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UM - Cardiovascular Conditions > Heart Failure > Flashcards

Heart Failure Flashcards

1
Q

What is heart failure

A

A clinical syndrome that occurs when the heart cannot provide enough blood flow to meet metabolic requirements or accommodate venous return

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2
Q

T or F: Heart failure incidence has declined over the past 20 years

A

False, but the survival rate has increased due to better risk ID, earlier intervention, and long term management

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3
Q

List 12 possible causes of HF

A
  • CAD
  • HTN
  • Diabetes
  • Valve Disease
  • cardiomyopathy
  • congenital heart disease
  • lung disease (cor pulmonale)
  • metabolic
  • anemia
  • drug toxicity
  • renal disease
  • aging
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4
Q

What are the two types of LV Dysfunction

A

Systolic (impaired contraction and ejection) [most common]
Diastolic (impaired relaxation and filling)

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5
Q

What contributes to Left Ventricular Dysfunction?

A

Excessive overload and increased afterload leading to an increased in LVEDP, causing increased P in the lungs, leading to congestion and dyspnea

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6
Q

What is the major clinical symptom of LV Dysfunction

A

Dyspnea

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7
Q

What is the consequence of LV Dysfunction?

A

Decreased CO leading to dec perfusion t/o the body

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8
Q

What is ejection fraction reduced heart failure?

A

Dec CO from impaired contraction of the ventricles (change in EF to <40%)

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9
Q

What is ejection fraction preserved HF?

A

Inefficient CO from thickening of the ventricular walls, leading to smaller chambers and less ability to accept blood (no/min change EF)

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10
Q

What is the etiology of LV HF

A
  • atherosclerosis
  • cardiomyopathy
  • HTN
  • Valve Disease
  • Drug toxicity
  • congenital defects
  • Diabetes
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11
Q

What are the s/s of LV HF?

A
  • dyspnea
  • dry cough
  • fatigue
  • orthopnea
  • fluid retention
  • pallor/cyanosis
  • crackles in lung base
  • mm weakness
  • dizziness
  • renal changes
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12
Q

What are the pulmonary effects of L HF?

A

Fluid acumm in pulmonary interstitium leading to early airway closure and a ground glass appearance on an x-ray

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13
Q

What are the stages of pulmonary edema?

A

1) Difficult to detect (weight gain > 3 lbs /day & difficulty lying flat)
2) Detectable via auscultation of lungs (crackles) and absence of air mvmt
3) Detectible via auscultation of lungs w/greater crackles and greater absence of air mvmt (blood-tinged foam, reduction of lung volumes, hypercapnia)

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14
Q

What stage of pulmonary edema is most commonly detected?

A

Stage 2, altho stage 1 would be better

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15
Q

What is the most common cause of RV HF

A

LV HF

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16
Q

What are the hallmark s/s of RV HF

A

LE Edema
Ascites

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17
Q

What occurs w/RV HF

A

inc in blood in ventricle leading to high RA & VC P, impairing venous flow in body, leading to inc P of the abdominal organs

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18
Q

What is the etiology of RV HF?

A
  • COPD
  • Pulm HTN
  • LV F
  • PE
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19
Q

What are the S/S RV HF?

A
  • periph edema
  • jugular vein distention
  • fatigue
  • ascites
  • liver engorgement
  • weight gain
  • cyanosis
  • decc exercise toelrance
  • anorexia
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20
Q

The following are s/s of a) LV or b) RV HF:

Jugular vein distention
Peripheral edema
Weight gain
Fatigue
Ascites

A

b) RV

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21
Q

The following are s/s of a) LV or b) RV HF:

Dyspnea
Renal changes
Crackles in lung base
Fluid retention

A

a) LV

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22
Q

Define cor pulmonale?

A

Alteration in the structure and function of the RV leading to Right sided HF

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23
Q

Progression to cor pulmonale from COPD

A

COPD –> chronic inflamm of lungs–> pulm vasoconstrict –> increased p and volm in pulm a –> pulm HTN –> RV hypertrophy –> R HF

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24
Q

What are compensatory mechanisms of HF? (LIST)

A
  • frank-starling mechanism
  • neurohumoral compensation
  • remodeling cardiac dysfunction
  • inc oxygen extraction
  • anaerobic metabolism
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25
Q

How does the Frank-Starling Mech work (HF)?

A

When preload increases (LVEDV), it causes an inc in pressure, causing an increase stretch, causing increase in CO to compensate for drop in CO w/HF

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26
Q

How does neurohumoral activation work (HF)?

A

Occurs when MAP drops w/HF, causing symp NS to release nor and epi which then increase HR and contractility to inc SV and TPR to inc MAP

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27
Q

What occurs w/ADH in HF?

A

It is increased so that BV can inc to inc CO but, long term, the increase in retention of fluid and pressure is bad as it contributes to the inc in afterload and preload already seen in HF

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28
Q

How does ventricular remodeling work in HF?

