HA oral: Induction (rest), CXR, EKG Flashcards

1
Q

How do you prevent recall?

A

Keep in mind the DOA of your induction agent in relation to the onset of your NMB
Induction dose of propofol will have a clinical effect/DOA = 10 minutes
May need additional induction drug available and administer as needed
Use inhalational/Volatile agent during ventilation
BIS monitoring

Recall is V-BAD
V.BAD
Volatile, Bis, Additional induction druv, DOA of agent in relation to NMB

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2
Q

RSI Definition

A

Rapid sequence intubation/induction (RSI) is an airway management technique that induces immediate unresponsiveness and muscular relaxation and is the fastest and most effective means of controlling the emergency airway.

Used in situations of full stomachs-at risk for aspiration

Adds the Sellick’s Maneuver and removes ventilation from the standard induction sequence

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3
Q

List standard induction steps

A
  1. MSMAIDS
  2. Position pt supine in sniffing position
  3. turn on oxygen flow & pre-oxygenate
  4. pre-induction medication
  5. lidocaine (+/-) induction agent
  6. wait until effect time. Check responsiveness & lash reflex
  7. Test ventilation (close APL valve, make sure can ventilate)
  8. check PNS
  9. paralytic drugs
  10. continue to ventilate until drug takes effect (recheck TOF)
  11. tape eyes
  12. scissor technique, laryngoscopy & intubate (inflate ETT, confirm placement, secure tube)
  13. continue ventilation by bag or switch to vent
  14. begin maintenance (overpressure va)
  15. check vent setting, observe expired VA and titrate down. give maintenance agent and abx
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4
Q

Rapid Sequence Induction (RSI):

A
Identify patient in need of RSI
Pre-operative prophylaxis for aspiration
 Bicitra/Reglan/Omeprazole/Pepcid or Zantac
Anxiolytic
Narcotic (avoid loss of consciousness to early)
Monitors on
Suction on and at head of bed
Supine-sniffing position
Pre-oxygenate (spontaneously breathing)
Sellick’s maneuver= cricoid pressure –gradually increase pressure as patient falls asleep
Induction agent
NO TEST VENTILATION
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5
Q

Extubation Criteria

A

Extubation Criteria – Respiratory Criteria for either fully awake or deeply anesthetized
TV >6mls/kg
VC >10 mls/kg
RR <30 breaths/min (Typically ~10 breaths/min. Make sure vent is turned off, pt must be spontaneously breathing!)
SaO2 >90%
EtCO2 <50 mmHg (COPD & asthmatics will naturally be higher, maybe 55 or 60 EtCO2 appropriate)
Sustained tetanic contraction with PNS

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6
Q

Nearly awake extubation

A

Muscle relaxant fully reversed and confirmed with PNS (if applicable)
All respiratory extubation criteria have been met
Anesthetic medications including volatile agents and infusions turned off
100% FiO2
Oropharynx suctioned
Patient is responsive to commands/purposeful movement
Sustained (5 second) head lift indicates clinically adequate reversal of NMB
Patient can maintain and protect own airway
ETT removed while positive pressure breath is given

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7
Q

deep extubation

A

Muscle relaxant fully reversed and confirmed with PNS (if applicable)
All respiratory extubation criteria have been met
Oropharynx suctioned
100% FiO2
Oral or nasal airway may be inserted
ETT removed while positive pressure breath is given
Volatile agents or infusions turned off
Mask airway maintained while patient spontaneously ventilating
Remain vigilant until patient is responsive and maintaining own airway

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8
Q

Cause & s/s of laryngospasm

A

Prolonged intense glottic closure
May present with high pitched squeak to total absence of sound (ominous sign)
Suprasternal and supraclavicular in-drawing,
increased diaphragmatic excursions
flailing of the lower ribs resembling a “rocking horse”
What muscles are involved in a laryngospasm
lateral cricoarytenoids
thyroarytenoids
cricothyroid
from stimulation of the vagus nerve
Most often seen during induction and emergence

