GI 1 Flashcards

1
Q

what is appendicitis?

A

the inflammation of the vermiform appendix & classified as simple, gangrenous, or perforated.

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2
Q

appendicitis is most common in who?

A

young adults, especially men

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3
Q

what are some common s/sx of appendicitis?

A

RLQ pain
McBurneys point
rebound tenderness
low grade fever
high WBC (10,000)
shift to the left (increase in bands)
-rupture is peritonitis

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4
Q

what is McBurney’s point?

A

halfway between the R anterior iliac crest & umbilicus

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5
Q

what are some s/sx of peritonitis?

A

abdominal pain (generalized)
high temp
higher WBC (20,000)
infection/sepsis

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6
Q

how do we treat appendicitis?

A

must be rapid, include antibiotics & may have drain in incision or open incision

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7
Q

what nursing interventions can be done for someone with appendicitis?

A
  1. Avoid laxatives, enemas, or heat as may cause rupture of appendix.
  2. pt is NPO. I & O & IV fluids to prevent dehydration.
  3. Monitor bowel movements & signs of return of peristalsis.
  4. Ambulate ASAP after surgery; usually within 8 to 12 hours.
  5. Semi-Fowler’s position to ↓ pain & promote drainage to prevent fluid accumulation.
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8
Q

what is cholecystitis?

A

inflammation of the gallbladder, either calculous or acalculous

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9
Q

what is calculous cholecystitis?

A

hemical irritation and inflammation from cholelithiasis (stones in the gallbladder, usually formed of cholesterol when bile becomes supersaturated with cholesterol) or choledocholithiasis (stones in the common bile duct)

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10
Q

what is acalculous cholecystitis?

A

inflammation occurring without gallstones, typically associated with biliary stasis.

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11
Q

cholecystitis is common in who?

A

4x more common in women (ages of 40-50):
-fair, fertile, overweight, forty or older

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12
Q

what are some s/sx of cholecystitis?

A

RUQ pain/ rebound tenderness (Blumberg’s sign)
Murphy’s sign
steatorrhea (fatty stool)
may have a fever

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13
Q

what is Blumberg’s sign?

A

RUQ - rebound tenderness – called Blumberg’s sign; may radiate to shoulder
-cholecystitis

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14
Q

what is murphy’s sign?

A

patient stops breathing during palpation just below right costal margin.
-cholecystitis

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15
Q

how do diagnose cholecystitis?

A

-Ultrasound determines gallstones and is best initial diagnostic test.

-HIDA (hepatobiliary) scan

-ERCP (endoscopic retrograde cholangiopancreatography) or MRCP

-Lab Values:
– WBC elevated - indicates inflammation.
–↑ amylase and/or lipase- indicates pancreatic involvement (stones in common bile duct).
– ALP (alkaline phosphatase), AST & bilirubin may be elevated (direct & indirect) if obstruction of the
common bile duct & liver involvement.

-Gallbladder X-ray test

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16
Q

what is a HIDA?

A

(hepatobiliary) scan: visualizes gallbladder and determines patency of biliary system.

17
Q

what is a ERCP? or MRCP?

A

(endoscopic retrograde cholangiopancreatography)
(magnetic resonance
cholangiopancreatography).

Pt. swallows a long, thick, lighted flexible tube connected to a computer and monitor. Dye is injected that stains the bile ducts. Stones can be removed. Assess gag reflex following test.

18
Q

what does gallbladder x-ray test tell us?

A

radiopaque tablets are taken the evening before so that the gallbladder will be visible on x-
ray examination; this test may be done if the ultrasonography is inconclusive.

19
Q

how do we differenciate between cholecystitis & biliary obstruction?

A

-cholecystitis/cholelithiasis
–Pain RUQ (2-4 hours after eating high-fat) that may radiate to the R shoulder or back. Discomfort is
experienced because bile flow into the intestine is obstructed.

-choledocholithiasis (stone in the common bile duct)
–pain
–Jaundice due to obstruction/hepatocellular damage. When the common bile duct is completely obstructed, the bile is unable to pass into the duodenum & is absorbed into the blood.
–lab values: amylase, ALP, bilirubin
–feces - clay colored
–pruritis – itching due to accumulation of bile salts under the skin

20
Q

how do we treat cholecystitis?

A

surgery
–cholecystectomy
–choledochotomy (remove stones)

ABO

antispasmodics, anticholinergics
–reduce the contraction of the gallbladder

morphine or another opioid

lithotripsy (breaks up stones)

biliary catheter drain

21
Q

what are some nursing interventions for cholecystitis?

A

-low fat healthy diet
-assess liver or pancreatic involvement
-assess bowel sounds
-NPO, NG, F & E balance, TCDB & IS
-semi-fowlers
-may have a t-tube

22
Q

what is the patho of pancreatitis? how does this cause problems?

