Gingivitis Flashcards

1
Q

OVERVIEW

review
describe
diagnose
correlate

A

REVIEW: anatomy of periodontium

DESCRIBE: biofilm induced or non-biofilm-induced gingivitis

DIAGNOSE: type and severity of gingivitis

CORRELATE: histological and clinical signs of biofilm induced gingivitis

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2
Q

PERIODONTIUM

the _______ system of tissues that ________ the teeth and attaches them to the _____

what are the four components

A

the FUNCTIONAL system of tissues that SURROUND the teeth and attaches them to the BONE

Gingiva, cementum, PDL, and alveolar bone

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3
Q

GINGIVA

provides a ________ around the cervical portion of the tooth and covers the __________

holds tissues against the tooth during __________

composed of thin outer layer of _________ and an underlying core of ___________

A

provides a TISSUE SEAL around the cervical portion of the tooth and covers the ALVEOLAR PROCESSES

holds tissues against the tooth during MASTICATION

composed of thin outer layer of EPITHELIUM and an underlying core of CONNECTIVE TISSUE

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4
Q

FREE GINGIVA

  • non __________ gingiva: fits snuggly around the _____ but it is not ________
  • free gingival tissue: meets the tooth in a ____, rounded edge called the _______, should be coronal to the ____
  • gingival margin follows the contours of the _____ (wavy outline)
  • the FGM can be pulled away from the tooth with a ____ > forms a soft tissue wall: the ____________
A
  • non ATTACHED gingiva: fits snuggly around the TOOTH but it is not ATTACHED
  • free gingival tissue: meets the tooth in a THIN, rounded edge called the GINGIVAL MARGIN, should be coronal to the CEJ
  • gingival margin follows the contours of the TOOTH (wavy outline)
  • the FGM can be pulled away from the tooth with a PROBE > forms a soft tissue wall: the GINGIVAL SULCUS
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5
Q

GINGIVAL SULCUS

__ shaped, shallow space around the tooth

normal sulcus =

base of the sulcus is formed by the __________

A

U-shaped, SHALLOW space around the tooth

normal sulcus = 1-3mm

the base of the sulcus is formed by the JUNCTIONAL EPITHELIUM

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5
Q

GINGIVAL BOUNDARIES

coronal edge

apical edge

which mucosa is darker

what demarcates the attached gingiva from the alveolar mucosa

A

coronal edge - gingival margin

apical edge - alveolar mucosa

which mucosa is darker - alveolar (BVs)

what demarcates the attached gingiva from the alveolar mucosa - mucogingival junction

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6
Q

ATTACHED GINGIVA

prevents _______ from being pulled away

located between ______ and ______

widest in _______; narrowest in _____

width is not measured in the ______ because it’s difficult to distinguish between ________ mucosa

the color is ____ or _______, may be _________

A

prevents FREE GINGIVA from being pulled away

located between FREE GINGIVA and ALVEOLAR MUCOSA

widest in INCISOR & MOLAR REGIONS; narrowest in PREMOLAR REGIONS

width is not measured in the PALATE because it’s difficult to distinguish between PALATAL mucosa

the color is PALE or CORAL PINK, may be PIGMENTED

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7
Q

What would be “inadequate attached gingiva”

why is it bad?

A

2 mm with bleeding or 1 mm with or without bleeding

makes teeth more mobile, indicates bone loss

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8
Q

equation for amount of attached gingiva

A

(FGM-MGJ) - PD = amount of attached gingiva

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9
Q

INTERDENTAL PAPILLA/GINGIVA

part of gingiva that fills the area between two _________ teeth

________ to the ________

prevents _________

how many papillae does each tooth have

what is the COL; when is it absent

A

part of the gingiva that fills the area between two ADJACENT teeth

APICAL to the CONTACT AREA

prevents FOOD IMPACTION

teeth consist of TWO interdental papillae: FACIAL AND LINGUAL

col: valley-like depression in the interdental gingiva that lies directly apical to the contact area; absent if teeth are missing or recession is present

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10
Q

PERIODONTAL LIGAMENT (PDL)

____ connective tissue that covers the _____ and attaches it to the _____ of tooth socket

Composed of _____ bundles

Fibers attach to the _______ on one side and the ________ of the tooth socket on the other side

A

SOFT connective tissue that covers the ROOT and attaches it to the BONE of the tooth socket

Composed of FIBER bundles

Fibers attach to the ROOT CEMENTUM on one side and the ALVEOLAR BONE of the tooth socket on the other side

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11
Q

what are the 5 functions of the PDL

A
  1. suspends and maintains the tooth in the socket
  2. provides pressure/pain/sensory to the tooth
  3. provides nutrients to the cementum and bone
  4. builds and maintains cementum and alveolar bone
  5. remodels alveolar bone in response to pressure
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12
Q

CEMENTUM

thin layer of ______, ________ tissue that covers the surface of the ____

what color is it

overlies and protects _____

“bone-like” but more resistant to _________ than bone

does it have a blood/nutrient supply?