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9
Q

laryngospasm triggers

A
Secretions (vomitus, blood, saliva)
Foreign body 
Pain 
Pelvic or abdominal visceral stimulation
Stimulating glottis in a light plane of anesthesia
Reactive airway disease
Loud noises (pediatrics)

S, F, P, V, L, R

Floppy Vocal Lips Shut Please Respond

Fuck! Vocal Lips Shut, Please Respond

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10
Q

laryngospasm prevention

A

Deep plane of anesthesia reached prior to surgical stimulation
Either fully awake or deeply anesthetized with extubation- not in-between (Avoid stage Two)
Suction oropharynx prior to extubation
Remove ETT with positive pressure breath

Prevent Sudden Airway Death
Positive P, Suction, Avoid st 2, Deepen

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11
Q

Laryngospasm treatment

A

Recognize the event!
Immediate removal of the offending stimulus
Larson maneuver
Retromandibular notch/ laryngospasm notch
condylar process of the mandibular ramus anteriorly, the mastoid process posteriorly, and the external auditory canal superiorly
Pressure for 3-5 seconds and released for 5-10 seconds
Administration OXYGEN (100% FiO2)
continuous positive pressure
Deepen anesthetic (propofol)
Small dose of short acting muscle relaxant: Succinylcholine 20-40 m

Pray LORRD Savior
Positive pressure continuous, Larsons, Oxygen 100%, Recognize, Remove, Deepen, Succs

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12
Q

justifications for CXR

A
pneumonia (confirmation)			immunosuppressed pt
COPD w/acute exacerbation			foreign body
CHF							aspiration pneumonia
blunt trauma					lung tumor	
chest pain						suspected pneumothorax
SOB (severe)					hemoptysis
pulmonary HTN					PE
interstitial lung ds					ICU pt (adm, inv lines, ETT
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13
Q

basic tissue densities**

A

Black = Air
Dark gray = Subcutaneous tissue, Fat
Light gray = Soft tissue (muscles, heart, blood vessels, pus, etc.)
Off white = Bone
Bright white = Metal (pacemakers, surgical clips, bullets, etc.)

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14
Q

What does CXR position affect?

A

Magnification
Organ position
Blood flow
Gravitational pull*

Position Makes Bad Organs Good

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15
Q

causes of pneumothorax

A
Causes: 
Trauma 
Subclavian venous catheter, Liver biopsy 
Spontaneous (result of a bleb rupture)
Metastatic tumors 

EMS - Emergency (trauma, liver puncture), metastatic tumor, Spontaneous bleb & SVC

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16
Q

tension pneumothorax definition

A

Tension pneumothorax = mediastinal shift occurs or there is depression of the hemidiaphragm with displacement of the heart and trachea to the unaffected side

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17
Q

pneumothorax on CXR

A

UPRIGHT is the best position
Where is the first place to look for pneumothorax? Right and left upper hemithoraces

Supine: Deep Sulcus Sign

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18
Q

pleural effusion : Definition, Looking for? Causes? Best CXR position

A

Definition: collection of fluid between the visceral and parietal pleura (~100 mL to detect on upright CXR)

Look for:

  • Blunting costophrenic angles
  • inc basilar density (whiteness)
  • loss of nL lung hemidiaphragm

Causes:
Malignancies
Pancreatitis (LEFT-sided pleural effusion)
Cirrhosis (RIGHT-sided pleural effusion)
CHF (BILATERAL pleural effusion, usually associated w/cardiomegaly)
Pneumonias

[PCCCC- Pancreatitis, CA(malignancies), Cirrhosis, CHF, Consolidation (PNA)]

Best XR position: Upright

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19
Q

mediastinal shift in tension pneumonia, atelectasis, airway obstruction?

