Horses Flashcards

1
Q

When were horses domesticated

A

Domestication of horses in Eurasia (8000-5000BC): may have saved species from extinction due to heavy hunting (prior to 8000BC)
- Horse become particular useful: 2000 BC (pull chariots) and by 1000BC complete domestication: Europe Asia and North Africa

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2
Q

True or False

Horses have never been food animals

A

False

Horses were initially used for food (30 000 BC)

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3
Q

Male intact adult horse

A

Stalion

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4
Q

Male sterilized (no testes) Adult Horse

A

Gelding

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5
Q

Baby Male Horse

A

Colt

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6
Q

Adult Female Horse

A

Mare

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7
Q

Baby Female Horse

A

Fille

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8
Q

Baby horse (gender neutral)

A

Foal

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9
Q

What is 1 Hand in inches

Horse industry

A

1 Hand = 4 Inches

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10
Q

Withers

Horse

A

What you measure for height in horses
- Located near front shoulders

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11
Q

What are the three broad catagories of horse? | Size

A
  • Ponies: less than 14.2 hands at withers and less than 8oolbs
  • Light Horse: 15-17.2 hands and 900-1200 lbs, majority of breeds
  • Heavy horse: 16-18 hands, heavy muscle, large bones and sturdy legs, greater than 1400 lbs
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12
Q

Hot Bloods

Horses

A

Hot Bloods: Lighter bodies, idea for speeds (racing):
- Temperament is high strung and flighty: Arabian, Thoroughbred, Quarter Horse

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13
Q

Warm Blood

Horses

A

Warm Blood: Named for the area the horse was located. No true breed of warm blood (as hot bloods - Arabians):
- Mild temperament and calm spirt:
- Dressage ,Jumping, 3 day eventing: Hanoverian, Irish Sport Horse (Irish Hunter), Trakehner

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14
Q

Cold Bloods

Horses

A

Cold Blood: Mild manner, docile good with children:
- Heavy work (Agriculture; pulling equipment) Draft
- Breeds; Percheron, Clydesdales, Shire
- intelligent

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15
Q

Arabian Horses

A
  • Oldest breed of horse in world (ancestry 5000 years old)
  • Classified by ancestry nation of origin: Polish, Spanish, Russian, Egyptian, Domestic
  • Agile and excellent stamina
  • Dish face prominent eyes and arched neck
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16
Q

Thoroughbred

Horse

A

Breed developed from 3 Foundation Arabian Stallions
- Fastest Horse: Intermediate distance: (three-quarter mile -1.5 mile races)
- Cross breeding: influenced the development of breeds (Quarter horse, Standardbred and some Warm bloods- Irish Sport Horse)

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17
Q

Quarter Horse

A

Created: Initially English Horse Breeds crossed with Arabians (Arabs and Barb descent)
- Later: These horses crossed with Thoroughbreds: stronger and faster
- In NA: these new horse breeds crossed with Mustangs- quick horses and used for cattle work
- Strong hind quarters
- Fastest Horse: Short distance (quarter mile races)

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18
Q

Standardbred Horses

A
  • Foundation: Sire from Thoroughbred (name): Messenger
  • Harness racing: Pacers and Trotter: what is the difference (Video)
  • Dressage and Hunter jumpers
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19
Q

Mule vs Hinny

A

Male Donkey x Female Horse = Mule

Female Donkey x Male Horse = Hinny

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20
Q

What do horses stand on

A

Their middle fingers
- Coffin bone
- DP or P3

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21
Q

White Coat Colors

Horses

A

True white horses valued throughout history
- Lethal White Foal Syndrome: Dominant white gene (homozygous): often lethal in utero and intestinal neurological abnormalities- colic first 12 hours of life
- Heterozygous: pink skin- colored eye

22
Q

Horse Teeth

A

Teeth: important of digest cellulose, helps in hind-gut fermentation
- In grazing animals: good dentition is key to survival
- Uneven wear of teeth mouth injuries
- Wolf teeth: extra set of small premolars in front of large grinding premolars- 70% have Wolf teeth
- vestigial tooth - forest dwellers eating for brush plant products to now plains dwellers
- not involved in fighting- unlike canine or swine

Teeth will grow throughout life
- Must be grinded down
- “Float teeth”

23
Q

Horse Digestion

A

Digestive system:
- Prehensile lips (Video)
- Hind gut fermenters (cecum and colon) for fiber digestion (similar bacteria populations as rumen in ruminants). Fermentation
releases volatile fatty acids- a major feed source for forage-based diets