A

THIN layer of HARD, MINERALIZED tissue that covers the surface of the ROOT

LIGHT YELLOW

overlies and protects DENTIN

“bone-like” but more resistant to RESORPTION than bone

DOES NOT HAVE ITS OWN BLOOD/NUTRIENT SUPPLY

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13
Q

ALVEOLAR BONE

surrounds and supports ______ in the upper and lower jaws

AKA alveolar bone ______

existence of alveolar bone is dependent on ________

extractions lead to ________

A

surrounds and supports ROOTS in the upper and lower jaws

AKA alveolar bone PROPER

existence of alveolar bone is dependent on the PRESENCE OF TEETH

extractions lead to RESORPTION

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14
Q

what are the 4 functions of alveolar bone

A
  1. protection - sockets for teeth
  2. attachment - PDL fiber
  3. support - tooth roots
  4. shock absorption - forces placed on the tooth
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15
Q

CLINICAL GINGIVAL HEALTH ON AN INTACT PERIODONTIUM

BOP
PD
probing attachment loss
RBL

A

BOP - less than 10%

PD - 1-3 mm

probing/clinical attachment loss - none

RBL - none

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16
Q

CLINICAL GINGIVAL HEALTH ON A REDUCED PERIODONTIUM (stable periodontitis patient)

BOP
PD
probing attachment loss
RBL

A

BOP - less than 10%

PD - 4mm or less (no site over 4 mm with bleeding)

probing attachment loss - yes

RBL - yes

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16
Q

CLINICAL GINGIVAL HEALTH ON A REDUCED PERIODONTIUM (non-periodontitis patient/recession/bad brushing habit/crown lengthening)

BOP
PD
probing attachment loss
RBL

A

BOP - less than 10% bleeding

PD - 1-3mm

probing attachment loss - yes

RBL - possible

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17
Q

COLOR IN HEALTH

  • uniformly _____ (lighter hair/skin = usually lighter gingiva)(darker hair/complexion = darker shade)
  • pink gingiva is easily distinguished from ________
  • tissue may be _______
A
  • uniformly PINK (lighter hair/skin = usually lighter gingiva)(darker hair/complexion = darker shade)
  • pink gingiva is easily distinguished from DARKER ALVEOLAR MUCOSA
  • tissue may be PIGMENTED
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18
Q

3 basic states of periodontal tissues

A
  1. health (fills embrasures, no inflammation, coral pink)
  2. gingivitis (reversible, inflammation, BOP, rolled, edema)
  3. periodontitis (mobility, inflammation, loss of attachment, irreversible damage, working toward stability)
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19
Q

t/f: periodontal disease and periodontitis are the same thing

A

FALSE periodontitis is a TYPE of periodontal disease

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20
Q

periodontal disease is a _________ of the periodontium

what are the 2 types of periodontal disease

A

periodontal disease is a BACTERIAL INFECTION of the periodontium

gingivitis and periodontitis

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21
Q

CLINICAL(3) vs HISTOLOGICAL(4) HEALTH

A

CLINICAL
- pink
- firm
- less than 10% bleeding

HISTOLOGICAL
- Junctional epithelium coronal to CEJ
- supragingival fibers intact
- alveolar bone intact
- periodontal ligament intact

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22
Q

Pristine gingival health VS clinically health VS incipient gingivitis

A

PRISTINE GINGIVAL HEALTH
- no BOP or attachment loss
- 1-3mm PD
- no clinical signs of inflammation

CLINICALLY HEALTHY
- absence/low levels of inflammation

INCIPIENT GINGIVITIS
- less than 10% BOP
- variant of gingival health
- if left untreated, can rapidly progress to gingivitis

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23
Q

clinical (3) VS histological (4)

dental biofilm-induced gingivitis

A

CLINICAL
- red
- swollen
- bleeding likely (more than 10% BOP)

HISTOLOGICAL
- JE is at the CEJ
- supragingival fiber destruction
- alveolar bone intact
- periodontal ligament

