Health Psychology 2301 Midterm #2 Flashcards
Health Compromsing Behaviors
- undermine current or future health
- usually addicitive
- can be modified with incentive and help
Addiction Definition
- condition produced by repeated consumption of a natural or synthetic psychoacitve substance
- person has become physically and psychologically dependent on the substance
Definition Physical Dependence
- body has adjusted to a substance and incorporated it into normal functioning (need it to function normally)
Psychological Dependence
- usually happens before the physical dependence
- compelled by desire for the substance’s effects
- like it and how it makes you feel
Definition “Substance Use Disorder”
- In DSM
- Show two characteristics of many:
- tolerance for the substance
- failing to fulfill important obligations
- putting yourself or others at risk of physical injury
- having substance-related legal problems
Definition Tolerance
- body increasingly adapts to the substance and requires larger doses to get the same effect
Definition Withdrawl
- unpleasant physical and psychological symptoms experienced when discontinuing or reducing using a substance on which you were dependent
Use: Pos / Neg
- consequence increases the behaviour
- Positive: buzz, elation (cigarettes, booze)
- Negative: remove tension, stress, anxiety (works quickly so hard to stop)
- Use it to avoid withdrawl symptoms (DT’s, from alcohol or shakes, nausea)
Substance-related Cues
- (classical conditioning)
- pairings of events, emotional state or cue with a substance can make you crave it
- the stimuli links to the feeling of using
- ex: friends who drink or use
Incentive Sensitization Theory
(can get dopamine release when see the cue - incentive sensitization theory
Expectencies and Abuse
- form them young (see parents) or older (see vodka and remember getting sick on it)
- can be negative or positive
- idea about the outcome of the behavior
- can influence whether someone will stop using
Personality and Substance Use
- High impulsivity
- Risk taker
- Sensation seeking
- Low self-regulation
Genetics and Abuse
- Identical twins similar addictive behaviour
- different genes for different substances
- parental involvement can counteract
- epigenics: environment can alter the genes involved
Smoking
- Single greatest cause of preventable death
Who smokes?
-
Age: begins around 13 (Grade 8)
- more risk if no post-secondary school
- fewer begin after 20’s
-
Gender:
- 75% men
-
Sociocultural:
- 80% smokers in developing countries (poor health promotion, more stress, no soc. engin)
- 18% pop in Canada
- 58% Indigenous pop, higher on reserve
- assoc with social class -(blue collar more likely to smoke)
Why smoke - psychological
- perceive low risk, high benefit (teens)
- impulsive or sensation-seeking
- low self esteem
- concern body weight
- want image of cool, rebellious, glamorous
Why start - Social Factors
- Peers: hang with other smokers
- Family: more likely if parents smoke, reduces belief it will be harmful
- Stressors: depression increases smoking
- School: poor regulation/enforcement
- Culture: standards of “thinness”
- Media: see pop bands smoking
- LGBTQ: if not supported can increase smoking
Why Keep Smoking
- Psychological:
-
Affect Management Model
- smoke to regulate emotional state
- Behav. Conditioning: habit (linked with cues)
- Personal Identity: ties to self image (know you are a smoker - becomes part of you)
-
Affect Management Model
- Social:
- choose friends who smoke, no support to quit
- Biological:
- exposure during pregnancy, heredity, (area in brain (insula)
Nicotine
- Addictive, fast acting drug, reinforcing each hit - after 2 hours half gone after metabolism but half life
- stimulates the nervous system (reward system)
- can relax as well
- makes you alert, helps concentration, arousal, psychomotor performance and can screen out irrelevant stimuli
- Nicotine regulation model: keep level of it in blood to avoid withdrawl so even if low or light cigs just smoke more as need nicotine
Smoking and Cancer
- lung, mouth, espophagus etc.
