Gastrointestinal System - Pedi Flashcards

1
Q

GASTROINTESTINAL SYSTEM

A

*Route to ingest, digest, and absorb foods, fluids, vitamins, and nutrients
*Excretion/elimination of solid waste
*GI tract:
*Mouth, esophagus, stomach, pancreas, small intestine, large intestine
*Helper organs:
*Liver, gallbladder, spleen

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2
Q

DIFFERENCES BW PEDI AND ADULT

A

*Mouth – infants/young children oral phase – risk for infectious agents
*Swallowing - involuntary until 6 weeks
*Esophagus – lower esophageal sphincter matures at 1mos – prior to this regurgitation common
*Stomach capacity – smaller
*2 mos – 200 mLs (most smaller amts)
*16yo – 1500mLs
*Adult – 2000 mLs – 3000 mLs
*Digestive enzymes – deficient until ~6 months
*Small intestine
*~250 cm at birth
*600 cm in adult
*Liver function
*bilirubin conjugation starts at 2-3 weeks of age
*Pancreatic enzymes at adult levels by 2yo
*Palpable at birth – relatively large
*Excretory function – control of defecation (awareness/control 18-24 months)
*Total Body Water: infants/children > adults

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3
Q

PEDIATRIC GI ASSESSMENT (Health History)

A

*Past history – birth history, hospitalizations
*Present illness – onset? Symptoms? Assoc symptoms?
*Growth and development – gaining/ growing
*Emesis – Spitting up vs vomit, when? Frequency? Color? Volume?
*Stool – color, consistency, passage of meconium, pattern – BF usually with every
feed, Bottle 1-2 per day, size, abdominal pain, leaking/staining
*Nutrition – intake, appetite
*Activity – more sleep? Changes in activity level? Mental status?
*Family hx: IBS? IBD? Food allergies?

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4
Q

PEDIATRIC GI ASSESSMENT, CONT.
(Physical Exam)

A

*General – lethargic, interactive, active,distress
*Hydration status – MM, tears, fontanelle, turgor, UO
*Weight
*Heart /Perfusion– rate, rhythm; murmur, pulses, cap refill
*Abdomen – Inspect, Auscultate, Palpate
*Stool – Color, consistency, size, frequency, pattern changes
*Assess rectum – tone, fissures
*Skin – color, texture, diaphoretic

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5
Q

GI NURSING GOALS/INTERVENTIONS

A

*Maintain adequate fluid balance/hydration
*Prevent hypovolemia
*IV fluids, strict I & O, perfusion, daily weight
*Maintain skin integrity
*Appropriate Nutrition
*Treat/prevent infection
*Promote pain relief, comfort & rest
*Administer/manage medications
*Maintain and promote growth and development/milestone achievements
*Provide support to child & parents / Educate

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6
Q

GI/GI TRACT DISORDERS
(Structural anomalies)

A

*Cleft lip & palate
*Esophageal Atresia (EA)/Tracheoesophageal Fistula (TEF)
*Meckel’s diverticulum
*Omphalocele
*Gastroschisis
*Congenital Diaphragmatic Hernia (CDH)
*Anorectal malformations
*Imperforate anus
*Rectovestibular fistula
*Cloacal exstrophy/malformation

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7
Q

GI/GI TRACT DISORDERS
(Acute GI Disorders)

A

*Dehydration
*Vomiting
*Gastroenteritis
*Hypertrophic Pyloric Stenosis
*Necrotizing Enterocolitis (NEC)
*Malrotation and Volvulus
*Intusussception
*Appendicitis
*Peptic Ulcer Disease (PUD)
*Rotavirus, Clostridium difficile (c.diff), Giardia

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8
Q

GI/GI TRACT DISORDERS
(Chronic GI disorders)

A

*Gastroesophageal Reflux (GER)
*Constipation
*Encopresis
*Hirschsprung Disease
*Short Bowel Syndrome
*Inflammatory Bowel Disease
*Crohn’s Disease
*Ulcerative Colitis
*Celiac Disease

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9
Q

GI/GI TRACT DISORDERS
(Hepatobiliary Disorders)

A

*Pancreatitis
*Hyperbilirubinemia (Jaundice)
*Biliary Atresia
*Hepatitis
*Cirrhosis & Portal Hypertension

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10
Q

STRUCTURAL ANOMALIES
(Cleft Lip and Palate)

