Headaches Flashcards

1
Q

Secondary HA characteristics

A

Intracranial, deep, aching, dull

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2
Q

Sinus HA pain

A

behinad browbone or cheek bones

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3
Q

Cluster HA pain

A

in and around one eye

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4
Q

Tension HA pain

A

band squeezing the head

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5
Q

Migraine HA pain

A

pain, nausea and visual changes, generally on one side

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6
Q

How to treat Primary HA:

1. Tension

A
  • Underlying Cause
  • Heat/massage of neck muscles
  • Minor analgesics; combo products
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7
Q

Migraine

A

Recurrent, paroxysmal attacks of throbbing, pulsating pain, usually unilateral, bombined with autonomic disturbance

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8
Q

Episodic migraines

Time definition

A

0-14/month

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9
Q

Episodic migraine contributing factors to conversion

A
Obesity
msiking
High Caffeine 
Sleep disorder
Opioid use
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10
Q

Migraine without Aura

Dx

A
  1. Recurrent headaches Cat least 5 in lifetime)
  2. Untreated of unsuccessful treatment duration of 4-72 hours
  3. And two of the following
    - Unilateral
    - pulsating
    - moderate to severe
    - aggravation by routine physical activity
  4. associated with one of the following
    - N/v
    - Photophobia or phonophobia
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11
Q

Migraine w/ Aura

A

Aura

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12
Q

Chronic Migraine

A

Tension-like or migraine-like HA >15 days/month for >3 months with migraine features > 15d/mo

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13
Q

Consecutive stages in migraine

A
  1. Cortical spreading depression
  2. Extracrania & intracranial arterial constriction (during aura)
  3. Extracranial & intracranial arterial dilation & decreased electrical activity (During attack)
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14
Q

Vascular theory of migrain

A
1. 5HT release preceding pain phase
5HT2 receptors cause vasoconstriction
Extracranial arterial constriction, regional decrease in flow, may increase prostaglandin production
2. 5HT deficiency during pain phase
Extracranial areterial dilation
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15
Q

Neurogenic theory of migraines

A
  1. Changes in activity of locus coeruleus leading to
  2. Excitation of efferent neurons at trigeminal nuclei
  3. Release of vasoactive substances from trigeminal nerve, activating prostaglandins and degranuation of mast cells
  4. Vasodilation of arteries of dura mater -> plasma protein extravasation
  5. pain and inflammation
  6. sometimes excessive contraction of posterior neck muscles -> contraction HA as well
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16
Q

Menstrual Migraine

A

2 days prior to last day of menses

  • may be due to decrease in estradiol
    2. Aura rare
    3. OC use increases likelyhood
    4. pregnancy d
17
Q

Migraine triggers

A
  1. Hormones
  2. substances
  3. Biorhythyms
  4. Environment
  5. Behavior
18
Q

3-step approach to migraine Tx

A
  1. Non-specific Tx for mild migraine
  2. Specific treatment for moderate-severe pain associated with some impairment of function
  3. Prophylaxis in a migrainer
19
Q
  1. Non-specific Tx for mild migraine
A

NSAIDS: ASA, APAP, Combinations with Caffeine, butalbital

20
Q
  1. Specific Tx for moderate-severe pain associated with some impairment of function (nausea)
A

Combinations of analgesics, triptans, ergots, anti-emetics

21
Q

3 Prophylaxis for who

A

a. recurring
b. frequent HA
c. Contraindicated or failure of acute therapy
d. advers effects with acute Tx
e. Pt preference
f. uncommon conditions