- lung cancer: deadliest form of cancer
- can lose all cilia lining bronchial tube (can’t clear foreign products including carcinogen cig ones)
Smoking and Cardiovascular Disease
- twice the risk of CVD
- more smoke, higher the risk
- Nicotine constricts blood vessels, increases HR and BP and cardiac output so wear and tear on heart
- higher plaque and risk artherosclerosis
- if smoke more cardiac reactivity to stress
- lower physical activity usually or other poor habits
Lung Disease
- Emphasyma - lung tissue breaks down so poor airflow and alveoli become damaged and die so not much oxygen into bloodstream
- COPD - high risk of pneumonia, colds , bad immunity
Who Drinks - Age and Culture
- 15-16 yrs old (63% drink) and 35% binge (8+ drinks)
- First Nation: more non-drinkers/ if do then heavy
- can be associated with social life
- Self concept: “I am a drinker”
Alcohol Use Disorder / Problem/ Binge
- Alcohol use disorder: physical addiction, withdrawl symptoms if stop, high tolerance, no control
- Problem drinker: might not have withdrawl effects but social, psychological and medical problems
- Binge drinking: 5+ drinks at least once in a 30 day period
Alcohol Continuum
Psychosocial differences:
- perceive fewer negative (sedation) and more positive (stimulation) consequences
- experience high stress/trauma and live in environments that encourage drinking
- heightened physiological reactions (pleasure)
- less likely to use control strategies to not overdrink
Biological Reasons
-
Stress dampening
- buffer stress (will decrease stress response) BUT after a few can become unstable, anxious
- Family history/genes - develop tolerance, higher reward (positive reinforcement) less plateau,avoid withdrawl symptoms
Acute Effects of Alcohol
- Enters via bloodstream, metabolizes in liver
- Starts stimulant then becomes depressant (if lots)
- Lots alcohol:
- slurred speech/vision
- interferes with complex though processes
- poor coordination
- loss of balance
- Neural: binds to GABA receptors (quieting effect) and can even stop or slow breathing - alc. poisening, faint
Moderate vs Heavy Drinking
- Heavy: perceive more + consequences, often have greater stress, live where encourage drinking, strong substance-reated cues
- Moderate: more control strategies
Long Term Heavy Drinking Effects
- Cirrhosis - liver cells die off replaced by scar tissue
- blocks metabolism of protein, fat and carbs
- liver cells engorge with fat and protein
- obstruction of blood flow through liver
- Impaired immune function
- Cancer - breast, head/neck/liver/esoph/colorectal
- acetaldehyde:toxic chem & carcinogen
- High BP
- Brain damage - perception, memory, decreased grey matter
- Heart damage:
- arrhythmia
- weak heart so bad blood flow
- increased cholesterol
- hypertension
FASD
- Drinking pregnant: raise risk of fetal alcohol syndrome or low birth weight or impaired learning, facial abnormalities, growth deficiencies
- Also: miscarriages, still births, CNS disorders
- especially dangerous in early months pregnancy
Capillery Hemorrhages
- alcohol vasodilator when metabolized (widens)
- vessels continue to enlarge
- blotchy skintone, redness, broken capilleries
Benefits of Drinking?