A

Most common congenital craniofacial defect:
frontonasal and/or maxillary processes did not
fuse
*~1 in 700 births
*Unknown cause. Contributing factors:
*Genetics, folic acid deficiency, advanced
maternal age, anticonvulsants, ETOH &
smoking, family hx
*25-30% also have syndrome/other
abnormalities
*Lip tissues fuse 5 – 6 weeks gestation
*Palate closes 7-9weeks gestation

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11
Q

STRUCTURAL ANOMALIES:
Complications cleft lip and palate

A
  • feeding difficulties
  • insufficient growth
  • altered dentition
  • delayed or altered speech
  • otitis media with effusion
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12
Q

STRUCTURAL ANOMALIES:
Treatment cleft lip and palate

A

Tx: Multidisciplinary team: plastic surgeon or
craniofacial specialist, oral surgeon, dentist or
orthodontist, psychologist, otolaryngologist, nurse,
LICSW, audiologist, and speech language pathologist
Surgical repair cleft lip and palate:
*Lip 2-3 months – early repair improves feeding and
appearance
*Palate 6-9 months – essential for speech
development

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13
Q

STRUCTURAL ANOMALIES:
Cleft Lip and Palate Nursing Management

A
  • Promoting adequate nutrition – may need special nipple, burp frequently
  • Aspiration risk – prior to repair for palate may have a prosthodontic device
  • to act as palate / breastfeeding may be more effective due to the soft breast tissue that is pliable and may cover opening
  • Post op some surgeons resume BF immediately / special nipples for bottle fed
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14
Q

STRUCTURAL ANOMALIES:
Cleft lip and palate, nursing management

A

*Parent – Infant bonding: appearance may affect
*Emotional support
*Post operatively:
*Feeding support, may need restraints to prevent rubbing / touching surgical site,
certain things in mouth may affect repair ie no pacifier, straws, suction catheters
*Avoid vigorous or sustained crying

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15
Q

STRUCTURAL ANOMALIES:
Esophageal Atresia

A

*proximal and distal ends of esophagus do not communicate
*upper esophagus ends in a pouch / lower portion ends above the
diaphragm

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16
Q

STRUCTURAL ANOMALIES:
Tracheoesophageal Fistula

A
  • abnormal communication between esophagus and trachea
  • BOTH Atresia and Fistula typically associated with other abnormalities:
  • VACTERL work up
  • Vertebral, Anal atresia, Esophagus, Trachea, Renal abnormalities, or limb differences
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17
Q

STRUCTURAL ANOMALIES:
Esophageal Atresia and Tracheoesophageal Fistula

A

*maternal hx of polyhydramnios
*neonate with copious, frothy bubbles in mouth and nose and drooling
*abdominal distension – air accumulates
*can not insert NGT
*newborn with rattling respirations, excess salivation
*3 C’s: cough, choking and cyanosis with feeding

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18
Q

STRUCTURAL ABNORMALITIES:
Nursing Management Atresia and Fistula

A

*Preop:
*NPO
*HOB elevated prevent reflux/aspiration
*Orogastric tube – low wall suction / avoid irrigation
*I+O’s – fluid and electrolyte
*Comfort measures avoid crying
*Postop:
*TPN
*Antibiotics
*PO /NGT feeds about a week postop
*No deep suctioning

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19
Q

STRUCTURAL ANOMALIES:
Omphalocele

A

*Congenital anomaly of anterior abdominal wall
*Defect of umbilical ring
*Vary in size
*Evisceration of abdominal contents into an external peritoneal sac
*Bowel / liver / entire GI tract

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20
Q

STRUCTURAL ANOMALIES:
Gastroschisis

A
  • congenital defect of anterior abdominal wall
  • Herniation of abdominal contents thru a defect, typically to left or right of umbilicus
  • NO peritoneal sac protecting organs, exposure to amniotic fluid - thickened, edematous and inflamed
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21
Q

STRUCTURAL ANOMALIES:
Nursing Assessment Gastroschisis and Omphalocele

A
  • size of deformity
  • appearance of organs
  • look for twisting of intestine
  • Full exam looking for other deformities ie VACTERL
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22
Q

STRUCTURAL ANOMALIES:
Nursing Management Omphalocele/ Gastroschisis

A
  • Prevent hypothermia
  • Prevent fluid loss
  • Prevent trauma to exposed contents
  • Prevent infection – sterile technique/antibiotics
  • NPO / orogastric tube / PN
  • Monitor exposed contents for vascular changes
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23
Q

STRUCTURAL ANOMALIES:
Omphalocele/ Gastroschisis
(Treatment, Surgery, Post op)

