head and facial disorders Flashcards

1
Q

etiolgy of bell’s palsy

A
  • idiopathic
  • might be associated with HSV
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2
Q

what conditions increase risk for bell’s palsy

A
  • pregnancy
  • diabetes
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3
Q

clinical presentation

  • sudden onset of unilateral facial paralysis
    • inability to close eye
    • facial drooping
    • decreased tearing
    • hyperacusis
    • +/- lost of taste to anterior 2/3 of tongue
A
  • bell’s palsy
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4
Q

bell’s palsy

A
  • acute facial nerve palsy
    • LMN lesion affecting ipsilateral side of face, involves entire side
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5
Q

ramsay hunt syndrome

A
  • cephalic zoster with facial nerve involvement or herpes zoster oticus
    • evaluate for vesicles near external meatus
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6
Q

what differentiates bell’s palsy symtpoms from lyme disease

A
  • lyme disease can affect facial nerve but typically bilateral involvement lasting less than 2 months
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7
Q

how would a central facial palsy (UMN lesion) present

A
  • UMN lesion affects contralateral portion of lower face
    • can still raise eyebrows
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8
Q

how is bell’s palsy diagnosed

A
  • clinical diagnosis
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9
Q

max severity of bell’s palsy should be reached within what time frame

A
  • progressive with max seveity within 3 weeks
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10
Q

when should diagnostic studies be considered in assessment of suspected bell’s palsy

A
  • atypical symptoms
  • no significant improvement in 4 months
  • progression beyong 3 weeks
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11
Q

treatment of bell’s palsy

A
  • prednisone x 5-7 days
  • +/- valacyclovir
  • best results if tx initiated within 3 days of symptoms onset
  • artificial tears applied hourly
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12
Q

duration of symptoms of bell’s palsy

A
  • majority improve within 3 weeks with normal function returning in 3-6 months
    • recurrence rate 7-15%
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13
Q

What is Trigeminal Neuralgia?

A
  • recurrent brief episodes of severe, unilateral pain along 5th cranial nerve
    • pain is electrical “shock-like” sensation
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14
Q

patient population affected in Trigeminal Neuralgia

A
  • W >M
  • > 50 yo
  • family association rare
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15
Q

Trigeminal Neuralgia can be classified into what two groups

A
  • classic: idiopathic or vascular compression of trigeminal nerve root
  • painful trigeminal neuropathy (secondary)
    • causes: multiple sclerosis
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16
Q

in Trigeminal Neuralgia, compression of trigeminal nerve causes

A

demyelination

17
Q

Trigeminal Neuralgia typically affects what part of 5th cranial nerve

A
  • unilateral
  • V2 > V3 > V1
    • V1 may be associated with autonomic symptoms
18
Q

tiggers of Trigeminal Neuralgia

A
  • light touch
  • chewing, talking, shaving, brushing teeth
19
Q

what is pretrigeminal neuralgia

A
  • continuous, dull ache in jaw prior to classic symptoms of Trigeminal Neuralgia
20
Q

how is Trigeminal Neuralgia diagnosed

A
  • at least 3 paroxysmal episodes of unilateral facial attacks: severe intensity, shock-like pain
  • affects trigeminal nerve distribution only
  • no neurological deficits

**may need to r/o other causes: MRI/MRA

21
Q

treatment of Trigeminal Neuralgia

A
  • carbamazepine
    • +/- Baclofen
22
Q

What is Temporal arteritis

A
  • giant cell arteritis
  • chronic vasculitis of large and medium vessels
    • wall thickening and decreased lumen
23
Q

Temporal arteritis can lead to what two serious conditions

A
  • ophthalmic artery occlusion: blindness
  • basilar artery occlusion: brain stem infarct
24
Q

Temporal arteritis commonly affects what patient population

A
  • > 50 yo
  • F>M
25
Q

clinical presentation

  • new headache, unilateral, severe
  • visual disturbances
  • polymyalgia rheumatica
  • jaw claudication
  • fever
A

Temporal arteritis

26
Q

physical exam for Temporal arteritis should include

A
  • vision evaluation
  • temporal artery palpation: will be tender with decreased or absent pulse
    • ausculate for bruits
  • check carotid bruits
27
Q

how is Temporal arteritis diagnosed

A

need 3/5

  • ESR > 50 mm/hr
  • age of onset > 50 y.o.
  • new, localized headache
  • temporal artery tenderness, decreased temporal pulse
  • temporal artery biopsy​
    • 2cm section of artery
      • ​presence of multi-nucleated cells” diagnostic
28
Q

treatment of Temporal arteritis

A
  • glucocorticoid treatment - prednisone
    • high suspicion, start tx prior to confirming dx
29
Q

what is temporomandibular joint dysfunction

A
  • pain associated with TMJ misalignment
    • muscular hypertrophy with malocclusion
    • arthritis
30
Q

what puts someone at a high risk for temporomandibular joint (TMJ) dysfunction

A
  • rheumatoid arthritis
  • ? mood and psychiatric disorder
31
Q

clinical presentation

  • periauricular pain and tenderness of TMJ and muscles of mastication
  • crepitus of jaw with movement
  • decreased jaw ROM
A

temporomandibular joint dysfunction

32
Q

treatment of temporomandibular joint dysfunction

A
  • NSAIDS, tylenol, TCAs