- Light drinking:
- decreased risk of MI & Death
- lower BP
- lower risk of dying from Heart Attack
- increase good cholesterol
- Amount
- 1-2 / week for women
- if don’t drink don’t start
Drugs - who uses
- Teens try - weed grade 7-8 20% students
- prescription drugs tried - opiods
- non-mainstream youth more vulnerable (street kids, those who are discrim. against, kids who had trauma)
Why teens use/keep using
- Psych: low self-control, high sensation-seeking
- Soc: friends use, family, celebrities - all have positive attitudes about the drugs
- Continue:
- makes them feel good
- reduces anxiety and tension
- drug-related cues
- social pressure
- rebellious, impulsive, illegal no problem
- less conforming
- less religious
Preventing Approaches
3 Approaches:
- Public Policy and legal
- Health Promotion and Education (address social refusal skills - modelling / role play) MADD speakers
- Family involvement
Public Policy/Legal
- Taxation and higher cigarette prices
- Age limits
- Ride programs
- Outlaw all use (posses, sell, use)
- More patrols
- Close bars
Health Promotion/Education
- don’t focus on harm - already know that/don’t care
- Current consequences: now: more $ if quit, more dates if don’t smell; later: fewer wrinkles, nicer teeth
- teach refusal skills, announce intention to quit (more likely to do it)
- Build Skills: assertiveness training, anxiety reduction, start really early then have booster sessions, involve parents; have facilitator be someone they can relate to (close to their age)
- Media Campaigns - MADD speakers
Family Involvement
- Many parents not aware kids are using
- less likely to use if know parents disapprove
- weekly sessions with parents
- monitor behavior and use discipline
- help teach kids to avoid peer pressure
- reduce family conflict
- follow up in grade 10 shows less likely to have begun
Quit NO Therapy
- Mature Out - use often peaks at 22 then declines (getting jobs, locial norms change, in relationships)
- Self efficacy determines if can quit on one’s own
- Cold turkey best if paired with a reward (might take a few tries)
- Early Intervention: screening by health prof. (check for binge drinking, crravings, other); information / advice / support
Treatment Methods
- Psychological
- Self-help groups
- Inpatient / Outpatient
- Chemical methods
- Multidimensional programs
Psychological Treatments
- motivational interviewing
- reduce negative reinforcement
- skills training - social anxiety, self-efficacy
- congnitive restructuring - new helpful thoughts
- Behavioural: gain control over environmental condit.
- self-monitor
- stimulus control
- behavioural contracting - reward/punishment
Self-Help Groups
- AA - largest one / Alanon, Narcon, Alateen, etc. which is “total abstinence model”
- good cuz with others making same commitment
- bit religious and deals with idea of powerlessness (12 step)
- Never recover - always recovering
- longer with AA better social functioning and less use
Controlled Use
- e.g. non-dependent problem drinking (controlled)
- cognitive behavioural techniques (goal setting, self-monitoring and self-control)
- for those who have not been drinking a long time, are high functioning, employed, living in supportive environments
- Advantages(?) maybe a more realistic goal and abstinence-based programs have high drop-out rates
Inpatient/Outpatient
- Inpatient: detoxification - withdrawal under medical supervision (average: 28 days), limited facilities
- Outpatient: discharge to follow-up sessions or supervised living, therapy, CB strategies
Chemical Methods
- Smoking: nicotine replacements
- decreases craving and withdrawal
- helps to quit
- Alcohol:
- Antabuse - daily, makes you nauseous, also not learning the skills to stay sober
- Naltrexone - blocks the “high” feelings
- Narcotics (another lecture)
Multidimensional Methods
- CBT + patch
- Methadone +reward system
- need to verify self-reports, daily phone calls, family involvement, physician involvement
Relapse
- takes 2 weeks for body to adjust then go into the world where there are stressors
- Bad moods, social cues
- Stress
- motivated reasoning / denial / decreased self efficacy after relapse
- irrational thinking - why relapse “okay” and less likely to endorse sober life
Harm Reduction
- Harm reduction: keep people safe cuz they’ll mess up (drugs and alcohol) - some students do use protective behavioral strategies: eat more, pick # drinks before going, alternate with water, pace yourself, know what you “can’t” drink
- focuses on risks / consequences and not just use
- promote safe substance use
- focus more on reducing substance use to help minimize social and physical harm
- normalize: relapse is not a failure, but normal/common
Antagonist / Agonist
- Agonist: Chemical/substance - activates a receptor and causes an action
- Partial-Agonist: -Chemical/substance -activates a receptor, with less affinity for the receptor
- Antagonist: Chemical/substance - inhibits action by blocking or inhibiting a receptor
Narcotic Analgesics
- produce an opiod effect in the brain
- binds to opiod receptors
- euphoria, relaxation, pain relief
- high dose: death or respiratory failure
Dopamine
- Dopamine reward pathway: ventral tegmental - limbic - nucleus accumbens
- rat experiment - kept pressing lever to slelf medicate, even when lesioned
Endogenous Opiods
- naturally produced in the body
- Endorphin, Enkephalin, Dynorphin, Endomorphin, - involved in analgesia - pain regulation
- Endorphins: sexualy behavior, pain, appetite, reward
- most potent opiod to agonist receptor
- Heroin and morphine (natural) bind to the same receptors
- Others (synthetic) fentanyl, methadone
- some 1/2 & 1/2 : oxycodone, hydromorphone
Prescription Opiod Use
- problem if long term as can lead to addiction / abuse
- pain subjective so doc takes patient’s word for it
- pain can get worse - continual mu receptor agonists
- can cause euphoria - bad if at risk for addiction
Fentanyl
- used for palliative care
- similar to herion
- lollipops, patches, sublingual sprays, tabs
- now mixed in with street drugs
- no quality control
- 1917 - 4 o.d.’s per day
- higher use in males and ppl under 29
- HIV more prevalent
Opiods and D.S.M. Disorder
- Craving, or a strong desire or urge to use opioids.
