Hip Flashcards

1
Q

Remind yourself of the bony anatomy of the hip

A
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2
Q

Remind yourself of the ligaments surround the hip

A

Intracapsular

Ligament of head of femur (from the acetabular fossa to the fovea of the femur)

Extracapsular

There are three main extracapsular ligaments, continuous with the outer surface of the hip joint capsule:

  • Iliofemoral ligament – anterior inferior iliac spine and then bifurcates before inserting into the intertrochanteric line of the femur. Y shaped. Prevents hyperextension of hip joint. Strongest.
  • Pubofemoral – superior pubic rami to the intertrochanteric line of the femur. Triangular shape. Prevents excessive abduction and extension.
  • Ischiofemoral– body of the ischium to the greater trochanter of the femur. Spiral orientation. Prevents hyperextension and holds the femoral head in the acetabulum.
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3
Q

Where does the capsule of the hip joint run from and to?

A
  • Capsule attached to acetabulum proximally
  • Distally, at the anterior it attaches to the intertrochanteric line and posteriorly it attaches ~1cm superomedial to the intertrochanteric crest
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4
Q

Remind yourself of the arterial supply to the hip

A

***Arterial supply is retrograde

  • Abdominal aorta bifurcates into common iliac
  • Common iliac bifurcates into internal & external iliac
    • Internal goes on to supply abdominal organs
    • External becomes the femoral artery (at the inguinal ligament)
  • Femoral artery divides into
    • Profunda femoris
    • Superficial femoral
  • Profunda femoris gives off:
    • Medial femoral circumflex
    • Lateral femoral circumflex
    • Perforating arteries (supply anterolateral muscles of thigh)
  • Medial & lateral circumflex form anastomoses at base of femoral neck to form a ring
  • Ring gives off branches; these branches contribute to the cruciate and trochanteric anatomoses of hip
  • In children, ligamentum arteriosum (branch of obturator) also acts as blood supply to femoral head
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5
Q

Where are the cruciate and trochanteric anastomoses of hip?

What arteries contribute to each?

A
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6
Q

Which blood vessel predominatly supplies hip?

What is the relevance of this in intracapsular #NOF?

A

Medial circumflex artery

Lies directly on intracapsular femoral neck therefore displaced intracapsular fractures disrupt blood supply to emoral head and femoral head will undergo AVN so pts need joint replacement rather than fixation

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7
Q

A #NOF is a fracture occuring between?

A

Subcaptial region of femoral head to 5cm distal to lesser trochanter

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8
Q

State some risk factors for #NOF

A
  • Age
  • Osteoporosis risk factors
  • Falls risk factors
  • Low BMI
  • Cancer (pathological fractures)
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9
Q

Discuss the different types of #NOF; include any subtypes

A

NOF can be intracapsular or extracapsulare:

  • Intracapsular: between subcapital region of femoral head to the basocervical region
    • Subcapital
    • Transcervical
    • Basocervical
  • Extracapsular: from intertrochanteric line to 5cm below lesser trochanter
    • Intertrochanteric: between greater trochanter and lesser trochanter
    • Sub-trochanteric: from lesser trochanter to 5cm distal to lesser trochanter
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10
Q

What classification can be used to further classify intracapsular fractures?

Describe this classification

A
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11
Q

Describe the typical presentation of #NOF

A
  • Pain (commonlly in groin & thigh- may be referred pain to knee)
  • Inability to weight bear
  • Leg is shortened, externally rotated
  • Pain on pin-rollling leg
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12
Q

Explain why the leg appears shortened, abducted and externally rotated in #NOF

A

Axis of rotation that usually passes obliquely through femoral head and down neck of femur now passes through greater trochanter and vertically down long axis of femur; this alters the actions the muscles have on the bone.

  • Externally rotated: short lateral rotators of hip e.g. pirformis, obturator internus, sup & inf gemelli, quadratus femoris all cause external rotration of femoral shaft. Iliopsoas also now pulls lesser trochanter anteriorly contributing to external rotation
  • Abducted: abductors e.g. gluteus medius & minimus abduct femur
  • Shortened: muscles of thigh e.g. rectus femoris, adductor magnus and hamstrings pull distal fragment of femur upwards
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13
Q

What investigations should you do in a suspected #NOF

A
  • Plain radiographs
    • Affected hip (AP, lateral)
    • Pelvis (AP)
    • ?Full length femoral if suspect pathological fracture
  • ?MRI if cannot see fracture on plain radiograph
  • Bloods
    • FBCs
    • U&Es
    • Coagulation
    • Group & save
    • ?CK (if long lie)
  • Other investigations to complete your assessment (e.g. dipstick, CXR etc… often useful if elderly pt who has fallen & need to determine cause)
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14
Q

Discuss the initial management of a pt with #NOF

A
  • A-E or ATLS (dependent of mechanism of injury/state of pt)
  • Analgesia (opiod or regional anaesthesia such as iliofascial or femoral nerve blocks)
  • Assess neurovascular status
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15
Q

Discuss the management of intracapsular #NOF

A

Management depends on factors such as whether fracture is displaced or non-displaced and pt factors:

