Headaches Flashcards
What are sinister causes of headache
VIVID
- Vascular (SAH/haematoma)
- Infection (meningitis/encephalitis)
- Vision threatening (temporal arteritis/acute glaucoma)
- Intracranial pressure (SOL, hydrocephalus)
- Dissection (carotid dissection)
In terms of SOCRATES how do tension headaches present
S: generalised + bilateral O: gradual or acute C: constant pressure, as if the head were squeezing by a vise R: can occur lower back of head, neck and eye A: usually none T: more than 30 mins. Usually 3-4 hours E: Analgesics help S: moderate
What causes tension headaches
- Stress
- Dehydration
- Lack of sleep
- Exertion
What is the management for tension headache
Simple analgesics
In terms of SOCRATES (SOCATES), how does migraine present
S: unilateral O: paroxysmal (sudden attack) C: pulsating/throbbing A: pre-monitoring phase (aura) T: 4-72 hours E: physical activity/stress/straining S: moderate to severe
What can migraine be proceeded by
- Irritability
- depression
- fatigue
- aura hallucinations
(strange smell, lights, visual disturbances)
What are the associated symptoms with migraine
- Nausea + vomiting
- photophobia
- phono-phobia
- osmo-phobia
In terms of SOCRATES, how does migraine present
S: unilateral O: paroxysmal (sudden attack) C: pulsating/throbbing A: pre-monitoring phase (aura) visual changes, aphasia, tingling numbness T: 4-72 hours E: physical activity/stress/straining S: moderate to severe
What acronym can be used to remember migraine symptoms
POUND Pulsating One day onset Unilateral Nausea Disabling
Who is more likely to have migraines, males or females
females
What are the possible triggers for migriaine
- Cheese
- OCP
- Caffeine
- alcohol
- stress
What are the investigations for migraine
None
unless other differentials suspected (meningitis/subarachnoid haemorrhage)
What is the conservative management for migraine
Headache diary: ask them to avoid precipitating factors
What is the acute medical treatment for migraine
- Sumatriptan (5HT-agonists)
- Analgesia (NSAID)
- Antiemetic (metoclopramide)
In terms of SOCRATES (SOATS), how do cluster headaches present
S: unilateral, behind the eye
O: acute onset, same time each day (alarm clock headaches)
C: shooting, stabbing
R:
A: swollen eyelid and forehead swelling, nasal congestion, Horner’s syndrome
T: 20-30 minutes
S: Severe: can be disabling and can wake individuals from sleep
What autonomic responses do people get in cluster headaches
- Swollen eyelid and forehead
- Red eye
- tearing
- Runny nose
- Horner’s syndrome
Who is affected by cluster headaches more, male or females
males
What is the pattern of symptoms in a cluster headache
Patients may have daily headaches for a 5-10 week cluster, and this repeats itself once a year
What are the investigations for cluster headaches
None
unless other differentials are suspected (meningitis, subarachnoid haemorrhage)
What is the acute medical treatment for cluster headaches
- 100% oxyegn via a non-rebreathable mask
2. Sumitriptan (5HT agonist) - subcut
What is the prophylactic medical treatment for cluster headaches
Verapamil
In terms of SOCRATES, how does raised intracranial pressure present (SOAE)
S: bilateral O: gradual, present in the morning A: Vomiting, drowsiness, irritability - seizures - bilateral papilloedema (disc swelling) - focal neurology E: worse when lying down, bending over and coughing
What are causes of raised intracranial pressure
- space occupying lesion (tumour, abscess, haemorrhage)
- hydrocephalus
- trauma
What are investigations for raised intracranial pressure
Urgent MRI (or CT) to determine underlying lesion
In terms of SOCRATES (SOATS), what is the presentation of subarachnoid haemorrhage
S: usually occipital
O: sudden onset, thunderclap headache
A: syncope, nausea, vomiting
Meningeal irritation can lead to meningism (triad: neck stiffness, nausea, headache)
Can present with signs of raised ICP
T: continuous
S: very severe - maximum intensity within minutes
What are the possible causes of subarachnoid haemorrhages
- berry aneurysm at the junction of the circle of Willis
Aneurysms risk increased with Hx or FHx of polycystic kidney disease - 20% idiopathic
- other risk factors include alcohol/smoking/hypertension
What are the investigations for a subarachnoid haemorrhage
- urgent CT scan within 12 hours
- Lumber puncture within 12 hours
- if CT is normal
- Xanthochromia oxyhaemoglobin
What is xanthochromia
Metabolised blood which is yellowish consisting of bilirubin
What is the surgical management for SAH
Refer immediately to neurosurgery
- surgical clipping
- endovascular coil embolization
What is the acute medical treatment for SAH
Cardiopulmonary support AB: maintain airway and breathing C: maintain cerebral perfusion keep well hydrated Maintain blood pressure
What are further supportive measures for SAH
- Reduce high ICP
- osmotic diuretic (mannitol) or hypertonic saline - Prevent cerebral artery vasospasm
- Nimodipine
What can be given to reduce cerebral artery vasospasm
Nimodipine
What causes cerebral artery vasospasm in subarachnoid haemorrhage
When the cerebral artery is bathed in blood from a subarachnoid haemorrhage, it causes vasospasm
In terms of SOCRATES (OAT), how does subdural haemorrhage present
O: gradual onset A: sleepiness, personality changes - diminished verbal and motor response - possible signs of raised ICP T: continuous
What are the risk factors for subdural haemorrhage
- Trauma
- coagulopathy, anti-coagulant use
- advanced age
What is a subdural haemorrhage
collection of blood between the dural and arachnoid coverings of the brain. the build up is gradual and is usually venous