A

Treatment:
Surgery: exact way repaired depends on size of defect/size of child/ stability of child
May due primary closure or staged closure
Possibly silo closure
Post op:
monitor closely for respiratory compromise
pain management
fluids
Initiation of feeds

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24
Q

STRUCTURAL ANOMALIES:
Anorectal Malformations

A

Anomalies Associated with:
* VACTERL syndrome
* Esophageal atresia
* Intestinal atresia
* Malrotation
* Renal agenesis
* Hypospadias
* Vesicoureteral reflux
* Bladder exstrophy
* Cardiac anomalies
* Skeletal anomalies

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25
Anorectal Malformations
**Imperforate Anus *** Rectum ends in a blind pouch * Level of defect influences outcome – fecal continence * Staged repair * Colostomy while waiting repair
26
Nursing Assessment Imperforate Anus
* newborn assess for normal anal opening * passage of meconium stool within 24hours of birth * assess UO to identify accompanying GU abnormalities * assess for signs of intestinal obstruction – distension/vomiting
27
Nursing Management: Imperforate Anus
* Preop: NPO, gastric decompression, hydration status, education * Postop: pain, NPO maintained, gastric decompression, colostomy care, if needed. * After intestinal pull thru initially stool will be watery – skin breakdown
28
STRUCTURAL ANOMALIES: Meckel’s Diverticulum
Congenital – 3-6 cm outpouching (bulge) of lower segment of small intestine *Most common birth defect of GI system – 2-3% population *Most children asymptomatic *If symptoms, usually age 1-2y *Painless rectal bleeding *“brick” or “currant jelly” – very red stool Can be associated with partial or complete bowel obstruction *Obstruction – younger children *Diverticulitis - older children (~ 8y) - looks like appendicitis
29
Meckel’s Diverticulum: Common Complications
* Anemia * Bleeding * Ulcer * Obstruction (volvulus / intussusception) * Typically occur in first 2 years of life
30
Presentation Meckel’s Diverticulum
* Rectal bleeding, painless * Abdominal pain * Bloody, mucusy stools
31
Meckel’s Diverticulum: Dx
* Meckels Scan (radionuclide scan), CBC and metabolic panel
32
Meckel’s Nursing Management
* possibly blood products * IV fluids * NPO * Surgery to repair
33
Inguinal Hernia
* Abdominal or pelvic viscera travel thru inguinal ring into inguinal canal *** Males**: bowel *** Females**: fallopian tubes/ovaries * **Nursing Assessment:** Bulging mass in lower abdomen/groin * ** Management: ** Surgical repair * Reduction until surgery/ teach family (NP/MD) * Education to family about incarcerated hernias and urgency
34
STRUCTURAL ABNORMALITIES: Umbilical Hernia
More common: Preterm infants / African Americans * Incomplete closure of umbilical ring * Most self resolve by 4yo * Surgery if large that fail to close by 4 yo
35
ACUTE GASTROINTESTINAL DISORDERS: Dehydration
*Infants in young children more at risk *Increased extracellular fluid percentage, increase in body water compared to adults *Increased Basal Metabolic Rate *Immature renal function *Increased insensible fluid loss thru temp elevation
36
ACUTE GASTROINTESTINAL DISORDERS: Dehydration Nursing Management:
* Restore fluid volume * Prevent shock * Mild to moderate ORT * Severe IVF * Refer to Teaching Guidelines 20.1 ORT * Pedialyte, 50-100mL/kg over 4 hours, reeval * reg diet after rehydrated
37
ACUTE GASTROINTESTINAL DISORDERS: Vomiting
*** forceful expulsion of gastric contents thru mouth * Many different causes – it is a symptom of some other condition * Infectious: gastroenteritis, OM, pharyngitis, sinusitis, pyelonephritis, meningitis * GI: intussusception, malrotation, appendicitis, cholecystitis, pancreatitis, GERD, pyloric stenosis * GU: ureteropelvic junction dysfunction * Endocrine: Diabetic ketoacidosis * Neuro: IICP * Other: food poisoning, toxic ingestion
38
Vomiting: Nursing Assessment
* Hx * onset, progression, timing, characteristics * color – bilious never normal * bloody – GI bleed or esophageal bleed * hx of trauma? * feeding pattern * assoc symptoms ie fever, diarrhea, abdominal pain * PE * hydration/perfusion, mental status, abdomen
39
Vomiting: Nursing management
* Simple vomiting: ORT – 0.5 – 2oz ORS every 15 min age and size dependent * Antinausea meds * IVF
40
ACUTE GASTROINTESTINAL DISORDERS, CONT. Diarrhea
