Brainscape's Knowledge GenomeTM

Browse over 1 million classes created by top students, professors, publishers, and experts.


See full index

Prince Med > HEPATITIS, Sle > Flashcards

HEPATITIS, Sle Flashcards

1
Q

hepatotropic viruses

A

VIRAL HEPATITIS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
2
Q

common tissue tropism

A

VIRAL HEPATITIS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
3
Q

most common cause of acute hepatitis

A

HEPATITIS A VIRUS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
4
Q

does not cause chronic hepatitis and rarely produces fulminant
hepatitis unlike Hepatitis B and C

A

HEPATITIS A VIRUS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
5
Q

appearance of Ig M anti-HAV in the blood during the acute
illness is the first detectable response to HAV infection

A

HEPATITIS A VIRUS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
6
Q

first detectable antibody response to this infection is the appearance of IgM

A

HEPATITIS A VIRUS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
7
Q

the IgG antihepatitis A virus is detected as the patient recovers and persists for life

A

HEPATITIS A VIRUS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
8
Q

DNA virus

A

HEPATITIS B VIRUS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
9
Q

HEPATITIS B VIRUS

transmission

A

o transfusion
o contaminated needles
o breaks in the skin or mucous membrane
o sexual contact
o perinatal

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
10
Q

o acute hepatitis with recovery and clearance of the virus

o nonprogressive chronic hepatitis

A

HEPATITIS B VIRUS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
11
Q

the 2nd circulating Ag to appear

A

HBeAg

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
12
Q

its presence correlates with a period of intense
viral replication

A

HBeAg

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
13
Q

→ indicates progression to chronic infection
→ high infectivity
→ chronic carrier state

A

HBeAg

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
14
Q

massive liver cell necrosis, hepatic failure

→ high mortality

A

FULMINANT HEPATITIS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
15
Q

presence of necrosis and inflammation in the liver for more than 6 months

A

CHRONIC HEPATITIS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
16
Q

→ in 5-10% of cases, HBs antigenemia does not resolve
→ 90% male

A

CHRONIC HEPATITIS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
17
Q

some chronic HBV carriers manifest circulating HBsAg-anti HBs
complexes that cause extrahepatic illness

A

CHRONIC HEPATITIS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
18
Q

3% never develop anti-HBs and suffer from relentless and
progressive chronic hepatitis that leads to cirrhosis

A

CHRONIC HEPATITIS

How well did you know this?
1
Not at all
2
3
4
5
Perfectly
19
Q

common cause of chronic hepatitis and cirrhosis

A

HEPATITIS C VIRUS

20
Q

Flavivirus, RNA virus

A

HEPATITIS C VIRUS

21
Q

assembly of this virus in the liver requires the synthesis of HBsAg

o cannot exist without Hepatitis B virus

A

HEPATITIS D VIRUS (DELTA AGENT)

22
Q

infection with HDV may occur simultaneously with HBV infection (coinfection) or following HBV infection (superinfection)

A

HEPATITIS D VIRUS (DELTA AGENT)

23
Q

Ig M anti-HDV

A

SUPERINFECTION

(HEPATITIS D VIRUS (DELTA AGENT))

24
Q

fulminant hepatic failure

A

o liver necrosis
o liver failure

25
high aminotransferases, prolonged prothrombin time, hyperglobulinemia, high alkaline phosphatase
chronic hepatitis
26
spider angiomas, palmar erythema, mild hepatomegaly, mild tenderness, mild splenomegaly
chronic hepatitis
27
councilman
ACUTE VIRAL HEPATITIS
28
acidophilic bodies
ACUTE VIRAL HEPATITIS
29
acidophilic bodies
ACUTE VIRAL HEPATITIS
30
piecemeal necrosis
CHRONIC VIRAL HEPATITIS
31
T/f Sle is a rheumatic disease because it causes rhematic fever
F
32
Type III hypersensitivity reaction
SYSTEMIC LUPUS ERYTHEMATOSUS
33
anaphylactic/allergic reaction; rapid and may occur in minutes after encounter with antigen A. Type I B. Type II C. Type III D. Type IV
Type I
34
associated with IgE (associated with allergic reaction) A. Type I B. Type II C. Type III D. Type IV
B
35
release of histamine mast cells A. Type I B. Type II C. Type III D. Type IV
A
36
fatal in cases of anaphylactic shock A. Type I B. Type II C. Type III D. Type IV
A
37
associated with IgM and IgG A. Type I B. Type II C. Type III D. Type IV
B
38
antibody-related hypersensitivity reaction A. Type I B. Type II C. Type III D. Type IV
B
39
associated with antigen-antibody immune complex deposition in certain organs such as the kidney, spleen, heart, skin and joints A. Type I B. Type II C. Type III D. Type IV
C
40
delayed type A. Type I B. Type II C. Type III D. Type IV
D
41
cell-mediated reaction A. Type I B. Type II C. Type III D. Type IV
D
42
the presence of autoantibodies suggest that there is a breakdown in the immune surveillance mechanism leading to loss of tolerance
SYSTEMIC LUPUS ERYTHEMATOSUS
43
T/f SYSTEMIC LUPUS ERYTHEMATOSUS antinuclear antibodies (ANA) - most important diagnostic Abs
T
44
splenomegaly, lymphadenopathy
HEMATOLOGIC
45
leukopenia (less than ____/uL) → lymphopenia (less than ____/uL) → thrombocytopenia (less than ___/uL)
4,000 1,500 100,000
46
AMERICAN COLLEGE OF RHEUMATOLOGY CRITERIA FOR THE CLASSIFICATION OF SLE at least (4) of the ff. are required:
o malar rash o discoid rash o photosensitivity o oral ulcers o arthritis o serositis o renal disorder o leukopenia (less than 4000/uL) o lymphopenia (less than 1500/uL) o neurologic disorder o anti-DNA antibody, anti-Sm antibody or evidence of antiphospholipid antibody o antinuclear antibody in raised titer
47
drug-induced lupus:
o procainamide o hydralazine

Prince Med (5 decks)

Brainscape helps you reach your goals faster, through stronger study habits.
© 2025 Bold Learning Solutions. Terms and Conditions