Headaches Flashcards

1
Q

What are important red flag signs associated with headaches and what could they indicate?

A

Fever, photophobia + neck stiffness= meningitis
Sudden severe onset= haemorrhage/stroke
Dizziness= stroke
Visual disturbances= glaucoma
Sudden occipital headache= subarachnoid
Exacerbated by coughing, straining, standing, lying or bending over = RICP
Vomiting= carbon monoxide poisoning or RICP
History of trauma= intracranial haemorrhage
Pregnancy= pre-clampsia

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2
Q

What examination is important to carry out when raised ICP suspected?

A

Fundoscopy

-can identify papilloedema= optic disk swelling which occurs secondary to raised ICP

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3
Q

What are the main different types of headache?

A
Tension 
Secondary 
Sinusitis 
Analgesic
Hormonal 
Association with trigeminal neuralgia
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4
Q

What are the key features of a tension headache?
What causes tension headaches?
How can they be treated?

A
  • Ache in band-like pattern due to association with frontalis, temporalis and occipitalis muscles
  • can be present all the time but doesn’t stop ADL
Causes:
Stress
Depression
Alcohol
Skipping meals
Dehydration 
Medication overuse or withdrawl 
Cervicogenic i.e. cervical osteoporosis spondylsis 
Straining eyes i.e. especially if might need glasses but doesn’t currently use 

TX:

  • analgesia
  • relaxation
  • hot towels
  • physio if cervical osteoporosis
  • TCA= nortriptyline
  • SSRI
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5
Q

How are secondary headaches different to tension headaches?

A

Associate with clear cause:

  • infection
  • obstructive sleep apnoea
  • head injury
  • carbon monoxide poisoning
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6
Q

Why are headaches associated with sinusitis? What feature can help to diagnose these form of headaches?

A

Inflammation in ethmoidal, maxillary, frontal and sphenoidal cause headache and facial pain

Type of headache indicated if there is tenderness over the affected sinuses

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7
Q

Why do hormonal headaches occur? When are they most likely to happen?

A

Low oestrogen produces generic, non-specific tension-like headache

Timing:

  • 2 days before and 3 days of menstrual period
  • menopause
  • pregnancy= worst in first few weeks
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8
Q

What causes trigeminal neuralgia?
How might someone with trigeminal neuralgia present?
What is the recommended treatment?

A

Demyelination of one branch of trigeminal leads to upregulation of sodium channels meaning there is increased amount of Na+ crossing membrane meaning the Em is at point of depolarising all time
THEREFORE= increased sensitivity of the nerve

PX

  • Severe stabbing facial pain which can present as headache
  • triggered by something aggrevating trigeminal distribution i.e. cold wind, eating or touch

TX:

  • carbamazepine
  • oxycarbamazepine
  • another AED
  • MRI= might be presentation but there might be demyelination further up the pathway
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9
Q

What is a cluster headache? How might someone present?

A

Severe unilateral headache around the eye which comes in clusters of attacks for few days and then disappears for 1-2 years

Termed suicidal headache due to severity of the pain 
Unilateral presentation:
-red, swollen, watering eye 
-pupil constriction (miosis )
-eyelid drooping (ptosis)
-nasal discharge 
-face sweating
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10
Q

Why do cluster headaches occur and who is at risk of developing one?

A
Exact cause unknown but tend to occur in 30-50 yo males who smoke 
Triggers:
-alcohol 
-strong smells 
-exercise
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11
Q

How are cluster headaches management acutely and how are recurrences minimised?

A

Acute:

  • triptans
  • high flow 100% oxygen for 15-20 mins

Prophylaxis:

  • verapamil
  • lithium
  • prednisolone to break cycle between clusters
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12
Q

What are the different types of primary headaches rating from most severe pain to least?

A

Trigeminal neuralgia
Trigemial autonomic cephalalgias (TACs)
Migraine
Tension type headache

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13
Q

What are TACs?
What types of headaches fall under this category?
How can these types of headaches be treated?

A

Trigeminal autonomic cephalalgias
-unilateral (side-locked) headaches with pain in the trigeminal distribution
-associated with autonomic activation on same side as headache
I.e. rhinorritis/miosis/ptosis
-agititation

SUNCT
Paroxysmal hemicrania
Cluster headaches
Hemicrania continuas

TX:

  • high flow O2
  • fast acting triptan
  • prednisolone
  • indometacin
  • MRI
  • prophylaxis to prevent being on long term steroids
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14
Q

What is an important differential for TAC/trigmenial neuralgia? How would a patient present?

A

Acute closed-angle glaucoma

Px:

  • intense eye pain
  • N+V
  • red eye
  • headahce
  • tenderness around the eye
  • rings around lights
  • blurred vision
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15
Q

What are secondary causes of headaches?

A

Raised intracranial pressure

Thunderclap headache

Vascular problems

Sinusitis

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16
Q

What are the possible causes of raised ICP?

A

Increased arterial blood pressure

  • malignant hypertension
  • pre-eclampsia
  • hypercapnia i.e. OSA= C02 determines blood flow to brain

Increased CSF pressure

  • obstruction to flow
  • failure to reabsorb i.e. meningitis
  • overproduction

Increased brain pressure
-SOL

Increased venous BP
-venous sinus thrombosis

Meningeal inflammation

17
Q

What are the red flag signs indicative of raised ICP?

A
Papilloedema 
Seizures 
Focal neurological signs 
Visual disturbance 
Headache worse when lying down 
Pregnancy i.e. hypercoagulable state 
N+V
Hx of vasculitis 
Diabetes i.e. at risk of skull base osteomyelitits 
Morning headache or causing to wake up from sleep i.e. indication that ICP is on edge of being raised
18
Q

What are the causes of a thunderclap headache?
How would someone present?
What investigations need to be done?

A
Subarachnoid haemorrhage 
Meningitis 
Migraine= most common cause 
Central venous thrombosis 
Pituitary apoplexy 
Spontaneous intracranial hypotension 
Exertional headache 
Post-coital 

Worst headache of life
Reaches maximum within 1-10 mins

Ix:
-CT head
If CT normal:
-lumbar puncture

19
Q

What would you do it a CT head came back normal in suspected subarachnoid? What are you looking for?

A

Lumbar puncture

Xanthochromia:

  • breakdown product of blood collects in CSF after bleeding into subarachnoid space
  • takes 6-12 hrs to accumulate

Bacterial meningitis:

  • increased protein
  • neutrophilia
  • decreased glucose
  • cloudy appearance

Viral meninigitis

  • not real increase in proteins
  • lymphocytosis
  • normal glucose

TB meningitis

  • increased protein
  • lymphocytosis
  • decreased glucose
20
Q

What vascualar problems can cause a headache?

How would patients present?

A

Carotid artery dissection

  • neck/facial pain
  • Horners syndrome

Vertebral artery dissection

  • neck pain
  • sudden onset vertigo/ataxia

Temporal arteritis

  • vasculititis
  • rare <60
  • raised ESR/CRP
  • jaw claudication
  • general malaise
21
Q

Why would carotid artery dissection present with Horners syndrome?

A

Due to sympathetic trunk running along side CA meaning that dissection causes compression of autonomic fibres

22
Q

How might someone with a medication-overuse headache present?
How are these patients managed?

A

Headache present at least 15 days a month
Developed or worsened since taking analgesia
I.e. occurs with codeine-based analgesia, paracetamol, aspirin and ibuprofen

Management:

  • withdrawal of medication for 3 months
  • counselling that will likely get worse before better because need to reset pain threshold