Heart Failure Flashcards
HF
Pathophysiology
Inadequate tissue perfusion inspite of adequate filling pressure
HF
Presentation
SOB
HF
DDs of SOB
- Heart disease- HF
- Lung disease- Asthma, COPD, Lung fibrosis
- Renal failure- Pleural effusion/ pericardial effusion/ cardiomyopathy/ anemia
- Metabolic acidosis
- Anemia
HF
Classifications of HF
- Acute/ Chronic
- Systolic/ Diastolic
- Left/ right
HF
Systolic HF
ejection fraction is less (<60%)
HF
Diastolic HF
Heart is stiff and unable to stretch
HF
In which can the ejection fraction be normal
Diastolic HF
HF
Pulmonary edema Sx
- SOB
- B/L coarse crepts
- Patient tries to sit up
HF
Immediate Mx of acute heart failure
- Admit the pt
- ABC
- Give O2, if SpO2 <94%
- IV cannula and collect the blood for Ix
- IV frusemide- 80- 120mg
- GTN infusion
- If no response CPPV
- Added measures- Dialysis, venesection
HF
IV frusemide function in acute LV failure Mx
Dilatation of veins occurs first,
diuretic effect comes later
HF
How long does it take for the diuretic effect of frusemide to come into action
around 30 minutes
HF
Function of GTN in acute HF Mx
Venodilation
HF
what needs to be done if the high stat frusemide dose cause the BP to crash
the pt may not respond to it so start an infusion (5mg/hr)
HF
In which situation should GTN be given in acute LVF
if the BP is normal or high
HF
Next step if Frusemide and GTN fails
CPPV
HF
In which situation should GTN be avoided
if the BP is low
HF
Added measures in acute LVF Mx
- dialysis
- venesection ( not done anymore)
HF
LV failure pathophysiology
- LV pumping reduced
- Blood accumulates in the left ventricle
- workload on the L/atrium to send blood to the ventricle is high
- pressure in the L/atrium is high
- pressure increase in the pulm vessels
- fluid leaks out of the pulm vessels giving pulm edema
HF
Why CPAP in acute LVF Mx
sending air in a positive pressure will push the fluid out of the airways. Reducing the pulm edema
HF
High output HF Examples
- Anemia
- Thyrotoxicosis
- Wet beri beri
- AV fistula
- Paget’s disease
HF
High output HF?
cardiac output is high but its still not enough to meet the demands
HF
Chronic HF
RHF secondary to LHF
HF
Chronic HF is also known as
congestive HF
HF
LHF clinical features
- exertional dyspnea
- orthopnea
- Paroxysmal nocturnal dyspnea
- B/L fine crepts
HF
why orthopnea in LHF
Abdominal organs will push the diaphragm + already SOB
HF
Paroxysmal nocturnal dyspnea
getting up w cough during sleep
HF
RHF clinical features
- elevated JVP
- Tender hepatomegaly
- B/L ankle edema
- Ascites
HF
RHF pathophysiology
- R/ ventricle pumping is reduced
- Blood accumulates in the RV
- Workload on the R/ atrium is high
- Blood backup in the R/atrium. Pressure increases
- Blood backup in the SVC and IVC. Pressure increases
- Fluid leaks out giving fluid overload Sx