Heart Failure Flashcards

1
Q

Define Dyspnoea

A

Dyspnoea is an overall term used to describe an unpleasant awareness of increased respiratory effort and is used synonymously with ‘breathlessness’

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2
Q

Pathophysiology of Dyspnoea

A
  • The pathophysiology of dyspnoea is poorly understood.
  • Normal breathing - respiratory centres - brain stem.
  • Intrapulmonary parenchymal stretch receptors
  • Carotid body and central medullary chemoreceptors, peripheral vascular receptors and pulmonary artery baroreceptors all contribute to the pathways leading to dyspnoea.
  • Stimulation of pulmonary stretch receptors has been shown to influence the intensity of breathlessness
  • There is a no direct relationship between the degree of hypoxia and the degree of breathlessness
  • PCo2 & Ph. has a casual relationship
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3
Q

List the most common causes of dyspnoea

A
  • Asthma
  • Cardiac failure
  • COPD
  • Pneumonia
  • Interstitial lung disease • psychogenic disorder
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4
Q

List four life threatening causes of dyspnoea

A

Airway obstruction Anaphylaxis Epiglottitis

Severe pulmonary oedema Severe asthma Tension pneumothorax Cardiac tamponade Massive PE

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5
Q

List four urgent but not lifethretetning causes of dyspnoea

A

Simple pneumothorax Asthma (less severe) Pneumonia Pulmonary oedema COPD Metabolic acidosis Poisoning Valvular heart disease Myocarditis Guillain-Barre Syndrome Pulmonary embolism

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6
Q

List three non-urged causes of dyspoonea

A

Pleural effusion Neoplasm Pneumonia (less severe)

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7
Q

How does asthma present in ED?

A
  • May have abrupt onset
  • Tachypnoea, tachycardia
  • Accessory muscle use
  • Diffuse expiratory wheeze • Cyanosis, exhaustion
  • Low Sats Late sign
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8
Q

How is asthma managed in ED?

A
  • Assesment-Mild/Moderate/Acute Severe/Near fatal Asthma • Salbutamol/Steroids
  • Atrovent –Acute severe/Life Threatening
  • Magnesium Sulphate 2g-20 minutes
  • Aminophylline 5mg/kg-20 minutes • Invasive Ventilation
  • BTS Guidelines
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9
Q

How does pneumonia present?

A
  • Progressive & Systemic symptoms • Fever
  • May have associated chest pain
  • Productive cough Pyrexia
  • Tachypnoea, tachycardia
  • Coarse / focal crepitations
  • Bronchial breath sounds
  • Focally reduced breath sounds
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10
Q

Outline CURB 65

A
  • CURB 65 score of 2 • Confusion
  • Urea >7
  • RR>30
  • Low BP <90 • Age >65
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11
Q

How dose a pneumothorax present?

A
Simple
• Sudden onset with Pleuritic pain
• Unilateral reduced breath sounds
• Hyper resonance to percussion
Tension - as simple plus
• Collapse
• Extreme respiratory distress
• Tracheal deviation
• Elevated JVP
• Cardiovascular collapse: tachycardia, hypotension
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12
Q

How does anxiety present?

A
  • May have abrupt onset
  • Previous episodes related to stress
  • Tingling/pins and needles,
  • Hyperventilation syndrome
  • Tachypnoea
  • Tachycardia
  • Tetany
  • No other abnormal physical findings
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13
Q

List the causes of pulmonary oedema

A
  • MI/IHD
  • Arrhythmias
  • Prosthetic Valve Failure
  • Cardiomyopathy
  • Exacerbation of HTN/Valve disease
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14
Q

How does pulmonary oedema present?

A
  • Usually progressive,may be abrupt in onset
  • Previous myocardial infarction (MI)
  • Risk factors for ischaemic heart disease (IHD) • Dyspnoea, tachypnoea
  • Elevated JVP if associated with CCF
  • Bilateral inspiratory crepitations
  • Gallop rhythm
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15
Q

How is pulmonary oedema managed in ED?

A
  • GTN Spray plus GTN Infusion (10mcg/min)
  • Frusemide 50 mg iv
  • Opiod iv
  • NIV (CPAP/BiPaP)
  • Invasive Ventilation
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16
Q

How does PE present?

A
  • Dyspnoea commonly no other symptoms
  • Pleuritic chest pain ( Minority)
  • Haemoptysis
  • Unexplained Hypoxia
  • History
  • Pleural friction rub
  • 30% - normal SpO2
  • Tachycardia/Tachypnoea
  • Pyrexia (post Infarct)
17
Q

What is the difference between a massive and sub massive PE?

A
  • Massive PE- marked Hypoxia/Cardiovascular collapse
  • Submassive PE is defined as an acute PE without systemic hypotension (systolic blood pressure >90 mm Hg) but with either RV dysfunction or myocardial necrosis
18
Q

What are the disadvantages of a CTPA and VQ scan?

A

• CTPA – High dose radiation Definitive answer
Differential Diagnosis defined
• V/Q Scan – Low dose Radiation
Reported as Low/Int/High Corelation with Clinical scoring

19
Q

Management of PE

A
  • Sub Massive/Massive – Thrombolysis • Alteplase 100mg over 120 minutes
  • Cardiac arrest - tPa 50 mg stat
  • Heparin Infusion to be started
  • IVC Filter/Clot Fragmenting • LMWH for stable PE
  • 7-12% Occult Cancer
20
Q

Hypoxia Causes

A
  • V/Q Mismatch (PNA, CHF, ARDS, atelectasis, etc) – Raised A-a Gradient
  • Shunt (PFO, ASD, PE, pulmonary AVMs) Raised A-a Gradient
  • Alveolar Hypoventilation (interstitial lung, environmental lung , PCP Infection)- Raised A-a Gradient
  • Hypoventilation (COPD, CNS drug overdose, neuromuscular Disease)-A-a Gradient Reduced
  • Low FiO2 (ex: high altitude)- A-a Gradient Reduced