A

-Tension pneumothorax: the mediastinum is shifted toward the unaffected side (PUSHING IT)

-Atelectasis: collapse of entire lung segment might result in severe volume loss.
Mediastinal shift toward the affected side (PULL TOWARDS, like suction)

-Airway obstruction: mediastinal shift toward the unaffected side (can’t get out, builds up, push it)

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20
Q

male vs female CXR

A

Nipple shadows  common in men and women
compare both sides
Overlying breast tissue  accentuate pulmonary vasculature
(careful, not to dx as infiltrate/pneumonia)
can tape metallic BB or other metal object and reshoot film

21
Q

silhouette sign on CXR

A

It helps to determine the location of an abnormality in relation to normal structures
Loss of a normal border occurs if an abnormality is contiguous with that structure

RML vs RLL (pneumonias, masses)

  • Loss of right heart border indicates that the infiltrate is in the RML (right middle lobe)
  • Loss of right hemidiaphragm indicates that the infiltrate is in the RLL (right lower lobe)
  • Loss of left heart border indicates that the infiltrate is in the lingula of LUL (left upper lobe)
  • Loss of left hemidiaphragm indicates that the infiltrate is in the LLL (left lower lobe)
22
Q

When would you perform a CXR in a suspected Aspiration PNA?

A

Aspiration: the inhalation of gastric contents (following seizure, cardiac resuscitation, anesthesia-related complication)

CXR usually performed immediately after incidence

Follow-up should be performed within 12 hrs.
May take several hours for the gastric contents to react with the lung to cause fluid exudate and an alveolar infiltrate

23
Q

What masses are found in the anterior mediastinum ?

A

Anterior Mediastinum (4 Ts): lesion filling in the space behind the top of the sternum and the ascending aorta

Thymoma
Thyroid lesions
Teratoma (germ cell tumors, most commonly seen in males)
T cell lymphoma

(4Ts)

24
Q

What is found in the middle mediastinum ?

A

Middle Mediastinum (any lesions associated w/the aorta should be considered an aneurysm until proven otherwise)

Thoracic aortic aneurysm
Neoplasms
Adenopathy
Diaphragmatic hernias

(another T-AND)

25
Q

What is found in the posterior mediastinum?

A

Posterior Mediastinum (lateral view CXR projecting over the spine and are also paraspinous on the frontal CXR)

Neurogenic (90%)
Neuroblastomas (children)
Neurofibromas (adults) Schwannomas (adults)
Ganglioneuromas (adults)

(Neuro shit)

26
Q

systematic approach for CXR interpretation?

A

Who (correct patient)
What (film orientation)  AP, PA, supine, upright
When (date)
Why (reason for X-ray)  history and PE are extremely important
Exposure?

Airway.
Bones.
Cardiac. Diaphragm/mediastinum. Everything else

27
Q

Conduction system

A

Sinoatrial node: 60-100 bpm
Atrioventricular node: delays conduction for ventricular filing; initiates impulse 40-60 bpm
Bundle of His: directs impulse to left/right bundle branches
Purkinje fibers: reaches into myocardium to stimulate ventricular depolarization/ contraction. Initiates impulse 20-40 bpm

28
Q

EKG basics and vectors

A

Electrocardiogram (EKG) is the graphic display of the flow of electrical activity (action potentials) generated by myocytes.
Action potentials travel in all directions but the average (mean) current (vector) is measured by the EKG.
There are two main vectors
Vector of depolarization = QRS complex
Vector of repolarization = T wave

29
Q

3 lead EKG placement and poles

A

Electrodes placed on the skin are used to identify these poles.
Right Arm is always negative
Left Leg is always positive
Left Arm is positive in lead I and negative in lead III

30
Q

standard limb leads

A

Lead I: negative electrode on right upper limb to the positive electrode on left upper limb
Corresponds to a view of the lateral wall of the heart and areas supplied by the circumflex artery
Lead II: negative electrode on right upper limb to positive electrode on left lower limb
Corresponds to a view of the inferior wall of the heart and the areas supplied by the right coronary artery
Lead III: negative electrode on left upper limb to positive electrode on left lower limb
Corresponds to a view of the inferior wall of the heart and the areas supplied by the right coronary artery

