Gingival recession Flashcards

1
Q

What is recession? (4)

A

Recession is the apical shrinkage of the gingivae
beyond the amelo-cemental junction (measured
from the ACJ to gingival margin)
-may be localised or generalised
-indicator of past disease - but does not mean that active disease is present
-also occurs following healing from effective perio tx

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2
Q

Predisposing factors (3)

A

Lack of bone
Thin gingival tissue
Role of fraenum? Unsure whether it is a cause or people just can’t clean there well

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3
Q

Predisposing factors: lack of bone - developmental/ acquired (4)

A
Thin cortical plates/ Prominent roots
Dehiscences/ Fenestrations
Long standing periodontitis
Occlusal trauma or excessive ortho force may cause dehiscences
-deep overbite
-rotation around retainers
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4
Q

Precipitating factors (6)

A

N.B: All causes of gingival inflammation – provoke recession if predisposing factors present
• Plaque induced gingival inflammation ie. gingivitis
and/or periodontitis
• Traumatic toothbrushing
• Direct repeated trauma (eg. complete overbite
impinging on lower gingivae)
• Parafunctional habits
• Iatrogenic cause eg. ortho. Bands, prostheses
• Food trauma

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5
Q

Problems

A
• Patient anxiety? appearance, crown
margins visible?
• Stagnation area – plaque/calculus
• Sensitivity – exposed dentine
• Root caries leading to:
• Possible pulp death
• Continued breakdown
• Subgingival restorations?
• Oral appliances?
• Orthodontic tooth movement ?
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6
Q

Management if recession (9)

A
• Explain and reassure
• Correct precipitating factors where possible eg. ensure
atraumatic plaque control
• Thorough scale and polish
• Restore carious root surfaces (with respect for periodontium)
• Control any sensitivity
• Fluoride application?
• Monitor
Where indicated;
• Aesthetics – e.g. gingival veneer
• Surgical options e.g. laterally repositioned flap, grafts, apically repostioned flaps with/without regenerative
techniques
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7
Q

Outcome of management (5)

A
• Stabilisation/control of breakdown
• Residual stagnation areas
• Management of sensitivity – long term
• Management of the exposed root surface
to prevent caries
• Importance of review to maintain good
standard of home care
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8
Q

Control of sensitivity (3)

A
• Plaque control
• Fluoride toothpastes
• Desensitisers eg:
SupaSeal, Duraphat, Seal and Protect,
Cervitec
Desensitising toothpastes
Dentine bonding agents
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9
Q

Clinical questions (3)

A
  • When to monitor a patient without surgical evaluation?
  • When is the patient a candidate for surgical intervention?
  • When should the patient be referred to a periodontal specialist?
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10
Q

Monitor how?

A
  • Measure in mm and record from CEJ to top of gingival margin – subject to error
  • Clinical photographs
  • Study models
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11
Q

Miller classification (4)

A
• Class I:recession does not
extend mucogingival
junction, no loss of
interdental tissue/bone
• Class II:recession to or
beyond mgj, no loss of
interdental tissues
• Class III: +interdental
tissue loss, not beyond
recession
• Class IV: +interdental loss
beyond recession
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12
Q

Indications for surgery (6)

A
  • Continued inflammation
  • Progressive breakdown
  • Aesthetics
  • Frenal pull
  • Pocketing beyond MGJ
  • Advanced restorative procedures planned
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13
Q

Pre-operative assessment for surgery (6)

A
  • Is surgical treatment warranted?
  • Is recession stable following monitoring?
  • Medical and social assessment
  • Tooth vitality
  • Radiographic examination
  • Informed consent and clinical records
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14
Q

Types of surgery (4)

A
Laterally repositioned flap
Free gingival graft
Free connective tissue graft
Coronally repositioned flap
Guided tissue regeneration
Emdogain - stimulates repair of enamel
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15
Q

Systematic reviews: which type of surgery

A

(not including Emdogain)

Coronally Advanced Flap alone or assoc. with supepithelial CT grafts –> significant improvement

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