Heart Failure (M4D) Flashcards

1
Q

heart failure

A
  • inability of the ventricles to pump enough blood to meet the body’s metabolic demands
  • clinical syndrome
  • fluid overload, inadequate tissue perf
  • d/t disorder that affects heart’s ability to receive and eject blood
  • chronic, progressive syndrome
  • manage by lifestyle changes & meds
  • no cure, only Tx
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2
Q

systolic HF

A
  • results in dec blood vol being ejected from the ventricles
  • where HF begins
  • dec CO, dec systemic BP, dec perf to kidneys
  • forces body to go into RAAS (inc aldosterone, activate baroreceptors, leads to diastolic HF)
  • ventricular remodelling (inc hypertrophy to meet demands but it makes heart work harder to pump)
  • heart does not pump sufficient blood through body –> stimulates heart to work harder –> heart cannot respond –> failure progresses and gets worse and worse
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3
Q

L sided HF

A
  • L side of heart pumps O2-rich blood around the loop, blood coming from the lungs
  • if L side of heart cannot keep up w amount of blood returning from the lungs, fluid backs up into pulm circulation
  • blood returned from pulm vessels dec as pressure in L side of heart inc
  • inc pressure of blood working to return L side of heart forces fluid from pulm capillaries to push into pulm tissues and alveoli –> pulm edema and impaired gas exchange
  • leads to R sided HF
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4
Q

R sided HF

A
  • R side of heart accepts deoxygenated blood from the body
  • if blood trying to return to R side but not able to accomodate it, it will back up and venous return congested
  • results in congestion in peripheral tissues and viscera
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5
Q

HF risk factors

A
  • CAD
  • cardiomyopathy
  • valvular heart disease
  • HTN
  • renal dysfx
  • age
  • DM
  • metabolic syndrome
  • hyperlipidemia
  • A-fib
  • smoking
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6
Q

L side HF S&S

A
  • dyspnea
  • SOB
  • orthopnea (difficulty breathing while lying flat)
  • dry-nonprod cough that leads to frothy pink blood-tinged sputum
  • crackles in lungs
  • fatigue
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7
Q

R side HF S&S

A
  • inc jugular vein distention
  • edema in lower extremities
  • enlargement of liver & spleen
  • anorexia
  • nausea
  • weakness
  • weight gain (d/t retention of fluid)
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8
Q

interventions for HF

A
  • nutrition (DASH diet)
  • fluid vol interventions
  • activity intolerance
  • control SOB
  • control anxiety
  • impaired sleep
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9
Q

fluid volume interventions

A
  • diuretic therapy
  • daily weight
  • fluid restrictions (in/out)
  • respiratory assessment
  • positioning to reduce preload (semi-fowlers)
  • assess for skin breakdown
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10
Q

activity intolerance interventions

A
  • rest during times of exacerbation (dec workload of heart)
  • individualized period of daily exercise gradually inc in duration
  • do not exercise in extreme weather (can inc workload on heart)
  • pt should be able to talk while exercising
  • stop exercising if experiencing SOB, pain, dizziness
  • cool-down activities after exercising
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11
Q

SOB interventions

A
  • supplemental O2; monitor O2 sats
  • rest when SOBOE
  • raise HOB (never let pt lay flat w/ HF)
  • complete respiratory assessment (use of accessory muscles, RR, WOB)
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12
Q

controlling anxiety interventions

A
  • admin O2 if required
  • promote physical comfort and psychological support
  • relaxation techniques
  • screen for depression
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13
Q

impaired sleep interventions

A
  • provide required pillows for easy of breathing
  • provide chair for pt if they cannot get comfy in bed
  • do not lie flat for pts w HF
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14
Q

assessments / monitoring for HF

A
  • assess Na, K levels and fluid balance
  • weigh daily
  • respiratory assessments
  • assess for JVD
  • monitor and evaluate severity of edema
  • examine skin turgor & monitor for signs of dehydration
  • monitor pulse, BP, signs of postural hypotension
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15
Q

evaluation for HF pts

A
  • demonstrates tolerance for inc activity
  • maintains fluid balance
  • demonstrates less anxiety
  • makes decision regarding Tx and care
  • adheres to self-care regimen
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16
Q

diagnostics for HF

A
  • echocardiogram (EF)
  • chest X-ray
  • angiogram
  • BNP
  • K+ and Na+ (electrolytes)
  • CBC, renal fx, LFT
  • thyroid stimulating hormone
  • urinalysis
  • digoxin therapeutic levels
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17
Q

lab tests for HF

A
  • CBC
  • electrolytes
  • GFR, creatinine
  • digoxin level
  • BNP
18
Q

electrolyte imbalances

A
  • hypokalemia: HTN, ventricular dysrhythmias, muscle weakness
    hyperkalemia: SE of ACE inhibitors and ARBs, spirolactone, dysrhythmias
  • hyponatremia: S&S = disorientation, fatigue, malaise, muscle cramps
19
Q

brain natriuretic peptide (BNP)