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24
Q

prevalence of gingivitis

____ in the population
____ in ages 65+
____ in men
_____ in women

A

47.2% of the population

70.1% in ages 65+

56.4% in men

38.4% in women

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25
Q

INFLAMMATORY PROCESS OF PATHOGENESIS (7 steps)

A
  1. invasion of pathogens, injury or irritant to tissues produces transient vasoconstriction of BVs
  2. biochemical mediators released causing vascular wall changes which increases BV permeability and flow
  3. delivers defense supplies
  4. fluid leakage - inflammatory exudate
  5. defense cells migrate
  6. plasma cells and leukocytes leak from capillaries into the tissues
  7. disease signs followed by tissue destruction and healing
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26
Q

VASOCONSTRICTION, VASODILATION, AND EXUDATION

________ and ______ chain of events

Injured or dying cell releases ______ breakdown products such as _______

Cause release of enzymes found along ______ cells

Enzymes are activated and cause break in mast cell wall and release of _______

Histamine granules escape into tissue and cause ________

A

INTERDEPENDANT and INTERRELATED chain of events

Injured or dying cell releases CHRONIC breakdown products such as POLYPEPTIDES

Cause release of enzymes found along MAST cells

Enzymes are activated and cause break in mast cell wall and release of HISTAMINE

Histamine granules escape into tissue and cause DAMAGE

27
Q

AS THE INFLAMMATORY PROCESS CONTINUES

_______ in vessel permeability and _______

carry ______ supplies

_______ of BV with ________ or pooling of blood in the capillaries

_______

________ causes a break in endothelial cells lining BV’s

A

INCREASE in vessel permeability and BLOOD FLOW

carry DEFENSE supplies

VASODILATION of BV with HYPERMIA or pooling of blood in the capillaries

ENGORGED

HISTAMINE causes a break in endothelial cells lining BV’s

28
Q

What’s really happening?

________ form

Blood enters the ____ carrying: ____, _____ cells, ______

Inflammatory _____ enters the tissue

A

MICROULCERATIONS form

Blood enters the SULCUS carrying: PMNs, PLASMA cells, AND MACROPHAGES

Inflammatory EXUDATE enters the tissue

29
Q

WHAT ARE PMNS

___ line of _____

engulf and destroy ______

type of ______

Most _______ circulating immune cell

Plays a key role in _______

A

FIRST line of DEFENSE

engulf and destroy PATHOGENS

type of LEUKOCYTE

Most ABUNDANT circulating immune cell

Plays a key role in INFLAMMATION

30
Q

gingival inflammation

___, ______, and ______

repaired by _______

A

redness, swelling, bleeding

repaired by firbroblasts

31
Q

tissue invasion by bacteria results in _____ to the body

inflammatory response: increased blood flow delivers defense cells:

  • _____ and _______ leak from blood vessel into the tissue
  • _______ fight invading bacteria
  • some tissue destruction occurs surrounding the infection site, this is a ________ of the inflammatory repsone
A

tissue invasion by bacteria results in INJURY to the body

inflammatory response: increased blood flow delivers defense cells:

  • LEUKOCYTES and PLASMA PROTEINS leak from blood vessel into the tissue
  • LEUKOCYTES fight invading bacteria
  • some tissue destruction occurs surrounding the infection site, this is a SIDE EFFECT of the inflammatory repsone
32
Q

ACUTE INFLAMMATION
- bacterial infection of ____ or less
- ____, heat, ______, swelling, loss of ______

CHRONIC INFLMMATION
- no ______
- ______ onslaught of bacteria

chronic inflammation causes an ______ host inflammatory repsonse

A

ACUTE INFLAMMATION
- bacterial infection of 2 WEEKS or less
- REDNESS, heat, PAIN, swelling, loss of FUNCTION

CHRONIC INFLMMATION
- no SYMPTOMS
- PERSISTANT onslaught of bacteria (longer than 2 weeks)

chronic inflammation causes an EXAGGERATED host inflammatory repsonse

33
Q

GINGIVAL INFLAMMATION

Biofilm stimulates _______

Inflammatory response results in _________

Changes involve ____ and ____ gingiva

_______ papilla also impacted

Patients are generally _____ as there is no _____ associated

A

Biofilm stimulates IMMUNE REPOSNE

Inflammatory response results in CLINICAL CHANGES

Changes involve FREE and ATTACHED gingiva

INTERDENAL papilla also impacted

Patients are generally UNAWARE as there is no PAIN associated

34
Q

what is considered the key risk factor for the onset of periodontitis

A

gingival inflammation in reponse to bacterial plaque accumulation

we must remove the irritant, control of gingival inflammation is essential for the primary prevention of periodontitis