A

End result = detrimental
- increased stress = inc size, shape, structure and fun = lead to inc SV and higher CO even w/dec EF (SHORT TERM) –> LT = hypertrophy = less effective pumping

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29
Q

What is the onset, course, and prognosis of HF?

A

On: >= 60s
Course: Gradual
Prog = progressive

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30
Q

What medical tests are done for HF?

A
  • Electrocardiogram (ID infarct, hypertrophy, arrhyth, conduction disturbances)
  • Chest x-ray (heart size)
  • Lab Values (ABGs, liver ensymes, ANP/BNP.CNP)
  • MUGA
  • Echo
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31
Q

What drugs are used for HF?

A
  • ACE Inhib to help renal fun
  • diuretics to dec fluid volume
  • BB to assist Sym NS
  • Digitalis to inc mm contract
  • aldosterone antago to dec BV
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32
Q

What procedures are done for HF

A
  • CABG
  • Intra-aortic balloon pump
  • Ventricular assist devices (VAD)
  • Heart Transplant
  • Pacemaker
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33
Q

What is an echo used for w/HF

A

to determine size/shape of heart, strength of contraction, valve function, thrombosis

34
Q

What is MUGA (multigated acquisition imaging)

A

A noninvasive technique that calculates LV EF and studies the electrical activity of the heart

35
Q

What are the s/s of digitalis toxicity?

A

Nausea, vomiting, diarrhea, confusion, arrythmias, & blurred vision w/green/yellow halo around an object

36
Q

What does an increase in BUN indicate?

A
  • kidneys are not functioning due to dec renal blood flow
37
Q

What does an increase in creatinine indicate?

A
  • kidneys are not functioning well
38
Q

What does a rise in BNP indicate?

A

Pt likely has HF

39
Q

T or F: Individuals w/HF have no change in capacity for exercise

A

False, it decreases leading to a poorer prognosis

40
Q

What does dec. exercise capacity effect?

A

ADLs
Health QOL
Hospital admission rate
mortality

41
Q

What are the two scales used to classify HF and the difference b/t them?

A
  • AHA Classification: Based on structural damage
  • NYHA: based on symptoms w/physical activity
42
Q

What is Stage I of NYHA HF

A

No limitation in phys activity & ordinary phys A does not cause fatigue, palpitations or dyspnea

43
Q

What is stage II of NYHA HF

A

Slight limitation of phys a, comfy at rest but ordinary activity results in fatigue, palpitations, or dyspnea

44
Q

What is stage III of NYHA HF?

A

Marked limitation of physical a; comfy at rest but less than ordinary activity results in fatigue, palpitations, or dyspnea

45
Q

What is stage IV of NYHA HF?

A

Symptoms at rest; unable to do any PA w/o symptomology

46
Q

What is a state of compensated HF?

A

When the condition is stable and the pt is not exhibiting pulmonary and venous congestion-associated s/s

47
Q

What is acute decompensated HF?

A

Presence of new or worsening s/s (related to inc congestion and inc ventricular filling P) of dyspnea, fatigue, or edema that lead to hospitalization or unscheduled medical care

48
Q

When are you in the green zone for HF and PT?

A

NO:
swelling
SOB
Weight gain
Chest pain
Dec in ability to maintain activity level

= Can do PT

49
Q

When are you in the yellow zone of HF and PT?

A
  • weight gain 2-3 lbs in 24 hrs
  • increase cough + SOB
  • periph edema
  • orthopnea

= communicate w/physician b4 exercise

50
Q

When are you in the red zone for HF and PT?

A
  • SOB @ rest
  • Unrelieved chest pain
  • wheezing or chest tightness at rest
  • paroxysmal nocturnal dyspnea
  • weight gain/loss of >5 lbs in 3 days
  • confusion

= immediate visit to ER or physicians office

51
Q

What are some HF Specific Qs?

A
  • are symptoms stable/worsening?
  • are they (sympt) provoked or at rest?
  • any accompanied symptoms?
  • orthopnea or PND?
  • how far can u walk and is it typical?
  • are you retaining fluid?
  • do you restrict sodium in ur diet?
  • are you losing/gaining weight?
  • how do you sleep?
52
Q

What do you do for a systems review of HF?
- CP
- integ
- MSK
- NeuroMusc
- Comm

A
  • CP: vitals at rest & w/activity + edema
  • Integ: LE skin & color
  • MSK: ROM, strength, posture, height, weight
  • Neuromusc: Gross mobility and sensation
  • communi: affect, ability, cognition, lang, learning style
53
Q

What are precautions and safety concerns for HF (aka what is the risk?)

A
  • risk of:
    rehospitalization, pneumonia, decompensation, & fall risk
54
Q

Goals of HF Rehab?

