Geriatrics I Flashcards

1
Q

What is frailty?

A

Frailty is a distinctive health state related to the ageing process in which multiple body systems gradually lose their in-built reserves. Around 10 per cent of people aged over 65 years have frailty, rising to between a quarter and a half of those aged over 85.

  • dyshomeostasis, alongside soco-environmental insults

syndrome of:
Falls
Delirium
Immobility
Incontinence

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2
Q

Presentations of diseases in frailty

A
  • in frailty may have diff presentations vs classic pres.

HYPERTHYR.
Depression
Cognitive impairment
Muscle weakness
Atrial fibrillation
Heart failure
Angina

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3
Q

Implications in Geriatrics

A
  • increasing co-morb.
  • increasing inter-variability in organ reserve and funct.
  • variable pres.
  • little drug evidence for 80yo+
  • POLYPHARM.
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4
Q

Be aware of the importance of a multi-dimensional and multi-disciplinary holistic approach to assess and address multi-component health problems in elderly.

A

a

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5
Q

Frailty Phenotype

A

3 out of 5
Unintentional weight loss
Exhaustion
Weak grip strength
Slow walking speed
Low physical activity

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6
Q

System Failure in Frailty

A

Compromise of functional systems that allow daily activity and functionality
* mobility = getting up
* comprehension = reacting and acting to every day
* self-care skills = independence

= frailty syndrome

> Hx taking = ADL = activities of daily living to assess function and needs

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7
Q

Comprehensve Geri Assess.

A

goal centred, holistic, multidisciplin.

mdt:
dr = broad overview and responsibility, medical contributors
physio = assess mobility
OT = ADLs
Nurses = care needs and assessments long-term
+ others

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8
Q

Explain the typical symptom pattern associated with each of the main types of incontinence andmgmt

A

1) STRESS INCONTINENCE
weak bladder outlet
- urine leak w/ pressure, movement
- women w/ children, post-menopausal

> physio
oestrogen cream + duloxetine
sx option

2) RETENTION + OVERFLOW. INCONTINENCE
strong bladder outlet
- poor flow, voiding, hesitancy, dribbling
- urethra blocking, BPH

> a-blocker - TAMSULOSIN relaxes sphincter
anti-androgen - FINASTERIDE shrinks prostate
suprapubic cath.

3) URGE
strong bladder muscle
- detrusor contracts low volumes = SUDDEN NEED
- bladder stones? or stroke

> anti-muscarinics - OXYBUTININ, TOLTERODINE, SOLIFENACIN relaxes detrusor
beta3 adrenoreceptor agonists - MIRABEGRON relaxes detrusor
bladder re-training

4) NEUROPATHIC BLADDER
underactive
- rare, 2º neuro. e.g MS, stroke, or prolonged cath.
- no awareness thus leads to overflow

> cath
or sometimes parasympathomimetics

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9
Q

Factors contributing to Incontiencne

A

extr. factors
- physical. state
- mobility
- confusion
- inappt. drinking pattern
- MEDICATIONS
- constipation
- circumstances

intr. factors
- bladder
- outlet
- too weak, too strong

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10
Q

when to refer urinary incontinence

A

Failure of initial mgmt
max 3mos of mgmt

OR
- fistula
- palpable bladder after micturition = RETENTION
- CNS disease
- organic pelvic disease

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11
Q

Local Innervation and Urinary Function

A

S2-S4 (para) = CONTRACTION strenght & freq

T10-L2 (symp.) = DETRUSOR RELAX b-adrenoreceptor

T10-S2 (symp.) = BLADDER NECK and INT. SPHINCTER CONTRACTION a-adrenorecept.

S2-S4 (somatic) = UROGENITAL DIAPHGRAGM CONTRACTION & EXT. SPHINCTEER

+CNS inhibit parasym tone promoting relaxation thus storage
+ sphincter closure mediated by a-adrenergic and somatic activity
+ pontine micturition centre storage progam
+ frontal cortex, caudal spinal cord part

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12
Q

Referring faecal incontinence

A

Faecal incontinence
Referral after failure of initial management:

Constipation or diarrhoea with normal sphincter
Referral necessary at onset:
Suspected sphincter damage
Neurological disease

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13
Q
  • Describe the main clinical features of delirium, its investigation and management
A
  • disturbed consciousness: hypo, hyper, mixed
  • change in cognition
  • acute onsent and fluctuant

+ disturbed sleep/wake cycle
+ disturbed psychomotor behaviour
+ emotional disturbance

may be precipitated by infection, dehydration, pain etc. = INSULT

*post sx
* EoLife

  • 4AT;
    4+ possible delirium ±cog impairment
    1-3 pos. cog impairment
    0 - unlikely

> treat cause
TIME bundle
orientation of pt. and independence

> !QUETIAPINE (oral) only if danger to themselves or others

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14
Q
  • Appreciate how to differentiate delirium from other types of cognitive impairment
A

a

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15
Q

Appreciate the impact delirium has on people, their relatives, and the staff looking after them

A
  • SIGNIFICANT FALLS RISK
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16
Q

Understand the main principles of capacity assessment and Adults with Incapacity legislation

A

a

17
Q

SIGN 88 GUIDELINES in Elderly Delirium & UTI

A
  • many bacteriuria present asymptomatically in elderly anyway
  • greatest prevalence is in 75yo+ institutionalised category but even then it is 57%
  • abx more harmful
  • DO NOT USE DIPSTICK TESTS for UTI dx
18
Q

Assessing geris for management at home or secondary care?

A

? IV
? scans
? specialist support/care

? social set up
? community support available for Carl

19
Q

Causes of delirium

A
  • urine retention
  • infection

HIGH RISK
- ZOPICLONE: sleep
- AMITRYPTILLINE: anticholinergic

20
Q

Causes of falls in older people

A
  • multi-systemic; overlapping
  • incontinence: rushing to toilet
  • drugs: syncope, hypos, reduced consciousness
    increased sedation, urine, hallucinations, dizzy

Collapse with no memory ?syncope or cognition
Clear history of trip – think sensory (eyes, nerves)
Palpitations preceding fall and no trip - think cardiac
On turning – think postural instability
Any ‘near misses’- unsteady on standing
Syncope on exertion think aortic stenosis

21
Q

How to assess an older person who presents with a fall

A

? LONG LIE
rhabdo = CK
pneumonia
skin injury

?glucose

?other falls

?HPC of fall

?head injury = CT

?CARDIAC CAUSE
- meds = hypotension dt propanolol
- AO STENOSIS! rule out

22
Q

Gait: Ataxic Association

A

cerebellar dmg

23
Q

Gait: High stepping association

A

Peripheral neuropathy