35
Q

what part of the periodontium does gingivitis affect first?

what is the number one risk factor of PD

A

gingivitis affect the interdental papilla and the margins first (bulbous)

individual host reposne to biofilm

36
Q

slight gingivitis can be described as:

moderate can be described as

severe can be described as

A
  • less than 30% BOP, confined to the margins/ acute range, bulbous papilla
  • more than 30% BOP, diffuse redness, bulbous, rolled margins
  • can’t see the MGJ, red/bulbous
37
Q

if inflammation AND recession is present on one tooth, how do we record this

if bulbous and blunted what do we do

A
  • record recession on the perio chart (+1)
  • describe the papilla as bulbous on the gingival descriptors

we would mark the blunted because that is attachment loss

38
Q

does color variation always mean disease

A

NO, color change can occur for reasons other than inflammation. injury such as brushing too hard can cause a color change. Intraoral tattoos.

39
Q

CONTOURS OF HELATHY GINGIVA

smooth, even _____ margin

at or slightly _____ to the ___

_____ margin slightly coronal to the CEJ

pointed papilla that fills the _____

FGM is ____

A

smooth, even SCALLOPED margin

at or slightly CORONAL to the CEJ

TAPERED margin slightly coronal to the CEJ

pointed papilla that fills the SPACE

FGM is 000

40
Q

tissue size in gingivitis

Increased tissue ____ enlarges _______ and _______ gingival tissue

Enlargement can be _____ or involve the ____ mouth

A

Increased tissue FLUID enlarges MARGINAL and INTERPROXIMAL gingival tissue

Enlargement can be LOCALIZED or involve the WHOLE mouth

41
Q

stippling

____ of health

Disappears with ______, giving tissue a more ____/____ appearance (Stretched plastic wrap)

____ Gingiva is still ____

Tissue is not ________ with air

A

SIGN of health

Disappears with INFLAMMATION, giving tissue a more SMOOTH/SHINY appearance (Stretched plastic wrap)

ATTACHED Gingiva is still BOUND

Tissue is not RETRACTABLE with air (FLACCID)

42
Q

healthy vs gingivitis texture

A

healthy:
- firm
- maybe stippled (varys)

gingivitis
- spongy
- stipping disappears (varies)

43
Q

what is each papilla assciated with

A

bulbous: gingivitis
blunted and cratered: periodontitis or trauma

44
Q

does bleeding = disease

A

not always, less than 10% is healthy, but still talk with the patient

45
Q

BLEEDING

  • sulcus lining becomes ______
  • BVs ______
  • direct relation between the ________ and _______
  • absence of bleeding indicates _____
  • ______ means active disease
  • _______ technique can bleeding
  • no ________ despite bleeding (gingivitis)
A
  • sulcus lining becomes ULCERATED
  • BVs ENGORGE
  • direct relation between the DEGREE OF INFLAMMATION and AMOUNT OF BLOOD
  • absence of bleeding indicates HEALTH
  • TYPICALLY means active disease
  • PROBING technique can bleeding
  • no ATTACHMENT LOSS despite bleeding (gingivitis)
46
Q

PSUEDOPOCKETS

  • no _______ of the ___, remains _____ to the CEJ
  • no permanent destruction of the ____ or _____
A
  • no APICAL MIGRATION of the JE, remains CORONAL to the CEJ
  • no permanent destruction of the PDL FIBERS or ALVEOLAR BONE
47
Q

PERIODONTAL POCKET

a _______ deepening of the gingival sulcus as a result of:
-__________ of the JE
-Destruction of ___ fibers and _____

The presence of a periodontal pocket does ___ indicate necessarily that there is ____ disease at the site.

The majority of periodontal pockets in most adult patients with periodontitis are ______ sites.

The pocket is an indicator of ____ destruction from periodontitis.

A

a PATHOLOGIC deepening of the gingival sulcus as a result of:
- APICAL MIGRATION of the JE
- Destruction of PDL fibers and ALVEOLAR BONE

The presence of a periodontal pocket does NOT indicate necessarily that there is ACTIVE disease at the site.

The majority of periodontal pockets in most adult patients with periodontitis are INACTIVE sites.