* increase in frequency or decrease in consistency of stool * Acute vs chronic (more than 2 weeks) ** Acute:** viral – watery, loose, fever, vomiting * bacterial – bloody stools or mucousy * Parasites – fever, watery ** Chronic:** infants: milk/soy intolerance, enteritis, hirschprungs, nutrient malabsorption * toddlers: nonspecific, enteritis, celiac, UC * school age: IBD, appendiceal abscess, lactase deficiency, constipation with encopresis
41
Oral Candidiasis (Thrush)
* Fungal infection of the oral mucosa * Most common infants/newborns * HR: immune d/o’s, corticosteroid MDIs, antibiotic use * May be transmitted bw BF mom and baby
42
Oral Candidiasis (Thrush): Nursing Management
* Meds and Education * Nystatin topically younger / swish and swallow older * Fluconazole – SE’s hepatotoxicity, N/V * BF mom’s much treat as well * Keep bottles and pacifiers clean * Wash toys
43
Hypertrophic Pyloric Stenosis
* The circular muscle of the pylorus becomes hypertrophied causing thickness in the luminal side of the pyloric canal * The thickness causes an gastric outlet obstruction * Nonbilious vomiting 3-6 weeks of life – becomes more frequent and forceful - - projectile * 2-3.5/1000 live births Males > Females
44
Hypertrophic Pyloric Stenosis: Treatment
Pylorotomy
45
Hypertrophic Pyloric Stenosis: Nursing Assessment
* Forceful, nonbilious vomiting * Hunger after vomiting * Wt loss * Dehydration * Hard moveable “olive” mass in RUQ
46
Intussusception
* Proximal segment of the bowel “telescopes” into a more distal segment * edema, vascular compromise and partial/total obstruction * Intermittent telescoping then reduces spontaneously
47
Intussusception: Treatment
Barium/Air enema or Surgery
48
Intusussception: Nursing Assessment
* Severe pain intermittent (drawing knees up) vomiting, diarrhea, currant-jelly stools, bloody, lethargy, may be able to palpate sausage shaped mass in upper mid abdomen
49
Intusussception: Nursing Management
IVF, support for procedure
50
Malrotation
* occurs from a disruption in embryonic development * intestine is abnormally attached * mesentery narrows twisting on itself (volvulus) * Main symptom: bilious vomiting * abdominal pain, distension, bloody stools * Most cases first few weeks of life – but may occur when older
51
Malrotation: Therapeutic Management
Surgery - Ladd procedure straighten out intestine and divide the bands that contribute to misalignment
52
Appendicitis
* Acute inflammation of appendix * Peaks second decade of life * Obstruction of the appendix: possibly from fecal matter * IF it perforates inflammatory fluid and bacteria leak into abdominal cavity = Peritonitis (more common younger children) * Older children more mature omentum – making an abscess
53
Nursing Assessment: Appendicitis
* Hx: may be gradual onset * typically sxs progress but are not intermittent * vague abdominal pain that localizes to RLQ over hours * N/V after onset of pain * Small volume, frequent stools * Fever (low grade) unless perforation (high grade)
54
Exam: Appendicitis
* ill appearance/uncomfortable guarded movements Pain > at McBurneys point RLQ Distenstion and diffuse pain - peritonitis
55
Appendicitis: Dx
* CBC * CRP * US vs CT scan
56
Appendicitis: Treatment
* nonruptured, nongangrenous appendix preop antibiotics – no post op antibiotics * suppurative or gangrenous appendix antibiotics post op 48-72hours perforated – antibiotics possible for 7-14 days (may wait on surgery) * IF abscess develops post op - drains
57
GERD
* reflux of gastric contents into esophagus * occurs during relaxation of LES during swallowing, crying, valsava * Normal first year of life – outgrow 12-18 mos of age * Sxs: vomiting, irritability with feeding or posturing after feeds, bradycardia
58
GERD: Therapeutic Management
* HOB elevated * upright 30” after feeds * small, frequent feeds * Medications: ie prilosec
59
Constipation
* Typically presents during TT years * Painful experience during defecation turns to fear of defecation * and withholding behaviors * Organic causes are rare: sign of disease ie spina bifida or sacral agenesis * affects 30% of pedi population * Failure to achieve complete evacuation of the lower colon * BF: stool with each feed, sometimes skip 2-3 days * Bottle fed: 1-2 per day, may skip 2-3 days
60
Functional Constipation at least two of the following over 1mos:
* less than 3 BM / week * at least one episode fecal incontinence/week (post Toilet trained) * excessive stool retention hx * hard/painful BM * large fecal rectal mass * stool passage volume clogs toilet * stool withholding behavior (retentive posturing)