A
  1. Auto-digestion: injury permits digestive enzymes to leak into pancreatic tissue, then break down
    tissue & cell membranes in the organ & causes vascular damage, edema, hemorrhage, & necrosis → trypsin (initiates pancreatic digestion & activates lactase, which dissolve the elastic fibers of the blood vessels within the pancreas causing hemorrhage), kinins (causes vasodilation and ↑ capillary/vascular permeability), & phospholipase (causes necrosis of the pancreas).
  2. As more cells are destroyed, more enzymes are released, causes ↑ inflammation of the peritoneum and the pancreas. Drainage may collect into abscesses, which can eat through the bowel & trigger sepsis.
  3. In severe acute pancreatitis, total auto-digestion & pancreatic destruction may lead to diabetic ketoacidosis, shock, coma, & death.
23
Q

what causes pancreatitis?

A

acute- Obstruction of pancreatic or common bile duct (e.g. biliary stones, stasis) and alcohol consumption

chronic- heavy alcohol consumption

24
Q

what are some s/sx of pancreatitis?

A

-pain (mid-epigastric or LUQ)
-jaundice
-N/V
-fever
-hypotension, hypovolemia, respiratory distress & electrolyte imbalances.
-hyperglycemia
-steatorrhea

25
Q

what is common result from chronic pancreatitis shown by grey turner’s and cullen’s sign? What are those?

A

pancreatic hemorrhage

-Grey Turner’s sign: a gray-blue discoloration of the flank

-Cullen’s sign: a gray-blue discoloration around the umbilicus

26
Q

how do we diagnose pancreatitis?

A
  1. Lab values
    –Pancreatic enzymes: amylase, lipase
    –Hyperglycemia
    –Calcium-forming complexes develop because fatty acids have been set free from retroperitoneal fat by lipase, which causes hypocalcemia. Pt may have s/sx of ↓ Ca++.
    –WBC elevation
    –Liver function tests (LDH, AST) may be elevated in alcoholic liver disease & acute pancreatitis
  2. Imaging
    –Abdominal ultrasound
    –Contrast-enhanced CT – more reliable
27
Q

how do we treat pancreatitis?

A

-NPO with gastric suction to suppress enzymes & ↓ pancreatic stimulation

-Pancreatic enzyme supplement for pancreatic insufficiency

-Low fat diet to reduce discomfort & steatorrhea

-Meds
a. Histamine blockers & antacids are used to ↓ gastric secretions
b. Anti-emetics & analgesics such as morphine or other opioid
c. Anticholinergic drugs to ↓ vagal stimulation, inhibit pancreatic enzyme secretion, & relieve spasms
6. Operative procedures - pancreatic resection of the head, body, tail, or total removal of the pancreas (pancreatotomy), which results in compromised pancreatic function.

28
Q

what are some nursing interventions for pancreatitis?

A

-assess for fluid deficit, F+E imbalance
-resp. assessment
-assess bowel tones
-Nutritional support - TPN (monitor glucose levels) or NG/Peg feedings. Clear liquids → small amts of CHO →
bland low-fat/protein while gradually ↑ CHO. Avoid large meals, alcohol & caffeinated beverages.
-rest periods
-monitor for complications

29
Q

what are the common complications from pancreatitis and or/ pancreatic surgery?

A

pancreatic abscess (most serious); the most common
fatal complications of severe pancreatitis are hypovolemia & hyperglycemia.

30
Q

what is diverticulosis and diverticulitis?

A

 diverticulosis: multiple diverticula.
 diverticulitis: inflammation of diverticula.

31
Q

who commonly has diverticulosis/itis?

A

common after age 65

2/3 of adults > 80 have diverticulosis

32
Q

what are the s/sx of diverticulosis/itis?

A

no s/sx unless complications occur
1. Constipation, diarrhea, & flatulence.
2. Acute LLQ abd pain - may be relieved by a BM or passing flatus.
3. S/sx may ↑ after a meal or activities causing ↑ intra-abd pressure.
4. May see rectal bleeding or fever if infection present.

33
Q

whata re common complications of Diverticulosis/Diverticulitis?

A

-retained, undigested food in diverticular sac, resulting in a hard mass, causing abscess, fistulas,
bowel obstruction, peritonitis, & hemorrhage.

34
Q

how do we disgnose Diverticulosis or Diverticulitis?

A
35
Q

how do we treat Diverticulosis or Diverticulitis?

A
36
Q

what are some nursing interventions for Diverticulosis Diverticulitis?

A
  1. Treat & watch for s/sx of complications
  2. Encourage:
     A soft high fiber diet if a diagnosis of diverticulosis
     NPO if a diagnosis of diverticulitis
     Fluids (at least 8 glasses per day)
     Maintenance: high vegetable fiber diet – seeds?
  3. Teach importance of:
     Avoid ↑ intra-abd pressure (e.g. constipation, tight belts, restrictive clothing & improper lifting
    techniques; however low-impact weight lifting is acceptable)
     Encourage activities that ↑ peristalsis (e.g. walking & ↑ fluids)
     Importance of having a daily BM & weight loss
     Notifying the physician of changes in bowel elimination patterns or characteristics, fever, or abd pain.