who is trying to detect large amount of free intraabdominal fluid. How would you position the patient to perform the Fluid Wave Exam?

A

1) Patient puts ulnar surface of their hand over belly button area
2) You than put one hand on flank and tap on the opposite flank

A + sign would be if you feel a moderate to strong fluid wave emanating into the opposite side

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9
Q

For some reason your attending wants you to perform the puddle sign on this patient. . . you denote that you get it. . . he vapes. . . but then proceed to perform it:

A

1) Patient gets on hands and knees for 5 mins
2) you listen with diaphragm while flicking the flank area (starting low and working up)

A + sign is if you hear a sudden intensity and clarity of a sound. (this is when it has passed the edge of the peritoneal fluid)

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10
Q

You have a patient that was diagnosed with CKD, and they ask you to help them with some of their symptoms with your magic osteopathic ways. You gladly agree.

What specific areas would you need to do an OSE on?

What specific chapman point locations do you need to check?

A

Kidney – evaluate T10-T11 / Vagus N

Ureters – evaluate T10-L2 / Vagus N & Pelvic Splan

Bladder – evaluate T12-L2 / Pelvic Splan

Kidney – (Ant) 1 inch up and lateral to belly button

(Post) Between TP of T12/L1 on ipsi side

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11
Q

You have a patient that was diagnosed with CKD, and they ask you to help them with some of their symptoms with your magic osteopathic ways. You gladly agree.

What specific things would you want to evaluate if you wanted to take care of the biomechanical approach to the patient?

A

SD of the OA, AA, T10-11 (kidneys), Psoas Muscles

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12
Q

You have a patient that was diagnosed with CKD, and they ask you to help them with some of their symptoms with your magic osteopathic ways. You gladly agree.

What specific things would you want to evaluate on the respiratory/circulatory method?

A

Give them O2 via Mask/Nasal Canula

Check LYMPHATICS –> Thoracic Inlet MFR, Diaphragm techniques, Rib Raising, Efflaurage/Petra, Pectoral traction, Thoracic Pump, Abdominal Pump, sacral rocking etc.

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13
Q

You have a patient that was diagnosed with CKD, and they ask you to help them with some of their symptoms with your magic osteopathic ways. You gladly agree.

Next you decide that they deserve your best approach to the neurological component – what are the best techniques?

A

You are going to work on the Vagus N/Pevlic Splanchnics for Parasymp

and their viscero-somatic levels for Kidney/Ureter/Bladder

Also look at Chapman Points!

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14
Q

You have a patient that was diagnosed with CKD, and they ask you to help them with some of their symptoms with your magic osteopathic ways. You gladly agree.

You want to help him out with the Metabolic/Energetic/Immune component of his healing process - -what is the best way?

A

Give them Loop Diuretics, Fluid Restriction,

Stop any offending meds (NSAIDS/PPI’s),

adjust their meds now that they have CKD

Monitor Food Intake/Weight

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15
Q

You have a patient that was diagnosed with CKD, and they ask you to help them with some of their symptoms with your magic osteopathic ways. You gladly agree.

What are some behavioral things you can help this patient with?

A

Excersize, Diet, Avoid Offending agents, help them with inciting cause of this dz

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16
Q

you have a patient that is having HF – you want to understand how someone with HF can possibly lead to AKI. Discuss the process you might see.

Neurohormonal Adapatation?

A

HF = Hemodynamic Derangements

Causing RAAS activation (from low blood output)

This increases ADH & Endothelin 1 Release –> Salt/Water retention and vasoconstriction

Leads to bad reabsorption of Urea

Leads to too much systemic vasoconstriction

this increases afterload –> further reducing cardiac output

further decreasing renal perfusion!

17
Q

you have a patient that is having HF – you want to understand how someone with HF can possibly lead to AKI. Discuss the process you might see.

Increased Renal Venous Pressure?

Associations with HFpEF?

A

If you increase Central Venous Pressure –> it decreases GFR (causing AKI)

Metabolic Derangements cause systemic inflammation –> causing myocyte stiffening, hypertrophy, and intersitital fibrosis

18
Q

Diane has AKI (dont be like Diane) – what are some treatment modalities that we can offer her?

A

Remove the offending agent

Loop Diuretics

Adjust Meds by renal fxn

Oxygen therapy

Monitor Weight/Electrolytes

Fluid Restriction

Dietary Consult

19
Q

Diane is noted to have an increase in serum creatinine of 125%, with a urine output that has been less than 0.5 mL/kg/hour for 15 hours.

What KDIGO stage is she at?

A

Stage 2