61
Encopresis
soiling of fecal matter into underwear beyond age of expected TT (4-5 yo)
62
Constipation: Nursing Management
* Toileting Behaviors: regular toileting times, positive reinforcement for toileting * Dietary changes * Medications: suppositories/enema, miralax, colace, senna
63
Hirschprung Disease (congenital aganglionic megacolon)
* disorder of motility of the intestinal track resulting in obstruction * most commonly characterized: Failure to pass a meconium within first 24 hours of life * Lack of ganglion cells in the intestine - inadequate motility * Can occur anywhere from rectosigmoid colon all the way to small intestine
64
Hirschprung Disease (congenital aganglionic megacolon): Nursing Assessment
Hx: Passage of meconium * did require rectal stimulation * Down syndrome or family hx higher incidence Exam: Abdominal distension * may stool with rectal stimulation / typically stool not in rectum until after stimulation Dx: rectal suction bx – aganglionic cells
65
Hirschprung Disease (congenital aganglionic megacolon): Therapeutic Management
* initially may do rectal dilations * Surgery: resection / ostomy while healing
66
Nursing Management post op: Hirschprung Disease (congenital aganglionic megacolon):
* Ostomy care, I+O’s, skin care * Observe for signs and symptoms of enterocolitis: fever, distension, rectal bleeding
67
Short Bowel Syndrome
* Nutrient malabsorption – excessive intestinal fluid and electrolyte loss * Occurs after massive small intestinal resection * Most common causes are NEC, small intestinal atresia, gastroschisis, malrotation or trauma to small intestine * Goals: minimize bacterial overgrowth and maximize nutrition * Management: possible antibiotics prophylactically, vitamin and mineral supplementation, antidiarrheals, TPN
68
Inflammatory Bowel Disease
* Crohns and UC * Cause: UK * 15-20% diagnosed in childhood/adolescence * Exacerbations & remissions; chronic, sometimes debilitating
69
Crohns
affects large and small intestines (can affect entire alimentary canal, mouth – anus) * Lesions full thickness, acute and chronic inflammation, noncaseating granulomas, extraintestinal fistulas, abscesses, strictures, and fibrosis * Abdominal pain, diarrhea with blood if colon involved, perianal disease, fistula, abscess, anorexia
70
Ulcerativecolitis
* affects only large intestine and rectal mucosa (proctitis) * guishing features Crohns and UC * Superficial, acute inflammation of mucosa * Abdominal pain, bloody diarrhea, urgency, tenesmus
71
Celiac Disease
* Immunologic Disorder * Gluten causes damage to the small intestine * Villi in small intestine damaged d/t immunologic response to digestion of gluten * Most common chronic GI d/o Europe and US * Prevalence > 3% - increased if family hx, autoimmune or genetic d/o’s
72
Celiac
Treatment – Gluten free diet (strict) Initial presentation: often by age 2 diarrhea, steatorrhea, constipation, FTT/wt loss, Abdominal distention, poor muscle tone, irritability, fatigue, dental d/o’s, anemia, amenorrhea, delayed puberty, nutritional deficiencies
73
Celiac: Nursing Management
* Gluten free- Diet – Strict * Dietitian * Support groups * Resources Teaching Guidelines for Dietary Considerations table 20.2
74
Functional Abdominal Pain
* Approx 2-4 % all pedi outpt visits are r/t recurrent abdominal pain * Etiology: UK * May occur as a result of: rectal distension, impaired gastric relaxation in response to pain, or heightened sensitivity to visceral pain * Psychological responses may occur if pain persistent or recurrent
75
Functional Abdominal Pain: Nursing Management
* Typically a battery of tests are completed blood, Xrays, other diagnostics r/t complaints to r/o other organic causes * Identifying triggers and increasing coping skills * Return to activities/school – keep routines
76
Biliary Atresia
* Cause: UK * Absence of all or some of the major biliary ducts * resulting in obstruction of bile flow * Cholestasis occurs
77
Biliary Atresia: Nursing Assessment
* Light beige stools (Normal stool color for infants is yellow, green or brown.) * Dark brown urine * Distended abdomen * Hepatosplenomegaly * Difficulty gaining weight * Ascites * Cirrhosis in several mos if untreated
78
Biliary Atresia: Management
* Surgery (Kasai procedure) most successful before 45 days old * if not successful, or later in age, may need liver transplant * Other D/o’s Hepatitis, Pancreatitis, Gallbladder Disease