- Recurrent opioid use resulting in a failure to fulfill major role obligations at work, school, or home.
- Continued opioid use despite having persistent or recurrent social or interpersonal problems caused or exacerbated by the effects of opioids.
Symptoms Opiod Withdrawal
- for Short acting opiates: first 6-12 hours
- for Long acting opiates: 30 hours
- yawning - trouble staying/falling asleep
- anxiety/agitation/sweating
- nose running, fever, muscle aches
- racing heart / hypertension
Bio Factors Opiod Use
Use
- History of chronic pain
- Impulsivity
Symptomology
- Sexual Dysfunction / Pinpoint Pupils / slurred speech/ HR, BP, Respir. and Temp up
Risks
- Altered learning and memory, HIV, Hepatitis, Sniffing causes perforations nasal septum, death
Psych Factors Opiod Use
History of drug use or traumatic experiences
Why use?
- Depression
- Motivated reason (downplay the negative, play up positive affective state)
- Personality: neuroticism
Social Impact Opioid Use
Social Impact:
stigma - users are devalued or rejected (not recognized as an illness, which it is)
marginalization of ppl with OUD leads to continuance - social exclusion leads to more use
Ppl with OUD thought to be in control over use, leading to blame, anger or punishment
Social Factors Opiod Use
Environment: partner, friends, family are users or are medical personnel and have access to opiods
Social Consequences: housing conditions, unemployment, marital difficulties
OUD Related Harms
Homelessness, prostitution, violent crime, Hep. C, HIV,
Biopsychosocial Model OUD
Bio: physiological, medications, neurochem., genetics
Psycho: emotions, attitudes, learning, beliefs, stress mgmt
Social: Family, peer, relationships, culture, socioeconomics
Dr. Gelbien: E.R. Physician / Back Pain - Percocet then crutch for: emot. pain, stress, escape. No Percocet left so then Fentanyl. Once tried that ‘s it. Addict mind stronger than rational mind. : divorce, geograph. separated from his kids, mother dies from patch. Addiction itself is a biol factor PLUS effect of stronger drug (Fentanyl). Social: police at his home, divorce. Psych: Stress.
Factors re Gabien’s addiction
- marital difficulties
- moved an hour from friends so isolated
- work stressful
- stressful relationships with staff at work
- drank - sometimes to excess on weekends
- Percocet presecription for hockey injury
- Another injury - back - more Percocet
- Recreational use of the pills when with friends
- Then daily use for work stress - relieve pain/anxiety
- Wife’s Ex moves closer- more drugs, withdrawal
- Mom gives him one of her Fentanyl patches
*
Opioid Stats
- 2016: opiod OD more common than fatal car crash
- major primary cause of preventable death for 19-35 year-olds in Canada
- 285% increase since 1985 (O.D.)
- 2436 opiod deaths in 2016
- BC: 7 ppl die a day
- 1013 in first 8 months in B.C.
Increased Opiod Use
- increased use of prescription opiods
- high bootleg synthesis and some with Fentanyl
- Strong drug- can block even horrible pain
- misuse can lead to coma, O.D.
- In paliative care they are used for pain - GOOD use as knocks them out (pain mgmt necessary)
- 2mg can cause o.d.
- 35% increase of Fentanyl in street drugs
- recommendation: treat it as an epidemic