Young,fit patient

  • Undisplaced: internal fixation with cannulated hip screws
  • Displaced: increased risk of AVN therefore total hip replacement

*NOTE: fixation may fail and may require total hip replacement later

Elderly patient

  • Hip hemiarthroplasty or total hip replacement regardless of if displaced or undisplaced: do total hip in pts who are medically fit for anaesthesia, abel to walk around independently or with stick and cognitively sound.
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16
Q

Discuss the surgical management of extracapsular #NOF

A
  • ORIF with dynamic hip screw or trochanteric femoral intramedullary nailing
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17
Q

State some immediate post-operative complications

A
  • Pain
  • Bleeding
  • Leg length discrepancies
  • Neurovascular damage
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18
Q

Discuss the long term management of #NOF

A
  • Physiotherapy
  • Occupational therapy input
  • Under care of orthogeritricians
19
Q

State some potential long term complications of #NOF

A
  • Joint dislocation
  • Aseptic loosening
  • Peri-prosthetic fracture
  • Deep infection//prosthetic joint infection
20
Q

What is the mortality of a #NOF at 1 year?

A

30%

21
Q

What internal fixation method has been used to internal fix this hip?

A

Dynamic hip screw

22
Q

What internal fixation method has been used to internally fix this hip?

A

Cannulated nail screws

23
Q

What internal fixation method has been used to internally fix this hip?

A

IM nail

24
Q

The pelvis containts the rectum, bladder and uterus in females aswell as iliac vessels & lumbosacral nerves; why is this relevant in pelvic fractures?

A

Pelvic fractures can be associated with:

  • life threatening haemorrhage
  • neurological deficit
  • urogential trauma
  • bowel injury

Therefore full neurovascular assessment of lower limb required including checking anal tone, sacral nerve roots & iliac vessels

25
Q

Describe the typical presentation of pelvic ring fractures

A
  • Obvious deformity to the pelvis
  • Pain
  • Swelling
  • Bruising
26
Q

What is the typical mechanism of injury for pelvic fractures?

A

Blunt truama e.g. RTA, fall from height

27
Q

Hypotensive pt with history of pelvic trauma has a _________ until proven otherwise

A

Pelvic ring fractures can cause significant blood loss resulting in hypovolaemic shock

28
Q

What investigations would you do if you suspect a pelvic fracture?

A
  • Plain radiographs of pelvis (AP, inlet view & outlet view)
  • OR CT scan (often pts are involved in high energy injuries so CT may be required anyway so don’t require x-rays aswell)
29
Q

What two classification systems are used to classify pelvic fractures?

A
  • Young & Burgess
  • Tile
30
Q

Low energy injuries in young pts can result in avulsion fractures of the pelvis. State which sites are commonly affected and how these avulsion fractures usually present

A
31
Q

Discuss the management of pelvic fractures, include:

  • Initial managmeent
  • Definitive management
A

Initial Management

  • ATLS
  • Pelvic binder (to stablise pelvis and therefore aid clot formation)

Definitive Managment

  • Conservative or operative
    • Indications for operative management: life-threatening haemorrhage, unstable fractures, open fractures, associated urological injury
    • Immediate surgery required for those who are haemodynamically unstable after fluid resuscitation
32
Q

State some potential complications of a pelvic ring fracture

A
  • Urological injury (more common in men)
  • VTE
  • Long standing pelvic pain
33
Q

What is the usual mechanism of injury for an acetabular fracture?

A
34
Q

Describe how an acetabular fracture typically presents

A
  • Pain
  • Inability to weight bear
35
Q

What is a Morel-Lavallee lesion?

A

Internal degloving injury whereby skin and subcutaneous tissue are abruptly separated from underlying fascia due to trauma resulting in creation of a potential space. May spontaneously resolve or become encapsualted & persistent

36
Q

What investigations are required for a suspected acetabular fracture?

A
  • Plain film radiographs (AP, Judet, obturatro oblique and iliac oblique)
  • Or CT scan (often occurs in traumatic injuries therefore CT often done anyway. ALSO GOLD STANDARD )
37
Q

What classification system is used to classify acetabular fractures?

A

Jduet & Letournel

38
Q

Discuss the management of acetabular fractures

A

Initial

  • ATLS
  • Reduce any dislocations

Definitive

  • Undisplaced or minimally displaced: conservatively with protected weight bearing for 6-8 weeks
  • Displaced: surgical fracture fixation (in elderly this is often pre-cursor to hip replacement)
39
Q

State some potential complications of acetabular fractures

A
  • VTE
  • OA
40
Q
A
41
Q

If a pt has a hip fracture, how soon should they be offered surgery?

A

Day of admission or day after

42
Q

What is the Nottingham Hip Fracture score?

A

Predicts 30 day mortality after hip fracture

43
Q

What is the relevance of serum lactate in pts with #NOF?

A

High serum lactate is marker for mortality

Conclusions from paper: Patients with an elevated venous lactate following hip trauma should be identified as being at increased risk of death and may benefit from targeted medical therapy.