GI 1 Flashcards
what is appendicitis?
the inflammation of the vermiform appendix & classified as simple, gangrenous, or perforated.
appendicitis is most common in who?
young adults, especially men
what are some common s/sx of appendicitis?
RLQ pain
McBurneys point
rebound tenderness
low grade fever
high WBC (10,000)
shift to the left (increase in bands)
-rupture is peritonitis
what is McBurney’s point?
halfway between the R anterior iliac crest & umbilicus
what are some s/sx of peritonitis?
abdominal pain (generalized)
high temp
higher WBC (20,000)
infection/sepsis
how do we treat appendicitis?
must be rapid, include antibiotics & may have drain in incision or open incision
what nursing interventions can be done for someone with appendicitis?
- Avoid laxatives, enemas, or heat as may cause rupture of appendix.
- pt is NPO. I & O & IV fluids to prevent dehydration.
- Monitor bowel movements & signs of return of peristalsis.
- Ambulate ASAP after surgery; usually within 8 to 12 hours.
- Semi-Fowler’s position to ↓ pain & promote drainage to prevent fluid accumulation.
what is cholecystitis?
inflammation of the gallbladder, either calculous or acalculous
what is calculous cholecystitis?
hemical irritation and inflammation from cholelithiasis (stones in the gallbladder, usually formed of cholesterol when bile becomes supersaturated with cholesterol) or choledocholithiasis (stones in the common bile duct)
what is acalculous cholecystitis?
inflammation occurring without gallstones, typically associated with biliary stasis.
cholecystitis is common in who?
4x more common in women (ages of 40-50):
-fair, fertile, overweight, forty or older
what are some s/sx of cholecystitis?
RUQ pain/ rebound tenderness (Blumberg’s sign)
Murphy’s sign
steatorrhea (fatty stool)
may have a fever
what is Blumberg’s sign?
RUQ - rebound tenderness – called Blumberg’s sign; may radiate to shoulder
-cholecystitis
what is murphy’s sign?
patient stops breathing during palpation just below right costal margin.
-cholecystitis
how do diagnose cholecystitis?
-Ultrasound determines gallstones and is best initial diagnostic test.
-HIDA (hepatobiliary) scan
-ERCP (endoscopic retrograde cholangiopancreatography) or MRCP
-Lab Values:
– WBC elevated - indicates inflammation.
–↑ amylase and/or lipase- indicates pancreatic involvement (stones in common bile duct).
– ALP (alkaline phosphatase), AST & bilirubin may be elevated (direct & indirect) if obstruction of the
common bile duct & liver involvement.
-Gallbladder X-ray test
what is a HIDA?
(hepatobiliary) scan: visualizes gallbladder and determines patency of biliary system.
what is a ERCP? or MRCP?
(endoscopic retrograde cholangiopancreatography)
(magnetic resonance
cholangiopancreatography).
Pt. swallows a long, thick, lighted flexible tube connected to a computer and monitor. Dye is injected that stains the bile ducts. Stones can be removed. Assess gag reflex following test.
what does gallbladder x-ray test tell us?
radiopaque tablets are taken the evening before so that the gallbladder will be visible on x-
ray examination; this test may be done if the ultrasonography is inconclusive.
how do we differenciate between cholecystitis & biliary obstruction?
-cholecystitis/cholelithiasis
–Pain RUQ (2-4 hours after eating high-fat) that may radiate to the R shoulder or back. Discomfort is
experienced because bile flow into the intestine is obstructed.
-choledocholithiasis (stone in the common bile duct)
–pain
–Jaundice due to obstruction/hepatocellular damage. When the common bile duct is completely obstructed, the bile is unable to pass into the duodenum & is absorbed into the blood.
–lab values: amylase, ALP, bilirubin
–feces - clay colored
–pruritis – itching due to accumulation of bile salts under the skin
how do we treat cholecystitis?
surgery
–cholecystectomy
–choledochotomy (remove stones)
ABO
antispasmodics, anticholinergics
–reduce the contraction of the gallbladder
morphine or another opioid
lithotripsy (breaks up stones)
biliary catheter drain
what are some nursing interventions for cholecystitis?
-low fat healthy diet
-assess liver or pancreatic involvement
-assess bowel sounds
-NPO, NG, F & E balance, TCDB & IS
-semi-fowlers
-may have a t-tube
what is the patho of pancreatitis? how does this cause problems?
- Auto-digestion: injury permits digestive enzymes to leak into pancreatic tissue, then break down
tissue & cell membranes in the organ & causes vascular damage, edema, hemorrhage, & necrosis → trypsin (initiates pancreatic digestion & activates lactase, which dissolve the elastic fibers of the blood vessels within the pancreas causing hemorrhage), kinins (causes vasodilation and ↑ capillary/vascular permeability), & phospholipase (causes necrosis of the pancreas). - As more cells are destroyed, more enzymes are released, causes ↑ inflammation of the peritoneum and the pancreas. Drainage may collect into abscesses, which can eat through the bowel & trigger sepsis.
- In severe acute pancreatitis, total auto-digestion & pancreatic destruction may lead to diabetic ketoacidosis, shock, coma, & death.
what causes pancreatitis?
acute- Obstruction of pancreatic or common bile duct (e.g. biliary stones, stasis) and alcohol consumption
chronic- heavy alcohol consumption
what are some s/sx of pancreatitis?
-pain (mid-epigastric or LUQ)
-jaundice
-N/V
-fever
-hypotension, hypovolemia, respiratory distress & electrolyte imbalances.
-hyperglycemia
-steatorrhea