The pocket is an indicator of PAST destruction from periodontitis.

48
Q

in gingivitis the JE stays ______ while the ________ moves coronally

A

in gingivitis the JE stays STILL while the GINGIVAL MARGIN moves coronally

49
Q

CHRONIC GINGIVITIS: GINGIVAL FLUID

Inflammatory product secreted from ____ that exhibits pathologically-induced increased _______

____ sign of inflammation

Appears prior to ________

Provides _____ for the growth of subgingival plaque biofilm causing disease to increase

Chronic: continues to feed and could progress to ________

A

Inflammatory product secreted from the SULCUS that exhibits pathologically-induced increased PERMEABILITY

FIRST sign of inflammation

Appears prior to BLEEDING

Provides NUTRIENTS for the growth of subgingival plaque biofilm causing disease to increase

Chronic: continues to feed and could progress to PERIODONTITIS

50
Q

BIOFILM INDUCED GINGIVITIS
- Gingival diseases with ______ factors
- Most ______ disease
- ____ relationship between inflammation and _____

NONBIOFILM INDUCED GINGIVITIS
- ____, ____, ___, induced gingival diseases (different bacteria from gingivitis, not ____ bacteria)
- ____ causes
- _____
- Patient _____

A

BIOFILM INDUCED GINGIVITIS
- Gingival diseases with MODIFYING factors
- Most COMMON disease
- LINEAR relationship between inflammation and BIOFILM

NONBIOFILM INDUCED GINGIVITIS
- BACTERIAL, VIRAL, FUNGAL, induced gingival diseases (different bacteria from gingivitis, not ORAL bacteria)
- SYSTEMIC causes
- MALNUTRITION
- Patient SYMPTOMS

51
Q

COMMON CHARACTERISTICS OF BOTH GINGIVITIS TYPES (4)

A
  • Signs of inflammation confined to the gingiva
  • No attachment loss bone loss associated with the inflammation
  • Inflammation is REVERSIBLE with biofilm removal
  • May progress to PERIODONTITIS if left untreated
52
Q

SUBTYPE: BIOFILM-INDUCED GINGIVITIS ON A REDUCED PERIODONTIUM (stable perio patient with over 10% bleeding)

  • BOP must be on _____ sites
  • when the gingivitis occurs, active ______ sites are NOT present
  • Preexisting _______ from a previous history of periodontitis
  • ______ at the gingival margin
  • very ____
A
  • BOP must be on NON PERIO sites
  • when the gingivitis occurs, active PERIODONTITIS sites are NOT present
  • Preexisting BONE AND ATTACHMENT LOSS from a previous history of periodontitis
  • PLAQUE at the gingival margin
  • very RARE
53
Q

LOCAL COMPLICATING FACTORS

  • factors that ____ or ______ localized areas of gingivitis
  • the most common local complicating factor for gingivitis would be ______
  • list 4 examples
A
  • factors that MODIFY or PREDISPOSE localized areas of gingivitis
  • the most common local complicating factor for gingivitis would be BIOFILM TRAPS:
  • malposition of teeth
  • overhanging restorations
  • orthodontics/braces
  • calculus
54
Q

MODIFIED BY SYSTEMIC FACTORS

_____ initiates the disease- _________ will modify/intensify the disease

________ system and fluctuations in ______ hormones cause more ______ species of microbes such as __________

Example causes:
(3)

A

PLAQUE/BIOFILM initiates the disease- HOST SYSTEMIC FACTORS will modify/intensify the disease

ENDOCRINE system and fluctuations in FEMALE hormones cause more BACTEROIDES species of microbes such as PREVETELLA INTERMEDIA

Example causes:
- PREGNANCY
- PUBERTY
- MENOPAUSE

55
Q

MODIFIED BY MEDICATIONS

________ inflammation of the gingiva related to plaque biofilm and systemic _____

The cause is likely overstimulation of _____

Biofilm _______ the disease and is _____ by medications

Xerostomia- over ___ meds

___________: an increase in the size of the gingiva resulting from systemic medications (e.g. ______)

A

EXAGGERATED inflammation of the gingiva related to plaque biofilm and systemic MEDICATIONS

The cause is likely overstimulation of FIBROBLASTS

Biofilm INITIATES the disease and is MODIFIED by medications

Xerostomia- over 400 meds

DRUG INFLUENCED GINGIVAL ENLARGMENT: an increase in the size of the gingiva resulting from systemic medications (e.g. DILANTIN)

56
Q

is plaque accumulation necessary for the initiation of gingival enlargement?

what can reduce (but not eliminate) gingival overgrowth, why doesn’t it eliminate it

which teeth are typically affected most, give 3 terms to describe the gingiva

A

Plaque accumulation is NOT necessary for initiation of gingival enlargement, but it will exacerbate the gingival disease.

