Human Sexuality And Reproductive Cycle Flashcards
Eccrine glands:
Apocrine glands:
Sebaceous glands:
Eccrine glands and apocrine glands increase secretions and become fully functional during puberty.
Eccrine glands are found over most of the body –
Apocrine glands in the axillae, anal and genital areas. Apocrine sweat is released in response to emotional stimuli.
Sebaceous glands become active under the influence of androgens in both sexes. Secreting sebum most active on face, neck, shoulders, upper back, chest and genitals.
What is gender typing?
children acquire the values, motives, and behaviors viewed as appropriate for males and females within a culture.
Describe the phases of male sexual response
Excitement phase- rapid erection, thickening of scrotal skin, elevation of the scrotal sac response of vasocongestion. Increased HR, increased BP, flushed skin, increased genitalia blood flow
Plateau phase- increased length and diameter of penis, testes size increases by 50%. Cowper’s gland produces lubrication at the urethral meatus. Orgasm is the climax of the plateau phase.
Ejaculation stage I- seminal fluid expulsed from the seminal vesicles into the prostatic urethra.
Ejaculation stage II- seminal fluid expulsed from the prostatic urethra to the urethral meatus.
Final phase- (resolution)- immediately after orgasm, rapid loss of vasocongestion and decrease in penis size.
Resolution – refractory periods- unable to be re-stimulated to another orgasm, even if the penis remains erect.
Describe phases of sexual response in women
Excitement phase- vasocongestion causes clitoral enlargement and vaginal lubrication. The vaginal orifice opens; Increases HR, BP, and increased blood flow to genitals.
Plateau phase- retraction of clitoris under clitoral hood, vagina expands in width and depth, rising of cervix and uterus.
Orgasmic phase- Increases HR, RR, BP.
Resolution phase- clitoris returns to normal size and position, vasocongestion dissipates.
No obligatory refractory period.
Describe the two types of Impotence:
Primary:never achieved an erection necessary for intercourse.
Secondary: has experienced erections in past but subsequent difficulty. Treatment with sildenafil citrate (Viagra)
Problems with ejaculation:
subjective response of both partners dependent on levels of satisfaction.
o (Primary) physiological disturbances.
o (secondary) interpersonal problems, lumbar sympathectomy, antiadrenergic drugs (guanethidine, methyldopa)
Dyspareunia:
Vaginismus:
Dyspareunia- (painful intercourse) lack of lubrication, inadequate sexual arousal, drugs, decreased estrogen and infections.
Vaginismus- involuntary contractions, painful penetration.
Age-related sexual problems-
climacteric- period between middle to old age which has significant sexual changes.
Thinning of vaginal tissues, slower arousal,
fewer spontaneous erections, chronic diseases, medication usage,
decreased mobility and body image changes.
Symptoms of menopause
ovarian secretion of the hormones estrogen and progesterone is halted, resulting in changes in tissues dependent on these hormones.
Estrogen-responsive tissues are found in the female genital organs, breast tissue, pituitary gland, hypothalamus.
Changes include decrease in thickness of vaginal wall, decrease in vaginal secretions, vaginal and urethral atrophy, loss of bone density, and vasomotor clinical manifestation such as hot flashes.
nonhormonal drugs to relieve the clinical manifestations of menopause such as osteoporosis (Fosamax), vaginal dryness (water-soluble lubricants), and hot flashes (vitamin E).
Some women choose to use herbal therapy with black cohosh, ginseng, and dong quai for relief of clinical manifestations.
Pros and cons of HRT
Estrogen reduces the risk of atherosclerosis, angina, coronary artery disease, and osteoporosis.
Estrogen increases risk for breast cancer and dysfunctional uterine bleeding.
When used alone without progesterone, there is an increased incidence of uterine cancer.
When used in combination with progesterone, may have menstrual periods.
Uses for estrogen therapy
Estrogen may be given for treatment of ovarian failure, female hypogonadism, postmenopausal osteoporosis, inoperable breast and prostate cancers, atrophic vaginitis, and uterine conditions resulting from hormonal imbalance.