24
Q

Horse Reproduction

A
  • Gestation: 340 days
  • Labours short: 10-15 minutes
  • 50-80% breeding efficiency
  • Seasonal polyestrous: short breeding season spring and summer
  • Estrus: 5-7 days and to improve breeding efficiency, breeding starts Day 3 of estrus and continues every other day until mare refuses breeding
  • Colostrum (first milk): critical first hours following delivery, if not foals often succumb to infection
25
Q

Horse Vision

A

Vision: wide field of vision and eyes see independently (monocular vision): cannot see directly in-front (forehead) or behind (tail)

26
Q

Horse Hearing

A

14 Hz - 25 Hz (20Hz-20KHz): responds well to low tone commands: Don’t like high or shrill commands
- Ears rotate 180º: 10 muscles (people 3 ear muscles)

27
Q

Horse Taste

A

Like salt and sweet. Dislike bitter and sour:
- protective against poisonous plants and water

28
Q

Horse Smell

A

better sense of smell than people: use smell for pheromone detection (stallion) and predator detection.
- Foals and mares bond through sense of smell

29
Q

Flehmen reaction

Horses

A

Horse rolls upper lip (expose sensitive membrane of lips and nose) to detect unusual smell.
- Allows ‘scent’ to interact with vomeronasal organ (important in pheromone detection)

30
Q

Colic

A

abdominal pain: many causes
- Horse is listless or varying degrees of distress
- Signs: curling lips, pawing, kicking at belly, lying down, rolling
- Pulse rate elevated, sweating, pink to blue gums
- Emergency in horses
- Stats: 4.2 colic/ 100 horses: 11% are fatal

31
Q

What are the 2 general catagories of cholic

A
  1. Colic by cuase
  2. Colic by location
32
Q

Cholic by cause

A
  • Digestive Colic: too much feed or change in feed
  • Spasmodic or gas colic: eating too fast or toxin in feed
  • Sand colic: sand in feed
  • Impaction colic: not enough water
  • Others: ‘Intestinal accident’
33
Q

Colic: Stomach

A
  • Gastric colic’ (gastric dilatation and rupture): stomach small (8-10 liters).
  • Stomach will rupture as fluids content increase -as horses can not vomit
  • Intestinal obstruction (fluid build up) or too much gas . Very common
  • Horse stomach rupture (Stomach Impaction): impacted with feed. Less common
34
Q

Colic: Intestine

A

Most common causes of colic:
- obstruction
- strangulation
- intussusception
- inflammation
- volvulus (180º rotation around mesenteric axis)
- ascarid impaction
- cranial mesenteric arteritis (verminous arteritis)
- Enteroliths and cecal impaction (many more)

35
Q

Strangulation: Pedeunulated lipoma

A

A type of cholic that occurs in old horses
- Fat gows on fiberous tissue
- Starts swinging →Wraps around and strangles intestines
- Must be surgicaly removed

36
Q

Colic: Prevention and Treatment

A
  • Mild exercise (walk or jog): gas colic
  • Pain meds (NSAIDS)
  • Stomach tube
  • Fluid rehydration
  • Better feed and access to water
  • Surgery
  • Deworm
37
Q

Pathology and Incubation

EIA

A
  • EIA: Acute – Chronic incidence of fever anemia (low RBC), edema and cachexia (wasting), jaundice,
  • Many horses are asymptomatic or only have mild clinical symptoms and thus are carriers
  • Virus: lives in leukocytes for life and in plasma during periods of viremia
  • Infects: different cell lineages: RBC ,
    macrophage, monocytes, endothelial cells: leads to destruction
  • incubation period: 7-45 days
37
Q

Equine Infectious Anemia (Swamp Fever) (EIA)

A

Reportible Disease
- Retrovirus (RNA virus)
- Lentivirus
- Associated with high morbidity and mortality
- Found worldwide
Is considered a sporadic disease and
incidence is low (CFIA, 2014)
- Hot Spot: Northeast Alberta: Cold Lake region

38
Q

EIA: Transmission

A
  • Mechanical transmission: Biting flies (Horse, Deer flies; painful), main method of transmission.
  • Local infection more likely than distant animals
  • Other transmission: contaminated equipment, milk, semen, in utero
39
Q

Diagnosis and Treatment

EIA

A

Use Clinical signs (above slides)