31
Q

augmented limb leads

A

aVR: Right arm electrode is positive, and the left arm and left leg electrodes are channeled together to form a common reference point that has a negative charge
aVL: Left arm electrode is positive, and the right arm and left leg electrodes are channeled together to form a common reference point that has a negative charge
Corresponds to a view of the lateral wall and areas supplied by the circumflex artery
aVF: Left foot electrode is positive, and the right arm and left arm electrodes are channeled together to form a common reference point that has a negative charge
Corresponds to a view of the inferior wall of the heart and areas supplied by the right coronary artery

32
Q

precordial leads

A

The positive poles are on the anterior and lateral chest and the negative poles are on the opposite side of the positive pole
V1: positive electrode placed directly over the right atrium
Corresponds to the septal wall and areas supplied by the LAD (left anterior descending artery)
V2: positive electrode placed just anterior to the AV node
Corresponds to the septal wall and areas supplied by the LAD
V3 and V4: positive electrode placed over the ventricular septum
Corresponds to the anterior wall of the heart and areas supplied by the LAD
V5 and V6: positive electrode placed over the lateral surface of the left ventricle
Corresponds to the lateral wall of the heart and areas supplied by the circumflex artery

33
Q

12 lead EKG view

A

Lead V1, V2 view the septal wall and LAD
Lead V3, V4 view the anterior wall and LAD
Lead I, aVL, V5, V6 view the lateral wall and circumflex.
Lead II & III and aVF view the inferior wall of the heart supplied by RCA

34
Q

5 lead EKG

A

Takes a standard three lead system (right limb electrode, left limb electrode and left leg electrode) and adds a right leg and chest electrode.
By adding the right leg lead electrode any of the six limb leads can be viewed.
The chest electrode (V1) can be moved to any of the precordial V positions to obtain all six precordial views

35
Q

vector of depolarization= QRS complex

A

Heart depolarizes from base to apex and endocardium to epicardium
Myocytes go from internally negative to internally positive  produces a positive electrical current
Positive deflection on the EKG occurs when vector of depolarization travels towards a positive electrode
Negative deflection on the EKG occurs when vector of depolarization travels away from a positive electrode
Biphasic deflection on the EKG occurs when vector depolarization travels perpendicular to positive electrode

36
Q

vector of repolarization= Twave

A

Heart repolarizes from apex to base and epicardium to endocardium
Myocytes go from internally positive to internally negative  produces a negative electrical current
Positive deflection occurs when the wave travels away from a positive electrode

37
Q

normal EKG measurement

A

PR interval measurement (Normal: 0.12 - 0.2)
QRS interval measurement (Normal: <0.12)
QT interval measurement (Normal: <0.45 men, <0.47 women

38
Q

left axis deviation

A
Left axis deviation has a positive R wave deflection in lead I but negative R wave deflection in aVF
Leaving each other = Left axis deviation
Causes of left axis deviation (conditions that make the left side of the heart work harder or hypertrophy) 
Chronic HTN
LBBB
Aortic stenosis
Aortic insufficiency
Mitral regurgitation
39
Q

right axis deviation

A

Right axis deviation has a negative R wave deflection in lead I but positive R wave deflection in aVF
Reaching each other = Right axis deviation
Extreme right axis deviation has negative R wave deflections in both lead I and aVF
2 thumbs down = BAD
Causes of right axis deviation (conditions that make the right side of the heart work harder or hypertrophy) – reaching for each other; or extreme; both down
COPD
Acute bronchospasm
Cor pulmonale
Pulmonary hypertension
Pulmonary embolism