A
  • secreted by ventricles of heart in response to excessive stretching of cardiac muscle cells
  • to determine if pt is in HF and severity of HF
  • also used as an indicator if Tx improving HF condition
20
Q

fluid balance

A
  • intravascular fluid (in vessels)
  • extravascular fluids (lymph or CSF)
  • interstitial spaces (spaces b/w cells, tissues & organs)
  • isotonic (soln equal conc of solns across membranes)
  • osmotic pressure (fluid shifting from low to high conc until soln equal conc)
  • colloid osmotic pressure (similar to osmosis but w proteins)
21
Q

drugs used to treat HF

A
  • diuretics
  • ACE inhibitors
  • ARBs
  • direct vasodilators
  • beta blockers
  • cardiac glycosides (digoxin)
22
Q

preload reducers

A
  • reduce myocardial workload by reducing amount of vol coming back to heart
  • ex. diuretics, vasodilators
23
Q

worst thing that can happen when taking preload reducers…

A
  • inadequate CO by pooling of blood in extremities
  • hypotension (orthostatic)/dehydration
  • electrolyte imbalance
24
Q

afterload reducers

A
  • reduce myocardial workload by dec resistance heart has to pump against
  • ex. ACE inhibitors, ARBs, diuretics
  • look for retained high K+ levels and low Na+ levels
  • given in combination w/ diuretics
  • assess HR, BP, blood values, and electrolytes
  • monitor urine output, dry mucous membranes, high HR, low BP, arrhythmias, blood values
25
Q

worst thing that can happen when taking afterload reducers…

A
  • hypotension, hypovolemia, hyperkalemia

- aggravated HF

26
Q

contractility (& afterload) reducers

A
  • reduce myocardial workload by reducing contractile force
  • reduce myocardial workload by reducing afterload
  • ex. beta blockers
  • assess apex 1 min (HR) and BP
  • look at pt’s normal HR & BP
27
Q

worst thing that can happen by taking contractility reducers…

A
  • inadequate CO from reduced contractility

- develop bradycardia, dec urinary output

28
Q

positive inotropes (cardiac glycosides)

A
  • inc the contractile force of myocardium
  • slow HR (gives heart more time to fill)
  • ex. cardiac glycosides (digoxin)
  • do not give digoxin if HR below 60
29
Q

worst thing that can happen by taking positive inotropes…

A
  • inc myocardial workload by inc O2 demands, digoxin toxicity
  • bradycardia (dec conduction b/w nodes)
30
Q

assessments/monitoring for positive inotropes…

A
  • assess HR (apex 1min)
  • monitor blood values of digoxin (avoid toxicity)
  • monitor renal Fx (excreted by kidneys)
  • look at last dosage of digoxin
  • be aware of S&S of digoxin toxicity (anorexia, nausea, visual disturbances, bradycardia, confusion)
  • careful of loop diuretics (dysrhythmias); watch for K+ levels
31
Q

pt teaching w/ digoxin

A
  • S&S of digoxin toxicity (anorexia, nausea, visual disturbances, bradycardia, confusion)
  • teach about dosing
  • med in liquid form
  • keep digoxin tablets in air-tight container protected from light
32
Q

pediatric considerations of HF

A
  • most frequently secondary to structural abnormalities
  • congenital heart defects (need Sx)
  • myocardial failure, contractility of ventricles impaired (d/t cardiomyopathy, dysrhythmias, severe electrolyte imbalances, ind demand on heart w/ sepsis or severe anemia)
33
Q

goals of HF therapy

A
  • treat underlying issue
  • improve cardiac Fx (inc contractility & dec afterload)
  • remove accumulated fluid and Na+ (diuretics, test electrolytes regularly, give fludi in small containers, daily weigh ins)
  • dec cardiac demands (neutral thermal enviro, treat infect, reduce WOB)
  • improve tissue oxygenation and dec O2 consumption (add supplemental O2)
34
Q

crystalloid fluid

A

supply water and Na+ to maintain osmotic gradient b/w extravascular and intervascular compartments
- if given large amounts –> dec colloid osmotic pressure

35
Q

why admin crystalloids

A
  • admin as maintenance IV fluid
  • pt NPO
  • dehydration
  • hypovolemic shock
  • hypotension
  • inc urinary flow
  • electrolyte imbalances
36
Q

negative SE of crystalloids

A
  • some will inc intracranial pressure

- peripheral edema (d/t overhydration, low albumin, can cause pulm edema)

37
Q

types of crystalloids

A
  • normal saline (isotonic soln; 0.9% Na)
  • hypertonic saline (3% Na)
  • some have dextrose in it (dextrose 5% water - hypertonic soln)
  • ringers lactate soln (have lactate, Na, K; isotonic soln)
38
Q

colloids

A

protein substances that inc the colloid osmotic pressure and move fluid from IS compartment to plasma in intravascular space by pulling fluid into blood vessels

  • ex. albumin, globulins
  • “plasma expanders”
  • maintain plasma vol longer
39
Q

why admin colloids

A
  • use w burn pts (damaged blood vessels; skin not intact –> leaky)
  • renal pts (give albumin during dialysis run)
40
Q

negative SE of colloids

A
  • more expensive
  • be aware of allergies (its a protein)
  • no O2 carrying capacity or clotting factors