METICULOUS PLAQUE CONTROL can reduce but will not eliminate gingival overgrowth (fibrotic tissue remains forever, and needs to be removed via surgery)

MAXILLARY TEETH are most affected; fibrotic, lobular, pink

57
Q

what drugs are associated with gingival overgrowth (3)

A
  • calcium channel blockers (nifedipine)
  • anti-convulsive (dilantin)
  • immunosuppressive (cyclosporine)
58
Q

does medication impact someone plaque level?

A

no, it alters the hosts immune response which sends more fibroblasts to the area making the tissue firmer and overgrown. The best thing we can do is remove the plaque and put the pt. on 3 month recare

59
Q

MODIFIED BY MALNUTRITION (3rd world countries typically)

-Dental biofilm aggravated by nutritional ________

  • although rare, vitamin __ def. predominates
  • ______ acid; spontaneous _____

-Vitamins __, __, __

-At risk for vitamin deficiencies:
(3)

A

-Dental biofilm aggravated by nutritional DEFICIENCEIES

  • although rare, vitamin C def. predominates
  • ASCORBIC acid; spontaneous BLEEDING

-Vitamins A, B, C

-At risk for vitamin deficiencies: INFANTS, INSTITUTIONALIZED ELDERLY, ALCOHOLICS

60
Q

NON BIOFILM INDUCED

Viral, fungal or non-periodontal associated bacterial origin, ______ process

Viral - ______
Fungal- _______
Fungal- _______
Reactive- _________

A

Viral, fungal or non-periodontal associated bacterial origin, FUNGAL process

Viral - PRIMARY HERPES
Fungal- CANDIDIASIS/THRUSH
Fungal- HISTOPLASMOSIS
Reactive- ALLERGIES (ex- toothpaste allergy would have no bleeding but inflammation)

61
Q

description of the mouth during PRIMARY HERPETIC GINGIVOSTOMATITIS (PHG) (6)

this occurs when you are first exposed to the herpes virus

A
  • fiery red marginal gingiva
  • swollen papilla that bleed
  • painful
  • yellowish ulcers with red halo
  • fever
  • dehydration
62
Q

non biofilm induced gingivitis: example of a viral origin

PHG
- usually affect _______
- HSV1: severe reaction to _______
- gingiva: rapidly ____, burst and form painful _____
- ____ manisfestations
- _______! use caution with touching
- after being infected you develop _______
- ______

A
  • usually affects YOUNG CHILDREN
  • HSV1: severe reaction to INITIAL EXPOSURE
  • gingiva: rapidly ERUPT, burst and form painful ULCERS
  • SYSTEMIC manisfestations
  • TRANSMISSIBLE! use caution with touching
  • after being infected you develop IMMUNITY
  • RECURRENT
63
Q

Dermatologic oral lichen planus

  • intense _____ of gingiva
  • u_______
  • interlacing _______ may be present
  • raised _____ may be present
  • disease of ________
  • characterized by ______, swollen ____ on skin and/or _____
  • cause:
A
  • intense REDNESS of gingiva
  • ULCERCATIONS
  • interlacing WHITE LINES may be present
  • raised WHITE LESIONS may be present
  • disease of SKIN AND MUCOUS MEMBRANES
  • characterized by ITCHY, swollen RASH on skin and/or MOUTH
  • cause: unknown, maybe allergic or immune reactoin
64
Q

ALLERGIC REACTION TO TOOTHPASTE

Reaction to _____ in toothpaste

what ingredient is most commonly seen with allergic reactions

Most common in those with _________

Fiery red ___

Tissue ______ of mucosa

is there bleeding?

what should the patient do?

A

Reaction to ADDITIVE in toothpaste

SODIUM LAURYL SULPHATE

Most common in those with OTHER ALLERGIES

Fiery red GINGIVA

Tissue SLOUGHING of mucosa

no

change brands

65
Q

IAG can only be determined on the _____

how to calculate IAG

A

facial

MGJ-GM - PD = IAG

66
Q

what are the 2 codes for planning gingivitis treatment

A

D1110: adult prophylaxis (slight)

D4346: scaling in the presence of mod/severe gingivitis