40
Q

Myocardial ischemia wall infarcts

A

Inferior Wall Ischemia (Right coronary artery & Posterior interventricular branch): II, III, and aVF
Lateral Wall Ischemia (Circumflex branch of the left coronary artery): I, aVL, V5, V6
Septal Ischemia (Left anterior descending artery): V1-V2
Anterior Wall Ischemia (Left anterior descending artery): V3 -V4
Anterior-septal (Left anterior descending artery): V1-V4

41
Q

preop assessment for cancer and general adverse effects

A

1) Surgery, (2) Chemotherapy, (3) Radiation therapy

Toxicities/Adverse Effects have the potential to affect nearly every organ system
Bone marrow suppression
CV toxicity
Pulmonary toxicity
Central and peripheral nervous system damage
Nutritional status (e.g., alopecia, N/V/D)

42
Q

systems affected by radiation and clinical effects

A

skin-acute-erythema, rash, hair loss; chronic-fibrosis, sclerosis, telangletasias
GI- acute-malnutrition, mucositis, n/v; chronic- adhesions, fistulas, strictures
cardiac- conduction defects, pericardial effusion, pericardial fibrosis, pericarditis
respiratory- airway fibrosis, pul. fibrosis, pneumonitis, tracheal stenosis
renal- acute-glomerulonephritis; chronic-glomerulosclerosis
hepatic- sinusoidal obstruction syndrome
endocrine- hypothyroidism, panhypopituitarism
hematologic- acute-bone marrow suppression; chronic- coagulation necrosis

43
Q

preop assessment for cancer patient

A

HISTORY & CO-MORBITITIES?
Malignancy in the head or neck
Airway exam and possible need for tracheostomy
Recurrent laryngeal nerve damage (S/S: hoarseness)
Mediastinal masses obstructing great vessels
Trachea compression manifestations: Dyspnea, dysphagia, stridor, wheezing, coughing  recumbent position
SVC compression manifestations: Compression of SVC  JVD & facial, chest, neck, upper extremity edema
Preop testing:
Ultrasound, CT, MRI
Flow volume loops to see how much their expiratory volume has been impaired
EKG, ECHO
Anesthetic concerns:
Radiation to neck can lead to tracheal stenosis  smaller airway may have trouble getting ETT in
If someone has a mass obstructing the trachea and you can’t lay them back, how are you going to get them to sleep?
Spontaneous awake and ENT at bedside
“Radical neck procedures” may need to be converted to a trach at the end of the case

44
Q

when is expiration CXR useful?

A

small pneumothorax: expiration will make the lung smaller and denser, and at the same time will relatively make the pneumothorax appear larger

lodged foreign body: “ball-valve phenomenon” – air can move past the object during inspiration, but during expiration (the bronchus gets smaller) and air can not exit around the obj. As a result, the expiration image will show air trapping in the affected lung and a mediastinal shift will occur toward the unaffected side

45
Q

compare AP vs PA view

A
AP (supine)
Xray beam entering anterior thorax, exit posterior
pt position: supine
detector position: on back
heart size magnified
diaphragm (cephalad)
PA view (upright)
x-ray beam enter posterior thorax, exit anterior
position: upright
detector position: front 
heart size (true to size)
diaphragm (caudad)
46
Q

Downfalls of a supine CXR

A
  • limits full inspiration
  • cephalic push
  • small pleural effusions layer in posterior pleural space and can easily be misssed
  • be careful when interpreting
47
Q

compare and contrast overexposure vs underexposure

A

overexposure:
Image is dark
Easy to see: Thoracic spine, clavicles, behind the heart, NG & ET tube placement
Cannot see: pulmonary vessels in the periphery, small nodules, or fine structures
(bones and hard stuff)

Underexposure: Image is white
Easy to see: pulmonary vasculature (don’t mistake for infiltrate)
Cannot see: behind the heart, spinal anatomy, or behind hemidiaphragms
(soft tissue and vasculature)

48
Q

Considerations when doing an upright CXR

A
  • inspiration is greater
  • domes of hemidiaphragm at posterior T10 level
  • Hypoinflation - dome at 7th rib