Heart Sounds Flashcards
mechanism for the production of the S1 heart sound
mitral and tricuspid valves close
systole
“Lub”
mechanism for the production of the S2 heart sound
closing of atrial and pulmonic valves
diastole
“Dub”
reason for normal splitting of the S1 heart sound
the mitral valve and tricuspid valve close at different times
reason for normal splitting of the S2 heart sound
AKA physiologic splitting
occurs during inspiration and disappears during expiration
pathologic splitting of S2
WIDE split
persists thru out cycle (can hear during inspiration and expiration)
pathophysiology behind the production of systolic ejection clicks
occurs in systole
caused by mitral valve prolapse
occurs between S1 and S2
pathophysiology behind the production of opening snaps
occurs during Diastole
caused by rough, calcified valve that opens up in early diastole
pathophysiology behind the production of S3s
occurs in diastole
“kentucky” murmur
occurs after S2
regurgitation
pathophysiology behind the production of S4s
atrial contraction, extra kick before the “Lub”
occurs in diastole
“tennessee murmur”
possible shapes of a murmur
crescendo
decrescendo
crescendo-decrescendo
plateau
grading the murmur includes:
intensity (x/6)
pitch (high, med, low)
quality (blowing, harsh, rumbling, musical)
holosystolic murmurs
begin at S3 and continue until S4
mitral regurgitation
tricuspid regurgitation
ventricular septal defect
mitral regurgitation
holosystolic
does NOT become louder with inspiration
tricuspid regurgitation
holosystolic
blowing
LOUD with inspiration
ventricular septal defect
wide radiation
found only in babies!!