HA Flashcards
primary causes of migraine
i. HA & associated features are the disorder
ii. Tension 69%
iii. Migraine 16%
iv. Cluster 0.1%
secondary causes of migraine in order
i. HA caused by other disorder
ii. Infection 63%
iii. Trauma 4%
iv. Vascular Disorder 1%
v. SAH <1%
vi. Tumor 0.1%
what types of infections cause headaches
sinus infection- pressure on sinuses, tops of the teeth
meningitis- nuchal rigidity, fever, rash, body aches
dental abscess
headaches in the population M:F
b. 5:9 M:F
cluster more common in males
migraines more common in females
TTH presentation
time
and location
pain is constant an daily an worse late in the day
bilateral
vise-like band
vi. Emanates from neck and shoulders
“Featureless” HA
TTH
NO associated sx like n/v
ix. S.C.A.L.P (PE)
- skin
- Connective Tissue
- Aponeurosis í connected to the skin
- Loose connective tissue
- Pericranium íoutside of the bony cranium
- Occipital nerves are superficial and attached to the capitis muscles
triggers for TTH
- Stress
- Depression/Anxiety
- Posture
- Jaw clenching when sleeping
- Female - get more tension HA’s than males do
- Middle age
how to differentiate TTH in a person with anxiety or depression
pts with mental health issues wake up with HA and they get worse throughout the day
chronic TTH
at least 10 episodes month at least 30 minutes location bilateral pressing or tigehtning no n/v no photophobia
best way to treat a HA
at the onset
on’t treat your HA like an earthquake
OTC analgesic snd drink a ton of water
less likely to progress and reoccur
caffeine can be helpful
caffeine helps HA becasue
is a vasoconstrictor
what to avoid with TTH
Opiates
amitriptyline (TCA)
works really good as a low dose for TTH
usually want to take it at night because it can cause drowsiness
EMG
biofeedback therapy that can be used to treat TTH
ALT to medication for TTH
acupuncture
maybe massage
TENS tx
Transcutaneous Electrical Nerve Stimulation
causes nerve stimulation of mm and can reset nerves and help mm to stay more relaxed in
TTH of in pts with chronic back pain
onset of migraines
6-8 in boys
after menses in women
Fhx in what % of pts with migraines
70%
Pathophysiology of migraines
vasonstriction followed by vasodilation theory kind of debunked
looks like more of a nuero vascular issue with spreading that leads to trigeminal vascular system triggering and cortical spreading depression that depolarizes the cortex –> leads to blood vessel dilation
triggers inflammatory mediators to be released
triggers for migraine
- FH - predisposing factor 70%
- Stress 80%
- Lack/excess of sleep>50%
- Missed meals 57%
- Foods (chocolate, EtOH) 27-38%
- Hydration status definitely a trigger for migraine HA
- Light 38%
- Noise, glare
- Menstruation (catamennial migraines) 65%
a. Raises a whole new tx option for hormones
b. Progesterone - related to HA
i. Estrogen was added to the progesterone only BC b/c HA was a huge SE
Unilateral
Throbbing
Episodic with early onset
migraine