Tests:
- Antibody tests Coggins test (AGID) and ELISA: Horses are seronegative for 2-3 weeks and thus the Coggins test usually doesn’t work in early infection
- Molecular tests: PCR: Good for testing foals to infected mares
- Vaccine: none
- Vaccine: 1975-1990 China developed vaccine, not recommended strategy (OIE, Govt. of Canada)
- Terminate animal or permanently isolate for other equids (is this good to isolate)

40
Q

Laryngeal Hemiplegia

A

Roaring/Wistling Horses

  • Exercise intolerance in horses
  • Left side is mainly affected.
  • Right side and bilateral nerves affected is uncommon
  • Left recurrent laryngeal nerve: Due to extended length of left recurrent nerve travelling around aortic arch (base of the heart)
  • Progressive loss in distal myelinated fibers that innervate that laryngeal muscles (left side)
  • Left arytenoid cartilage and left vocal fold (chord) becomes weak and can progress to paralysis.
  • Arytenoid cartilage collapsed medial (into the tracheal lumen) and the vocal fold will not open.
  • Reduced glottal cross-section area (airway area is smaller)

The greater the exercise greater the air resistance and the louder the ‘roaring’
- More exercise will further collapse the
trachea and more difficulty breathing
- Greater exercise intolerance and poorer horse performance
- Most horses with laryngeal hemiplegia sound normal (no roaring) at rest

41
Q

Treatment and Complications

Roaring Horses

A

Treatments
- Prosthetic laryngoplasty: best for performance horses- horses usually never reach potential
- Laryngeal ventriculectomy: reduces roaring

Complications:
- Chronic cough
- Aspiration of feed (aspiration pneumonia)
- Implant failure/infection

42
Q

How many horses will get laminitis?

A

1:7 horses will have an incidence of laminitis in its life-time

43
Q

What does Founder mean?

Laminitis

A

Founder: means to give away or collapse

44
Q

Laminitis

A

Inflammation of structures attaching the hoof to the 3rd phalanx bone (coffin bone)
- Failure or loss of attachment of the laminar basal epithelial cells of the epidermal laminae with dermal laminae
- The 3rd phalanx rotates toward the sole of the foot and can penetrate through the sole: ‘Sinking’

45
Q

Mechanisms of Laminitis

A

1) Matrix molecules of the basement membrane and dermis breakdown by metalloproteinases
2) dysregulation of hemidesmosomes-adhesion molecules that used to attach epithelial cells to matrix molecules

46
Q

Causes of Laminitis

List all 5 Common types (5 bonus types)

A
  1. Endocrine Disease: Cushing’s disease (Hyperadrenocorticism) or Equine Metabolic Syndrome: Possibility due too much insulin produced, this may injure laminae
  2. Excessive grain intake (common)
  3. Grazing on pasture (grain overload): of high sugar content such as spring/fall growth (very common)
  4. Excessive or concussive exercise in adult horses (common)
  5. Obesity - risk increases greatly as body score increases (common)
  6. Stress from disease or trauma: excessive bearing of weight on 1 leg - post injury to other limb(s)(common)
  7. Use of corticosteroid drugs
  8. walnut shavings in a load of bedding: 1% walnut in shaving in bedding will lead to laminitis
  9. Small hooves or poor hoof quality
  10. Infections: diarrhea (bacterial[salmonella], rickettsia [Potomac Horse fever], retained placenta, septicemia etc (sporadic)
47
Q

Diagnosis of Laminitis

A
  • Clinical exam and history
  • Radiographs
  • Nerve blocks
48
Q

What are the types of laminitis

A
  • Acute: <3 days; hot hoofs, painful to walk, depressed, decrease appetite , prominent digital pulse: only severe incidence leads to 3rd phalanx rotation and sinking
  • Subacute:> 3 days, similar to symptoms and manifestation as acute laminitis
  • Chronic: intermittent periods of acute and subacute laminitis, will eventually lead to rotation and sinking of 3rd phalanx
49
Q

Acute Laminitis

A

<3 days;

hot hoofs, painful to walk, depressed, decrease appetite , prominent digital pulse:

only severe incidence leads to 3rd phalanx rotation and sinking

50
Q

Subacute Laminitis

A

> 3 days

similar to symptoms and manifestation as acute laminitis
- depressed
- low appitite
- prominent digital puls

51
Q

Chronic Laminitis

A

Intermittent periods of acute and subacute laminitis, will eventually lead to rotation and sinking of 3rd phalanx