Side effects of estrogen
Edema Spotting between menstrual periods Thromboembolism Hypertension Increase in weight Breast tenderness Dark skin patches that darken when exposed to the sun
Caffeine and smoking increase risks
Contraindications and precautions of estrogen
Contraindicated in clients with history of breast cancer, except in cases when cancer is not dependent on estrogen
History of thromboembolitic disorders, hepatic or renal disorders, and undiagnosed vaginal bleeding (may increase risk of endometrial cancer)
Pregnancy
Hypersensitivity
Drug interactions of estrogen
Anticoagulants: decrease anticoagulant
Antidiabetics: impair glucose tolerance that may change antidiabetic drug requirements
Anticonvulsants: estrogen-induced fluid retention may precipitate seizures
Barbiturates: decrease effect of estrogen
Corticosteroids: increase risk of toxicological effect of corticosteroids
Rifampin: decreases effect of estrogen
Why should tetracycline be avoided during pregnancy
Tetracycline hydrochloride should not be given to pregnant women in the last half of their pregnancy (or not at all) because it may permanently discolor the teeth, cause enamel defects, or retard bone growth of the fetus.
Adnexa:
ovaries and fallopian tubes together are referred to as the Adnexa
Vas deferens —
Ejaculatory ducts —
Seminal vesicles —
Vas deferens —a long, muscular tube that travels from the epididymis into the pelvic cavity, to just behind the bladder. The vas deferens transports mature sperm to the urethra.
Ejaculatory ducts —formed by the fusion of the vas deferens and the seminal vesicles; empty into the urethra.
Seminal vesicles —sac-like pouches that attach to the vas deferens near the base of the bladder. The seminal vesicles produce a sugar-rich fluid (fructose) that provides sperm with a source of energy and helps with the sperms’ motility. The fluid makes up most of the volume of a man’s ejaculate.
Follicular phase
1 Follicular phase - This phase starts on the first day of your period. During the follicular phase of the menstrual cycle, the following events occur:
- Two hormones, follicle stimulating hormone (FSH) and Luteinizing hormone (LH) are released to travel to the ovaries. The hormones stimulate growth of about 15-20 eggs in the ovaries each in its own “shell,” called a follicle. These hormones also trigger an increase in the production of the female hormone estrogen. As estrogen levels rise, like a switch, it turns off the production of FSH.
- As the follicular phase progresses, one follicle in one ovary becomes dominant and continues to mature. This dominant follicle suppresses all of the other follicles in the group. As a result, they stop growing and die. The dominant follicle continues to produce estrogen.
Ovulatory phase -
2 The ovulatory phase, or ovulation, starts about 14 days after the follicular phase started. The ovulatory phase is midpoint of menstrual cycle, with next menstrual period starting about 2 weeks later. During this phase, the following events occur:
• The rise in estrogen from the dominant follicle triggers a surge in amount of Luteinizing hormone (LH) that is produced by the brain.
This causes the dominant follicle to release its egg from the ovary.
- As the egg is released (a process called ovulation) it is captured by finger-like projections on the end of the fallopian tubes (fimbriae). The fimbriae sweep the egg into the tube.
- Also during this phase, there is an increase in the amount and thickness of mucus produced by the cervix (lower part of the uterus.) If a woman were to have intercourse during this time, the thick mucus captures the man’s sperm, nourishes it, and helps it move towards the egg. Fertilization usually takes place in the middle portion of the fallopian tube (ampulla).
- Secretory- (progestational, luteal, premenstrual) dominance of progesterone from the corpus luteum causes the endometrium to become dilated with glycogen and mucin.
Luteal phase of menstration
The luteal phase begins right after ovulation and involves the following processes:
- Once it releases its egg, the empty follicle develops into a new structure called the corpus luteum.
- The corpus luteum secretes the hormones estrogen and progesterone. Progesterone prepares the uterus for a fertilized egg to implant.
- If intercourse has taken place and a sperm has fertilized the egg, the fertilized embryo will travel through the fallopian tube to implant in the uterus. If egg not fertilized, it passes through uterus. Not needed to support a pregnancy, the lining of the uterus breaks down and sheds, and next menstrual period begins.