A
  • self-MGT improved w/behav for healthy habits acquired
  • ability to perform phys tasks inc
  • disability w/chronic illness reduced
  • perform and independence in ADL inc
  • phys response to inc o2 demand improved
55
Q

What is required to start exercise in Pt w/HF?

A
  • compensated
  • speaking w/o dyspnea and RR <30
  • < mod fatigue
  • crackles in < 1/2 lungs
  • Resting HR < 120 bpm

If on invasive monitors:
- Cardiac index > 2.0 L/min
- CVP < 12 mmHg (RAP)

56
Q

What happens to skeletal mm w/exercise in HF

A
  • Inc mitochondria
  • II –> I fibers
  • Inc capillary density
  • Inc strength and endurance
57
Q

What happens to vasculature w/exercise in HF

A
  • Improved endothelium-mediated vasodilation
  • favorable remodeling of conduit arteries
  • inc. diameter of arterioles
  • dec. TPR
58
Q

What happens to ventilatory system w/exercise in HF

A
  • inc MIP (max inspiratory P)
  • enhanced efficiency of ventilation
  • reduced lactate levels during exercise
  • improved gas diffusion across alveolar capillaries
59
Q

What are the ACSM Exercise recommendations for stable HF?

A
  • Aerobic b/t 55-75% HRR and RPE 11-14
  • Strength 30-40% 1RM progressed to 70% 1RM
60
Q

What are the parameters for inspiratory mm training?

A

Class II and III w/HFrEF at: 30 min/day @ >30% MIP (max inspiratory pressure) 5-7x/week for 8-12 weeks

61
Q

What is an abnormal physiological response to exertion?

A

Drop in SBP below standing rest or abnormally exaggerated rise in SBP

62
Q

When should exercise be terminated in pts w/HF?

A
  • dyspnea or fatigue
  • RR > 40
  • development of S3
  • inc pulmonary crackles
  • dec HR or BP > 10 bpm/mmHg
  • diaphoresis, pallor, or confusion
  • poor pulse pressure
  • inc PVC
63
Q

Define cardiomyopathy

A

Disease of the heart mm itself

64
Q

List the 3 types of cardiomyopathies

A
  • dilated (weak + enlarged heart)
  • hypertrophic ( thickened + stiff heart mm)
  • Restrictive (unusual w/stiff but not thickened heart)
65
Q

What is the most common type of cardiomyopathy

A

Dilated

66
Q

What is the cause of dilated cardiomyopathy

A

1/3 = idiopathic
2/3 = other heart disease, thyroid disease, viral infections, poor diet + alcohol, toxins

67
Q

What occurs in dilated cardiomyopathy

A

LV dilates and myocardial wall thins, weakening the pump ability that can lead to HF, valve disease, arrythmias, and blood clots

68
Q

What is a common cause of sudden cardiac arrest in young people?

A

Hypertrophic cardiomyopathy

69
Q

What occurs in hypertrophic cardiomyopathy

A

Myocardial cells enlarge causing walls of LV or interventricular septum to thicken that can cause mitral value insufficiency or dangerous arrythmias

70
Q

What are the symptoms of hypertrophic cardiomyopathy?

A

Chest pain, dizziness, SOB, fainting

71
Q

Which cardiomyopathy looks like:
a) EF preserved HF
b) EF reduced HF

A

a) hypertrophic Cardiomyopathy
b) Dilated cardiomyopathy

72
Q

What occurs in restrictive cardiomyopathy?

A

The ventricles are rigid from scar tissue, leading to dec filling and atrial hypertrophy that can eventually lead to HF and arrythmias

73
Q

What are the risk factors of cardiomyopathies?

A
  • chemo
  • obseity
  • LT ETOH abuse
  • smoking
74
Q

S/s of cardiomyopathies?

A
  • chest pain
  • dyspnea
  • orthopnea
  • tachycardia
  • palpitations
  • periph edema
  • distended jugular vein
  • fatigue
  • weakness
75
Q

What is the onset of cardiomyopathies

A

Any age

76
Q

What is the course of cardiomyopathies

A
  • depends on type, but could progress to: HF, arrythmias, or valve issues
77
Q

What is the prognosis of Cardiomyopathies?

A
  • Idiopathic dilated cardio = Poor
  • hypertrophic = relatively normal life bc EF presereved
78
Q

What are precautions should you take for cardiomyopathies?

A
  • limited activity
  • diminish stress
  • take vitals
79
Q

What medical tests are done w/cardiomyopathies

A
  • chest x-ray
  • echo
  • PET
  • blood chemistry
  • EKG
80
Q

What is the medical MGT of cardiomyopathies?

A
  • lifestyle changes (be active and eat healthy)
  • drugs (BB, Ca+ Blockers)
  • Sx (mechanical circulatory support or transplant)

UM - Cardiovascular Conditions (5 decks)

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