HF Flashcards

1
Q

Pathophysiology

A

Inability of the heart to achieve adequate cardiac output

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2
Q

Presentation

A

SOB
Fatigue

Right:
- peripheral oedema (pitting)
- raised JVP
- hepatomegaly

Left:
- pulmonary crackles
- PND
- orthopnoea
- displaced apex beat - cardiomegaly

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3
Q

Risk factors

A

IHD - commonest cause
Coronary artery disease
Previous MI
Valvular heart disease
Chronic lung disease - cor pulmonale
HTN
AF
DM
Family history

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4
Q

Investigations

A

Basic obs
Bloods- BNP, FBC, U+Es, LFTs, CRP, Trop I, lipids, HbA1c
ECG
CXR
Echocardiogram - EF

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5
Q

BNP

A

400+

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6
Q

BNP may be reduced by

A

BMI
diuretics
ACEi
Beta blockers
Aldosterone
African Caribbean

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7
Q

BNP may be elevated by

A

70+ yo
LVH, MI
Hypoxia
Pulmonary hypertension
PE
Sepsis
COPD
DM
Liver cirrhosis

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8
Q

Ejection fraction calculation

A

SV/EDV

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9
Q

Ejection fraction percentages

A

Normal - 50% +
HFrEF - less than 40%

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10
Q

Management

A

Loop diuretic - furosemide +
1. ACEi/ARB + Beta blocker - started 1 at a time
2.Spironolactone
3. Digoxin

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11
Q

NYHA classification

A

I - no limitation of physical activity
II - some limitation with physical activity relieved by rest
III - breathless with minor activity relieved by rest
IV - breathless at rest

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12
Q

Acute management of pulmonary oedema

A

Sit upright
Oxygen if required
Diamorphine - decrease tachypnoea
Furosemide
GTN
May need CPAP

  • treat precipitating cause e.g. MI
  • manage fluid overload - fluid restriction and furosemide
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13
Q

Loop diuretic monitoring

A

Monitor renal function
Weight

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14
Q

CXR findings (ABCDE)

A
  • alveolar oedema - bat wing sign
  • Kerley B lines
  • cardiomegaly
  • dilated upper lobe blood vessels
  • pleural effusion
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15
Q

End stage HF

A

High risk of dying within 6 - 12 months

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16
Q

What does BNP do

A
  • Promotes Na+ excretion which takes water with it to decrease blood pressure.
  • causes vasodilation of afferent arteriole
  • synthesised and stored in atrial myocytes
  • released in response to atrial stretch
17
Q

ECG signs

A

Ischaemia
Hypertrophy
AF

18
Q

Conservative Mx

A

Stop smoking
↓ salt intake
Optimise weight - dietician
Supervised group exercised based rehab programme

19
Q

Digoxin

A

Enhances vagal activity
Increases K+ efflux
Decreases Ca2+ influx

20
Q

Signs of pulmonary oedema

A

Distressed, sweaty, cyanosed
↑HR, ↑RR
↑JVP
S3
Bibasal creps
Pleural effusions
Wheeze

21
Q

Symptoms of pulmonary oedema

A

Dyspnoea
Orthopnoea
Pink frothy sputum
PND

22
Q

Third line drugs for HF

A

Ivabradine
- inhibits funny currents
- sinus rhythm > 75/min and LV fraction < 35%

Sacubitril valsartan
- LV fraction < 35%
- if symptomatic on ACEi

Digoxin
- no proven benefit on mortality
- symptomatic relief by inhibiting Na+/K+ ATPase
- used if also have AF

Hydralazine
- if Afro - Caribbean

Cardiac desynchronisation therapy
- if broad complex QRS - LBBB

23
Q

Vaccinations

A

Annual influenza
One off pneumococcal